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scid
    All of the 26 SCID mouse were successfully transplanted human bladder cancer; The T24 human bladder cancer cells and tumor xenograft expressed KDR positively.
    26只SCID小鼠均成功长出人膀胱癌移植瘤,人膀胱癌T24细胞及移植瘤瘤体组织阳性表达KDR。
    The GMT of human IgG/VCA and the level of human IgG in the sera were 1∶108 and 96 2±56 4 μg/L in 14 SCID hPLNL mice and 1∶7 9 and 13 84±6 0μg/L in 8 SCID hPBL mice,respectively.
    14只SCID-hPLNL小鼠血清内人IgG/VCA的几何平均滴度(GMT)和血清IgG浓度在1∶108和96.2±56.4μg/L; 在另8只SCID-hPBL中为1∶7.9和13.84±6.0μg/L。
    The GMT (geometric mean titer) of human IgG/VCA and the level of human IgG in the sera were 1∶108 and (96.2±56.4) μg/L in 14 SCID hPLNL mice and 1∶7.8 and (13.84±6.0) μg/L in 8 SCID hPBL mice respectively.
    IgG/VCA的几何平均滴度(GMT)和血清IgG浓度,在14只SCID-PLNL小鼠中为1∶108和(96.2±56.4)μg/L; 在8只SCID-PBL小鼠为1∶7.8和(13.84±6.0)μg/L。
    e the suspcnsion of tumorcclls was implanted into one site,and the tumor fragments were implanted into opposed site in same mouse:All tumor-bcar-ing scid mice were sacrificed on 68th day.
    24只scid小鼠分48个部位同时移植,动物均于肿瘤移植后68天全部处死。
    Methods The tumor mass which was formed by injecting human breast cancer cell line MDA MB 231 subcutaneously was orthotopically transplanted into breast fat pad of 6 SCID mice and the metastasis lymphynodes were passaged with same method.
    方法 将人乳腺癌细胞系MDA MB 2 31瘤块原位移植于 6只SCID鼠乳房的脂肪垫上 ,用其癌转移淋巴结传代共传 1 8代 68只。
    Twelve male SCID mice of 4 week old were divided into 2 groups at random and both YCD-K562B and K562B cells were implanted to each mice. 5-FC or saline was given i.p for 10 days after tumor developed, and relative tumor volume was measured every 3 days.
    12只SCID小鼠分为治疗及对照组 ,在小鼠左右两侧近前肢处腹部皮下注射YCD K5 6 2B及K5 6 2B细胞 ,成瘤后治疗组腹腔注射 5 0 0mg kg 5 FC共 10d ,对照组腹腔注射生理盐水 ,观察瘤体相对体积变化及病理变化。
    Many large colonies grew in soft agar (4%) and the transplanted tumors were found in all 4 nude and 4 SCID mice, with strong invasive potency demonstrated in vitro and in vivo .
    软琼脂培养多细胞大集落形成率 4 %。 接种的 4只裸小鼠和 4只SCID小鼠皆成瘤。
    Of 38 survival SCID mice,24 mice developed tumors in their body cavities.
    渡过急性GVHR期而存活的 38只SCID小鼠中 ,共有 2 4只形成肿瘤。
    Methods The SCID mice were reconstituted by intraperitoneal injection(i.p)of5×10 7 human lymphocytes from Epstein-Barr virus(EBV)seronegative individuals.
    方法将SCID小鼠随机分为4组,每组7只,分别为:(1)实验组:在每只SCID小鼠的腹腔内接种人外周血单个核细胞(peripheral-bloodmononuclearcells,PBMC)1周后,腹腔接种EB病毒(Epstein-Barrvirus,EBV)悬液;
    Raji cell lines which were injected into 3 SCID mice grew up into neoplasms, and the neoplasms were observed by light and electron microscope.
    同时用Raji细胞系接种 3只SCID鼠 ,在光镜、电镜下观察成瘤组织结构变化。
    All the 22 SCID mice were intraperitoneally injected with human peripheral blood lymphocytes (PBL) to establish immune reconstituted model.
    以上22只SCID小鼠均用人外周血淋巴细胞腹腔注射进行人免疫功能重建。
    tissue were placed subdermally into 15 severe combined immunodeficient(SCID) mice. The mice were randomly divided into three groups. The implant was saved for pathology,immunohistochemistry and HPV DNA examination at 2nd、4th、6thweeks.
    方法获取正常子宫颈组织的移行带标本,接种于12只SCID鼠皮下,随机分成3组,分别在2、4、6周取出其皮下移植物行HE染色、免疫组化及人乳头瘤病毒(HPV)DNA的检测。
    Subsequently, the tumor cells from fi rst-generation mice model were respectively subcutaneously implanted into 61 se cond-generation SCID mice and 8 BALB/CA-nude mice and the rate of the tumor f ormation and the latent period of the tumor formation were observed.
    动物传代,观察第二代61只SCID小鼠和8只裸鼠的成瘤率和成瘤潜伏期。
    Methods RA synovium and normal human cartilage under the kidney cap- sule of the SCID mice were engrafted,and were maintained for 4~16 weeks.
    方法70只SCID小鼠,3~6周龄。
    Methods Sixty SCID mice were randomized into six groups,individual tumor growth was observed,and drug related systemic toxicity was also evaluated after administration of 5-fluorouracil-loaded polyactic acid sustained-release preparation.
    方法36只SCID小鼠随机分为6组,分别是瘤内注射生理盐水的对照组、瘤内注射5-Fu-NPs(包括低剂量和高剂量)组、瘤内注射无载药NPs组、瘤内注射5-Fu组及腹腔内注射5-Fu组。
 

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