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chronic severe hepatitis b
    Results The RBC,Hb,PLC and PCT were lower in patients with chronic hepatitis B of severe degree and with chronic severe hepatitis B than in control ( p <0.01),while MCV,RDW,MPV and PDW higher in the two patient groups than in control( p <0.01);
    结果 两组病例组RBC、Hb、PLC、PCT值均显著低于健康对照组 (p <0 .0 1) ,而MCV、PLC、PCT值均显著低于慢性重型肝炎组 ,MCV、RDW、MPV值均显著高于慢性重型肝炎组 (p <0 .0 1) ;
    The difference between two groups with Dunnett T3 analysis showed there are statistically difference between the groups of the normal group and the chronic severe hepatitis B group (P=0.014), the acute hepatitis B group and the chronic hepatitis B group (P=0.009), and the acute hepatitis B group and the chronic severe hepatitis B(P=0.003).
    组间两两比较,正常对照组与慢性重型肝炎组、急性乙型肝炎组与慢性乙型肝炎组及慢性重型肝炎组之间差异均有显著性(P值分别为0.014、0.009和0.003)。
    IL-4 was positively correlated with IL-18(r= 0.6,P< 0.01),and IL-4 was positively correlated with IFN-γ(r= 0.54,P< 0.01) in chronic severe hepatitis B group.
    慢性重型肝炎组IL 4与IL- 18呈正相关( r=0.6,P<0.01); IL- 4 与IFN -γ呈正相关( r=0.54, P<0.01)。
    Conclusion:Lipo-PGE_1 injection treats chronic severe hepatitis B by reducing the serum TNF-α level.
    结论:Lipo-PGE1注射液可降低慢性乙型重型肝炎患者血清TNF-α的水平,是其治疗慢性重型肝炎的作用机制之一。
    No significant difference of C allele frequency was found in chronic hepatitis B (44.1), chronic severe hepatitis B (45.8), and liver cirrhosis (45.0) (P>0.05).
    在慢性肝炎、慢性重型肝炎和肝炎肝硬变3组人群中,C等位基因频率分别为44.1%、45.8%和45.0%,彼此间相差不显著(P>0.05)。
    Treatment effect of lamivudine on chronic severe hepatitis B
    拉米夫定治疗慢性重型肝炎疗效观察
    Some red blood cell and platelet parameter changes in patients with chronic hepatitis B of severe degree and with chronic severe hepatitis B
    慢性肝炎重度和慢性重型肝炎患者红细胞及血小板参数变化及意义
    The Study on the Effects of Colon Herbal Dialysis in Patients with Chronic Severe Hepatitis B
    肠透析治疗慢性重型肝炎内毒素血症临床研究
    CLINICAL CHARACTERISTICS OF JAUNDICE IN PATIENTS WITH CHRONIC SEVERE HEPATITIS B
    慢性重型肝炎不同黄疸临床特点的研究
    It has not been reported that how the endotoxin activated the systemic inflammatory response syndrome and what role the endotoxin receptor on the mononuclear macrophages and the expression of induced downstream introcellular signaling molecular played in chronic severe hepatitis B. Our experiment was trying to explain the mechanism of the severity of hepatitis by the core signal conduction pathway of CD 14 and TLR4,and by the induceded systemic inflammatory response syndrome.
    在肝炎重型化过程中,内毒素如何激活单核细胞诱导失控性炎症反应,体内单核巨嗜细胞表面内毒素受体及其引发胞内下游信号分子的表达情况在慢性重型肝炎中的作用尚未见报道。 本实验力求阐明肠源性内毒素血症通过“CD14、TLR4”为核心的信号传导通路并诱导失控性炎症反应最终导致肝炎重型化的机制;
    Serum levels of soluble turnor necrosis factor receptor (sTNFR) were detected with ELISA in 30 patients of chronic active hepatitis B and 24 patients of chronic severe hepatitis B. The results showed that the serum levels of sTNFR in chronic severe hepatitis B and chronic active hepatitis B were obviously increased,especially in patients with chronic severe hepatitis B. It's degree was positively correlated with serum total bilirubin concentration and negatively correlated with prothrombin activity.
    应用酶联免疫吸附试验法测定了30例慢性活动型肝炎及24例慢性重型肝炎患者血清可溶性肿瘤坏死因子受体(sTNFR)水平。 结果显示:慢性重型肝炎及慢性活动型肝炎患者sTNFR水平显著升高,尤以慢性重型肝炎患者为甚,升高程度与总胆红素呈正相关,与凝血酶原活动度呈负相关。
    The further analysis for 24 patients with chronic severe hepatitis B showed that serum levels of sTNFR was more higher in patients accompanied by infection or hepatorenal syndrome than those unaccompanied by.
    对24例慢性重型肝炎患者进一步分析表明;
    Serum levels of soluble tumor necrosis factor receptor (sTNFR) were detected with ELISA in 32 patients of chronic severe hepatitis B.The results showed that the serum levels of sTNFR in chronic severe hepatitis B were obviously increased and the sTNFR level was positively correlated with serum total bilirubin concentration, negatively with prothrombin time activity.
    应用酶联免疫吸附试验法测定了32例慢性重型乙型肝炎患者血清可溶性肿瘤坏死因子受体(sTNFR)水平。 结果 慢性重型肝炎患者sTNFR水平显著升高,升高程度与总胆红素呈正相关,与凝血酶原活动度呈负相关。
    These results indicate that detection of serum sTNFR in patients with chronic severe hepatitis B is significant for evaluating the clinical severity and for assessing the prognosis of the disease.
    伴有感染或肝肾综合征时,sTNFR水平显著高于无感染或无肝肾综合征患者,最终死亡者其水平也显著高于存活者。 提示测定慢性重型肝炎患者血清sTNFR水平对判断病情及预测转归有一定的价值。
    Methods Levels of plasmic LPS and serumal sFasL were detected,and expression of FasL in liver tissue were investigated synchronously in 12 patients with chronic severe hepatitis B. 20 normal subjects and 16 patients with chronic hepatitis B were detected as controls.
    方法 检测 1 2例慢性重型肝炎患者血浆内毒素LPS的水平 ,同步观察肝组织FasL表达与血清sFasL的水平 ,并与 1 6例慢性乙型肝炎患者进行对照。
    Results The blood levels of LPS,sFasL in patients with chronic severe hepatitis B were significantly higher than those in patients with chronic hepatitis B and normal subjects.
    结果 慢性重型肝炎组LPS水平、sFasL、FasL表达阳性率及阳性强度均高于慢性乙型肝炎组 ,P <0 .0 1。
    Results The positive prevalence of HBeAg and/or HBV DNA in chronic severe hepatitis B was much lower the level of ALT,TB and the prognosis(P<0 01).
    结果 慢性重型肝炎病人的HBeAg和 /或HBVDNA阳性率明显低于非重型慢性肝炎 (P <0 0 1) ; HBV复制状态与血清转氨酶、总胆红素水平及预后无明显相关性。
    but it in creased obviously after PHA stimulation in vitro ( P <0.01) , In comparison with normal controls, the apoptosis of PBLs was significantly higher in patients with chronic hepatitis B ( P <0.01) and lower in patients with chronic severe hepatitis B ( P <0.01).
    但与正常对照组相比 ,活化诱导的淋巴细胞凋亡在慢性乙型肝炎患者明显增高 ,而在慢性重型肝炎患者则降低 ,差异有显著性 (P <0 .0 1)。
    RBC,Hb,PLC and PCT in patients with chronic hepatitis B of severe degree were lower than in patients with chronic severe hepatitis B,while MCV,RDW and MPV higher than in chronic severe hepatitis B patients( p<0.01 );
    慢性重型肝炎早期PLC、PCT值均显著低于慢性重型肝炎中期 ,PDW值均显著高于对照组 (P <0 .0 1) ,并与肝脏损害程度呈正相关。
    Results In comparison with normal controls, the apoptosis of PBL was significantly higher in patients with chronic hepatitis B (P<0.01)and lower in patients with chronic severe hepatitis B (P<0.01)after PHA stimulation in vitro.
    结果 与正常对照组相比 ,活化诱导的淋巴细胞凋亡在慢性乙型肝炎患者明显增高 ,而在慢性重型肝炎患者则降低 ,差异有显著性 (P<0 .0 1)。
 

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