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    ②Totally 30 adult SD rats were divided into 3 groups at random: injury control group, early transplantation group and postpone transplantation group, 10 in each group.
    ②将30只成年SD大鼠按随机数字表法分为3组,即损伤对照组、早期移植组和延期移植组,每组10只。
    The injured groups as control:to replace Shenmai injection by NS in each group as described above,and marked the new groups as A',B',C',D',E' and F'.
    用生理盐水替换参麦为油酸损伤对照组,分别为A′、B′、C′、D′、E′、F′组。
    Method:Seventy-two SD rats were randomly divided into sham group(A),normal saline group(B) and ChABC group(C) with each group of twenty-four.
    方法:SD大鼠72只,雌雄不限,随机分为假手术组(A组)、损伤对照组(B组)和ChABC治疗组(C组),每组24只。
    Group 3(n=24) was divided into 3 subgroups: non-injured group(n=8), vehicle-treated group (n=8)to whom Saline was applied and CsA - treated group(n=8).
    第3组大鼠(n=24)随机分为正常对照组(n=8)、损伤对照组(n=8)和CsA治疗组(n=8),以Morris水迷宫实验和避暗实验测定伤后不同时间点 DA大鼠学习记忆能力的变化,研究 CsA的治疗作用。
    3. Behavior rehab of experimental group of implanted HHK was well than that of no implanted HHK.
    3 证实了植入HHK实验组的恢复早于单一损伤对照组,其恢复程度也比同一时期单一损伤对照组好。
    (2) sham liver injury group;
    (2)假肝损伤对照组
    3. Compared with the ACI control, ET concentration in plasma and injuried cortex at each time group is decreased after HBO therapy(p<0.05).
    3.动物急性颅脑损伤经高压氧治疗后6h,24h,48h各时间组血浆及损伤皮层组织内皮素含量均明显低于相应急性颅脑损伤对照组(p<0.05);
    Results 1.In positive groups, polylysine, damaged by Fenton reaction , the endothelial cell proliferation dereased remarkably (P<0.01) .
    1.分别经Fenton体系、多聚赖氨酸作用的损伤对照组,内皮细胞增殖活性明显下降(P<0.01)。
    3. Compare with positive control group damaged by oxygen-derived free radidicals , the excretion of endothelins decreased remarkably in SS-GAG group (P<0.01) , and decreased with increase of SS-GAG dose.
    3.SS-GAG保护组与经氧自由基损伤的损伤对照组对比,内皮素的分泌明显减少(P<0.01),且随SS-GAG剂量增加而降低;
    4. Compare with positive control group, the number of VCAM-1 expression descended obviously in SS-GAG groups (P<0.01) .
    4.经SS-GAG保护的HUVEC表达VCAM-1的量,较氧自由基损伤的损伤对照组明显减少(P<0.01)。
    The activity of ACHE in the Hypoxia group was lower significantly than that in other groups except the VC-L, M groups (P<0.01).
    缺氧损伤对照组乙酰胆碱酯酶(ACHE)活性低于除VC-L、M组外的其它各组,差异有显著性(P<0.01);
    METHODS:120 mice were divided randomly into normal control group(I group), merely light damage group(II group) and experimental group(III group).
    方法:120 只小鼠随机分为正常对照组(I 组)、光损伤对照组(II 组)和实验组(III 组)。
    The cell proliferation of the H2O2 injuried control groupswas much lower than those of the normal control groups, it was higher inthe GPC groups than those of the H2O2 injuried control groups(p<0.05 ).
    H_2O_2损伤对照组细胞增殖活性均明显低于正常对照组,3个GPC剂量组细胞增殖活性均明显高于H_2O_2损伤对照组,GPC对照组细胞增殖活性均高于正常对照组,各组间差异均具有显著性(p<0.05)。
    The expression of Bax was much higher in the injuried controlgroup than that of the normal control groups, it was much lower in thehigh-dose and middle-dose GPC groups than that of the injuried controlgroups(p<0.05) The cell proliferation of the irradiating control groups
    H_2O_2损伤对照组细胞Bax的表达明显高于正常对照组,高、中GPC剂量组细胞Bax的表达明显低于H_2O_2损伤对照组,各组间差异均具有显著性(p<0.05)。 辐射对照组细胞增殖活性明显低于正常对照组,GPC剂量组细胞增殖活性均高于辐射对照组,各组间差异均具有显著性(p<0.05)。
    Control group(C);
    损伤对照组(C);
    control groups ( spinal cord injury lday group, 3 day group , 7 day group, 14 day group, 21 day group , 28 day group).
    (2)脊髓损伤骨髓基质干细胞非移植组(损伤对照组)(再分为6组:术后1天组、3天组、7天组、14天组、21天组和28天组);
    The results demonstrated that blood glucose level was much lower after peritonitis injection of ZTL(25g/kg)and STS(12.5g/kg)at 3,6,9,12 days than that of the control group at the same time(P<0.01),with a dose—dependent relation,while there was no significant difference blood glucose following EHL injection compared with control group(P >0.05).
    腹腔注射卷柏(12.5g/kg)后的3天、6天、9天的血糖值明显低于损伤对照组(P<0.01)。 腹腔注射木贼(12.5g/kg)后大鼠血糖水平呈下降趋势,无统计学差异(P>0.05)。
    It showed that the axon recovery of Angelica therapy groups at both time points was better than that of control injury group respec- tively.
    提示2个当归治疗组轴突恢复均较各自损伤对照组好。
    Methods Experimental animals were divided into three groups: the group of microgene pSVPoMcat implanted to genetically modify SC (group A), SC implanted group (group B) and the control group (group C).
    方法将实验动物分为pSVPoMcat微基因修饰SC组(A组),SC移植组(B组),损伤对照组(C组)。
    The AChE activity in the experimental group and the control group was reduced 1,3 day after nerve injury (P>0 05).
    结果表明术后1,3天实验组(指损伤给药组)和对照组(指损伤对照组)AChE活性均下降(P>005);
 

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