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    Preliminary Study on Mechanisms of Hematoporphyrin Monomethyl Ether-Photodynamic Therapy Induced Hela Cells Death
    血卟啉单甲醚光动力治疗诱导Hela细胞死亡机制的初步研究
    THERAPY-INDUCED ACUTE LEUKEMIA ( REPORT OF 1 CASE )
    治疗诱导性急性白血病(附1例报告)
    Objective To induce renal transplantation tolerance by CTLA4-Ig adenovirus gene therapy in allogeneic rats and study the relative mechanisms of transplantation tolerance.
    目的通过CTLA4-Ig腺病毒基因治疗诱导异基因大鼠之间的肾脏移植免疫耐受,并探讨相关机制。
    Induction of cardiac transplantation tolerance by CTLA4Ig adenovirus gene therapy in allogeneic rats
    CTLA4Ig腺病毒基因治疗诱导异基因大鼠心脏移植耐受
    Study on accumulations of mitochondrial 4977bp deletion of skin with PUVA treatment
    PUVA治疗诱导皮肤线粒体DNA 4977 bp缺失突变累积的研究
    Adenovirus-mediated gene transfer of CD40Ig gene induced long-term survival of cardiac allograft in rats
    腺病毒介导的CD40Ig基因治疗诱导异基因大鼠心脏移植物的长期存活
    Apoptosis and Lipolysis of White Adipocytes Induced by Neuropeptide Y- Y5 Receptor Antisense Oligodeoxynucleotides
    神经肽Y-Y5受体反义基因治疗诱导大鼠脂肪细胞凋亡研究(英文)
    Experimental study on apoptosis induced by CPD4 photodynamic therapy of human ovarian cancer cells
    CPD4光动力学治疗诱导人卵巢癌细胞凋亡的实验研究
    Induction of long-term good glycemic control in newly diagnosed type 2 diabetic patients by transient intensive insulin treatment
    短期胰岛素强化治疗诱导初诊2型糖尿病患者血糖长期良好控制的临床试验
    Influence of interleukin 12B (IL12B) polymorphisms on spontaneous and trea tment induced recovery from hepatitis C virus infection
    白介素12B的多态性对HCV感染后自然康复和治疗诱导康复的影响
    Apoptosis Induction Effect of pEgr-P16 Genetherapy Combined with Low Dose Irradiation on Esophageal Cancer Cells
    低剂量辐射联合pEgr-P16基因治疗诱导食管癌细胞凋亡的研究
    Verteporfin photodynamic therapy induced apoptosis in choroidal neovascular membranes
    维替泊芬光动力治疗诱导脉络膜新生血管凋亡
    Study on relationship between cervical cancer hela cell apoptosis inducing by electrochemical therapy and changes of different ionic concentration in external environment
    直流电治疗诱导宫颈癌HELA细胞凋亡与外环境离子浓度变化关系的研究
    (4) Perfusion in situ with Ad-CTLA4-Ig gene and Ad-FasL gene into the recipient kidneys through the renal artery during cold preservation , observed the survival time of the kidney transplantation in rats and searched the possible mechanism of immune tolerance induced by transfecting CTLA4-Ig gene and FasL gene therapy.
    (4)将CTLA4-Ig 基因和FasL 基因,在冷保存时经供肾动脉局部灌注转染至供肾体内,观察其对肾移植大鼠生存期的影响,并探讨转CTLA4-Ig 基因和Fasl 基因的转基因治疗诱导大鼠肾移植免疫耐受的可能机理。
    The average serum level of IL-4 from B group and C group was significantly less than the level of the A group (B group:49.8+ 6.7 pg/ml and C group:47.9+5.3pg/ml,p= 0.013 and 0.008,respectively).
    而血清IL-4水平与对照组比较则显著下降m组49.8士6.7 pg/ml,C组 47.9士5.3Pg/ml,均显著低于A组70.3土4.5 Pg加1中值分另为0.013和0.008)。 说明荷红白血病小鼠syn于见后,CpG仆CSF免疫治疗诱导了W 型免疫反应。
    Methods 105 first renal transplant recipients were divided into daclizumab group (n=35), basiliximab group (n=35) and contral group(n=35).
    方法:首次接受尸体肾移植受者105例随机分成3组,每组35人,分为接受2剂basiliximab、2剂daclizumab治疗的诱导组和对照组。
    Objective: To induce cardiac transplantation tolerance by CTLA4Ig adenovirus gene therapy in allogeneic rats, and study the relative mechanisms of transplantation tolerance.
    目的 :通过CTLA4Ig腺病毒基因治疗诱导异基因大鼠之间的心脏移植耐受 ,并探讨相关机制。
    Conclusion: Bax proteins were associated with apoptosis induced by radiotherapy.
    结论:放射治疗诱导宫颈癌肿瘤细胞凋亡,可能与凋亡调节基因bax基因表达的诱导密切相关。
    Immunohistochemical stain proved increased expression of NFκB after taxol single treatment while combination with AdIκBα intratumoral injection eliminated this expression accompanied with the slightly decreased expression of VEGF and stable p53 status.
    免疫组化染色证实泰素治疗诱导了H460移植瘤细胞的NFκB表达增加,AdIκBα转染则消除了其表达,同时伴有轻度增加的VEGF表达,而p53表达不受影响。
    The newest research about radioactivity inducing tumor cell apoptosis.
    放射治疗诱导肿瘤细胞凋亡的近期研究动态
 

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