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    Human metallolastase (MMP-12) is a MMP, the abnormal expression of which has so far been documented in macrophages associated with many diseases, such as rheumatoid arthritis (RA) and osteoarthritis (OA).
    人类金属弹性蛋白酶(Human metallolastase, MMP-12)是MMPs的一种。 现已证实由巨噬细胞分泌MMP-12的异常表达与许多疾病有关,如类风湿性关节炎(Rheumatoid arthritis, RA),骨性关节炎(Osteoarthritis, OA) 等。
    TNF-α is mainly produced by activated macrophages. Many inducers can activate macrophages and induce expression of TNF-α gene transcription.
    TNF-α主要由巨噬细胞分泌产生,有许多体内、外物质可激活巨噬细胞、诱导TNF-α基因转录表达。
    Interleukin 12 (IL-12) is produced by antigen presenting cell, for instance, macrophage cell eg. IL-12, a cytokine composed of heterodimers has phenotropic bioactivity and contain two subunits, p35 and p40. And IL-12 can stimulate NK ( Natural Killer ) cells and maturate CTLS.
    白细胞介素—12(IL-12)是由巨噬细胞等抗原提呈细胞产生的具有多种生物学活性的异二聚体细胞因子,含有p35和p40两个亚单位,能刺激NK细胞增殖,促进细胞毒T淋巴细胞(CTL)成熟。
    Background: TNF-a, a cytokine with complex bioactivity, is mainly secreted by macrophages and T cells.
    研究背景TNF-α是一种具有复杂生物活性的细胞因子,它主要由巨噬细胞和T细胞产生。
    It has been proved that TNF—α(17kd,157aa) produced by macrophages and TNF—β (25kd,17aa) , a glucoprotein produced by activated T—lymphocytes, may have the same receptors, with which they combine and put into effect.
    现已证明,TNF—α(17kd,157aa)是由巨噬细胞产生的一种单核因子,TNF—β(25kd,171aa)是由活化型T淋巴细胞产生的一种淋巴因子糖蛋白,两者可能具有相同受体,并通过与受体结合发挥效应。
    Conclusion: u-PA produced by macrophages immediately activate proMMP-2 and proMMP-9,and control their activity,may play a vital role in AAA formation,dilation and rupture.
    结论 由巨噬细胞产生的u-PA直接激活,并调节MMP-2和MMP-9的活性,在AAA的形成、扩张和破裂中起着关键性的作用。
    Results:The epilentic cells were mainly composed of macrophages and fibroblasts.
    结果 :人工晶状体表面主要由巨噬细胞和成纤维细胞组成。
    Conclusion u-PA produced by macrophages may immediately activate proMMP-2 and proMMP-9, which play a pivotal role in the formation and development of AAA.
    结论由巨噬细胞产生的u-PA直接激活原MMP-2和原MMP-9,在AAA形成和扩张中起着关键性作用。
    At 3 and 6 h after lesion, iNOS was mainly produced by neurons. At 12 and 24 h after lesion, most of iNOS was produced by macrophages. But at 72 and 120 h, most of the iNOS was produced by glia cells.
    伤后iNOS阳性细胞数量也增多,伤后3、6h主要由神经细胞表达iNOS,在12、24h主要由巨噬细胞表达iNOS,而72、120h则主要由胶质细胞表达iNOS。
    In terms ofits biological effects, IL- 18 is thus closely related to andacts synergistically withIL- 12. Analysis of amino acid sequence and structural motifs,however, classify' IL-18 as a member of the IL-i family of cytokines.
    人和小鼠的IL-18主要是由巨噬细胞样细胞产生,如单核巨噬细胞,肝博士论文《白细胞介汞18的重组表达及其抗肿泊作用的研究》中的Kllpffer细胞等。
    Human Tumor Necrosis Factor a (abbreviated as hTNFa) was selected as target forign gene and it is a sort of multi-functional protein cytokine, which was generated by macrophage or monocyte under stimulus.
    本研究用人肿瘤坏死因子α(Human Tumor Necrosis Factor α,简称hTNFα)作为外源目的基因。 它是由巨噬细胞和单核细胞受到刺激后产生的一种多功能蛋白质细胞因子。
    Objective TNF-a, a cytokine with wild-ranging bioactivity, is produced by macrophage and activated T cell.
    研究背景 TNF-α是一种生物学活性十分广泛的细胞因子,它主要由巨噬细胞和活化的T细胞产生。 它通过与细胞膜表面的受体结合而发挥效应。
    Since 1962, when Gross and Lapiere discovered the first MMPs-collagenase, man had discovered various of MMPs in succession, where MMP-2 has a strong degradational effect on extracellular matrix, it is secreted bymacrophage, smooth muscle cells and endothelial cells and so on. MMP-2 will degrade the fibrous cap and collagen in the basilar membrane when it is increased in the plaque, and it is one of the important factor to make the fibrous cap thin and accelerate atherosclerosis's development.
    自从 1962年 Gross和 Lapiere发现第一种基质金属蛋白酶一胶原酶以来,人类陆续发现了很多种基质金属蛋白酶,其中的MMP—2对细胞外基质具有极强的降解作用,它由巨噬细胞、平滑肌细胞以及内皮细胞等分泌,在动脉粥样硬化斑块中憎多时降解纤维帽及基底膜的胶原等基质成分,是使纤维帽变薄及加速动脉粥样硬化的重要因素之一。
    The HHK scaffold material was degraded into small particles by ubiquitin system at first, and then those particles phagocytosed by macrophages.
    在整个降解的过程中首先是通过体内的Ub系统将HHK材料降解成微小的碎片,再由巨噬细胞等通过吞噬完成清除。
    TNF - a, produced in macrophage, T lymphocyte, mast cell , etc, is dose effective in tissue. Low level of TNF - a is beneficial to tissue repair and inflammatory response, while, chronic stimulation and massive release of TNF ?
    它由巨噬细胞、肥大细胞、T淋巴细胞等多种细胞产生,TNF-α作用与其在组织中的水平有关,低水平的TNF-α在组织修复、炎症应答中起作用,对机体有利;
    It causes degradation of extracellar matrix compound. MMPs is mainly produced by macrophage cells, T, B lymphocyte and fibroblast cells, smooth muscle cells.
    MMP主要由巨噬细胞、T、B淋巴细胞,或纤维细胞及平滑肌细胞产生,巨噬细胞产生MMP依赖于前列腺素E-2(prosostaglandin E-2,PGE2)的作用。
    In patients with fetal death in early pregnancy IAP levels were closely correlated with ratios of TNF-p/ IL-4.DiscussionIAP has a molecular weight of 50000, an isoelectric point of 3. 0, a darbo-hydrate 31.5%.
    IAP是分子量为50000,等电点为3 .0,含糖31 .5%的一种糖蛋白,主要由巨噬细胞、粒细胞等产生。 当机体受到免疫复合物或致病因子刺激时,体内IAP水平显著升高。
    Tumor necrosis factor (TNF), the cytokine secreted by macrophage and lymphocyte, has a close relationship with keratinocyte.
    肿瘤坏死因子(TNF)是一类由巨噬细胞和淋巴细胞等分泌的细胞因子,与角质形成细胞有密切的关系。
    The activation and wandering of macrophages of milky spots were observed when Chinese ink was injected into the peritoneal cavity of the mice,The resultes of the experiment showed that: the milky spots of diaphragmatic peritoneum of human were mainly composed of macrophages, The shape and size of the milky spot were not the same, The diameter of the milky spot was from 90. 5 to 361.2μm.
    用墨汁小鼠腹膜腔内注射,观察乳斑主要由巨噬细胞组成,其形态不一,多为圆形或椭圆形,直径为90.5~361.2μm。
 

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