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by macrophages
    Background: TNF-a, a cytokine with complex bioactivity, is mainly secreted by macrophages and T cells.
    研究背景TNF-α是一种具有复杂生物活性的细胞因子,它主要由巨噬细胞和T细胞产生。
    It has been proved that TNF—α(17kd,157aa) produced by macrophages and TNF—β (25kd,17aa) , a glucoprotein produced by activated T—lymphocytes, may have the same receptors, with which they combine and put into effect.
    现已证明,TNF—α(17kd,157aa)是由巨噬细胞产生的一种单核因子,TNF—β(25kd,171aa)是由活化型T淋巴细胞产生的一种淋巴因子糖蛋白,两者可能具有相同受体,并通过与受体结合发挥效应。
    Conclusion: u-PA produced by macrophages immediately activate proMMP-2 and proMMP-9,and control their activity,may play a vital role in AAA formation,dilation and rupture.
    结论 由巨噬细胞产生的u-PA直接激活,并调节MMP-2和MMP-9的活性,在AAA的形成、扩张和破裂中起着关键性的作用。
    Conclusion u-PA produced by macrophages may immediately activate proMMP-2 and proMMP-9, which play a pivotal role in the formation and development of AAA.
    结论由巨噬细胞产生的u-PA直接激活原MMP-2和原MMP-9,在AAA形成和扩张中起着关键性作用。
    At 3 and 6 h after lesion, iNOS was mainly produced by neurons. At 12 and 24 h after lesion, most of iNOS was produced by macrophages. But at 72 and 120 h, most of the iNOS was produced by glia cells.
    伤后iNOS阳性细胞数量也增多,伤后3、6h主要由神经细胞表达iNOS,在12、24h主要由巨噬细胞表达iNOS,而72、120h则主要由胶质细胞表达iNOS。
    The HHK scaffold material was degraded into small particles by ubiquitin system at first, and then those particles phagocytosed by macrophages.
    在整个降解的过程中首先是通过体内的Ub系统将HHK材料降解成微小的碎片,再由巨噬细胞等通过吞噬完成清除。
    In the endometriosis patients,the amount of peritoneal fluid and the concentration of macrophages are increased. Thus the cytokines secreted by macrophages in peritoneal(fluid) and serum are increased remarkably.
    子宫内膜异位症患者腹腔液增多,腹腔液中巨噬细胞浓度增加,腹腔液和血清中由巨噬细胞分泌的细胞因子也明显增多。
 

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