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    Pathological Observation of Schistosomiasis Hepatic Fibrosis Coincident with Hepatocellular Carcinoma
    血吸虫病肝纤维化并发肝细胞癌的病理观察
    Methods:Twenty hepatic fibrotic rabbit models were randomly divided into the HGF group(n=10) and control group(n=10).
    方法:20只肝纤维化新西兰大白兔模型,随机分为两组(HGF组,对照组),每组各10只。
    Objective To evaluate the clinical effect and the indexes of hepatic fibrosis by TACE (Transcather arterial chemoembolization) with different chemotherapy dose in advanced hepatic carcinoma.
    目的:对比观察不同化疗药物剂量经动脉导管灌注化疗栓塞术(transcather arterial chemoembolization,TACE)治疗原发性肝癌的疗效及对肝纤维化指标的影响。
    PATHOLOGICAL OBSERVATION OF SCHISTOSOMIASIS HEPATIC FIBROSIS COINCIDENT WITH HEPATOCELLULAR CARCINOMA
    血吸虫病肝纤维化并发肝细胞癌的病理观察
    The Content Changes of Serum Liver Fibrosis Indicators after Transarterial Chemoembolization for Primary Hepatic Carcinoma and Their Clinical Significance
    肝癌化疗栓塞后血清肝纤维化指标的含量变化及临床意义
    A Sutdy of Three Intervention Therapies Influence on the Five Hepatofibrosis Indexes of Middle and Advanced Hepatocellular Carcimona Serum
    三介入治疗对中晚期肝癌血清五项肝纤维化指标影响的研究
    The New Method in Inspecting Liver Injure After Repeated Transcatheter Arterial Embolizationfor Treating Hepatocellular Carcinoma:The Liver Fibrotic Indicator Determined
    监测肝癌经多次肝动脉化疗栓塞术后肝损伤的新方法——肝纤维化指标的测定
    The Significance of The Measurement of Serum Fibrosis Markers in Liver Fibrosis for The Hepatocellular Carcinoma
    原发性肝癌患者血清肝纤维化指标检测的意义
    Effect of Hepatic Fibrosis on Liver Metastasis in Cancer of Colon and It's Mechanisms
    肝纤维化对结肠癌肝转移的影响及其作用机制的研究
    27 cases of viral-induced cirrhosis with Child-Pugh A, 23 cases of viral-induced cirrhosis with Child-Pugh B, 15 cases of viral-induced cirrhosis with Child-Pugh C.
    正常对照组34例,肝炎肝纤维化组21例,肝硬化Child-Pugh A级27例,肝硬化Child-Pugh B级23例,肝硬化Child-Pugh C级15例; 共5组120例。
    1.3 Construction of liver metastasis model:After the formation of hepatic fibrosis,we divided randomly GA mice into two groups:normal liver group(group A1, GA1) and normal liver inoculated with CT26 group (group A2,GA2);
    1.3转移癌模型的构建:肝纤维化模型制成后,将A组和B组小鼠再分别分为两组,即A1组(正常肝组)、A2组(正常肝种瘤组)、B1组(肝纤维化组)和B2组(肝纤维化种瘤组),每组16只小鼠。
    Objective To develop a radioimmunoassay (RIA) of type Ⅳ collagen (CⅣ) for diagnosis of liver fibrosis and cirrhosis.
    目的建立血清Ⅳ型胶原(CⅣ)放射免疫测定法,辅助诊断肝纤维化及肝硬化。
    RESULTS These 9 cases accout for 39% of 231 cases of hepatocellular carcinoma and 46% of 195 cases of schistosomiasis hepatic fibrosis respectively diagnosed by histopathology between 1959 to 1995.Microscopically,schistosomiasis hepatic fibrosis in the early stage was found to be coexistent with hepatocellular carcinoma.
    结果该9例分别占1959~1995年间病理诊断肝细胞癌的3.9%(9/231)和血吸虫病肝纤维化的4.6%(9/195)。 镜下见早期血吸虫病肝纤维化与肝细胞癌并存。
    Result HSP47-positive cells were observed in the capsule of HCC and the fibrous septa of fibrostic liver around HCC.
    结果 人肝癌包膜、癌旁肝纤维化纤维间隔内的纤维细胞以及在纤维间隔附近肝细胞周围的星状细胞表达HSP47蛋白。
    after third TACE. the indicators of fibrosis rose significantly (P < 0. 01 ). the most significantly one is collagen type Ⅳ, It could rise significantly (P< 0. 05 ) after the first curation of TACE.
    结果:TACE治疗前后对比,三次肝纤维化指标均有不同程度的升高,以第三次TACE术后升高最明显(P<0.01),三项肝纤维化指标中,以Ⅳ·C变化最显著,第一次TACE术后即有统计学意义(P<0.05)。
    Methods By using histochemistry method to study the distribution of collagen I(Col Ⅰ), collagen Ⅲ(Col Ⅲ), desmin(DM) and α smooth muscle actin (α SMA) in hepatic fibrosis, HCC and CC.
    方法 用免疫组织化学方法分别显示I型胶原 (ColⅠ )、Ⅲ型胶原 (ColⅢ )、结蛋白 (DM)和α 平滑肌肌动蛋白 (α SMA)在肝纤维化、HCC和CC的分布。
    Results The distribution of DM, α SMA positive cells were consistent with that of Col Ⅰ and Col Ⅲ in hepatic fibrosis and liver cancer, namely, within the DM and α SMA positive cells zone, one could see collagen deposition.
    结果 在肝纤维化和肝癌中 ,DM及α SMA阳性细胞与ColⅠ、ColⅢ的分布具有一致性 ,即DM及α SMA阳性细胞的区域可见胶原沉积。
    Objective To summarize the experience in extraction of total RNA from the tissues of human hepatocellular carcinoma (HCC) and experimental hepatic fibrosis (EHF).
    目的总结一步法抽提人体肝细胞癌(HCC)癌组织及实验性肝纤维化肝组织总RNA的体会。
    Conclusions TNF α is able to induce the expression of CTGF in HSC, and this may be one of the mechanisms for TNF α involving in the liver fibrosis.
    结论 TNF α可诱导 HSC的 CTGF mRNA表达,可能是其参与早期肝纤维化形成的作用机理之一。
    Objective To investigate the change of liver fibrotic indicator in the hepatocellular carcinoma(HCC)patients after repeated transcatheter arterial infusion chemotherapy and embolization(TAE).
    目的 探讨原发性肝癌 (HCC)经多次肝动脉化疗栓塞术 (TAE)后肝纤维化指标的变化。
 

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