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    2. Alleviate the fat denaturatiDn of the liver cells.
    2、减轻肝细胞脂肪变性
    In rats treated with 45 and 150 mg/kg/day, sperm counts were decreased and there was a greater incidence of sperm morphological abnormalities.
    氟吗啉使45mg/kg/day和150 mg/kg/day剂量组的雄鼠、雌鼠发生肝脏毒性,引起肝细胞脂肪变性
    Levels of serum ALT,AST,MDA were much lower in GSH group than in NASH group and levels of liver SOD,GSH were much higher in GSH group than NASH group (P﹤0.05).
    肝脏广泛肝细胞脂肪变性,仅有轻微的炎性细胞浸润,未见坏死灶及纤维组织增生。 (4)GSH组大鼠血清TG、FFA及肝组织TG、FFA、ATP、TNFα与NASH组相比无明显变化,血清AST、ALT及MDA显著低于NASH组(P﹤0.05),肝组织GSH、SOD显著高于病理对照组(P﹤0.05);
    Results: With TFGs treatment, contents of serum TC、 LDL-C、T Bili of AFL rats were significantly decreased;
    结果:TFGs能显著改善AFL大鼠肝细胞脂肪变性; 显著降低AFL大鼠血清TC、LDL-C(p<0.01)、T Bili(中剂量p<0.05,大剂量p<0.01)水平,但大剂量升高血清TG水平;
    To establish a model of hepatocyte steatosis, we incubate the normal human L-02 hepatocyte in RPMI-1640 complete medium with 50% fetal bovine serum for 24 hours.
    用50%胎牛血清的RPMI-1640完全培养基孵育人肝L-02细胞24小时,制备肝细胞脂肪变性模型。
    In high fat-induced fatty liver quails, GSH-PX in liver was significantly improved (P<0.05 or P<0.01) .
    显微镜检查结果也显示,给予蛇床子素治疗的肝细胞脂肪变性程度明显减轻(P<0.05或P<0.01)。
    Results Plasma PAI 1 activity increased in model group at 6 and 12 weeks, and was significantly higher than normal group at 12 weeks. PAI 1 mRNA relative values of hepatic tissue were also higher than normal group (3.474±0.051 vs 1.210±0.031, P <0.01).
    结果 与正常组相比 ,模型组家兔血浆脂质和PAI 1活性在实验 6周时即显著增高 ,实验结束时模型组肝脏呈重度肝细胞脂肪变性 ,肝组织PAI 1mRNA表达较正常组显著增强。
    Results HE staining and electron microscopic examinations revealed that in the model group, the normal hepatic architecture disappeared and pseudo-lobule formation was observed.
    结果 HE染色及电镜观察显示 :模型组肝脏失去正常结构 ,部分假小叶形成 ,而肝炎平组肝细胞脂肪变性和肝纤维化明显减少。
    the concentrations of BUN and Cr, activities of ALT and AST decreased (P< 0.05); an d the fatty degeneration of hepatocytes improved (P< 0.01).
    降低病鼠ALT、AST活性和BUN、Cr含量 (P<0 .0 5 ) ,改善肝细胞脂肪变性 (P <0 .0 1)。
    RESULTS: Light microscopy in HE staining showed that liver fatty score was significantly lower in the breviscapine group compared with model ani mals (0.55±0.43 vs 1.54±0.65, P<0.01).
    结果:HE染色显示模型组部分肝细胞脂肪变性,评分为1 .5 4±0 . 6 5 ,灯盏花素给药组评分为0 . 5 5±0 . 4 3,差异高度显著(P <0 . 0 1)。
    and the expression of TLR-4 protein was determined by immunohistochemistry method. ResultsRSM could alleviate the fatty degeneration and adiponecrosis of hepatic cells induced by alcohol, down-regulate the expressions of TLR-4 mRNA and HO-1 mRNA, and significantly decrease the number of TLR-4 positive cells.
    结果丹参能减轻酒精所引起的肝细胞脂肪变性、坏死,下调TLR4mRNA表达及HO 1mRNA表达,并能使TLR4阳性细胞数明显减少。
    However, TP and EGCG treatment improved histological changes, and significantly decreased the levels of plasma endotoxin and serum TNF-α, IL-1, IL-18, as well as the expression of CD14, LBP, TNF-α, IL-1 and IL-18 mRNA (P < 0.05 or P < 0.01). Fatty degeneration was still observed in TP and EGCG treatment group, but focal necrosis and inflammatory cell infiltration disappeared.
    茶多酚、EGCG高低剂量分别同时处理显著降低了酒精性肝损伤大鼠血清ALT、TNF-α、IL-1、IL-18与血浆内毒素水平及肝组织CD14、LBP、TNF-α、IL-1、IL-18mRNA的表达(P<0.05或P<0.01),肝组织仍可见肝细胞脂肪变性,但未见坏死及炎性细胞浸润.
    ①Lobules of liver in the rats of model group was disorder with a pile of deposition of fibrous tissue, inflammatory cell infiltration and fatty degeneration of hepatocytes.
    ①模型组大鼠肝小叶结构紊乱,有大量纤维组织沉积,炎症细胞浸润,肝细胞脂肪变性
    After administerd repeatedly for 180 days,in the 9.0 g/kg dose groups,the TP and Crea increased(P<(0.01)orP<0.05);
    2只大鼠肝细胞脂肪变性。 给药180 d时,9.0 g/kg和4.5 g/kg剂量组肌酐升高(P<0.01或P<0.001);
    Histopathologically, betaine significantly decreased the degree of steatosis (P<0.05 or P<0.01),and betaine 0.8 g/kg had a tendency to normalize basically the structure of hepatocyte.
    组织病理学检查结果显示,甜菜碱可使肝细胞脂肪变性程度明显减轻(P<0.05或P<0.01),0.8g/kg组的肝细胞结构趋于正常。
    Compared with the controls, pathological results showed that betaine significantly decreased the degree of steatosis(P<0.05 or P<0.01).
    病理检查结果显示给予甜菜碱后可使肝细胞的脂肪变性程度明显减轻(P<0.05或P<0.01)。
    ⑤The hepatic steatosis of the model group was more severe compared with the control group (P < 0.05),the training group was obviously lighter than the model group (P < 0.05),but still more serious than the control group (P < 0.05).
    ⑤模型组肝细胞脂肪变性程度显著重于对照组(P<0.05),运动组显著轻于模型组(P<0.05),但仍然显著比对照组严重(P<0.05)。
    Results Spotty hepatic steatosis and simple fatty liver were observed in the model group at 4th and 8th weeks,respectively. From 12th week to 24th week,the livers gradually progressed from steatohepatitis to fibrosis.
    结果模型组大鼠4周肝脏可见散在性肝细胞脂肪变性,8周为单纯性脂肪性肝炎,12~24周从脂肪性肝炎进展为脂肪性肝炎伴肝纤维化。
    and morphological changes of hepatocytes were observed under microscope with Oil-Red-O staining.
    采用Oil-Red-O染色观察肝细胞脂肪变性
    (4)Oil-Red-O staining showed that significant lipid vacuoles primarily occurred in periportal areas of liver lobule in rats treated with VPA or VPA added with PB. The number of hepatocyte containing lipid droplets in infant rats was 9.2 times higher than that in control,and 15.7 times higher than that in VPA added with PB group.
    (4)Oil-Red-O染色显示,VP/VPA+PB组均出现门管区为主的肝细胞脂肪变性,脂肪细胞数分别高出对照组9.2倍和15.7倍,差异非常显著(P<0.01)。
 

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