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    Results: In RAFE and AA Groups, the expression of CK on renal tubular cells is declined comparing with the Control Group, and the enhanced expression of α-SMA and Vimentin can be observed on tubular cells.
    结果:经图像分析,RAFE 和AA组肾小管上皮细胞CK较对照细表达减弱,而α-SMA在肾小管上皮细胞及肾间质细胞呈弱阳性表达、Vimentin表达较对照组明显增多;
    Effect of Shen Luo-ning on Extracellular Matrix Metabolism of Kidney Interstitial Tissue in IgA Nephrosis Rats
    肾络宁对IgA肾病大鼠肾间质细胞外基质代谢的影响
    These findings strongly suggest that renal interstitial fibrogenesis is mediated by CTGF that is expressed in the tubular epithelial cells.
    因此,我们在进一步探寻更有效的基因治疗方法来下调病理状态下肾小管上皮细胞CTGF的表达,以期延缓肾间质细胞外基质的积聚。
    In renal fibrotic tissues, the expression pattern of TGF- 1 was similar to HSP47. (3) The expression level of HSP47 was positively correlated with COL-IV, TGF- 0 1 and a -SMA.
    其中,TGF-β1表达特点与HSP47相似,亦主要定位于小管上皮细胞、肾间质细胞及肾小球内。
    Compared with the inactivation of ERK and p38, phosphorylation of JNK was observed in glomeruli, renal tubules and interstitial cells in PAN rats, which was also inhibited by A&A treatment.
    PAN大鼠肾小球、肾小管及肾间质细胞的JNK均明显活化 ,A&A可明显抑制上述部位的JNK活化。
    The a-SMA expression in interstitium increased progressively starting at day 7. By using consecutive renal tissue slicing, the enhanced expression of a-SMA in renal interstitium can be observed in the circumference of renal tubular cells with vimentin-positive.
    通过连续切片观察到,vimentin阳性的肾小管周围肾间质细胞a-SMA表达增强,并与肾小管间质纤维化程度基本一致。
    ② The number of Bcl-2 positive interstitial cells in LN was increased,and was positively correlated with the degree of interstitial infiltration of inflammatory cells.
    ②LN肾间质细胞Bcl 2表达增高 ,与间质炎症细胞浸润程度正相关。
    Results The expression of CTGF was detected in tubular epithelial cells and interstitial cells in chronic damaged tissues.
    结果CTGF主要表达于肾小管上皮细胞和一些肾间质细胞胞浆,CTGF表达量与肾小管间质病变程度成正比。
    After administration of A&A(12 g·kg·d~(-1)) for 0,3,7 and 10 d,renal damage including interstitial cell infiltration,tubular atrophy and interstitial fibrosis were quantified on HE and Masson stained tissue sections.
    在给药(A&A 12 g.kg-1.d-1)后d 0、3、7、10处死动物,观察肾功能变化,对肾间质细胞浸润、肾小管萎缩和肾间质纤维化情况进行半定量评分。
    Result: In RAFE and AA Groups,the expression of CK on renal tubular cells is declined comparing with the Control Group,and the enhanced expression of α-SMA and Vimentin can be observed on tubular cells.
    结果:经图像分析,RAFE和AA组肾小管上皮细胞CK较对照组表达减弱,而α-SMA在肾小管上皮细胞及肾间质细胞呈弱阳性表达、V im entin表达较对照组明显增多;
    EFFECTS OF SINOMENINE ON TRANSDIFFERENTIATION OF RENAL INTERSTITIAL CELLS IN MOUSE WITH UNILATERAL URETERAL OBSTRUCTION
    抗风湿药青藤碱对UUO小鼠肾间质细胞表型转化的影响
    The semi-quantitative analysis results for immunohistochemical staining showed that TGF- 3 1, a -SMA and collagen I expression remarkbly increased in the obstructed kidneys of both group B and C compared with group A(P<0.01).
    a石MA只在正常大鼠肾组织血管壁上表达,小球和小管中无阳性染色:B组和C组除血管壁上有表达外,可见大量QEMA阳性染色出现在肾间质细胞胞浆中,同时部分小管上皮细胞胞浆内也出现阳性染色颗粒; 与A组比较显著增多0<0刀1),但C组明显少于B组0<0刀5)。
    Results: The increased expression of Bax and PCNA proteins in the tubular andinterstitial cells, and the increased expression of TGF-pl proteins in the renal interstitium and glomeruli of the obstructed kidneys were detected compared with the sham-operated group.
    结果:UUO大鼠肾小管上皮细胞、肾间质细胞中Bax、PCNA蛋白表达显著增多,肾间质、肾小球中TGF-β1蛋白表达显著增多。
    Light microscopy shows that there are vacuolation, granular degeneration , necrosis in renal tubule epithelial cells at the end of week 6 and getting worse at the end of week 12. Electron microscope shows that there were the degeneration , necrosis on the microvillus and edema in mitochondrion of proximal tubule cells.
    12周可见肾小管上皮细胞中/重度空泡变性或颗粒变性,大量坏死,肾间质明显水肿,可见轻/中度纤维化; 电镜可见近曲小管管腔微绒毛大面积变性坏死,线粒体高度水肿,肾间质细胞稀疏排列,毛细血管增多、充血,间质胶原纤维轻/中度增生。
    Incontrast, there was high HSP47 expression in the tubular epithelial cells, renal interstitial tissue, glomerular epithelial and mesangial cells of trial groups. The intensity of HSP47 expression was positively correlated with the degree of glomerulosclerosis and tubulointerstitial injury(P<0.01).
    而在慢性肾脏疾病患者的病变肾组织中,HSP47表达显著增强,主要表达于病变区小管上皮细胞、肾间质细胞及肾小球系膜细胞和上皮细胞,其表达量随肾组织纤维化程度加重而明显增加(P<0.01)。
    It’s not difficult to see that cytokine and renal interstitial cells play a important role in RIF and recent study shows that too.
    从上述过程不难看出细胞因子和肾间质细胞在肾间质纤维化中的重要作用,晚近大量研究资料也证实了这一点。
    Ultrastructures in renal interstitial cells of two groups of different-aged rats were observed in this paper. The results showed that there were changes with age in renal interstitial cells between two groups,Those charges mainly appear as an increase in the number of cells, cytoplasmic processes organelles, and in cytoplasm, in the adult group,The narrowness of perinuclear cisternae
    本文观察了二组不同年龄大鼠肾间质细胞的超微结构,结果表明,肾间质细胞有增龄变化,主要包括:细胞数量、胞质、胞质突起、细胞器增多,核周池缩小。
    Renal tissues of 14 patients with acute renal tubulointerstitial disease were studied by using immunohistochemical method. Cells infiltrated in interrenal tissue were observed.
    对14例病理诊断为急性肾间质小管性疾病的肾活检组织标本采用免疫组化法对肾间质细胞浸润进行了研究。
    Objective To investigate the way of interstitial cell loss and its possible relation to Fas expression during the course of renal interstitial fibrosis.
    目的 探讨肾间质纤维化形成过程中的肾间质细胞凋亡及其与Fas 表达的关系。
    Conclusion The apoptosis in this model may be pathogenetically related to interstitial cell loss in the course of renal interstitial fibrosis and the apoptosis is probably mediated by Fas/FasL system.
    结论 细胞凋亡可能为该模型中肾间质细胞丢失的原因,且Fas/FasL系统可能介导了该细胞凋亡。
 

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