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severe viral hepatitis
    A Discussion on the Causes of Death in Severe Viral Hepatitis A Clinical and Pathologic Analysis of 223 Fatal Cases
    重症病毒性肝炎的死亡原因探讨(223例死亡病例的临床和病理分析)
    Analysis of Plasma Amino Acids in Patients with Severe Viral Hepatitis and Its Clinical Significance
    重症病毒性肝炎患者血浆氨基酸分析及其临床意义
    Analysis of the Causes of Death in 281 Cases of Severe Viral Hepatitis
    281例重症病毒性肝炎死亡原因分析
    Efficacy of glucagoninsulin therapy in severe viral hepatitis.
    胰高糖素——胰岛素治疗重症病毒性肝炎疗效观察
    Analysis of Prognostic Factors in Severe Viral Hepatitis
    影响重症病毒性肝炎预后有关因素的分析
    Severe viral hepatitis complicated with MSOF: Clinical analysis of 102 cases
    重症病毒性肝炎并发多系统脏器衰竭102例临床分析
    Relationship Between Complement Activity and Liver Necrosis in Severe Viral Hepatitis
    重症病毒性肝炎补体系统激活与肝坏死的关系及意义的研究
    The Changes of Serum Levels of TNF-α and IL-6 and Its Significance in Patients with Severe Viral Hepatitis
    重症病毒性肝炎患者血清TNF-α和IL-6水平变化及其意义
    Alprostadil in the Treatment of Azotemia Resulted from Severe Viral Hepatitis or Cirrhosis of the Liver
    前列地尔治疗重症病毒性肝炎肝硬化所致氮质血症
    Expression of Collagen and PDGF-1 of Liver Tissue in the Patients with Severe Viral Hepatitis
    重症病毒性肝炎肝组织胶原蛋白和血小板衍生生长因子-1的表达
    Fuzzy Pattern Recognition in Prediction of Severe Viral Hepatitis Progression
    模糊模式识别在重症病毒性肝炎病情转归中的应用
    In this paper the serum levels of TNF-a and IL-6 in 30 patients with severe viral hepatitis (SH) were measured by ELISA.
    应用双抗体夹心法检测 30例重症病毒性肝炎(SH)患者血清TNF-α和IL-6水平。
    Objective To study the molecular mechanism of fibrosis in the patients with severe viral hepatitis by detecting the expressions of procollagen I mRNA and platelet-derived growth factor-1 (PDGF-1) mRNA and type Ⅰ ,Ⅳ collagen.
    目的 了解重症病毒性肝炎患者肝组织I型前胶原mRNA、血小板衍生生长因子-1(PDGF-1)mRNA及Ⅰ、Ⅳ型胶原蛋白的表达情况,探讨重症病毒性肝炎纤维化发生的分子机制。
    Results The expressions of procollagen I mRNA and PDGF-1 mRNA and type Ⅰ , Ⅳ collagen were significantly higher in the patients with severe viral hepatitis than those in controls.
    结果 与正常肝组织相比,重症病毒性肝炎患者的肝组织Ⅰ型前胶原和PDGF-1 mRNA的表达显著增加,Ⅰ、Ⅳ型胶原蛋白大量沉积。
    Conclusion The increase of PDGF-1 mRNA and procollagen I mRNA expression which leads to collagen deposition is one of molecular mechanisms of liver fibrosis in severe viral hepatitis.
    结论 PDGF-1 mRNA与Ⅰ型前胶原mRNA表达的增加是导致肝组织胶原蛋白合成增加,重症病毒性肝炎发展成肝纤维化乃至肝硬化的原因之一。
    The comparison of the seven parameters of liver function of the 69 patients with severe viral hepatitis which were divided into 2 groups according to curable or incurable,suggested that the levels of serum total bilirubin and albumin be significant for judging the necrosis of liver celt and the progno- sis of the patients.
    通过69例重症病毒性肝炎有效无效两组病例的肝功能7项检测指标对比研究结果认为血清总胆红素、白蛋白对判断肝坏死程度及预后有较大意义。
    NI in 100 cases with severe viral hepatitis were re-corded.
    本文对1986年1月至1989年12月我院收治的重症病毒性肝炎100例作院内感染调查,总发生率为70%。
    The microcircuiation of nailbed in 60 case of patients with childhood viral hepatitis were observed. The results showed that there were abnomal in 37 case of patients,the incidence of the changes of the microcirculation in chronic and severe viral hepatitis were more than others.
    本文观察了60例各型小儿病毒性肝炎的甲襞微循环,查出甲襞微循环异常者37例,以慢性、重症病毒性肝炎的微循环障碍发生率为高,且黄疸的深度与微循环障碍发生率成正相关。
    Objective To study the therapeutic effect of blood activing polypeptide on chronic severe viral hepatitis (CSH) and the method used.
    目的 探讨血活素治疗慢性重症病毒性肝炎 (CSH)的疗法。
    Methods The specimens of liver tissues in 20 cases of severe viral hepatitis and 6 cases without liver disease were detected for procollagen Ⅰ mRNA and PDGF-1 mRNA by situ hybridization, and for type Ⅰ , Ⅳcollagen by immunohistochemical staining. The total collagen was detected by Sirius red staining.
    方法 将重症病毒性肝炎肝组织标本20例,非肝病患者肝组织6例,用原位杂交法检测肝组织I型前胶原和PDGF-1 mRNA的表达,用免疫组化法观察肝组织Ⅰ、Ⅳ型胶原蛋白的沉积、分布和含量,用天狼红染色观察肝组织总的胶原含量。
 

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