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    So, modulating the metabolism and effects of PAF (e.g., blocking the PAF receptor) may become important strategies of intervention of Alzheimer's disease, HIV-associated dementia or post-ischemic neuronal death.
    调控PAF的代谢及其效应(如阻断PAF受体)将成为干预阿尔茨海默病、HIV相关性痴呆以及脑缺血的重要策略。
    Effects of naoyikang on capability of learning and memory in Alzheimer’s disease model mice
    脑益康对阿尔茨海默病模型小鼠学习记忆的影响
    Effects of olanzapine and quetiapine on the amyloid beta-protein 42 secretion in transfected N2a cells of Alzheimer disease related genes
    奥氮平与喹硫平对阿尔茨海默病相关基因共转染N2a细胞分泌淀粉样β蛋白42的影响(英文)
    Protective Effects of Dipfluzine on the Brain in Alzheimer's Disease Rats and Naturally Aged Rats
    双苯氟嗪对阿尔茨海默病大鼠及自然衰老大鼠的脑保护作用
    Antagonistic Effect of Melatonin on Alzheimer's Disease-like Cell and Phosphorylated Tau Protein
    褪黑素抗阿尔茨海默病样细胞效应和磷酸化tau蛋白
    Experimental Study on Prevention and Treatment of Chemical Drugs Induced Alzheimer's Disease with Vitamin E and Tetramethylpyrazine
    维生素E和川芎嗪防治化学药物诱导的阿尔茨海默病的实验研究
    Alzheimer's disease (AD) is a disorder associated with progressive degeneration in memory and in the recognition functions of old people.
    早老性痴呆即阿尔茨海默病(Alzheimer's Disease,AD)是一种以进行性高级认知功能障碍和记忆功能丧失为特征的老年性疾病。
    Objective To observe the expression of nicotinic acetylcholine receptor(nAchR) in the brain's fimbria/fornix transected rats,the effect of ectogenic estrogen on it,and its relation with Alzhelmer's disease(AD).
    目的以双侧穹隆-海马伞切断制作阿尔茨海默病(AD)大鼠模型,观察脑内海马CA1区和皮层区烟碱型乙酰胆碱受体(nAchR)表达的变化,探讨与AD相关的发病机制,并观察雌激素的干预作用。
    The current hypothesis for the cause of this disease is that it is the result of aberrant production of β-amyloid (Aβ) and plaque deposition in the brain. Aβ_ 42 , less soluble and forming the major component of the amyloid plaques, is generated via the cleavage of β-amyloid protein precursor (β-APP) by γ-secretase, a key enzyme in the production of Aβ.
    近来研究认为阿尔茨海默病的产生主要起因于β淀粉样蛋白(β-amyloid,Aβ)在大脑内的沉积,而由γ-分泌酶切割β淀粉样前体蛋白产生的疏水性Aβ42是形成Aβ的主要原因,γ-分泌酶是防治阿尔茨海默病很有潜力的靶点。
 

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