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异常释放
    100% in MODS group. Conclusions The abnormal release of the TNF-α? IL-1β?
    结论 细胞因子TNF -α、IL - 1β、IL - 6、IL - 8、IL - 10的异常释放可能参与了危重病MODS的病理过程。
    Hypothalamic Orphanin FQ Participates in Neuroendocrinologic Mechanism of Electroacupuncture Normalizing Abnormal Release of Pituitary LH in Ovariectomized Rats
    下丘脑孤啡肽参与电针调整去卵巢大鼠LH异常释放的神经内分泌机制
    Findings of this serial studies strongly suggested that the uncontrollable "mediator disease" resuIted from ischemic-reperfusion damage, swelling and edema of visceraI tissues, the endothelial cell damage and abnormal release mediators might be of importance in the pathogenesis of early PB organ damage。
    缺血和再灌流损伤、脏器水肿、内皮细胞损伤及多种炎症介质的异常释放,导致难以控制的介质病,是脏器损害的重要因素;
    Objective To study the effect of extracorporeal circulation on the abnormal release of proinflammatory and anti inflammatory cytokines, and explore their effect on systemic inflammatory response after operation.
    目的 研究体外循环术对致炎性细胞因子和抗炎性细胞因子异常释放的影响 ,并探讨其在术后炎症反应中的作用。
    Conclusions:Ionic channel antagonist ketamine can inhibit partly the abnormal release of NO in cerebral ischemia,and the change of NO induced by early cerebral ischemia might be the stress of brain on environmental stimulation.
    结论 :一定量的离子通道拮抗剂氯胺酮能部分抑制缺血性中枢神经元一氧化氮的异常释放 ,且早期脑缺血诱生一氧化氮的异常升高很可能是脑组织对外界刺激的应激反应
    This paper briefly presents the sources,release amount and reasons of abnormal release of 14C in Heavy Water Reactor based on the control strategies of operating CANDU plants. This paper also places emphasis on describing some control measures for 14C applied in Third Qinshan Nuclear Power Company.
    本文依据加拿大已运行的CANDU堆型核电站对14 C排放的管理策略 ,简要介绍了重水堆核电站14 C的来源 ,向环境的释放量以及异常释放的原因和控制释放的经验并着重介绍了秦山三期 (重水堆 )核电站对14 C排放的管理所采取的控制措施。
    On the study of such abnormal release,an technology would be found to pre-warm the outburst of coal and gas effectively.
    通过研究氢气氦气的异常释放,有可能建立一种联测技术有效地预警煤与瓦斯的突出
    Conclusions The abnormal release of TNF-α,IL-1β,IL-6,IL-10 play an important role in the pathophysiology of MODS with elderly patients. To maintain the balance of cytokines may be a method of treatment in future.
    结论 细胞因子TNF -α、IL - 1β、IL - 6、IL - 10的异常释放参与了老年MODS的病理生理过程,选择不同时机以纠正细胞因子的失衡可能是今后的救治方向。
    BACKGROUND: As unspecific antagonist of opiate receptor, naloxone is widely used for multiple diseases which are related with abnormal release of endogenous opium.
    背景:纳洛酮作为阿片受体的非特异性拮抗剂,已被用于治疗内源性阿片类物质异常释放有关的多种疾病。
 

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