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谷氨酰胺
    Methods Thirty nine severe burn patients with total burn surface area of 30%~60% and full thickness burn area of 20%~50% were randomly divided into 2 groups: control (C, 19 patients) and glutamine treatment group (GLN group, 20 patients).
    方法 采用随机对照双盲试验 ,将烧伤总面积在 30 %~ 60 % ,Ⅲ度烧伤面积在 2 0 %~ 50 %的 39例患者随机分为谷氨酰胺治疗组 (GLN组 ,2 0例 )和安慰剂对照组 (C组 ,1 9例 )。
    Methods:39 severe burn patients (total burn surface area was 30%~60%,full thickness burns area was 10%~30%) were randomly divided into two groups:control group(C group,19 patients) and glutamine treatment group (Gln group,20 patients).
    方法 :采用随机双盲对照法 ,将烧伤总面积在 30 %~ 6 0 %、三度烧伤面积在 10 %~ 30 %的 39例病人 ,随机分为谷氨酰胺治疗组 (Gln组 2 0例 )和安慰剂对照组 (C组 19例 )。
    METHODS:39 severe burn patients(total burn surface areas 30%~60%,full thickness burn areas 20%~50%) were randomly divided into two groups:control group(C group,19 patients) and glutamine treatment group(GLN group,20 patients).
    方法 :采用随机双盲对照法 ,将烧伤总面积在30 %~60 %、Ⅲ°烧伤面积在20 %~50 %的39例患者随机分为谷氨酰胺治疗组 (20例 )和安慰剂对照组 (19例 )。
    Methods Eighty-eight Wistar rats were inflicted with 30% total body surface area full thickness burns and randomly divided into two groups: namely burned control (B) and glutamine treatment(GLN) group.
    方法 采用 3 0 %体表面积Ⅲ度烧伤大鼠模型 ,将 88只Wistar大鼠随机分为烧伤对照 (B)组和谷氨酰胺 (GLN)组。
    Methods 50 male Wistar rats were randomly divided into three groups:control(SL,n=10),obstructive jaundice group(OJ,n=20) and glutamine treatment group(TG,n=20).
    方法:健康雄性Wistar大鼠50只,随机分成空白对照组(SL,n=10)、阻塞性黄疸组(OJ,n=20)和谷氨酰胺治疗组(TG,n=20)。
    [Methods] Fifty male wistar rats were randomly divided into three groups: control group(n=10)、obstructive jaundice group(n=20) and glutamine treatment group(n=20).
    [方法]健康雄性Wistar大鼠50只,随机分成空白对照组(SL,n=10)、阻塞性黄疸组(OJ,n=20)和谷氨酰胺治疗组(TG,n=20)。
    Methods Eighty-eight Wistar rats were inflicted with 30% total body surface area full thickness burns and then randomly divided into burned control (B) group and glutamine treatment (GLN) group.
    方法采用30%体表面积Ⅲ°烧伤大鼠模型,将88只Wistar大鼠随机分为烧伤对照组和烧伤后补充谷氨酰胺(GLN)组。
    Methods Fifty male Wistar rats were randomly divided into three groups: Control group (n=10), obstructive jaundice group (n=20) and glutamine treatment group (n=20).
    方法健康雄性Wistar大鼠50只,按随机数字表随机分成空白对照组(n=10)、阻塞性黄疸组(n=20)和谷氨酰胺治疗组(n=20)。
    Conclusion Growth hormone or glutamine treatment prevents intestinal mucosal atrophy possibly by up regulating expression of c fos and c jun in intestinal mucosa to promote intestinal cell proliferation. Moreover combined use of these two gut trophic factors can produce synergistic effects.
    结论 生长激素或谷氨酰胺单独应用均可通过上调小肠粘膜上皮细胞c fos和c jun基因的表达 ,促进细胞分裂增殖 ,从而防止小肠粘膜萎缩的发生 ,且两者联合应用具有协同增效作用
    Conclusions There are hypermetabolism reaction after severe burn injury, and glutamine treatment could reduce the hypermetabolic reaction. It may be the mechanism of glutamine effect that reducing catabolism hormone release and promoting anabolism hormone secretion.
    结论 烧伤大鼠代谢率明显增高 ,服用谷氨酰胺能有效降低烧伤后高代谢反应 ,其机制与谷氨酰胺能促进合成激素分泌 ,降低分解激素释放有关
    Methods Wistar rats inflicted with 30 % total body surface area(TBSA) full thickness thermal injury were randomly divided into three groups, i.e. burn with enteral nutrition(EN), burn with glutamine treatment (GLN), and normal control(C) groups.
    方法 采用 30 %体表面积 度烧伤大鼠模型 ,随机分成伤前对照 (C)组、普通肠道营养 (EN)组及谷氨酰胺强化的肠道营养 (GL N )组。 EN和 GL N组除是否给予谷氨酰胺外 ,其它条件均相同。
    Methods Eighty-eight Wistar rats were inflicted with 30% total body surface area full thickness burns and randomly divided into two groups of normal enteral nutrition (B) and enteral nutrition supplemented with glutamine treatment(Gln) group.
    方法:将88只Wistar大鼠随机分为烧伤后常规肠内营养支持组(B)和谷氨酰胺(Gln)强化的肠内营养组,并与烧伤前指标作对照(PBD0)。
    Methods Twenty-four male SD rats were randomly divided into three groups as control group(n=8),endotoxin group(n=8)and glutamine treatment group(n=8). Heart was isolated from rats and perfused on Langendorff apparatus with Krebs- Henseleit(KH)buffer(Saturation 95%+5% CO_ 2)at a constant pressure( 8.33 kPa)and temperature( 37 ℃).
    方法健康SD雄性大鼠24只,随机分成3组:正常对照组,内毒素组,谷氨酰胺+内毒素组,在Langendorff装置上用KH液对大鼠离体心脏行主动脉逆灌注。
    INTERVENTIONS Only nutritional support was given in the control group while the treatment group received also glutamine treatment.
    干预:对照给予单纯营养支持,治疗组给予谷氨酰胺治疗。
    During the first and second week after administration of glutamine, the serum TNF-α of glutamine treatment group was statistically higher than that in control group and obstructive jaundice group. Meanwhile, glutamine treatment group showed statistically lower serum level of IL-10, TBIL, ALT and AST than obstructive jaundice group.
    而在应用谷氨酰胺后1、2周血清TNFα较空白对照组和阻塞性黄疸组有明显升高,IL10、TBIL、ALT及AST较阻塞性黄疸组明显下降,细菌移位较阻塞性黄疸组也明显减少。
    There were statistically less bacteria translocations in glutamine treatment group than those in obstructive jaundice group. Conclusion Glutamine can increase the immune function by changing serum concentration of TNF-α, IL-10 and decrease the bacteria translocation.
    结论谷氨酰胺能够改变阻塞性黄疸大鼠血清TNFα、IL10功能的变化,增强大鼠免疫功能,减少肠道细菌移位。
    Conclusions Glutamine treatment increased Bcl-2 mRNA expression and decreased Bax mRNA expression, as a result, the ratio of Bax and Bcl-2 mRNA expression decreased.
    结论谷氨酰胺使内毒素血症小肠Bcl-2基因表达增强,明显高于正常; Bax基因表达减弱至接近正常;
 

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