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亚低温
    Experimental Study of Moderate Hypothermia After Traumatic Injury of Rat Spinal Cord
    亚低温治疗大鼠脊髓损伤的实验研究
    Establishment of Severe Traumatic Brain Injury Model Induced by Fluid Percussion in Rabbits and the Effects of Posttraumatic Mild Hypothermia Therapy
    兔液压冲击重型脑创伤模型的建立及亚低温治疗的影响
    A Study of Cell Membrane Damage after Severe Traumatic Brain Injury and the Effect of Hypothermia on It
    重型脑创伤后细胞膜损伤及亚低温对其影响的研究
    Effects of hypothermia for experimental brain injury and edema
    亚低温对实验性颅脑伤中神经功能的保护作用
    Effect of Mild Hypothermia on the Content of Malondialdehyde (MDA) and Activities of Superoxide Dismutase (SOD) in Brain Tissue after Complete Cerebral Ischemia Reperfusion in Dogs
    亚低温对完全性脑缺血再灌注后丙二醛含量和超氧化物歧化酶活性的影响
    Methods Seventy-five neonate rats(subjected to unilateral left carotid artery ligation and given 8%oxygen for 2h) were randomly allocated to five groups:sham operation group(A),model group(B),GM1 group(C),hypothermia group(D),GM1 and hypothermia group(E).
    方法用75只新生7 d的SD大鼠制作缺氧缺血(HIBD)模型,随机分为假手术(A)组、模型(B)组、神经节苷脂GM1(C)组、亚低温(D)组、神经节苷脂与亚低温联合干预(E)组共5组,每组15只。
    Results:Compared with normothermia group,the neurological grade in hypothermia group greatly decreased at 4 ~8 d(P<0.05).
    结果:亚低温组于4~8d神经功能评分减低与常温组相应时间点比较有显著性差异(P<0.05);
    The use of mild hypothermia may decrease the brain damage,which might be related to the buildup of expression of MAP-2 and S100β.
    缺血早期应用亚低温可减轻脑组织损伤,促进脑缺血后神经功能恢复,其机制可能与增强脑组织中MAP-2和S100β表达有关。
    The dogs in the hypothermia group were treated by intravenous infusion of cold physiological saline at(1.2±0.34)℃(5% body weight) during the operation and their body temperatures were decreased to 33~35℃ with ice blanket after the operation for 48 hours.
    亚低温组在术中采用静脉内输入1.2℃±0.34℃冷冻生理盐水,输液量为体重的5%,术后采用降温毯降温,体温控制在33~35℃,持续48h。
    Results The dogs' vital signs were stable and the mortality was 40%(12/30) in mild hypothermia group.
    结果亚低温组动物生命体征平稳,死亡率40%(12/30);
    group C hypothermia (31.5-32℃) + LPS (5 mg·kg-1) and group D hyperthermia (39.5-40℃) + LPS (5 mg·kg-1) .
    亚低温+内毒素组(C组)静脉注射LPS 5 mg·kg-1,维持直肠温31.5~32℃; 高温+内毒素组(D组)静脉注射LPS 5 mg·kg-1,维持直肠温39.5~40℃。
    ResultsThe percentage of recanalization was 87.5% in the hypothermia group, 75% in the TCD+UK group, significantly higher than that in the UK group (37.5%)(P<0.05).
    结果局部亚低温组血管再通率为87·5%,TCD+尿激酶组血管再通率为75%,均高于尿激酶组(P<0·05);
    Objective To study the effects of mild hypothermia on aquaporin 4 (AQP4) expression and blood brain-barrier permeability in rat focal infarcted cerebral tissues,and investigate the mechanisms of mild hypothermia on ischemic brain edema.
    目的观察脑梗死后亚低温干预对大鼠水通道蛋白4(aquaporin 4,AQP4)表达及血脑屏障(blood brain-barri- er,BBB)通透性的影响,探讨亚低温对缺血性脑水肿的作用机制。
    Results The death cases in room temperature group were 16 (47.5%),which in hypothermia group were 14 (33.3%) and hypothermia combined with artificial ventilation group were 19 (29.7%).
    结果常温组死亡16例(47.5%),亚低温组死亡14例(33.3%),亚低温+机械通气组死亡19例(29.7%),3组间较好疗效依次为:亚低温+机械通气组、亚低温组、常温组,P均<0.05;
    The neuro-function impairement score (0~4) of the sacrificed animals was noted. Results Neurological impairement score was significantly lower in the mild hypothermia group than that in the normothermia group on 4d, 6d and 8d (P<0.05).
    结果:亚低温组于4d、6d、8d神经功能评分减低与常温组相应时间点比较有显著性差异(P<0.05);
    Ice bags were used for total body hypothermia,and body temperatures were maintained between 31 to 33℃ in the mild hypothermia group;
    亚低温组用冰袋全身降温,维持体温在31~33℃;
    Results The levels of glucose in the brain microdialysate:(0.44±0.08)mmol/L in the normal temperature group<(0.53±0.03)mmol/L in the mild hypothermia group<(0.74±0.06)mmol/L in the control group;
    结果脑微透析液中葡萄糖水平:常温组(0.44±0.08)mmol/L<亚低温治疗组(0.53±0.03)mmol/L<对照组(0.74±0.06)mmol/L;
    L/P ratio: 28±12 in the mild hypothermia group<35±10 in the control group<108±26 in the normal temperature group.
    L/P比值:亚低温治疗组(28±12)<对照组(35±10)<常温组(108±26)。
    Results Compared with the normothermia group,focal mild hypothermia greatly increased the expression of Survivin and obviously decreased the expression of NF-κBP65 in penumbra(P<0.05orP<0.01).
    结果相同缺血再灌注时间点,亚低温组比常温组缺血侧Survivin表达水平显著增高(P<0.05),NF-κBP65核内移明显降低(P<0.01)。
    Methods Cases were divided into 2 groups: mild hypothermia treatment group and control group.
    随机分为亚低温组与对照组,亚低温组患者入院后行亚低温治疗,对照组行常规治疗。
 

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