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后细胞
    Expression of Survivin protein in the cells treated with SB at 2.500 mmol/L was detected by immunohistochemistry SP method.
    采用免疫组织化学SP法检测2.500mmol/LSB作用24h、48h、72h后细胞抗凋亡蛋白Survivin表达的变化。
    The cell VDRas3 which stably expressed VDR antisense mRNA was used to observe the effect of 1,25(OH) 2 D 3 on the proliferation of HOS-8603 cells and the induction of p21 mRNA, one of the VDR target genes, when the VDR in the cells was blocked.
    并进一步利用稳定表达VDR反义mRNA的细胞株VDRas3细胞研究VDR被阻断后细胞增殖以及基因转录的变化。
    The arrangement of microfilament in the cells became regular and was similar to nonmalignant normal cells, but there was no effect on microtubule when the bel 7402 cells were treated with lidamycin 10 nmol/L for 8 h.
    10nmol/L力达霉素处理细胞8h后,细胞伸展,微丝排列整齐,向非恶性细胞转变,但对微管没有影响。
    The fluorescence emission spectrum of the cells incubated with He ALA was identical to that of PpIX, indicating that He ALA could induce PpIX in the cells.
    细胞的荧光显微图象显示 ,经He ALA培育后 ,细胞中生成了PpIX。
    RESULTS:The PGE 2 level in the cells cultured supernatant decreased by NIM in a concentration and time dependent manner.
    结果 :NIM处理A 5 49细胞后 ,细胞培养上清液PGE2 水平下降 ,呈浓度和时间依赖性 ;
    In the cells treated with 25 μmol/L of doxazosin for 48h,the percentage of distribution in each phase of the cell cycle was similar to that of controls;
    25μmol/L多沙唑嗪作用48h后,细胞周期分布情况与对照组相比差异无统计学意义;
    The expression of cylin D1,CDK4 and phosphorylated pRb were down-regula ted in the cells which transfected with p16 gene.
    免疫细胞化学显示外源性p16表达可下调CDK4及cylinD1的表达(P <0 .0 5 ) ,Western -Blot提示转染外源性p16基因后细胞中磷酸化pRb表达明显降低。
    Results:The grey level ratio of RANKL mRNA expression to β-actin in untreated OBs was 0.32,that in the cells infected by 107 and 109 CFU/ml of Pe for 24 h was increased to 1.73 and 2.24 respectively.
    结果以107和109CFU/mlPe作用成骨细胞24h后,细胞表达RANKLmRNA与βactin的mRNA灰度值的比值分别为1.73、2.44。 大鼠头盖骨细胞基础表达RANKLmRNA为0.32。
    Results The recombinant adenovirus Ad/siRNA/COX-2 was successfully constructed. Ninty six hours after Ad/ siRNA/COX 2 transfecting into Eca 109 cells,COX-2 mRNA was reduced by 71.7%,and PGE2 concentration in the cells culture supernatant was decreased by 62.0%.
    结果重组腺病毒 Ad/siRNA/COX-2构建成功,转染 Eca-109细胞后,细胞 COX-2 mRNA 水平下降71.7%(P<0.01),培养液上清 PGE2浓度下降62.0%(P<0.01);
    RESULTS: The result show that hypoxia and reperfusion increased the necrosis rate and apoptosis rate, enhanced the concentration of LDH in the medium and the [Ca 2+]i overload in the cells (P<0.05), but there was no difference between these groups (P>0.05).
    结果:实验表明,缺氧复氧损伤可以造成不同部位间心肌细胞坏死率和凋亡率明显升高,培养液中LDH的含量增加,细胞内钙离子超载,但各组细胞间并没有明显区别(P>0.05)。 给予缺氧预适应后,细胞损伤的各种指标均显著低于缺氧复氧组(P<0.05)。
    The expression of P53 protein in the cells was obviously higher in transfection group than in non-transfection group.
    转染组细胞在转染48h后,细胞中P53蛋白表达量高;
    The levels of IL-2 and IFN-γ se- cretion enhanced significantly in the cells with lactadherin functional peptide in comparison with those in the untreated cells (P = 0.0394, P = 0.0082, respectively), but IL-4 secretion had no marked change.
    给予乳凝集素处理后细胞培养上清中IL-2(P=0.0394)和IFN-γ(P=0.0082)的含量高于未处理细胞组,而IL-4没有显著的增高;
    ALA-PDT induced 16% of the apoptotic cells. But in the cells with CoCl-2-induced expression of HIF-1α,ALA-PDT only induced 4% of the apoptotic cells.
    PDT后高表达HIF-1α的细胞具有较高的细胞存活率,不同浓度CoCl2诱导的不同HIF-1α蛋白质水平与PDT后细胞存活活性相一致,且细胞凋亡率由未诱导时的16%降低至4%。
    The proliferation of cells and the relationship between survivaland dose were investigated in Chinese hamster lung(CHL)cells grown atstationary phase and irradiated with ~(60)Co gamma-rays. The ultrastructuralchanges and chromosome aberration in the cells after irradiation were alsoobserved.
    本文介绍中国仓鼠肺(CHL)细胞受0.087—10.0Gy 剂量~(60)Coγ射线照射后,细胞群体倍增数、存活率、染色体畸变率和超微结构的变化等观察结果。
    The myocardial cells of one-to three day-old postnatal SD rats were cultured. The culture medium was fluoridated (25mg F-/L),after 30-minute cultivation the cells pulsated rapidly, then slowly,and stopped pulsating in no time,the survival rate significantly decreased,release rate of (51)Cr from injured membrane significantly, continuously increased, the total content of Ca(2+),Na+ in the cells increased,and Mg(2+),K+ decreased.
    采用SD大鼠乳鼠心肌细胞进行培养、培养基中加氟(26mgF-/L),30分钟后细胞搏动加速,而后停搏,细胞比活率显著下降,细胞(51)Cr释放率持续显著性升高,细胞Ca(2+)、Na+含量增加,Mg(2+)、K+降低。
    Addition of anti-uPAR antibody in the cells remarkably down-regulated the phosphorylation level of p44 as compared with the control group, suggesting that uPA-ATF participates cell signal transduction and this reaction can be inhibited by anti-uPAR antibody;
    用uPAR抗体处理后 ,细胞p4 4蛋白磷酸化水平明显降低。 说明uPA -ATF参与细胞信号转导 ,而且受uPAR拮抗剂的影响 ;
    DATA SYNTHESIS:After opioid drugs acted on opiate receptor,the transduction of extracellular signals by receptor into cells was mainly adjusted by G protein, there were more than 20 kinds of G protein in the cells, different opiate recep tors had interactions with different kinds of G protein.
    资料综合:阿片类药物作用于阿片类受体后,细胞外信号主要经受体传入胞内是由G蛋白来调节的,胞内有20种以上的G蛋白,不同类型阿片受体与不同种类的G蛋白相互作用。
    In order to investigate the survival of mouse B16 melanoma cells suspended in different solutions and exposed to ultrasound of different frequencies, MTT test which had been proven to be effective to measure the survival of cells was applied to study the activity of succinate dehydrogenase of the mitochondria in the cells and to evaluate the lesion effect of ultrasound in the cells.
    采用MTT法检测,经超声、超声+血卟啉分别作用后细胞线粒体内琥珀酸脱氢酶的活性,研究超声激活血卟啉对体外培养的B16小鼠黑色素瘤细胞的杀伤作用。
    Addition of anti-uPAR antibody in the cells remarkably down-regulated the phosphorylation level of P44 as compared with the control group, suggesting that uPA-ATF participates cell signal transduction and this reaction can be inhibited by anti-uPAR antibody.
    用uPAR抗体处理后,细胞p44蛋白磷酸化水平明显降低。 说明uPA-ATF具有细胞信号转导活性,该活性受uPAR拮抗剂的影响。
    The adipose drops began to cumulate in the cells,and to the most quantity until the 21 st day.
    无血清分化培养基培养4d后细胞形态逐渐变圆,并出现球性脂滴,脂滴的数量逐渐增多至分化培养的第21天到达顶峰。
 

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