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血性脑损伤
    DYNAMIC CHANGES OF LEVELS OF VASOACTIVE INTESTINAL PEPTIDE AND SUBSTANCE P IN PLASMA AFTER HYPOXIC ISCHEMIC BRAIN INJURY IN NEONATAL RATS
    新生大鼠缺氧缺血性脑损伤后血浆血管活性肠肽和P物质水平的动态变化
    Dynamic changes in levels of vasoactive intestinal peptide and adrenocorticotropic hormone in the brain after hypoxic ischemic brain injury
    新生大鼠缺氧缺血性脑损伤后脑内血管活性肠肽和促肾上腺皮质激素的动态变化
    Possible Mechanism of Electroacupuncture Preconditioning for Hypoxia/Ischemic Brain Injury Protection Effect in Neonatal Rats
    电针预处理对新生大鼠缺氧缺血性脑损伤保护作用的可能机制
    Protective effects of activin on the neurons of neonatal rats with hypoxic ischemic brain injury
    激活素对新生大鼠缺氧缺血性脑损伤后神经元的保护
    The Change of Insulin-like Growth Factor-1 and the Affection on Neurogenesis after Ischemic Brain Injury
    胰岛素样生长因子-1在缺氧缺血性脑损伤后的变化及对神经生成的影响
    Objective To explore the role of β endorphin and opioid μ receptor on the experimental treatment of hypoxic ischemic brain injury(HIBI)by intracerebral transplantation of genetically modified myoblasts expressing and secreting brain derived neurotrophic factor(BDNF)in neonatal rats.
    目的 探讨 β 内啡肽及阿片 μ受体在脑内移植脑源性神经营养因子 (BDNF)载体细胞治疗新生大鼠低氧缺血性脑损伤 (HIBI)中的作用。
    Objective:To observe dynamic changes in levels of vasoactive intestinal peptide and adrenoeorticotropic hormone immunoreactivities,as immunoreactivities vasoactive intestinal peptide(irVIP),immunoreactivities adrenocorticotropic hormone(irACTH) in the brain of neonatal rats subjected to hypoxic ischemic brain injury(HIBI).
    目的 :观察新生大鼠缺氧缺血性脑损伤 (HIBI)后不同时间皮质、海马和纹状体血管活性肠肽和促肾上腺皮质激素免疫活性物质 (ir VIP、ir ACTH )水平的变化。
    Objective: To explore the possible mechanism of electroacupuncture preconditioning (EAPC) and combined with ATP-sensitive potassium channel (K ATP) blocker preconditioning for hypoxia/ischemic brain injury protection by observing the changes of the immediate genes (c-fos and c-jun protein content) in brain at the early stage after cerebral hypoxia/ischemic injury, and the effect of EAPC on these changes.
    目的 :观察新生大鼠缺氧缺血后大脑早期即刻基因c fos和c jun蛋白含量的变化 ,电针预处理及合用ATP敏感钾通道 (KATP)阻滞剂对其影响 ,以探讨电针预处理对缺氧缺血性脑损伤保护作用的可能机制。
    Objective:To explore the mechanisms of dynorphinA 1-13 in hypoxic ischemic brain injury in neonatal rats.
    目的 :探讨强啡肽A1-13 在新生鼠缺氧缺血性脑损伤中的作用机制。
    Objective To explore the effects of brain derived neurotrophic factors (BDNF) on concentrations of cerebral endogenous opioid peptides(EOP)in neonatal rats subject to with hypoxic ischemic brain injury (HIBI).
    目的 观察新生大鼠缺氧缺血性脑损伤 (HIBI)后 ,外源性脑源性神经营养因子 (BDNF)对脑内源性阿片肽 (EOP)水平的影响。
    [Conclusion] β-APP is a sensitive marker for axonal disruption in the white matter of the immature brain ,FDP could down regulation the expression of β-APP , from what lighten the ischemic brain injury , and protect the white matter of brain.
    【结论】 β APP是未成熟脑白质损伤的敏感指标。 FDP可减少 β APP的表达水平 ,从而减轻缺血性脑损伤 ,对脑白质具有保护性作用。
    METHODS: The experiment was conducted in the Medical College of Shandong University from July 2004 to July 2005. Totally 60 seven-day-old Wistar rats were randomly divided into 6 groups with 10 rats in each group: Normal control group, hypoxic ischemic brain injury model group (model group), sham-operation group and low, middle, high dose of activin treatment group (low, middle, high activin group).
    将60只新生7d龄Wistar大鼠随机分为6组,每组10只。 即:正常对照组、缺氧缺血性脑损伤模型组、假手术组及高剂量激活素治疗组、中剂量激活素治疗组、低剂量激活素治疗组。
    Specimens of each group were collected at different time-point after hypoxic ischemia (0,2,6,24,48 hours with 2 rats in each time-point), so as to observe the pathological changes and ultrastructure changes of brain tissues in rats with hypoxic ischemic brain injury The cell apoptosis was analyzed with flow cytometer, and the effect of activin on cells apoptosis in rats with hypoxic ischemic brain injury was observed.
    各组于缺氧缺血结束后不同时间点(0,2,6,24,48h,每个时间点2只大鼠)采集标本,观察激活素对缺氧缺血性脑损伤模型鼠的脑组织病理学变化及脑组织超微结构变化的影响; 应用流式细胞仪做细胞凋亡分析,观察激活素对缺氧缺血性脑损伤后脑细胞凋亡的影响。
    Conclusion β-APP is a sensitive marker for axonal disruption in the white matter of the immature brain ,FDP can down regulation the expression of β-APP , from what lighten the ischemic brain injury , and protect the white matter of brain.
    结论β-APP是未成熟脑白质损伤的敏感指标。 FDP可减少β-APP的表达水平,从而减轻缺血性脑损伤,对脑白质具有保护性作用。
 

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