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脑缺血损伤
    Conclusion The expressions of STAT3 increased might indicate the transmission course in ischemic nerve cell signal,and play a key role in neuron apoptosis of ischemic brain injury.
    结论STAT3蛋白表达增强可能介导缺血性脑损伤的信号转导过程,其在脑缺血损伤神经元凋亡过程中起关键作用。
    Conclusions: Ginsenoside Rb1,Rb3,Rg1,Rh2,F11 have the protective effects against ischemic brain injury.
    结论:人参皂甙Rb1、Rb3、Rg1、Rh2、F11有明显的抗脑缺血损伤作用。
    Effects of Angelica Sinensis Injection on the Expression of Vascular Endothelial Growth Factor (VEGF) and Its Receptors (Flt-1 and Flk-1) after the Ischemic Brain Injury in Rats
    当归注射液对脑缺血损伤后血管内皮生长因子及其受体表达的影响
    In order to investigate the role of NMDA-NO-cGMP pathway in ischemic brain injury, We have established a method of measuring the constitutive and inducible NOS through the conversion of L-~3H-arginine to L-~3H-citrulline, and a method of measuring NMDA receptor activity through ~3H-MK801 binding.
    为了探讨NMDA受体-NO-cGMP通路在脑缺血损伤中的作用机制,我们利用大鼠全脑缺血模型,应用L-~3H-精氨酸转化法测定cNOS和iNOS活性,~3H-MK801结合试验测定NMDA受体活性,观察了脑缺血再灌损伤后海马等脑区的NMDA受体、NOS活性和cGMP含量变化。
    Conclusion Ginsenoside Rb1 has protective effects against ischemic brain injury.
    结论 人参皂甙Rb1有明显的抗脑缺血损伤作用
    Effects of glucoside and aglycone parts of rhubarb on the metabolism of free radicals in rats with ischemic brain injury
    大黄苷及苷元对脑缺血损伤大鼠自由基代谢的影响
    The protective effect of nine ginseng saponinmonomers on ischemic brain injury in rat
    九种人参皂甙单体抗脑缺血损伤作用研究
    Changes of Nitric Oxide Synthase and Nitric Oxide content in serum and brain tissue after intracerebral hemorrhage in rats and ischemic brain injury
    实验性大鼠脑出血后血清、脑组织一氧化氮合酶、一氧化氮含量变化与脑缺血损伤
    There are huge cytokine expression and proinflammatory infiltration in damage area after ischemic brain injury, which aggravate brain injury especially in reperfusion.
    脑缺血损伤后在受损区有大量细胞因子的表达和炎细胞的浸润,可加重脑组织损伤,尤其在再灌注中更加明显。
    (3) Caspase-3 plays a key role in neuronal apoptosis and ischemic brain injury.
    3.C卿ase习 tnRNA在介导脑缺血损伤神经元凋亡过程中起关键作用。
    Conclusion: MCAO and reperfusion of rat brain represents a good model to study focal brain ischemia. The computer assisted imaging is an objective and quantitative technique to evaluate ischemic brain injury and to assess the neuroprotective effect of drugs.
    结论 :缺血 30 min和再灌注 2 4 h是诱导局灶性脑缺血的合适条件 ,计算机辅助技术客观定量综合评价脑缺血损伤及药物作用是可行的方法。
    Conclusion Ginsenoside Rb 1 has the protective effects against ischemic brain injury. The mechanism is related to the antagonist effect of ginsenosides on Glutamate excitory toxicity.
    结论 人参皂甙 Rb1 有明显的抗脑缺血损伤作用 ,其作用机制与抗谷氨酸的神经毒性作用有关。
    Conclusions The neuronal apoptosis after focal cerebral ischemia reperfusion was a dynamic ongoing process. The expression of Bcl-2 might inhibit apoptosis. Caspase-3 might play a key role in neuronal apoptosis of ischemic brain injury.
    结论 脑缺血再灌流后 ,缺血周围区神经细胞的凋亡是一个动态的渐进过程 ,Bcl- 2基因表达可能抑制细胞凋亡 ,caspase- 3基因在介导脑缺血损伤神经元凋亡过程中起关键作用。
    Conclusion These results suggest that the neuronal apoptosis is a dynamic ongoing process. The expression of Caspase-3 might play a key role in neuronal apoptosis and ischemic brain injury.
    结论 脑缺血再灌流后 ,缺血周围区神经细胞的凋亡是一个动态的渐进过程 ,Caspase - 3基因在介导脑缺血损伤神经元凋亡过程中起关键作用。
    Objective To study the effects of rehabilitative training on the proliferation of the neural stem cells located in hippocampal formation (HF) after focal ischemic brain injury (cerebral infarction) in adult rats, and explore the basis or mechanism on which the rehabilitative training after cerebral infarction can promote neural function recovery of patients.
    目的 研究成年大鼠局灶性脑缺血损伤(脑梗死)后康复训练对海马结构中内源性神经干细胞增殖的影响,探讨脑梗死后康复训练使神经功能改善的理论基础。
    CONCLUSION: SPECT is capable of detecting ischemic brain injury immediately. It is proved by histological evidence that a more-than-5-hour ischemia will cause irreversible changes in neurons.
    结论:单光子放射型计算机断层显像可在脑缺血损伤即刻发现脑损伤表现,并经组织学验证脑缺血5h后神经元即就进入不可逆的状态。
    The comprehension of mechanism of cerebral ischemic tolerance highlights new avenues for future prevention and treatment of ischemic brain injury
    对脑缺血耐受发生机制的理解有助于预防和治疗脑缺血损伤
 

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