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血性脑损伤
    Effect of Dynorphin A 1 ̄13 on Hypoxia Ischemic Brain Injury in Neonatal Rats
    强啡肽A_(1~13)在新生鼠缺氧、缺血性脑损伤中的作用
    AIM: To observe influences of ischemic brain injury on proliferation and transference of endogenous neural stem cells.
    目的:观察缺血性脑损伤对内源性神经干细胞增殖、迁移的影响。
    Conclusion Hyperbaric oxygen treatment had an obvious effect on ischemic brain injury,and increasing the expression of Neuroglobin might be one of the possible mechanisms of the therapy.
    结论高压氧对缺血性脑损伤有显著治疗作用,提高脑红蛋白的表达水平可能是其治疗的机制之一。
    Objectives To investigate the role of neural cell adhesion molecule on the neural plasticity after the ischemic brain injury in rat. Method Twenty-six male Wistar rats were subjected to an episode of right middle cerebral artery occlusion( MCAO) for two hours(n=20), sham operation(n=3) and no operation(n=3).
    目的 明确神经细胞黏占附分子(neural cell adhesion molecule,NCAM)的表达对缺血性脑损伤神经可塑性的作用. 方法 Wistar大鼠26只,体重170—200g,分为空白对照组(n=3)、假手术组(n=3)和损伤组(n=20).
    Study on the Changes of Transmitter Amino Acids in Hippocampi and Cortex of Ischemic Brain Injury Rats
    缺血性脑损伤大鼠海马皮层递质氨基酸变化的研究
    C-FOS GENE EXPRESSION AND MEDICINAL REGULATION IN HYPOTHALAMUS OF RAT FOLLOWING ISCHEMIC BRAIN INJURY
    缺血性脑损伤大鼠下丘脑c-fos基因表达及药物调控
    The protective mechanism of fructose-1, 6-diphosphate on ischemic brain injury
    1,6-二磷酸果糖对缺血性脑损伤保护机制的研究
    DYNAMIC CHANGES OF LEVELS OF VASOACTIVE INTESTINAL PEPTIDE AND SUBSTANCE P IN PLASMA AFTER HYPOXIC ISCHEMIC BRAIN INJURY IN NEONATAL RATS
    新生大鼠缺氧缺血性脑损伤后血浆血管活性肠肽和P物质水平的动态变化
    Excitatory Amino Acid Toxicity and Ischemic Brain Injury
    兴奋性氨基酸毒性与缺血性脑损伤
    EXPRESSION OF LIM HOMEOBOX GENE Lhx3 AND Lhx4 AFTER THE CEREBRAL ISCHEMIC BRAIN INJURY IN ADULT RATS
    同源盒基因Lhx3和Lhx4在成年大鼠缺血性脑损伤后的表达变化
    Different spatio-temporal expression of poly(ADP-ribose) polymerase for repair ing the cells after ischemic brain injury
    多聚(ADP-核糖)聚合酶不同时空的表达特征对缺血性脑损伤细胞修复的影响
    Effect of Kang Naoxueshuan Tablet on Protecting Ischemic Brain Injury in Rats
    抗脑血栓片对大鼠缺血性脑损伤保护作用的实验研究
    JAKs-STATs signal transduction and ischemic brain injury
    JAKs-STATs信号转导通路与缺血性脑损伤
    Mitochondria calcium overload and ischemic brain injury
    线粒体Ca~(2+)超载与缺血性脑损伤
    Effects of Angelica polysaccharide on the expression of angiopoietin after the ischemic brain injury in rats
    当归多糖对大鼠缺血性脑损伤后血管生成素表达的影响
    Heat shock protein 70 and ischemic brain injury
    热休克蛋白70与缺血性脑损伤
    Effects of IGF-1 on the Proliferation, Migration and Differentiation of Neural Stem Cells after Ischemic Brain Injury in Adult Rats
    IGF-1对缺血性脑损伤大鼠神经干细胞增殖、迁移和分化的影响
    Effects of Angelica sinensis on the neural plasticity following ischemic brain injury by MRI
    MRI研究当归对缺血性脑损伤神经可塑性的影响
    Apoptosis is one of the patterns of cell death during cerebral ischemia. As an antiapoptotic gene, it has been proved in many studies that bcl-2 gene ( B-cell lymphoma/leukemia-2 gene) can protect neural cells from ischemic brain injury. Bax gene (bcl-2 associated x gene) has been proved to be a proapoptotic gene, colaberating with bcl-2 to adjust apoptosis.
    脑缺血以后神经细胞死亡的形式之一即是细胞凋亡,bcl-2基因(B-cell lymphoma/leukemia-2 gene)作为一种抗凋亡基因,在大量研究中证明对缺血性脑损伤后神经细胞具有保护作用,bax基因(bcl-2 associated x gene)被认为是一种凋亡促进基因,与bcl-2共同调节凋亡。
    Conclusion: EA can recover the function of limbs and upregulate the expression of GDNF and bFGF, that may contribute to the protective action of EA in reducing ischemic brain injury.
    结论 电针能显著改善局灶性脑缺血再灌注大鼠的神经体征,对缺血性脑损伤具有一定的保护作用,这一作用可能是通过提高脑组织GDNF、bFGF的表达来实现的。
 

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