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    (2) Mild to moderate hypothermia could reduced the expression of ICAM 1 and leukocyte infiltration(P<0.01).
    (2 )缺血早期进行亚低温治疗能明显减轻缺血再灌注后 ICAM- 1的表达及减少白细胞浸润 (P <0 .0 1)。
    Objective To study the effect of moderate hypothermia on cerebral mitochondrial succinate dehydrogenase (SDH) activities, mitochondrial complex Ⅱ activity and the capacity of ATP synthesization in newborn rats with hypoxic-ischemic brain damage (HIBD).
    目的 研究亚低温治疗对缺氧缺血脑损伤 (hypoxic -ischemicbraindamage ,HIBD)新生大鼠的脑线粒体琥珀酸脱氢酶 (succinatedehydrogenase ,SDH)复合酶Ⅱ的活性及ATP合成能力的影响 ,以探讨亚低温治疗的神经保护机制。
    Method Patients with acute severe head injuries (GCS≤8)were assigned into two groups:Group I,30 cases(average GCS≤ 5.38) received standard large trauma craniotomy and moderate hypothermia for 5-7 days;
    方法 对急性重型颅脑损伤患者 (GCS≤ 8) ,治疗组采用外伤性标准大骨瓣开颅术 ,术后即给予亚低温冬眠治疗 5~ 7d ;
    The study included 44 with deep hypothermia circulatory arrest (DHCA), 7 with deep hypothermia low flow rate (DHLF), 23 with DHCA+DHLF, 37with moderate hypothermia bypass and another 18 underwent NCPB. RESULTS CPB times were (75.7±41.5)min, (48.9±424.1)min, and (33.9±15)min.
    5 2例采用改良超滤 (MUF)方法 ,4例进行常规超滤 (CUF)方法 ,90例运用常规超滤结合改良超滤方法。 结果 CPB时间为 ( 75 .7± 41.5 )min ,主动脉阻断时间是 ( 48.9± 2 4.1)min,停循环时间为 ( 3 3 .9± 15 .1)min,低流量时间为 ( 62± 2 8.9)min。
    Moderate hypothermia, high flow rate 2.4~2.8L/(min.m 2), MAP at about 70 mmHg, Hct at 22%~25% were used.
    采用低温、2 .4~ 2 .8L/ (min·m2 )高流量灌注 ,平均动脉压在 70mmHg以上 ,Hct保持在 2 2 %~ 2 5 %。
    There were 31 cases with moderate hypothermia high flow rate, 11 cases with deep hypothermic low flow rate, 9 cases with deep hypothermic circulatory arrest, and 2 cases with normothermia high flow rate.
    中低温高流量灌注 3 1例 ,深低温低流量 (DHLF) 11例 ,深低温停循环 (DHCA) 9例 ,常温体外循环 2例。
    Results In moderate hypothermia group the ICP gradually decreased and PO2 increased after 24 hours of hypothermia, with obvious difference from the control group (P<0.05).
    结果亚低温治疗24h后ICP逐渐下降,脑组织PO2逐渐上升,与对照组比较差异有显著性(P<0.05)。
    Methods: From March 1996 to April 2003, 208 infants were operated under moderate hypothermia and high perfusing flow, the blood flow was 80~150 ml/(kg·min), 200 ml/(kg·min) less than 10 kg weight, critical congenital heart disease were applied with modified ultrafiltration (MUF), perfusion pressure kept 30~80 mmHg.
    方法回顾该院1996年3月~2003年4月208例婴幼儿先心病术中体外循环情况,采用中度低温、中高流量灌注,灌注流量80~150 ml/kg·min,10 kg以下高达200 ml/kg·min。 预充液以胶体为主,晶胶比为0.06~0.6。
    Objective To probe the alteration and significance intracranial press(ICP)and brain tissue PO 2(PbtiO 2) in severe head injury treated by moderate hypothermia(33~35℃).
    目的探讨重型脑损伤患者亚低温(33~35℃)治疗前后颅内压(ICP),脑组织氧分压(PbrO2)的变化及临床意义。
    Methods Twenty-four rabbits were randomly segregated into five groups:control group normal temperature group(groupⅠ), system mild hypothermia group(groupⅡ), system moderate hypothermia group(groupⅢ) and wound regional hypothermia group(group Ⅳ); Serum levels of IL-1β,IL-6,IL-10,TNF-α were determined with ELISA.
    方法30只新西兰大白兔,随机分为正常对照组、常温组(Ⅰ组)、全身浅低温组(Ⅱ组)、全身中低温组(Ⅲ组,)、创伤局部降温组(Ⅳ组),采用ELISA法测定血清IL1β、IL6、IL10、TNFα含量。
    CPB adopted moderate hypothermia ASCP (nasopharyngeal temperature 23.1~25℃,perfusion flow 10~15ml/kg/min);
    中度低温末ASCP的灌注流量为10~15ml/(kg·min),鼻咽温为23.1~25℃;
    38 adopted moderate hypothermia moderate flow perfusion,and another 2 underwent deep hypothermia low flow perfusion(DHLF). The urine during CPB was 50~1 000(324.7±268.9) ml.
    38例采用中低温中低流量灌注,2例采用深低温低流量CPB,CPB中尿量50~1000(324.7±268.9)毫升(ml)。
    Objective To observe the effects of moderate hypothermia on the expression of HSP70 mRNA in focal model of transient cerebral ischemia in rats.
    ①目的观察亚低温对大鼠局灶性脑缺血后热休克蛋白70(HSP70)mRNA表达的影响。
    Methods Thirty rabbits were randomly divided into normal control group and traumatic hemorrhagic shock group that was randomized into normal temperature group, mild hypothermia group, moderate hypothermia group and regional hypothermia group.
    方法30只新西兰大白兔,随机分为正常对照组和创伤失血性休克组,创伤失血性休克组又根据不同的体温干预措施分为常温组、浅低体温组、中低体温组和局部降温组,采用ELISA法测定肠组织匀浆上清液TNFα、IL-6、IL-8和IL-10含量。
    Objective To investigate the effects of moderate hypothermia on the permeability of alveolar capillary membrane in acute lung injury (ALI) induced by lipopolysaccharide (LPS) .
    目的探讨中度低温对脂多糖(LPS)诱导急性肺损伤(ALI)大鼠肺泡毛细血管膜通透性的影响。
    control group(n=22) received operation on arrested heart with moderate hypothermia(28℃~30℃) CPB. Blood samples were taken from artery.
    对照组在冷晶体停跳、中度低温(28℃~30℃)CPB下手术。
    Methods 60 patients with cerebral hemorrhage were divided into two groups,moderate hypothermia group(30 cases) and control group(30 cases).
    方法将60例脑出血患者分为亚低温治疗组和对照组,每组30人。
    Objective To observe the dynamic changes in the DNA binding activity of nuclear factorκB (NFκB) in rat pancreatic and lung tissues in acute hemorrhagic necrotizing pancreatitis (AHNP), and investigate the effects of moderate hypothermia on AHNPinduced NFκB binding activity in a timedependent fashion.
    目的观察急性出血坏死性胰腺炎(AHNP)后不同时间点胰腺和肺组织中核转录因子κB(NFκB)的DNA结合活性动态变化,以及胰腺炎后应用中度低温对NFκB活性的影响。
    NFκB binding activity was inhibited at 2 and 5 hours of moderate hypothermia in lung tissue (P<0.01), but there was no difference in pancreatic tissue between normothermic and hypothermic rats at each time point.
    与AHNP常温组相比,低温组AHNP后2h和5h肺组织NFκB活性均显著降低(P均<0.01),而胰腺组织NFκB活性与常温组差异不明显。
    Methods 36 cases undergoing long time CPB from September 2003 to September 2005 were analyzed retrospectively, deep hypothermia circulatory arrest was emploied in 2 cases, moderate hypothermia in 34 cases;
    方法2003年9月至2005年9月老年人长时间体外循环(CPB)心内直视手术36例,其中采用深低温停循环2例、中度低温34例。
 

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