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    group B 26 cases, nerve toxicity of 1~3 grade occurred in 9,4,3 cases respectively ,develop ratc was 61.5%.
    3级1例,总发生率28.5%。 B组26例,发生神经毒性1级9例;
    Study on Nerve Toxicity and Oxidation Damage to Antioxidant Defenses of Mice Induced by Pentachloronitrobenzene (PCNB)
    五氯硝基苯对小鼠的神经毒性和抗氧化防御系统损伤研究
    Conclusion GIK can lighten the nerve toxicity of Glu,Asp and NO,and protect on the ischemic organisation.
    应用GIK后较缺血组有明显降低。 结论GIK能减轻Glu、Asp、NO的神经毒性,起到对缺血脑组织的保护作用。
    The diversity of the two groups’(Ⅰ,Ⅱ) perisensory nerve toxicity is also obvious. 62.9 percent toxicity group and 31.6 percent of HCPT group appear the toxicity (P< 0.05), but the diversity of adverse reaction is not obvious (P> 0.05).
    毒副反应两组Ⅰ~II外周神经毒性有显著性差异,Oxaliplatin组为62.9%,HCPT组为31.6%(P<0.05),其他毒副反应的差异无显著性(P>0.05)。
    Clincal observation of the prevention and treatment of the nerve toxicity by combining potassium aspartate and magnesium aspartate and calcium gluconate
    门冬氨酸钾镁与葡萄糖酸钙联用防治奥沙利铂神经毒性的临床观察
    An individual with CYP2C9*3 allele caused the central nerve toxicity after orally routine dose of phenytoin. Therefore CYP2C9 polymorphisms are particularly relevant to the metabolism of drugs with narrow therapeutic indices such as warfarin, tolbutamide and phenytoin.
    其中CYP2C9~*3发现的较早,研究的也最广泛,
 

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