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四唑氮蓝试验
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  “四唑氮蓝试验”译为未确定词的双语例句
     Methods Clinical features and laboratory data were collected from the pedigree with X-CGD and the proband was hospitalized in Shenzhen Children Hospital.
     方法根据患儿临床症状、体征及四唑氮蓝试验(NBT)分析诊断,采集家系成员外周血抽提基因组DNA,扩增CYBB基因并检测基因突变。
短句来源
  相似匹配句对
     NITROBLUE TETRAZOLIUM REDUCTION TEST IN DIAGNOSIS OF SPONTANEOUS PERITONITIS IN CIRRHOSIS
     四唑还原试验在肝硬化自发性腹膜炎诊断中的应用
     NBT reduction test showed functional maturity of the differentiated HL-60 cells.
     NBT(Nitroblue tetrazolium,四唑)还原试验显示其功能亦渐超成熟。
短句来源
     TESTING
     试验
短句来源
     BLUE
    
短句来源
     AIMS:To investigate implication of nitroblue tetrazolium reduction test (NBT) in diagnosis of spontaneousperitonitis (SBP) in cirrhosis.
     目的:探讨四唑还原试验(NBT)在肝硬化自发性腹膜炎(SBP)诊断中的应用意义。
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  nitroblue tetrazolium test
Practical use of nitroblue tetrazolium test in febrile disorders of children
      
The generation of reactive oxygen species in leukocytes from 73 healthy donors is studied using the luminol- and lucigenin-dependent chemiluminiscent methods and the nitroblue tetrazolium test.
      
Hydrocortisone decreased the count of peritoneal mononuclear phagocytes and mast cells and total phagocytic activity of peritoneal phagocytes in rats, but had no effect on the nitroblue tetrazolium test.
      
β-Adrenoceptor blockade abolished the suppressive effect of hydrocortisone on phagocytosis and prevented the decrease in the count of mast cells, but markedly reduced the number of neutrophils and parameters of stimulated nitroblue tetrazolium test.
      
A nitroblue tetrazolium test was normal, ruling out chronic granulomatous disease and leaving the possibility of a subtle defect in the NRB.
      
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Objective Chronic pulmonary aspergillosis is different from acute pulmonary asp ergillosis in clinical picture, radiogram, diagnostic procedures and prognosis. Four patients with chronic pulmonary aspergillosis had been misdiagnosed as havi ng pneumonia or pulmonary tuberculosis for a long time before admission to the h ospital. The purpose of this report was to summarize the clinical manifestations and laboratory findings for correct diagnosis of chronic pulmonary aspergillosi s. Methods Four patients with...

Objective Chronic pulmonary aspergillosis is different from acute pulmonary asp ergillosis in clinical picture, radiogram, diagnostic procedures and prognosis. Four patients with chronic pulmonary aspergillosis had been misdiagnosed as havi ng pneumonia or pulmonary tuberculosis for a long time before admission to the h ospital. The purpose of this report was to summarize the clinical manifestations and laboratory findings for correct diagnosis of chronic pulmonary aspergillosi s. Methods Four patients with chronic pulmonary aspergillosis seen between Octo ber 2002 and October 2004 were retrospectively studied. Their clinical manifesta tions, chest radiographic feature, immune status, diagnostic procedure, therapy and prognosis were reviewed. Results The chief complaints of these patients were chronic cough and fever for 3 to 12 months. Chest wall abscess developed in the late course in case 1 a nd 4. Fine moist rales were heard and hepatosplenomegaly was found in case 1 and 2. No abnormal sign was found in case 3 and 4. Chest radiographic feature: loba r consolidation with adjacent pleural thickening was present in all cases. In ea rly phase, solitary or multiple small nodules were found in 2 cases. Case 1-3 ha d normal IgG, IgM, IgA, IgE, T Cell subsets and NBT test. Case 4 had chronic gra nulomatous disease. Etiologic evidences: culture was positive for Aspergillus (A.) fulmigatus in sputum and in chest wall abscess in case 1 and 4; for A. niger in sputum and spore existing in lung tissue in case 2; for A. fulmigat us in sputum and hypha existing in lung tissue in case 3. All patients were tr eated with combination of amphotericin B and itraconazole. Their symptoms were c ontrolled 10-30 d after treatment. In case 1 the disease relapsed 6 months later and the patient died at last due to giving up treatment by his parents. Case 2 was free of symptom for 12 months and his chest radiographic lesion disappeared completely 6 months later. Treatment of case 4 was given up. Case 3 continued t o receive treatment and observation. Conclusion Chronic pulmonary aspergillosis should be considered in children with long period fever and cough and lobar consolidation associated with adjacent pl eural thickening or with nodular infiltration. The diagnosis of chronic pulmonar y aspergillosis depended on identification of aspergillus from sputum or lung ti ssue. Combined amphotericin B and itraconazole might control the disease.

 目的 探讨儿童慢性肺曲霉菌病的诊断和治疗。方法 分析 4例儿童慢性肺曲霉菌病的表现、诊断和治疗,并复习有关文献。结果  ( 1 ) 4例均表现为长期或间断发热、咳嗽,病程3月~1年。其中 2例合并胸壁脓肿。(2)2例肺部闻及细湿啰音并肝脾肿大,另 2例肺部及其他部位检查无异常。(3)2例患儿发病前无基础疾病史, 1例患慢性肉芽肿病, 1例曾患原发性肺结核。4例患儿IgG、IgA、IgM、IgE, T细胞亚群、总补体和C3、C4、中性粒细胞数量均正常。3例四唑氮蓝试验正常, 1例异常。(4)胸部影像学表现: 4例在病程中均表现为单侧肺叶实变伴胸膜肥厚。2例病初表现为多发结节影。(5)4例痰液培养均有曲霉菌生长, 2例行肺活检,在肺组织中发现曲霉菌菌丝或孢子。2例合并胸壁脓肿者,脓液培养也有曲霉菌生长。(6)4例患儿均联合应用二性霉素B和伊曲康唑治疗, 10d~1个月症状控制。结论 对于有长期发热、咳嗽,胸部影像表现为肺叶实变伴胸膜肥厚或为结节性阴影,病情进展缓慢的儿童, 应考虑慢性肺曲霉病的可能。确诊依赖于多次痰液培养或肺组织培养或在肺组织中发现曲霉菌生长。一旦确诊,联合应用二性霉素B和伊曲康唑可...

 目的 探讨儿童慢性肺曲霉菌病的诊断和治疗。方法 分析 4例儿童慢性肺曲霉菌病的表现、诊断和治疗,并复习有关文献。结果  ( 1 ) 4例均表现为长期或间断发热、咳嗽,病程3月~1年。其中 2例合并胸壁脓肿。(2)2例肺部闻及细湿啰音并肝脾肿大,另 2例肺部及其他部位检查无异常。(3)2例患儿发病前无基础疾病史, 1例患慢性肉芽肿病, 1例曾患原发性肺结核。4例患儿IgG、IgA、IgM、IgE, T细胞亚群、总补体和C3、C4、中性粒细胞数量均正常。3例四唑氮蓝试验正常, 1例异常。(4)胸部影像学表现: 4例在病程中均表现为单侧肺叶实变伴胸膜肥厚。2例病初表现为多发结节影。(5)4例痰液培养均有曲霉菌生长, 2例行肺活检,在肺组织中发现曲霉菌菌丝或孢子。2例合并胸壁脓肿者,脓液培养也有曲霉菌生长。(6)4例患儿均联合应用二性霉素B和伊曲康唑治疗, 10d~1个月症状控制。结论 对于有长期发热、咳嗽,胸部影像表现为肺叶实变伴胸膜肥厚或为结节性阴影,病情进展缓慢的儿童, 应考虑慢性肺曲霉病的可能。确诊依赖于多次痰液培养或肺组织培养或在肺组织中发现曲霉菌生长。一旦确诊,联合应用二性霉素B和伊曲康唑可使病情控制。

Objective: To study the inhibitory effect of celecoxib on proliferation of two human lung cancer cells(Anip973?AGZY83-a) and the effect on cell cycle, attachment, invasion and chemotaxied-motion Methods: Methyl thiazolyl tetrazolium (MTT) assay was used to observe the growth inhibition rate of human lung cancer cell line Anip973, AGZY83-a that were exposed to the various concentration of the celecoxib (5, 10, 20, 40, 80 and160?滋mol/L) at the different times. Cell cycles phase distribution of cells was measured...

Objective: To study the inhibitory effect of celecoxib on proliferation of two human lung cancer cells(Anip973?AGZY83-a) and the effect on cell cycle, attachment, invasion and chemotaxied-motion Methods: Methyl thiazolyl tetrazolium (MTT) assay was used to observe the growth inhibition rate of human lung cancer cell line Anip973, AGZY83-a that were exposed to the various concentration of the celecoxib (5, 10, 20, 40, 80 and160?滋mol/L) at the different times. Cell cycles phase distribution of cells was measured by flow cytometry(FCM) assay. The ability of attachment to Lamininin and ECM gel was measured by MTT assay. The ability of invasion and of chemotaxied-motion through reconstituted basement membrane were investigated in transwell chambers. Results: Celecoxib could inhibit the growth of Anip973, AGZY83-a. Effects of inhibition depended on the action time and a certain concentration of celecoxib (P<0.01). Two cells after treated by celecoxib showed morphological changes characteristic of cells. Celecoxib(40?滋mol/L )could reduce abilities of attachment, invasion and chemotaxied-motion in lung cancer cells significantly(P<0.01). Conclusion: Celecoxib with certain concentration can inhibit the proliferation of human cancer cells in concentration -depended and time-depended patterns and can reduce ability of attachment, invasion and chemotaxied-motion in lung cancer cells.

目的:探讨Celecoxib对高、低不同转移能力的肺腺癌细胞株(Anip973、AGZY83-a)增殖的抑制作用及对细胞周期、形态、粘附、浸润、趋化运动能力的影响。方法:用不同浓度的Celecoxib培养液终浓度为(5、10、20、40、80、160滋mol/L)处理不同肺癌细胞株,采用四唑氮蓝试验(MTTAssay)法测定其对细胞增长的抑制情况,采用碘化丙啶(PI)染色,流式细胞(FCM)技术,观察Celecoxib作用不同肺癌细胞株后对细胞生长周期的影响,并应用Transwell小室观察对细胞运动、浸润能力及对粘附能力的影响。结果:Celecoxib在一定范围内(10、20、40、80、160滋mol/L)可有效地抑制Anip973、AGZY83-a肺癌细胞生长,并存在时间和剂量依赖性(P<0.01);Celecoxib在40滋mol/L浓度时培养24h对细胞周期无明显影响(P>0.01);对肺癌细胞粘附、浸润、趋化运动能力明显抑制(P<0.01);Celecoxib作用不同肺癌细胞后形态学有明显的改变。结论:Celecoxib对不同转移能力的肺癌细胞系Anip973、AGZY83-a具有明显...

目的:探讨Celecoxib对高、低不同转移能力的肺腺癌细胞株(Anip973、AGZY83-a)增殖的抑制作用及对细胞周期、形态、粘附、浸润、趋化运动能力的影响。方法:用不同浓度的Celecoxib培养液终浓度为(5、10、20、40、80、160滋mol/L)处理不同肺癌细胞株,采用四唑氮蓝试验(MTTAssay)法测定其对细胞增长的抑制情况,采用碘化丙啶(PI)染色,流式细胞(FCM)技术,观察Celecoxib作用不同肺癌细胞株后对细胞生长周期的影响,并应用Transwell小室观察对细胞运动、浸润能力及对粘附能力的影响。结果:Celecoxib在一定范围内(10、20、40、80、160滋mol/L)可有效地抑制Anip973、AGZY83-a肺癌细胞生长,并存在时间和剂量依赖性(P<0.01);Celecoxib在40滋mol/L浓度时培养24h对细胞周期无明显影响(P>0.01);对肺癌细胞粘附、浸润、趋化运动能力明显抑制(P<0.01);Celecoxib作用不同肺癌细胞后形态学有明显的改变。结论:Celecoxib对不同转移能力的肺癌细胞系Anip973、AGZY83-a具有明显的抑制作用,存在时间依赖性和在一定范围内剂量依赖性,对肺癌细胞粘附、浸润、趋化运动能力有明显抑制作用。

Objective The X-linked form of the chronic granulomatous disease (X-CGD) arises from mutations in the CYBB gene, which encodes the 91-KD glycoprotein gp91phox, the component of flavocytochrome b558. We confirm a X-CGD case recently and explore the mutation of CYBB gene in this family. Methods Clinical features and laboratory data were collected from the pedigree with X-CGD and the proband was hospitalized in Shenzhen Children Hospital. All of the exon and exon-intron boundaries of CYBB gene including proband...

Objective The X-linked form of the chronic granulomatous disease (X-CGD) arises from mutations in the CYBB gene, which encodes the 91-KD glycoprotein gp91phox, the component of flavocytochrome b558. We confirm a X-CGD case recently and explore the mutation of CYBB gene in this family. Methods Clinical features and laboratory data were collected from the pedigree with X-CGD and the proband was hospitalized in Shenzhen Children Hospital. All of the exon and exon-intron boundaries of CYBB gene including proband and 3 family members were analyzed by direct sequencing of PCR product from genomic DNA. Results The patient was diagnosed as X-CGD according to the clinical features and the nitroblue tetrazolium (NBT) test. The sequencing results of the proband revealed 914 T to A in the exon 9 of CYBB gene which caused Met312Lys missing mutation, which was also found in his mother and his sister. Conclusion X-CGD in this family is caused by Met312Lys in the gp91phox of flavocytochrome b558 and it is the first reported Cc.se in China.

目的对1例X连锁慢性肉芽肿病(X-CGD)家系进行临床表型和基因突变分析,揭示X- CGD的发病机制。方法根据患儿临床症状、体征及四唑氮蓝试验(NBT)分析诊断,采集家系成员外周血抽提基因组DNA,扩增CYBB基因并检测基因突变。结果①NBT显示,患儿未刺激组、大肠埃希杆菌内毒素刺激组及佛波乙酸酯刺激组中,中性粒细胞NBT还原率为0,患儿母亲的分别为4%,10%,25%,患儿父亲分别为2%,97%,97%,结合患儿临床症状、体征初步确诊为X-CGD;②基因突变分析显示,先证者位于Xp21.1编码NADPH细胞色素b558的gp91phox亚基的CYBB基因第9外显子第949位的碱基由T 突变为A,由此引起编码序列第312个氨基酸错义突变为Met312Lys,患儿母亲及姐姐均存在同位点基因突变,但患儿父亲CYBB基因无突变,证实该错义突变来源于母系。结论 CYBB基因氨基酸错义突变,是引起该家系X-CGD的原因。

 
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