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鸦片受体
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  opiate receptor
     THE CHANGES OF OPIATE RECEPTOR BINDING IN RAT BRAIN UNDER ELECTROACUPUNCTURE TOLERANCE CONDITION
     电针耐受时大鼠脑内鸦片受体结合的改变
短句来源
     The excitement effect was effectively blocked by the specific opiate receptor blocker-naloxone.
     鸦片受体阻断剂纳洛酮能有效地阻断吗啡和亮啡肽的兴奋作用。
短句来源
     (2) The hyperglycemic effect of morphine could be blocked by prior injection of the opiate receptor blocker naloxone similarly administered into cerebral ventricles.
     (2)吗啡的高血糖效应可由预先注入侧脑室的鸦片受体阻断剂纳洛酮所阻断。
短句来源
  opioid receptor
     δ_2 Opioid Receptor Mediates Immunocyte Activation
     类鸦片受体δ_2介导的免疫细胞激活作用
短句来源
     Antipruritic therapy with the oral opioid receptor antagonist naltrexone. Open, non-placebo controlled administration in 133 patients (Germ)
     口服类鸦片受体拮抗剂纳曲酮治疗133例瘙痒患者的开放、非安慰剂对照研究(德语)
短句来源
     Therefore , we conclude that there is only one kappa opioid receptor gene in the human genome .
     计数细胞染色体时没有发现有其它位置的杂交点,因此认为:在人类基因组中,仅存在一个k-鸦片受体基因。
短句来源
  opiate receptors
     AUTORAIOGRAPHIC LOCALIZATIOM OF OPIATE RECEPTORS OF CENTRAL NERVOUS SYSTEM (CNS) IN NORMAL AND ACUPUNCTURED RATS
     鸦片受体在大鼠脑内的分布及针刺时的变化
短句来源
     Acute experiments were performed on rabbits anesthetized with pentobarbi-tal and arterial blood pressure(BP)was recorded. 1.Intracerebroventricular(ivt)injection of 50μg of β-endorphin causedlowering of BP by 12.2±2.0 mmHg,which could be reversed by ivt injection ofnaloxone,an antagonist of opiate receptors.
     本工作应用家兔作急性实验发现:(1)脑室注射50微克β-內啡肽,可使血压下降12.2±2.0毫米汞柱。 此效应可被脑室注射鸦片受体阻断纳络酮所拮抗。
短句来源
  “鸦片受体”译为未确定词的双语例句
     Sequence Analysis and Localization of the Human Kappaopioid Receptor Gene
     人类k-鸦片受体的基因分析和定位
短句来源
     It is suggestedthat endorphins were related to these sympathetic inhibition.
     表明此种抑制作用有脑内鸦片受体与吗啡样物质参与。
短句来源
     the excitement action of morphine and leu-enkephalin on the sphincter of oddi might not be mediated by norepinephrine; the endogenous opiate-like substances and their receptors may play an important role in the regulation of the motility of the sphincter of oddi.
     实验结果表明:豚鼠总胆管括约肌存在鸦片受体,吗啡和亮啡肽对离体豚鼠总胆管括约肌的兴奋作用并非通过去甲肾上腺素起作用,并提示内源性鸦片样物质及其受体在总胆管括约肌运动调节中可能具有重要作用。
短句来源
     After injecting naloxone(1.8μg)into bilateral nucleus accumbens, the discharging from LHPE was no longer inhibited by electro-acupuncture (P<0.05) ; and the discharging from LHPI induced did not appear (P<0.05) at all. Cerebrospinal fluid was injected into bilateral nucleus accumbens for control, and this did not cause significant change of discharging from lateral habenular pain-units.
     双侧伏核内注射鸦片受体阻断剂纳洛酮(1.8μg)后、电针“足三里”不再抑制LHPE放电(P<0.05)、电针“足三里”使LHPI放电兴奋作用消失(P<0.05),而向伏核内注射人工脑脊液对外侧缰核痛单位放电无显著影响。
短句来源
     (3) Nucleus Raphe Magnus and its opiate recerptors as well as 5-HT in brain participate in the inhibitory action.
     中缝大核及其鸦片受体和中枢5-HT 参与上述的针刺抑制效应。
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  opiate receptor
Preliminary intravenous injection of δ-opiate receptor (OR) agonists DSLET (0.1 mg/kg) or DTLET (0.1 mg/kg) increased tolerance of isolated perfused myocardium to damage by oxidative stress simulated in vivo with FeSO4 + ascorbic acid.
      
Influence of Dalargin, a Peripheral Opiate Receptor Agonist, on Exercise Tolerance in Patients with Coronary and Peripheral Arte
      
Microinjection of opiate receptor antagonist naloxone 3 nmol into CNTS did not itself affect the level of blood pressure and heart rate, but could markedly attenuate the cardiovascular effects of NE in CNTS.
      
Opiate receptor blockade with naltrexone completely prevented the EtOH-induced delay in vaginal opening.
      
The opiate receptor antagonist naloxone affected fight and escape similarly as AMTP.
      
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  opioid receptor
The dissociation constant of the Dalargin-opioid receptor complex was found to be 4.3 nM.
      
The role of opioid receptor agonists and antagonists in the treatment of Parkinson's disease
      
Opposing effects on infarction of delta and kappa opioid receptor activation in the isolated rat heart: implications for ischemi
      
48-h pretreatment with TAN-67 (30 mg/kg), a δ1-opioid receptor agonist, maximally reduced IS/AAR (31.2 ± 6.5).
      
Acetylcholine (ACh) and opioid receptor agonists trigger the preconditioned
      
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  opiate receptors
Role of Opiate Receptors and ATP-Dependent Potassium Channels of Mitochondria in the Formation of Myocardial Adaptive Resistance
      
Role of δ1 Opiate Receptors in Regulation of Contractility in Isolated Rat Heart during Normal Oxygenation and Ischemia/Reperfus
      
Stimulation of μ- and δ-Opiate Receptors and Tolerance of Isolated Heart to Oxidative Stress: the Role of NO-Synthase
      
The immunomodulating effects of hydrocortisone and adrenaline against the background of blockage of opiate receptors
      
The immunomodulating effects of hydrocortisone and adrenaline against the background of blockage of opiate receptors
      
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Rabbits under urethane-chloralose or local anaesthesia were immobilizedby Flexidile and ventilated by artificial respiration.The ECG and blood pres-sure were recorded on the polygraph.To induce ventricular extrasystole,anelectrical stimulation was given to the hypothalamus in the vicinity of DMH,VMHor fornix for 5 sec.every 5 min..The number of ventricular extrasystoles after eachstimulation didn't vary significantly for more than one hour.But when the deepsomatic nerves such as the median nerve or peroneus...

Rabbits under urethane-chloralose or local anaesthesia were immobilizedby Flexidile and ventilated by artificial respiration.The ECG and blood pres-sure were recorded on the polygraph.To induce ventricular extrasystole,anelectrical stimulation was given to the hypothalamus in the vicinity of DMH,VMHor fornix for 5 sec.every 5 min..The number of ventricular extrasystoles after eachstimulation didn't vary significantly for more than one hour.But when the deepsomatic nerves such as the median nerve or peroneus profoundus nerve were stimula-ted with a current of low frequency and low intensity,the ventricular extrasystolecould be inhibited,while the blood pressure was still raised during hypothalamicstimulation.Section of the vagus nerve couldn't block such inhibition.So itis assumed that the somatic nerve stimulation might exert some kind of inhibitoryaction on the cardiac sympathetic center.By microinjection of morphine into the periaqueduct central gray of the mid-brain,the ventricular extrasystoles induced by hypothalamic stimulation could be in-hibited instantaneously.On the other hand,if naloxone was injected into the centralgray,the hypothalamus-indueed ventrienlar oxtrasystoles increased significantly,and could no longer be inhibited by somatic nerve stimulation.It is suggestedthat endorphins were related to these sympathetic inhibition.

1.以低频、低强度方波刺激腓深神经或正中神经可以抑制由刺激下丘脑引起的期前收缩。若电刺激过强或刺激腓浅、挠浅、前臂外侧皮神经则无此作用。2.刺激躯体神经对期前收缩的抑制作用与迷走神经及心室后负荷无密切关系,很可能是通过抑制心交感中枢紧张性来实现的。3.刺激下丘脑诱发的期前收缩可为中央灰质内微量注入吗啡所抑制,而为微量注入纳洛酮所增多,微量注入纳洛酮并可阻断躯体传入冲动对期前收缩的抑制作用。表明此种抑制作用有脑内鸦片受体与吗啡样物质参与。

In 13 conscious dogs,stainless steel cannulae were previously implanted intovarious areas of the brain.The heart rate and blood pressure measured by the indirectmethod were recorded on the polygraph.When normal saline(2μl/2min.)was mi-croinjected into the central gray(CG),dorsal area of the hippocampus(HPC)and sup-ramammillary area(SMA),the experimental hypertension produced by intravenousinfusion of noradrenaline(NA)t a constant rate was not affected,and the de-pressor effect of electroacupuncture at Tsu San-Li...

In 13 conscious dogs,stainless steel cannulae were previously implanted intovarious areas of the brain.The heart rate and blood pressure measured by the indirectmethod were recorded on the polygraph.When normal saline(2μl/2min.)was mi-croinjected into the central gray(CG),dorsal area of the hippocampus(HPC)and sup-ramammillary area(SMA),the experimental hypertension produced by intravenousinfusion of noradrenaline(NA)t a constant rate was not affected,and the de-pressor effect of electroacupuncture at Tsu San-Li still existed.Morphine 4μg/2μl/2 min.)microinjected into these areas caused a depressor effect for about 15minutes on NA-induced hypertension,with no significant changes in the heart rate.But when naloxone(4μg/2 μl/2 min.) was microinjected into these areas,the de-pressor effect of electroacupuncture was blocked.In the medial aspect of parietalcortex,morphine microinjection(4 μg/2 μl /2 min.)didn't produce any depressoreffect,and naloxone microinjection didn't influence the depressor effect of electro-acupuncture on NA-induced hypertension.It is suggested that the inhibitory effect on sympathetic vasomotor neurons byelectroacupuncture was due to the release of endogenous opiate-like substance andthe activation of opiate receptors in CG,HPC and SMA,etc..

在清醒犬的中脑中央灰质、海马背侧、乳头体上区等中枢部位微量注射生理盐水(2微升/2分钟),对血压、心率无明显影响,此时电针“足三里”穴对匀速注射去甲肾上腺素所致的实验性高血压具有显著的降压效应。在这种高血压的基础上,在以上三个部位微量注射吗啡(4微克/2微升/2分钟)可见血压短暂下降后又回升到原高血压水平,心率无显著变化。在这些部位微量注射纳洛酮后(4微克/2微升/2分钟)则以上电针降压效应消失。而在大脑顶叶内侧皮层微量注射吗啡对血压、心率无明显影响;在该处微量注射纳洛酮后,电针对实验性高血压仍有明显的降压作用。结果表明:电针对交感缩血管中枢的抑制与电针时中枢内产生的内(脑)啡肽对中脑中央灰质、海马、乳头体上区等部位鸦片受体的激活有关。

Acute experiments were performed on rabbits anesthetized with pentobarbi-tal and arterial blood pressure(BP)was recorded.1.Intracerebroventricular(ivt)injection of 50μg of β-endorphin causedlowering of BP by 12.2±2.0 mmHg,which could be reversed by ivt injection ofnaloxone,an antagonist of opiate receptors.2.During hemorrhagic or toxic shock there was significant increase of en-dogenous opiate-like substances in the whole brain,especially in the lower brainstem.3.The hypotensive state in hemorrhagic shock or...

Acute experiments were performed on rabbits anesthetized with pentobarbi-tal and arterial blood pressure(BP)was recorded.1.Intracerebroventricular(ivt)injection of 50μg of β-endorphin causedlowering of BP by 12.2±2.0 mmHg,which could be reversed by ivt injection ofnaloxone,an antagonist of opiate receptors.2.During hemorrhagic or toxic shock there was significant increase of en-dogenous opiate-like substances in the whole brain,especially in the lower brainstem.3.The hypotensive state in hemorrhagic shock or toxic shock could bepartially reversed by ivt injection of 80μg of naloxone,the effect of which couldinturn be antagonized by ivt injection of 100μg of serotonin.The results indicated that endogenous opioids in the brain may play an impor-tant role in the hypotensive effect of hemorrhagic and toxic shock and that theblockading of opiate receptors may prove to be one of the effective measures forthe prevention and treatment of hypotensive shock.

本工作应用家兔作急性实验发现:(1)脑室注射50微克β-內啡肽,可使血压下降12.2±2.0毫米汞柱。此效应可被脑室注射鸦片受体阻断纳络酮所拮抗。(2)在出血性和中毒性休克时,脑内鸦片样物质的活性显著升高。其中以脑干部位升高得更为明显。(3)脑室注射80微克纳络酮可以部分桔抗出血性和中毒性休克的低血压效应,延长中毒性休克动物的存活时间。(4)脑室注射100微克的5-羟色胺可以逆转纳络酮的抗休克作用,使休克低血压效应加剧。这些实验结果提示:中枢鸦片样物质在休克发生过程中具有重要作用。阻断鸦片样物质的作用,可能是防治休克低血压效应的一个途径。

 
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