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心脏电生理特性
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  “心脏电生理特性”译为未确定词的双语例句
     Effects of the RPV Fat Pad on Heart Electrophysiology of Canine
     犬右肺静脉脂肪垫对心脏电生理特性影响的实验研究
短句来源
     Effect of Inferior Vein Cava Fat Pad on Electrophysiological Characteristics of Canine's Heart
     犬下腔静脉脂肪垫对心脏电生理特性影响的实验研究
短句来源
     The first generation of H1 antagonists had been reported to have a beneficial effect on allergic arrhythmias.
     因此H:受体拮抗剂致心律失常作用的离子机制并未完全阐明,第一代H:受体拮抗剂与第二代H:受体拮抗剂对心脏电生理特性影响的区别以及第二代拮抗剂易于引发致死性心律失常的原因都是有待阐明的问题。
短句来源
     In the heart-torso model, the heart excitation propagation depends on a set of model parameters, so the heart excitation propagation should be determined if we can deduce the model parameters from the body surface potentials.
     在心电仿真模型中,心脏电兴奋传播过程是由一系列心脏电生理特性模型参数决定的,因此只要能从体表电位数据推知心电仿真模型参数,就可以确定心脏的电兴奋传播过程。
短句来源
  相似匹配句对
     Effects of pethidine on cardiac electrophysiological properties
     哌替啶的心脏生理效应
短句来源
     To study electrophysiological properties of Deiters′ cells.
     探讨该细胞的基本生理特性
短句来源
     CARDIAC ELECTROPHYSIOLOIC EFFECT OF DAURICINE
     蝙蝠葛碱的心脏生理作用研究
短句来源
     EFFECT OF CLONIDINE ON HEART ELECTROPHYSIOLOGY
     可乐定对心脏生理的影响
短句来源
     Diagnosis of Drought From Maize Physiological Electrical Characteristics
     从玉米生理特性诊断旱情
短句来源
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  cardiac electrophysiologic characteristic
Changes in myocardial stretch or load have been association with changes in cardiac electrophysiologic characteristics.
      


The electrophysiologic properties of dauricine 4mg/kg given iv were assessed in 11 patients with arrhythmia during intracardiac electrophysiological study. Effects of dauricine on ECG and BP were evalua-ted in 6 anaesthetized dogs. Dauricine increased significantly PA interval (15.8% ), AH interval(6. 1 %) and HV interval (21. 5%), lengthened QRS-width (14. 3%) withJTc being unchanged,prolonged atrial effective and functional refractoryperiod, but did not change A-V nodal refractory period, cSNRT andSACTc. In...

The electrophysiologic properties of dauricine 4mg/kg given iv were assessed in 11 patients with arrhythmia during intracardiac electrophysiological study. Effects of dauricine on ECG and BP were evalua-ted in 6 anaesthetized dogs. Dauricine increased significantly PA interval (15.8% ), AH interval(6. 1 %) and HV interval (21. 5%), lengthened QRS-width (14. 3%) withJTc being unchanged,prolonged atrial effective and functional refractoryperiod, but did not change A-V nodal refractory period, cSNRT andSACTc. In the anaesthetized dogs, dauricine prolonged PR, QTc and JTcintervals, lengthened QRS-width and lowered BP,these effects were mark-edly dose-dependant. However, heart rate was increased in a dose of1mg/kg and decreased significantly in doses of 6mg/kg and 9mg/kg. The data suggested that dauricine may be classified as an antiarrhyth-mic agent of class Ic type of the Harrison's modification of classification,that its partly important mechanism of antiarrhythmic effect may be rela-ted to slowing couction, specially His-purkinje system conduction, thatdauricine slowed significantly conduction with the function of sinus node being unchanged

本文研究了Dau 4mg/kg iv对11例心律失常患者的心脏电生理的影响,以及不同剂量的Dau(1,3,6,9mg/kg)iv对麻醉犬ECG和BP的影响。临床研究中观察到Dau使PA间期、AH间期和HV间期分别延长15.8,6.1和21.5%;QRS增宽14.3%,HR加快4.1%,而JTc间期无明显改变;使心房ERP和FRP分别延长4.6%和2.5%,房室结不应期无明显改变;对cNRT和SACTc无明显影响。麻醉犬实验中,Dan使PR间期延长,QRS增宽、BP下降,作用与剂量正相关。小剂量HR加快,较大剂量使HR减慢,JTc间期延长。结果表明,Dau的心脏电生理特性与Ic类抗心律失常药物很相似,Dau延缓心脏传导速度,尤其对希蒲系传导的延缓,可能在其抗心律失常效应中起着重要作用。在明显延缓心脏传导同时,对功能正常的窦房结无明显影响。

The purpose was to observe the effects of ibutilide on canine electrophysiological characters, and further more, to explore the mechanism of ibutilide′s converting atrial flutter(AFL). 15 mongrel dogs were anesthetized and intubated with a tracheal tube. The heart was exposed through a thoracotomy and then electrodes were fixed by suture. The electrophysiologic variables were measured with and without ibutilide(for the 10-minute infusions the dose was 0.10mg/kg, and 30 minutes later, the maintaining dose was...

The purpose was to observe the effects of ibutilide on canine electrophysiological characters, and further more, to explore the mechanism of ibutilide′s converting atrial flutter(AFL). 15 mongrel dogs were anesthetized and intubated with a tracheal tube. The heart was exposed through a thoracotomy and then electrodes were fixed by suture. The electrophysiologic variables were measured with and without ibutilide(for the 10-minute infusions the dose was 0.10mg/kg, and 30 minutes later, the maintaining dose was 0.01mg/min). These variables included the heart rate, the conduction of intraatrium and interatrium, the conduction ratio of isthmus, the effective refractory period, and so on. Results:Ibutilide had the significant suppressing effect for the sinus atrial node. The peak response time was 20~30 min and the heart rate recovered without drug administration. The atrial, ventricular and pulmonary vein effective refractory period were significantly prolonged(P<0.05~0.001).However, there were no significant difference on the conduction of intraatrium, interatrium and isthmus(P>0.05). Conclusion:The mechanism of ibutilide′s rapidly terminating AFL is speculated on the prolongation of the refractory period which could cause the whole excitable gap of the reentrant circuit to be affected by refractoriness, thus to terminate the AFL due to the reentrant wavefront′s inability to proceed.

观察伊布利特对犬心脏电生理特性的作用特点,初步探讨其终止心房扑动(简称房扑)的机制。15只成年健康雄性杂种犬,麻醉后气管插管,开胸并缝合电极,伊布利特按0.10mg/kg静脉推注给药(10min缓推,给药30min后按照0.01mg/min静脉滴注维持),观察给药前后心率、房内房间传导时间、峡部缓慢传导区传导速度、各部位不应期的变化。结果:伊布利特对心率有明显的抑制作用,作用的高峰时间在给药后20~30min,2h后心率基本恢复到用药前的水平;伊布利特能显著延长心房肌、心室肌以及肺静脉的不应期(P<0.05~0.001),但对房内、房间以及峡部的传导作用影响不明显(P>0.05)。结论:伊布利特终止房扑的机制可能是由于该药造成不应期延长,使折返环可激动间隙兴奋性降低,进而使波锋不能向前推进,终止房扑。其房扑折返环传导的减慢可能不是房扑终止的主要作用。

 
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