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单胺囊泡转运体
相关语句
  vesicular monoamine transporter 2
     Objective : To study the antitoxic role of vesicular monoamine transporter 2 (VMAT2) in transgenic Chinese Hamster ovary (CHO) cell.
     目的:研究转基因中国仓鼠卵(CHO)细胞中单胺囊泡转运体(VMAT_2)的抗毒性作用。
短句来源
     A Study on Antitoxic Role of Vesicular Monoamine Transporter 2 in Transgenic Chinese Hamster Ovary Cells
     转基因中国仓鼠卵细胞中单胺囊泡转运体抗毒性作用的研究(英)
短句来源
  “单胺囊泡转运体”译为未确定词的双语例句
     Objective To study the mithridatism of VMAT 2 in transgeneic CHO cell.
     目的 研究转基因中国仓鼠卵 (CHO)细胞中单胺囊泡转运体 (VMAT2 )的抗毒性作用。
短句来源
     The study on toxicity of dopamine and neurotoxin such as 6-OH-dopamine,MPP+ ,which is metabolic production of MPTP and can lead to experimental model of PD,has focus on two transporters in dopaminergic cell. They are dopamine transporter (DAT) and vesicular monoamine transporter(VMAT2).
     对多巴胺毒性作用机制的研究以及能产生实验性PD模型的毒物如6-羟多巴胺、MPTP代谢产物MP旷的研究已集中在多巴胺能神经元两种转运体上—神经元细胞膜上多巴胺转运体(DAT)、细胞囊泡膜上单胺囊泡转运体(VMAT_2)。
短句来源
  相似匹配句对
     MOLECULAR CLONING OF VESICULAR MONOAMINE TRANSPORTER-2 cDNA FROM HUMAN FETAL BRAIN
     人Ⅱ型单胺转运基因的克隆
短句来源
     The Role of Distribution of Vesicular Monoamine Transporter in Brain Tissue in the Pathogenesis of Parkinson Disease
     单胺转运的分布特征在帕金森病发病机制中的作用
短句来源
     A Study on Antitoxic Role of Vesicular Monoamine Transporter 2 in Transgenic Chinese Hamster Ovary Cells
     转基因中国仓鼠卵细胞中单胺转运抗毒性作用的研究(英)
短句来源
     Influence of Paraquat on Vesicular Monoamine Transporter 2 in Nigra-striatum of Mice
     百草枯对小鼠黑质纹状通路单胺转运2的影响
短句来源
     Distribution of Vesicular Monoamine Transporter in Parkinson Disease Model of C57BL Mice
     单胺转运在帕金森病小鼠脑组织中的分布特征
短句来源
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  vesicular monoamine transporter 2
Methamphetamine-induced locomotor activity and sensitization in dopamine transporter and vesicular monoamine transporter 2 doubl
      
Vesicular monoamine transporter 2 as a marker of gastric enterochromaffin-like cell tumors
      
The latter showed intense vesicular monoamine transporter 2 immunoreactivity consistent with its origin from the enterochromaffin-like cells of the gastric oxyntic mucosa.
      
A Metabolomic Study of Brain Tissues from Aged Mice with Low Expression of the Vesicular Monoamine Transporter 2 (VMAT2) Gene
      
The vesicular monoamine transporter 2 (VMAT2) sequesters monoamines into synaptic vesicles in preparation for neurotransmission.
      
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Objective To study the mithridatism of VMAT 2 in transgeneic CHO cell.Methods Using technology of transgene from PC 12 to CHO, MTT reduction assay was used to detect the toxic effect on MPP + to wtCHO and cDNACHO,meanwhile the role of reserpine was observed,including the toxic effect to MPP + on specific blocking agent of VMAT 2.Results cDNACHO to the sensitivity of MPP + was much less than that of wtCHO over concentration of 0.5 mmol/L MPP +; cDNACHO had the same...

Objective To study the mithridatism of VMAT 2 in transgeneic CHO cell.Methods Using technology of transgene from PC 12 to CHO, MTT reduction assay was used to detect the toxic effect on MPP + to wtCHO and cDNACHO,meanwhile the role of reserpine was observed,including the toxic effect to MPP + on specific blocking agent of VMAT 2.Results cDNACHO to the sensitivity of MPP + was much less than that of wtCHO over concentration of 0.5 mmol/L MPP +; cDNACHO had the same sensitive as wtCHO to rotenon;after the reserpine was added,the above role disappeared,but wtCHO reserpine was given alone,it couldn't change its sensitivity to MPP +.Conclusion VMAT 2 has protective effect on cDNACHO by transporting MPP + to vesicles; PC 12 possesses the antitoxic components.

目的 研究转基因中国仓鼠卵 (CHO)细胞中单胺囊泡转运体 (VMAT2 )的抗毒性作用。方法 利用转 PC1 2 细胞基因到 CHO细胞中形成的转基因 CHO细胞 (c DNACHO) ,采用 MTT比色法检测 1-甲基 -4 -苯基吡啶离子 (MPP+ )对 CHO细胞野生株 (wt CHO)和 c DNACHO细胞的毒性作用 ,并观察利血平——VMAT2 的特异性阻滞剂对 MPP+毒性作用的影响。结果 在 MPP+ 0 .5 m mol/ L 以上浓度 c DNACHO细胞对 MPP+ 敏感性比 wt CHO低得多 ;c DNACHO和 wt CHO对鱼藤酮 (rotenon)的敏感性无显著差异 ;加入VMAT2 的特异性阻滞剂利血平后 ,上述保护作用消失 ,c DNACHO对 MPP+ 敏感性与 wt CHO细胞无差异 ,而单独予以 wt CHO细胞利血平则不能改变它对低浓度 MPP+的敏感性。结论 此保护机制是由转基因细胞中 VMAT2 引起的 ,VMAT2 在转基因的非神经细胞系 (CHO细胞系 )中也能将 MPP+转运至囊泡内 ,从而保护细胞 ;同时也提示 PC1 2 细胞内具有抗毒...

目的 研究转基因中国仓鼠卵 (CHO)细胞中单胺囊泡转运体 (VMAT2 )的抗毒性作用。方法 利用转 PC1 2 细胞基因到 CHO细胞中形成的转基因 CHO细胞 (c DNACHO) ,采用 MTT比色法检测 1-甲基 -4 -苯基吡啶离子 (MPP+ )对 CHO细胞野生株 (wt CHO)和 c DNACHO细胞的毒性作用 ,并观察利血平——VMAT2 的特异性阻滞剂对 MPP+毒性作用的影响。结果 在 MPP+ 0 .5 m mol/ L 以上浓度 c DNACHO细胞对 MPP+ 敏感性比 wt CHO低得多 ;c DNACHO和 wt CHO对鱼藤酮 (rotenon)的敏感性无显著差异 ;加入VMAT2 的特异性阻滞剂利血平后 ,上述保护作用消失 ,c DNACHO对 MPP+ 敏感性与 wt CHO细胞无差异 ,而单独予以 wt CHO细胞利血平则不能改变它对低浓度 MPP+的敏感性。结论 此保护机制是由转基因细胞中 VMAT2 引起的 ,VMAT2 在转基因的非神经细胞系 (CHO细胞系 )中也能将 MPP+转运至囊泡内 ,从而保护细胞 ;同时也提示 PC1 2 细胞内具有抗毒性作用的成分

Objective : To study the antitoxic role of vesicular monoamine transporter 2 (VMAT2) in transgenic Chinese Hamster ovary (CHO) cell. Methods :With the technology of trans-gene from PC 12 to CHO, MTT reduction assay was used to detect MPP+ toxic effect on wild type CHO (wtCHO) and transgenic CHO. Meanwhile, the role of reserpine was also observed in MPP+ toxic effects. Results :The sensitivity of transgenic CHO to MPP+ was much less than that of wtCHO with 0. 5 mmol/L MPP+. Transgenic CHO had the same sensitivity...

Objective : To study the antitoxic role of vesicular monoamine transporter 2 (VMAT2) in transgenic Chinese Hamster ovary (CHO) cell. Methods :With the technology of trans-gene from PC 12 to CHO, MTT reduction assay was used to detect MPP+ toxic effect on wild type CHO (wtCHO) and transgenic CHO. Meanwhile, the role of reserpine was also observed in MPP+ toxic effects. Results :The sensitivity of transgenic CHO to MPP+ was much less than that of wtCHO with 0. 5 mmol/L MPP+. Transgenic CHO had the same sensitivity as wtCHO if rotenone was given. WtCHO, by given reserpine alone, didn't change its sensitivity to MPP+. Conclusions :VMAT2 has protective effect on transgenic CHO by transporting MPP+ to vesicles.

目的:研究转基因中国仓鼠卵(CHO)细胞中单胺囊泡转运体(VMAT_2)的抗毒性作用。方法:利用转PC12细胞基因到CHO细胞中形成的转基因CHO细胞(cDNA-CHO),采用MTT比色法检测1-甲基-4-苯基吡啶离子(MPP~+)对CHO细胞野生株(wtCHO)和cDNA-CHO细胞的毒性作用,并观察利血平——VMAT_2的特异性阻滞剂对MPP~+毒性作用的影响。结果:在MPP~+ 0.5mmol/L以上浓度cDNA-CHO细胞对MPP~+敏感性比wtCHO低得多;cDNA-CHO和wtCHO对鱼藤酮(rotenon)的敏感性无显著差异;加入利血平后,上述保护作用消失,cD-NA-CHO对MPP~+敏感性与wtCHO细胞无差异,而单独予以wtCHO细胞利血平则不能改变它对低浓度MPP~+的敏感性。结论:此保护机制是由转基因细胞中VMAT_2引起的,VMAT_2在转基因的非神经细胞系(CHO细胞系)中也能将MPP~+转运至囊泡内,从而保护细胞,同时也提示PC12细胞内具有抗毒性作用的成分。

 
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