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阴性刺激
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  “阴性刺激”译为未确定词的双语例句
     The successive order was: the DR antigen positive stimulating cell group > ABC antigen positive stimulating cell gooup > both ABC and DR antigen negative stimulating cell group .
     结果 ①各病例的各实验组刺激细胞诱导的CTL杀伤活性均显著低于对照组 ,依次为 :DR抗原阳性刺激细胞组 >ABC抗原阳性刺激细胞组 >ABC和DR抗原均阴性刺激细胞组 ,抗ABC单抗可完全阻断CTL特异性分泌TNF α ;
短句来源
     In the system the 40W light and sound sianal are as positive stimulus and the 15W light signal as neqative stimulus. A light reaction key and a sound reaction key are mounted.
     系统以40W灯光和声音信号为阳性刺激,15W灯光信号为阴性刺激,并配置了光反应、声反应按键.
短句来源
     ②Mixed lymphocyte reaction(MLR): mitomycin C-treated donor peripheral blood lymphocytes were used as stimulator, and proliferative reaction of peripheral blood lymphocyte of patients to donor transplanted antigen was detected with the incorporation of 3H-TDR method (Negative: There was no significant difference between the mean value of stimulation index and 1, conversely positive).
     ②混合淋巴细胞反应:以丝裂霉素C处理的供体外周血淋巴细胞作刺激细胞,3H-TDR掺入法检测患者外周血淋巴细胞对供体移植抗原的增生反应(阴性:刺激指数的均值与1无显著性差异,反之为阳性)。
短句来源
  相似匹配句对
     Results The results of mucosa stimulating test were negative.
     结果 :粘膜刺激试验表现为阴性
短句来源
     The cells in the negative control group were exposed to EMF.
     阴性对照组不进行电磁场刺激;
短句来源
     in troducing stimulus;
     诱导刺激
短句来源
     PAS was negative.
     PAS阴性
短句来源
     The Age of Thrills
     刺激时代
短句来源
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  negative stimulus
The response was considered positive if the fish took the food without touching the negative stimulus.
      
The treatments consisted of an increasing positive stimulus, a decreasing positive stimulus, an increasing negative stimulus, or a decreasing negative stimulus.
      
Animals adjusted their diet choice in response to the changing intensity of the negative stimulus, but not the positive stimulus.
      
Animals avoided foods associated with the negative stimulus to a greater extent when the stimulus was increasing each period compared to when it was decreasing.
      
As each additional negative stimulus is presented, the chance of delinquent activity increases.
      
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An operational food motion conditioned reflex experiment system controlled by apple-Ⅱ microcomputer is reported. In the system the 40W light and sound sianal are as positive stimulus and the 15W light signal as neqative stimulus. A light reaction key and a sound reaction key are mounted. A food-disk is controlled by a step-motor. After setting-up the experiment conditions, three stimulus signals appear automatically under the computer control. As soon as the experiment ends, the result is printed out. Real operations...

An operational food motion conditioned reflex experiment system controlled by apple-Ⅱ microcomputer is reported. In the system the 40W light and sound sianal are as positive stimulus and the 15W light signal as neqative stimulus. A light reaction key and a sound reaction key are mounted. A food-disk is controlled by a step-motor. After setting-up the experiment conditions, three stimulus signals appear automatically under the computer control. As soon as the experiment ends, the result is printed out. Real operations have proving that this system not only makes experimentors get rid of tedious manual works, but also makes the experi- ment results more accurate and more reliable.

介绍了用APPLE-Ⅱ微型计算机控制的操作式食物运动性条件反射实验系统.系统以40W灯光和声音信号为阳性刺激,15W灯光信号为阴性刺激,并配置了光反应、声反应按键.喂食的食盘由步进电机控制.预先设置实验条件后,在计算机控制下自动给出3种刺激信号,动物若正确操作按键,则食盘转动,自动喂食.实验结束时自动记录实验结果,最后将各项数据在打印机上打印出来.实际运行表明,该实验系统不仅能使实验者从繁琐的手工操作中解放出来,而且使实验的结果更精确,更可靠.

Objective To investigate the role of major histocompatibility complex(MHC) molecules in the induction of cytotoxic T lymphocyte(CTL) from oral squamous cell carcinoma. Methods ①The treated autologous tumor cells served as stimulating cells,on which the MHC mAb was used to block the HLA ABC and/or HLA DR antigens.The specific cytotoxicity of CTL induced by stimulating cells and the level of released TNF α were then detected. ② By blocking the HLA ABC and/or HLA DR antigens on target cells , the inhibition...

Objective To investigate the role of major histocompatibility complex(MHC) molecules in the induction of cytotoxic T lymphocyte(CTL) from oral squamous cell carcinoma. Methods ①The treated autologous tumor cells served as stimulating cells,on which the MHC mAb was used to block the HLA ABC and/or HLA DR antigens.The specific cytotoxicity of CTL induced by stimulating cells and the level of released TNF α were then detected. ② By blocking the HLA ABC and/or HLA DR antigens on target cells , the inhibition effect of MHC mAb on CTL cytotoxicity induced by autologous stimulating cells (not treated with mAb) was measured. Results ①The cytotoxicity induced by stimulating cells in experimental groups was significantly lower than that of the control group. The successive order was: the DR antigen positive stimulating cell group > ABC antigen positive stimulating cell gooup > both ABC and DR antigen negative stimulating cell group . The specific TNF α released by CTL was completely blocked by anti ABC mAb.②The average inhibition rate by mAb on cytotoxicity of CTL was: 52%,27% and 72% by anti ABC mAb, anti DR mAb, and anti ABC mAb plus anti DR mAb respectively. Conclusion Both MHC class I and class Ⅱantigens on OSCC cells play an important role in inducing CTL.

目的 探讨肿瘤细胞上的主要组织相容性复合体 (majorhistocompatibilitycomplex ,MHC)分子在诱导自体口腔鳞癌细胞毒T淋巴细胞 (cytotoxicTlymphocyte ,CTL)中的作用。方法 对 9例口腔鳞癌中的肿瘤细胞表面人类白细胞抗原 (humanleukocyteantigen ,HLA) ABC和HLA DR抗原进行检测 ,将其中 5例HLA ABC抗原表达 >5 0 %和HLA DR抗原表达 >35 %的病例作为研究对象 :①自体肿瘤细胞经处理后作刺激细胞 ,并用MHC单抗阻断刺激细胞上的HLA ABC和 (或 )HLA DR抗原 ,检测自体肿瘤细胞诱导的CTL的特异性杀伤活性及肿瘤坏死因子 (tumornecrosisfactor,TNF) α的分泌水平。②阻断靶细胞上HLA ABC和 (或 )HLA DR抗原 ,观察MHC单抗对自体刺激细胞 (未加单抗处理 )诱导的CTL细胞毒的抑制作用。结果 ①各病例的各实验组刺激细胞诱导的CTL杀伤活性均显著低于对照组 ,依次为 :DR抗原阳性刺激细胞组 >ABC抗原阳性刺激细胞组 >ABC和DR抗原均阴性刺激$...

目的 探讨肿瘤细胞上的主要组织相容性复合体 (majorhistocompatibilitycomplex ,MHC)分子在诱导自体口腔鳞癌细胞毒T淋巴细胞 (cytotoxicTlymphocyte ,CTL)中的作用。方法 对 9例口腔鳞癌中的肿瘤细胞表面人类白细胞抗原 (humanleukocyteantigen ,HLA) ABC和HLA DR抗原进行检测 ,将其中 5例HLA ABC抗原表达 >5 0 %和HLA DR抗原表达 >35 %的病例作为研究对象 :①自体肿瘤细胞经处理后作刺激细胞 ,并用MHC单抗阻断刺激细胞上的HLA ABC和 (或 )HLA DR抗原 ,检测自体肿瘤细胞诱导的CTL的特异性杀伤活性及肿瘤坏死因子 (tumornecrosisfactor,TNF) α的分泌水平。②阻断靶细胞上HLA ABC和 (或 )HLA DR抗原 ,观察MHC单抗对自体刺激细胞 (未加单抗处理 )诱导的CTL细胞毒的抑制作用。结果 ①各病例的各实验组刺激细胞诱导的CTL杀伤活性均显著低于对照组 ,依次为 :DR抗原阳性刺激细胞组 >ABC抗原阳性刺激细胞组 >ABC和DR抗原均阴性刺激细胞组 ,抗ABC单抗可完全阻断CTL特异性分泌TNF α ;②各实验组单抗对CTL的杀伤活性的平均抑制率分别为 :抗ABC单抗为 5 2 % ;抗DR单抗为 2 7% ;抗ABC单抗 +抗DR单抗为 72 %。 结论口腔鳞癌细胞上的MHC I、II类抗原在诱导自体CTL中起重要作用

Aim To investigate the relationship bet ween the damage of cognitive capability of the patient s with delayed encephalopathy after acute carbon monoxide poisoning and laten cies as well as the amplitude of each component of event related potentia l(P300),moreover the frequency of oc-currence of P300and prognosis assessment.Methods Prior to the test,Mi-ni-Mental State Examination(MMSE)and Blessed Behavior Scale were adop ted in order to rate scores in 20patients.The"detachment stimulation"being used as the"target"stimulation,and...

Aim To investigate the relationship bet ween the damage of cognitive capability of the patient s with delayed encephalopathy after acute carbon monoxide poisoning and laten cies as well as the amplitude of each component of event related potentia l(P300),moreover the frequency of oc-currence of P300and prognosis assessment.Methods Prior to the test,Mi-ni-Mental State Examination(MMSE)and Blessed Behavior Scale were adop ted in order to rate scores in 20patients.The"detachment stimulation"being used as the"target"stimulation,and passive tasks as we ll as auditory"OB"sequence would elicit P3a element for the pati ents.Results The frequency of P300oc-currence was 55%,and that of N2occur rence was 70%.Comparing the patient s with P300occurrence with the normal control:the latencies of N2and P3we re significantly prolonged,but there was no difference in the ampli-tude.Comparing the patients with ab sent P300with the normal subjects:t he latencies N1and P2were prolonged.T here was certain linear relationshi p be-tween N2latency and such coma.There was noticeable difference in the cli nical Blessed Scale scores between the patients with present P300or absent P300.In visiting cases,when there was P300,there was improvement in clinical symptoms.Conclusion P300may become an objective criterion testing the damage of cognitive capability of th e patients suffering from the delaye d en-cephalopathy after acute carbon mon oxide poisoning.

目的通过分析一氧化碳中毒后迟发脑病患者认知功能损害与事件相关电位(P3a)各成分潜伏期及波幅的关系,探讨P3的出现率及对预后的判断。方法对20例一氧化碳中毒后迟发脑病患者测试前应用简易精神状态检查量表(MMSE)及Blessed行为量表评分;采用阴性刺激,被动方式,听觉“oddball”刺激序列诱发P3a成分。结果P3出现率为55%,N2出现率70%;引出P3者与对照组相比,P3、N2潜伏期显著延长,未引出P3者与对照组相比,N1、P2潜伏期延长;急性一氧化碳中毒时昏迷时间长短与N2潜伏期之间有线性关系;引出P3和未引出P3患者临床Blessed量表评分之间有显著性差异;随访病例中,多数引出P3者,临床症状好转。结论P300可能成为测试一氧化碳中毒后迟发脑病患者认知损害严重性和判断预后的一项客观标准。

 
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