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咽鼓管软骨
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  “咽鼓管软骨”译为未确定词的双语例句
     The epithelial cells of the eustachian tube damaged slightly and only a few infilitrated inflammation cells were found in the submucosa.
     咽鼓管粘膜上皮细胞胞浆淡染 ,粘膜下少量炎症细胞浸润 ,咽鼓管软骨镜下未见病理改变。
短句来源
     Conclusion:The cartilaginous portion,muscle and fascia of the eustachian tube could be displayed clearly on MRI.
     结论 :MRI可较好显示咽鼓管软骨部及其肌肉、筋膜 ;
短句来源
     Objective To explore and assess the use of NiTi shape memory alloys in the eustachian tube to prevent and treat adhesive middle ear.
     目的 为探讨咽鼓管软骨段钛镍合金支架置入治疗和预防粘连性中耳炎的可行性并报告初步结果。
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  相似匹配句对
     Clinical application of CT and MRI of the Eustachian tube
     咽鼓管影像学
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     Conclusion:The cartilaginous portion,muscle and fascia of the eustachian tube could be displayed clearly on MRI.
     结论 :MRI可较好显示咽鼓管软骨部及其肌肉、筋膜 ;
短句来源
     the third is disfunction of Eustachian.
     ③咽鼓管功能障碍。
短句来源
     ③epiphyseal cartilage;
     ③骨骺软骨 ;
短句来源
     Tissue Engineered Cartilage
     软骨组织工程
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Slitting sling,fixed insertion and overlapping with inferior wall of eustachiantube were observed of the levator veli palati muscle(LVP)in cleft palate children.In such cases the LVP muscle was not able to contract and raise the eustac-hian cartilage effectively.Thus the incidence of ottis media with effusion maybecome significantly high.In order to obtain good ventilatory function of eustach-ian tube,the LVP muscle should be retrogradely placed and its sling reconstructedduring the repair of cleft palate.

解剖了30例正常婴幼儿,7例腭裂婴幼儿的头部标本。结果显示腭裂标本咽鼓管软骨的质地较同龄者软,腭帆提肌吊带中断且前移位,止点相对固定。该肌的咽鼓管软骨部几乎位于咽鼓管下壁下方。经分析比较,作者认为腭裂患者的腭帆提肌不具有力收缩和上抬咽鼓管软骨的能力,造成鼓咽管通气障碍,导致中耳炎。腭裂修复应恢复良好的咽鼓管通气功能,术时须将腭帆提肌后移并重建肌肉吊带。

AbstractThe purpose of this study was to obtain more infor-mations about the levator veli palatini (LVP) musclethrough the dissection of head and neck cadaver samplesin 20 sides of normal adults, 4 sides of normal newbornsand 4 sides of cleft palate newborns and histological sec-tions in 6 normal fetuses. The results shown that themiddle part of LVP is 7. 9 mm in width, 3. 4mm inthickness and 29mmz on cross-section area. There aretwo angle relationship between LVP and the soft palate ,which are 44. 5° angle...

AbstractThe purpose of this study was to obtain more infor-mations about the levator veli palatini (LVP) musclethrough the dissection of head and neck cadaver samplesin 20 sides of normal adults, 4 sides of normal newbornsand 4 sides of cleft palate newborns and histological sec-tions in 6 normal fetuses. The results shown that themiddle part of LVP is 7. 9 mm in width, 3. 4mm inthickness and 29mmz on cross-section area. There aretwo angle relationship between LVP and the soft palate ,which are 44. 5° angle to midline sagittal section inwardand 33. 0°angle to coronal section forward. The LVPinsertion is between the 40% to 70% of the soft palatelength and only 25% insertion of LVP is located at thejunction area ofpalatal aponeurosis and fibromuscularcomponent of palatine raphe. Other parts of LVP insertion are merged into the midline fibromuscular component. The muscle has a close relationship with the medial parr of the carrilaglnous poriion of Eustachian tube.The LVP is supplied by the palatal branches of ascend-ing palatine artery and ascending pharyngeal artery,which are into the LVP at or above the level of the hardpalate. The LVP nerve branches coming from the pha-ryngeal plexus pass through the lateral pharyngealspace. The LVP reconstruction in cleft palate repair isdiscussed.

腭裂手术中,恢复腭帆提肌(levatorvelipalatinimuscle,LVP)的位置及其两侧的完整性极为重要,为给临床提供有价值的参考数据,作者对20侧健康成人、4侧健康新生儿及4侧唇腭裂新生儿头颈标本LVP的解剖和6例胎儿头颈部标本组织学连续切片进行观察,证实LVP位于软腭长度的40%~70%之间,在咽侧有肌纤维与咽上缩肌、咽腭肌相连续。该肌中份宽7.9mm,厚3.4mm,截面积29mm ̄2。成人LVP与软腭的前倾斜角为33.0°±5.1°,内倾斜角为44.5°±4.6°。LVP肌腹与咽鼓管软骨部内侧基底关系密切。该肌止点前部与咽腭肌一起止于腭正中筋膜起始处,其余与腭正中筋膜、悬雍垂肌周筋膜及对侧LVP相融合。该肌血管来自腭升动脉及咽升动脉腭支,均由硬腭水平或该水平之上进入LVP。LVP由咽丛神经分支支配,该分支经咽旁间隙常与LVP血管伴行,呈网状或干线状。

Purpose:To observe the morphological change under microscope and study the pathologic basis of radiation damage of middle ear.Methods:16 healthy adult guinea were exposed to a fractionated gamma radiation (6 Gy per day) with a total dose of 60 Gy on one side of the temporal bone. Every four animals were killed on the second, fourth, eighth and twelfth week after radiation and their irradiated eustachine tube and temproal bone were taken to study using microscope. Four animals not treated with γ ray were selected...

Purpose:To observe the morphological change under microscope and study the pathologic basis of radiation damage of middle ear.Methods:16 healthy adult guinea were exposed to a fractionated gamma radiation (6 Gy per day) with a total dose of 60 Gy on one side of the temporal bone. Every four animals were killed on the second, fourth, eighth and twelfth week after radiation and their irradiated eustachine tube and temproal bone were taken to study using microscope. Four animals not treated with γ ray were selected as contrasting group.Results:Nine of the sixteen experimental animals had tympanic membrane perforation on the irradiated side. In the irradiated tympanic cavities of the 16 animals there existed accumulations which were proved by light microscopy to be large amounts of pyocytes at second, fourth and eighth week after radiation and the pyocytes disappeared,plasma cells increased, granulation tissue were formed and produced new fibrous tissues in the twelfth week. The epithelial tissue of the tympanic cavity peeled off. The connective tissue under the mucosa become edema, capillaries dilate or even rupture, the blood vessel walls were thickened and the lumen narrowed. The epithelial cells of the eustachian tube damaged slightly and only a few infilitrated inflammation cells were found in the submucosa.Conclusions:After being exposed to γ ray to a total dose of 60 Gy the epithelial tissues of eustachian tube and middle ear mucosa were damaged and the latter was more serious. Bacteria infection and suppurative inflammation occurred and then granulation and fibrous tissues were produced in the middle ear. The connective tissue under the mucosa become edematous. The walls of the capillaries thickened and the lumen narrowed. This would be disadvantageous in the repair of the epithelium.

目的 :观察豚鼠咽鼓管鼓室粘膜受γ射线照射后损伤的显微形态学改变 ,对其放射损伤的病理学基础作初步研究。方法 :选择正常成年豚鼠 16只 ,耳颞部接受 6 0Gyγ射线照射。照射结束后 2、4、8及 12周分别处死动物各四只 ,用显微镜观察照射侧的中耳腔、咽鼓管、鼓室粘膜 ;对照组动物 4只不作照射 ,取鼓室粘膜和咽鼓管作正常对照。结果 :实验组全部照射鼓室腔均出现大量脓细胞 ,随后局部出现肉芽组织及纤维组织 ;鼓室粘膜上皮完全脱落 ;粘膜下组织水肿 ,血管壁增厚 ,管腔变窄 ;咽鼓管粘膜上皮细胞胞浆淡染 ,粘膜下少量炎症细胞浸润 ,咽鼓管软骨镜下未见病理改变。结论 :豚鼠耳颞部受 6 0Gyγ射线照射后 ,中耳腔内发生化脓性炎症 ,随后局部出现肉芽组织及纤维组织 ,对中耳传音功能造成难以恢复的损伤 ;照射后鼓室粘膜比咽鼓管粘膜的损伤更严重 ;咽鼓管、鼓室粘膜下组织微循环减少 ,不利于上皮组织细胞的修复

 
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