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幽门周围的
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     ANECDOTE PYLORUS
     “幽门螺”轶事
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     Therapie der Helicobacter-pvlori-Infektion
     幽门螺旋菌感染的治疗
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     DIFFRACTION PATTERN AROUND SINGULAR POINT
     亮点周围的衍射模式
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     The Stress Field Around A T-Shaped Crack of Thin Plate
     薄板在T型裂纹周围的应力场
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  peripyloric
The standard operation should be used in the presence of any sign of tumor infiltration of the duodenal bulb or peripyloric lymph nodes.
      
Some groups have argued against PPPD for cancer of the pancreatic head, because the pylorus-preserving procedure is likely to compromise the field of resection and does not allow lymph node dissection of the peripyloric and perigastric groups.
      


Objective To investigate the wide tropism of hepatitis B virus (HBV) and its relationship to Helicobacter pylori (Hp) infection. Methods Seventy-two patients were selected , among them 28 with chronic hepatitis B (chronic hepatitis B group) , 44 with hepatitis B associated with cirrhosis (cirrhosis group) ;30 patients with gastritis but without liver disease were used as control . In all patients diagnostic en-doscopy was performed. Three gastric biopsies were taken within 3 cm around the pylorus of gastric...

Objective To investigate the wide tropism of hepatitis B virus (HBV) and its relationship to Helicobacter pylori (Hp) infection. Methods Seventy-two patients were selected , among them 28 with chronic hepatitis B (chronic hepatitis B group) , 44 with hepatitis B associated with cirrhosis (cirrhosis group) ;30 patients with gastritis but without liver disease were used as control . In all patients diagnostic en-doscopy was performed. Three gastric biopsies were taken within 3 cm around the pylorus of gastric antra. U-rease detection, HE, Fuchsin staianing and immunohistochemical analysis of Hp IgG antigen were conducted. Results Different extent of chronic inflammation in cirrhosis group and chronic hepatitis B group was 95.5% (42/44) and 92. 9% (26/28) respectively. It indicated that in chronic hepatitis group most patients were complicated with chronic inflammation, while in cirrhosis group most patients with gland atrophy and intestinal metaplasia. In these two groups the positive rate of Hp in the inflammatory gastric mucosa was 69.0% (29/42) and 76.9% (20/26) respectively . No obvious difference existed between these two groups and control group. The rates of HBV antigen expression in the Hp positive, and Hp negative gastric mucosa were 69.8% (37/53) and 73.7% (14/19) respectively (P >0.05). Conclusions (l)There are obvious expression of HBV antigen in the gastric mucosa of patients with chronic liver disease and cirrhosis . Its role in the pathogenesis of gastric mucosal lesion is the worthful subject to be further studied. (2) No relationship between HBV antigen and Hp antigen expression was observed in this study.

目的 探讨乙型肝炎病毒(HBV)的泛嗜性及与幽门螺杆菌(Hp)感染的关系。方法选择慢性乙型肝炎(慢肝)28例、乙型肝炎后肝硬化(肝硬化)44例,共72例作为观察组,无肝病的胃病患者30例作为对照组。受检者常规胃镜检查,取胃窦幽门周围3cm以内活体组织3块,除普通病理检查外,分别做乙型肝炎病毒表面抗原(HBsAg)、乙型肝炎病毒核心抗原(HBcAg)检测及快速尿素酶、品红染色和免疫组化法检测Hp。结果 慢肝组有不同程度的胃粘膜慢性炎症者达92.9%(26/28)、肝硬化组达95.5%(42/44),其中慢肝组以单纯慢性炎症为多,而肝硬化组以伴萎缩和肠化者为多。72例慢性肝病者中有51例胃粘膜HBV阳性,其中HBsAg、HBcAg双阳性16例;肝硬化组HBV抗原表达高于慢肝组,而HBsAg、HBcAg双阳性者低于慢肝组(P均<0.05)。在慢肝和肝硬化组有炎症的胃粘膜中Hp阳性率分别为76.9%(20/26)、69.0%(29/42),与对照组相比无显著差别。慢性肝病Hp阳性、阴性者胃粘膜HBV抗原表达率分别为69.8%(37/53)、73.7%(14/19),亦无统计学差异(P>0.05)。结论 (1)HBV在...

目的 探讨乙型肝炎病毒(HBV)的泛嗜性及与幽门螺杆菌(Hp)感染的关系。方法选择慢性乙型肝炎(慢肝)28例、乙型肝炎后肝硬化(肝硬化)44例,共72例作为观察组,无肝病的胃病患者30例作为对照组。受检者常规胃镜检查,取胃窦幽门周围3cm以内活体组织3块,除普通病理检查外,分别做乙型肝炎病毒表面抗原(HBsAg)、乙型肝炎病毒核心抗原(HBcAg)检测及快速尿素酶、品红染色和免疫组化法检测Hp。结果 慢肝组有不同程度的胃粘膜慢性炎症者达92.9%(26/28)、肝硬化组达95.5%(42/44),其中慢肝组以单纯慢性炎症为多,而肝硬化组以伴萎缩和肠化者为多。72例慢性肝病者中有51例胃粘膜HBV阳性,其中HBsAg、HBcAg双阳性16例;肝硬化组HBV抗原表达高于慢肝组,而HBsAg、HBcAg双阳性者低于慢肝组(P均<0.05)。在慢肝和肝硬化组有炎症的胃粘膜中Hp阳性率分别为76.9%(20/26)、69.0%(29/42),与对照组相比无显著差别。慢性肝病Hp阳性、阴性者胃粘膜HBV抗原表达率分别为69.8%(37/53)、73.7%(14/19),亦无统计学差异(P>0.05)。结论 (1)HBV在慢肝及肝硬化患者胃粘膜表达明显,应重视其在胃粘膜病变中的作用,加强防护措施。(2)在胃粘膜中HBV与Hp表达未见相关关系。

To investigate the etiology of chronic hepatitis B(HB)post cirrhosis gastritic mucosal lesion.In 102 patients diagnostic endoscopy was performed.Three gastric biopsies were taken within 2-3cm around the pylorus of gastric antra,urease detection,HE fuchsin staining and immunohistochemical analysis of Helicobacter pylori(HP) IgG antigen,HBsAg and HBcAg were performed separately.In cirrhosis group,95.5% of 44 patients (42/44) showed different extent of chronic inflammation of gastric mucosa 92.9%(26/28)in chronic...

To investigate the etiology of chronic hepatitis B(HB)post cirrhosis gastritic mucosal lesion.In 102 patients diagnostic endoscopy was performed.Three gastric biopsies were taken within 2-3cm around the pylorus of gastric antra,urease detection,HE fuchsin staining and immunohistochemical analysis of Helicobacter pylori(HP) IgG antigen,HBsAg and HBcAg were performed separately.In cirrhosis group,95.5% of 44 patients (42/44) showed different extent of chronic inflammation of gastric mucosa 92.9%(26/28)in chronic hepatitis B group.In addition to chronic gastric mucous inflammation,the date of existence of gland atrophy or/and intestinal epithelialization is 33.3%(14/42) and 11.5%(3/26)respectively(P<0.05).The positivity of HBVAg in these two groups is 81.8%(36/44)and 53.6%(15/28)respectively(P<0.05).In these two groups the positive rate of HP in inflammatory gastric mucous is 69.0%(29/42)and 76.9%(20/26)respecctively,there is no obvious defference between them. Among the 42 patients with cirrhosis associated gastritis,19 were found to be esophageales varicosis positive,but the other 23 were not,the positivity of HP of these two groups was 73.7%(14/19)and 65.3%(15/23)(P>0.05).The result of analysis of the severity of HB associated gastritis indicated that the portal hypertensive cirrhosis in later stage of liver disease plays an important role in the development of this disease.The relationship between hepatitis B virus and this disease needs further investigation.HP is not the pathogeny of this disease.

探讨乙型肝炎后肝硬化胃粘膜病变发病因素。 10 2例肝病或非肝病因伴有胃病行胃镜检查的患者 ,取胃窦小弯距幽门周围 ( 2 - 3 )cm处粘膜 3块 ,分别做快速尿素酶试验及嗜伊红、品红染色 ,并行免疫组化检测幽门螺旋杆菌HPlgG型抗原。发现肝硬化组 (LC组 )与慢性肝炎组 (HB组 )有不同程度胃粘膜炎症达 95 5 % ( 42 / 44 )、5 3 6% ( 15 / 2 8) ;前者萎缩、肠化多于后者 (P <0 0 5 )。上述两组乙型肝炎病毒 (HBV)抗原表达率分别为 81 8% ( 3 6/44 )、5 3 6% ( 15 / 2 8) ;前者多于后者 (P <0 0 5 )。与非肝病胃病患者 3 0例相比 ,LC组、HB组HP阳性率无明显差别(P >0 0 5 )。说明门脉高压及HBV是LC胃粘膜病变的主要发病因素

Objective:To investigate the wide tropism of hepatitis B and C virus(HBV?HCV).Methods:Seventy-two patients were selected, among them 28 with chronic hepatitis B (chronic hepatitis B group), 44 with hepatitis B associated with cirrhosis (cirrhosis group); 30 patients with gastritis but without liver disease were used as control. In all patients diagnostic endoscopoy was performed. Two gastric biopsied were taken within 3 cm around the pylorus of gastric antra. Urease detection, HE, and immunohistochemical analysis...

Objective:To investigate the wide tropism of hepatitis B and C virus(HBV?HCV).Methods:Seventy-two patients were selected, among them 28 with chronic hepatitis B (chronic hepatitis B group), 44 with hepatitis B associated with cirrhosis (cirrhosis group); 30 patients with gastritis but without liver disease were used as control. In all patients diagnostic endoscopoy was performed. Two gastric biopsied were taken within 3 cm around the pylorus of gastric antra. Urease detection, HE, and immunohistochemical analysis of HBV and HCV antigen were conduted. Results:Different extent of chronic inflammation in cirrhosis group most patients with gland atrophy and intestinal metaplasia. The positivity of HBVAg in these two groups was 81.8%(36/44)?53.6%(15/28), respectively (P<0.05). The positivity of HCVAg in 51 patients with chronic liver disease is 64.7%(33/51).Conclusion: There are obvious expression of HBV antigen in the gastric mucosa of patients with chronic liver disease and cirrhosis. Its role in the pathogenesis of gastric mucosal lesion is the worthful subject to be further studied.

目的 :探讨乙、丙型肝炎病毒 (HBV、HCV)的泛嗜性。方法 :选择慢性乙、丙型肝炎 (慢肝组 ) 2 8例、肝炎肝硬化 (肝硬化组 ) 44例 ,共 72例作为研究对象。受检者常规胃镜检查 ,取胃窦幽门周围 3cm以内活体组织两块 ,除普通病理检查外 ,分别做乙型肝炎病毒表面抗原 (HBsAg)、乙型肝炎病毒核心抗原 (HBcAg)、丙型肝炎病毒抗原 (HCVAg)免疫组化法检测。结果 :慢肝组有不同程度的胃黏膜慢性炎症者达 92 9% ( 2 6/2 8)、肝硬化组达95 5 % ( 4 2 /4 4) ,排除年龄影响因素外 ,慢肝组以单纯慢性炎症为多 ,而肝硬化组以伴萎缩和肠化者为多。慢肝组与肝硬化组患者分别有 5 3 6% ( 15 /2 8)、 81 8% ( 3 6/4 4)胃黏膜HBVAg阳性 ,其中HBsAg、HBcAg双阳性 3 1例。在5 1例患者胃黏膜HCVAg检测中有 3 3例 (占 64 7% )阳性表达、 66 7% ( 2 2 /3 3 )与HBVAg同时表达。肝硬化组HBVAg及HBsAg、HBcAg双阳性者均高于慢肝组 (P值均 <0 0 5 )。 结论 :HBV...

目的 :探讨乙、丙型肝炎病毒 (HBV、HCV)的泛嗜性。方法 :选择慢性乙、丙型肝炎 (慢肝组 ) 2 8例、肝炎肝硬化 (肝硬化组 ) 44例 ,共 72例作为研究对象。受检者常规胃镜检查 ,取胃窦幽门周围 3cm以内活体组织两块 ,除普通病理检查外 ,分别做乙型肝炎病毒表面抗原 (HBsAg)、乙型肝炎病毒核心抗原 (HBcAg)、丙型肝炎病毒抗原 (HCVAg)免疫组化法检测。结果 :慢肝组有不同程度的胃黏膜慢性炎症者达 92 9% ( 2 6/2 8)、肝硬化组达95 5 % ( 4 2 /4 4) ,排除年龄影响因素外 ,慢肝组以单纯慢性炎症为多 ,而肝硬化组以伴萎缩和肠化者为多。慢肝组与肝硬化组患者分别有 5 3 6% ( 15 /2 8)、 81 8% ( 3 6/4 4)胃黏膜HBVAg阳性 ,其中HBsAg、HBcAg双阳性 3 1例。在5 1例患者胃黏膜HCVAg检测中有 3 3例 (占 64 7% )阳性表达、 66 7% ( 2 2 /3 3 )与HBVAg同时表达。肝硬化组HBVAg及HBsAg、HBcAg双阳性者均高于慢肝组 (P值均 <0 0 5 )。 结论 :HBV、HCV在慢性及肝硬化患者胃黏膜表达明显 ,应重视其在胃黏膜病变发病中的作用 ,并加强防护措施

 
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