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   肾小管细胞 的翻译结果: 查询用时:1.595秒
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肾小管细胞     
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  renal tubular cell
     The combination at the subtherapeutic dose of RPM ( 0.5?mg/kg) and CsA (4?mg/kg every day) resulted in the slight increase of glycemia,and the slight degree of renal tubular cell impairment and tubulointerstitia fibrosis, P< 0.05 versus RPM 1? mg/kg each day + CsA 4?
     而亚治疗剂量的RPM (0 .5mg·kg-1·d-1)与CsA(4mg·kg-1·d-1)联用只引起轻度的血糖上升及肾小管细胞受损与间质纤维化 ,与前者相比 ,P <0 .0 5。
短句来源
     Methods Establish the model of renal tubular cell injury by H_2O_2,and then SDH and LDH in cultured renal tubularepithelial cell was demonstrated with histochemiatry methlds.
     方法建立H2O2致鼠肾小管细胞损伤模型,用组织化学方法显示培养小管细胞琥珀酸脱氢酶(SDH)和乳酸脱氢酶(LDH)。
短句来源
     Objective To investigate the effect of SMI on the aerobic metabolism of the H_2O_2-induced renal tubular cell injury.
     目的探讨参麦注射液(SMI)对H2O2导致的肾小管细胞氧化性损伤的有氧代谢的影响。
短句来源
     CONCLUSION: TNF-α induced by anoxia/reoxygenation may participate in the process of renal tubular cell injury.
     结论:肾小管缺氧-复氧后,TNF-α的产生增多,并参与了肾小管细胞损伤的过程。
短句来源
     Results SMI and coenzyme Q_(10) can not only reduce SDH and LDH activity loss in oxidative damaged renal tubular cell,with distinguished significance(P<0.01) compared with control group,but also pretect renal tubular cells those are not damaged.
     结果参麦注射液和泛醌都具有减轻氧化性损伤肾小管细胞SDH及LDH活性丧失的作用,与模型组比较有显著性差异(P<0.01),且参麦注射液对未损伤的小管细胞有明显的保护作用(P<0.01)。
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  renal tubular cells
     Effects of GS Rb_1 and Rg_1 on Cultured Renal Tubular Cells Injured by Hypoxia-reoxygenation
     人参皂苷Rb_1与Rg_1对肾小管细胞缺氧复氧损伤模型的影响
短句来源
     Methods: Human renal tubular cells were cultured in the culture medium containing different PDS concentrations (0μg.ml -1 ,5μg.ml -1 ,10μg.ml -1 ,25μg.ml -1 ,50μg.ml -1 ,75μg.ml -1 ,100μg.ml -1 ).
     方法 :用含有 7种不同浓度的人参二醇组皂甙 (0ug .ml-1、5ug.ml-1、10ug .ml-1、2 5ug .ml-1、5 0ug .ml-1、75ug.ml-1、10 0ug.ml-1)的培养基 ,在96孔培养板中培养人肾小管细胞 96小时后 ,以MTT法判定细胞的增殖情况。
短句来源
     CONCLUSION:Rb1 and Rg1 have protective effects on renal tubular cells injured by hypoxia-reoxygenation.
     结论:人参皂苷Rb1和Rg1对HRO性肾小管细胞损伤模型具有保护作用。
短句来源
     Effects and mechanisms of panaxadiol saponins(人参二醇组皂甙) promoting the proliferation of scathing cultured human renal tubular cells
     人参二醇组皂甙促进受损的培养肾小管细胞增殖的作用及机制研究
短句来源
     STUDY ON THE EFFECT AND MECHANISM OF IL - IRA ON CULTURED HUMAN RENAL TUBULAR CELLS
     IL-1受体拮抗剂对培养人肾小管细胞的影响及机制研究
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  renal tubule cells
     The results suggested that the ET1 effect on renal function impairment was mediated mainly through its effect on renal hemodynamics and might also perhaps through its direct effect on the renal tubule cells.
     结果表明:再灌注后ET-1很可能主要通过对肾脏血液动力学的作用发挥对肾功能的损害作用,对肾小管细胞也可能有直接作用。
短句来源
  human renal tubular cell
     EFFECTS OF PANAXADIOL SAPONINS ON PROLIFERATION OF HUMAN RENAL TUBULAR CELL
     人参二醇组皂甙对人肾小管细胞增殖的影响
短句来源
     Effect of Postasphyxial-Serum of Neonate in Inducing Injury of Human Renal Tubular Cell
     新生儿窒息后血清在诱导人肾小管细胞损伤中的作用
短句来源
     Role of Erythropoietin in Relieving Injury of Human Renal Tubular Cell Induced by Postasphyxial-Serum of Neonates
     促红细胞生成素减轻窒息新生儿血清诱导人肾小管细胞损伤
短句来源
     Methods:From the cultured human renal tubular cell strain the IR model was made and the drugs were tested. The proliferative activity of tubular cell was measured with the method of MTT. The content of ATPase and LDH on cell membrane and mitochondrion were detected by the histochemistry method.
     方法 :用传代培养的肾小管细胞复制缺血再灌注模型 ,MTT法测定细胞增殖活性 ,酶组化法测定细胞膜和线粒体中ATP酶和LDH含量的变化 ,用生化法检测小管细胞内SOD活性和MDA含量 ,观察参麦注射液 (SMI)对培养的鼠肾小管细胞的增殖和对缺血再灌注性小管细胞损伤的作用。
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  renal tubular cell
Cilastatin increases the bioavailability of imipenem, probably as a result of competitive displacement at the renal tubular cell.
      
However, it is not possible to measure to what extent the renal tubular cell is protected by cilastatin from the toxic effect of imipenem as cilastatin itself is also tubulotoxic.
      
The protective effect of FN against renal tubular cell injury by exposure to oxalate and COM crystals was examined by measuring the lactate dehydrogenase (LDH) activity and using a non-radioactive proliferation assay.
      
FN was found to have an inhibitory effect on renal tubular cell injury caused by exposure to oxalate and COM crystals.
      
We speculate that the inhibitory effect of FN on the adhesion of COM crystals is related to the inhibitory effect of exposure to COM crystals on renal tubular cell injury.
      
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  renal tubular cells
Cubilin was required for handling a greater amount of protein in nephrotic status and albumin-induced production of MCP-1 and RANTES by renal tubular cells, which further initiated tubulointerstitial inflammation in proteinuric disease.
      
The dystrophy developing in the kidney after cutting the sciatic nerve blocks the compensatory increase in specific accumulation of (3H)aldosterone in the cytoplasm of the renal tubular cells.
      
These results are additional evidence that the patients in group 2 have abnormal calcium metabolism, the abnormality being in the postadenylate cyclase-cAMP pathway in the renal tubular cells.
      
The nocturnal excretion of renal tubular cells was determined.
      
Effect of sodium fluoride on the expression of Bcl-2 family and osteopontin in rat renal tubular cells
      
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  renal tubule cells
In this review cellular transport systems for oxalate will be summarized with special attention for the progress that has been made to study oxalate transport in a model of cultured renal tubule cells.
      
Immunoenzyme localization of cathepsins in the Golgi region of rat hepatocytes and renal tubule cells
      
Studies were undertaken to define the effect of acute metabolic alkalosis (hypertonic sodium bicarbonate i.v.) on the chemical gradients for potassium, sodium and chloride across the apical membrane of individual renal tubule cells.
      
Effect of acute metabolic acidosis on transmembrane electrolyte gradients in individual renal tubule cells
      
Comparable results were derived from lymphoblastoid cells and renal tubule cells isolated from a patient's overnight urine confirming that cell lines provide a good model for mRNA analysis.
      
更多          
  human renal tubular cell
The binding of Shiga toxin (Stx) to Gb3Cer? in detergent-insoluble microdomains (DIM)/raft of the ACHN human renal tubular cell line causes the temporal activation of the Src-family kinase Yes [1].
      
  其他


The electron microscopical study of acute experimental poisoning of pterygium wilfordii revealed definitely ultrastructural changes of myocardium,kidney,spleen and liver.The myocardium showed formation of multiple characteristic microfcci cf myo-lysis,the kidney showed degeneration and necrosis of tubular cells,when the outstanding damage of the spleen was consisted mainly of diffuse necrosis of lymphocytes.The liver was only slightly damaged.According to the preliminary analysis of the observed ultrastructural...

The electron microscopical study of acute experimental poisoning of pterygium wilfordii revealed definitely ultrastructural changes of myocardium,kidney,spleen and liver.The myocardium showed formation of multiple characteristic microfcci cf myo-lysis,the kidney showed degeneration and necrosis of tubular cells,when the outstanding damage of the spleen was consisted mainly of diffuse necrosis of lymphocytes.The liver was only slightly damaged.According to the preliminary analysis of the observed ultrastructural changes of different organs,the authors concluded that the myocardium is quite sensible to the poisonous plant pterygium wilfordii,and as a significant organ of immunity,the spleen will be markedly damaged under poisonous doses of this drug.

本文对实验性急性雷公藤中毒进行了电子显微镜观察,发现明显的心肌病变,其主要特征为多发性微小肌溶灶的形成;肾脏主要病变为肾小管细胞的变性及坏死;脾脏的突出损伤为淋巴细胞的广泛崩解;肝脏损伤则较轻微。本文对各脏器病变进行了分析,认为心肌对雷公藤具有较高的敏感性,中毒剂量雷公藤对重要免疫器官脾脏有明显的损伤作用。

The excretory sy tom of snail Bradybaeua maacki consi t of two parts…kidney and ureter. The kidney is a gland,it is made up of threo parts: 1.Renal cap ule: it is made of cuboidal epithelium. 2.Uretral part. 3.Renal paronchyma: it is compo ed of many ronal tubules,the wall of each tubulo is covered with sceretory opithelium,the cell body of this opithelium links oach othor to form.a reticular tructuro in the renal tubule~-.The blood cells of this kind of snail scatter in the roticular structure.The shaps of...

The excretory sy tom of snail Bradybaeua maacki consi t of two parts…kidney and ureter. The kidney is a gland,it is made up of threo parts: 1.Renal cap ule: it is made of cuboidal epithelium. 2.Uretral part. 3.Renal paronchyma: it is compo ed of many ronal tubules,the wall of each tubulo is covered with sceretory opithelium,the cell body of this opithelium links oach othor to form.a reticular tructuro in the renal tubule~-.The blood cells of this kind of snail scatter in the roticular structure.The shaps of these blood cell is spocial,met of them are round,but a fw aro,irregular.The brooding way of the blood oell is direct coll divi ion. The snail kidnoy is not only an exoretory orgen,but also a storing and making blood cells orgon.There are no rich artris and veins in the kidney,the blood cells scatter completely among the ll bodies of the renal tubules.

马氏巴蜗牛的肾脏是一个腺体,它是由三个部分组成;肾被膜,肾输尿管部和肾实质部。肾脏和辅尿管构成蜗牛的排泄系统。它的肾脏并不仅仅是一个排泄器官,同时又是一个储存和制造血液的器官。肾脏中无丰富的动、静脉,血球完全分散存在于组成肾小管的细胞与细胞之间。

Gossypol has been shown to cause a side effect hypokalemia. This study was designed to evalulated the possible role of gossypol in relation to renal potassium loss by using histochemical method for comparative analysis of gossypol and gentamicin, a well known tubulo-toxic agent, as positive control with respect to nephrotoxic demage in rats and guinea pigs. The results indicated that gossypol had obvious effect on reabsorption function by inducing transitional decrease of membrane enzymes activities of renal...

Gossypol has been shown to cause a side effect hypokalemia. This study was designed to evalulated the possible role of gossypol in relation to renal potassium loss by using histochemical method for comparative analysis of gossypol and gentamicin, a well known tubulo-toxic agent, as positive control with respect to nephrotoxic demage in rats and guinea pigs. The results indicated that gossypol had obvious effect on reabsorption function by inducing transitional decrease of membrane enzymes activities of renal tubular cells but had no effect as comparable with gentamicin to cause structureal damage of tubular cells.

棉酚可导致低血钾症已有实验证明.本文应用组织化学方法分析大鼠和豚鼠喂服棉酚及已知肾小管毒剂的庆大霉素(作为阳性对照)后肾的毒性损伤,着重研究在有关肾性失钾中,棉酚所起的作用。结果表明棉酚使肾小管膜酶暂时性减弱,影响重吸收功能.但和庆大霉素相比,棉酚不会引起肾小管细胞的结构损伤.

 
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