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钾通道     
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  potassium channels
     Compared to the expression of potassium channels Kv9.1, Kv9.2, Kv9.3 mRNA on the 7 day after myocardial infarction, potassium channels Kv9.1, Kv9.2, Kv9.3 mRNA remarkably decreased on the 30 day (P<0.05).
     心肌梗死组30天时与7天时比较钾通道Kv9.1、Kv9.2、Kv9.3mRNA量下降(P<0.05)。
短句来源
     Roles of the Kv1.2, Kv1.5, Kv2.1 potassium channels in hypoxia pulmonary vasoconstriction
     Kv1.2、Kv1.5、Kv2.1钾通道在缺氧性肺血管收缩中的作用
短句来源
     Compared to MI groups, potassium channels Kv9.1, Kv9.2, Kv9.3mRNA in the metoprolol group remarkably increased on the 30 day (P<0.05).
     与心肌梗死组比较,30天时美托洛尔组钾通道Kv9.1、Kv9.2、Kv9.3mRNA明显升高(P<0.05);
短句来源
     Results: Compared to the sham-groups, potassium channels Kv9. 1, Kv9.2, Kv9.3 mRNA in the MI groups remarkably decreased regardless of 7 days or 30 days (P<0.05 or P<0.01).
     结果:与假手术组比较,无论7天或30天,心肌梗死组钾通道Kv9.1、Kv9.2、Kv9.3mRNA均显著降低(P<0.05或P<0.01)。
短句来源
     Compared to the sham-groups, potassium channels Kv9.1, Kv9.2, Kv9.3 mRNA in the metoprolol groups remarkably decreased regardless of 7 days or 30 days (P<0.05).
     与假手术组比较,无论7天或30天,美托洛尔组钾通道Kv9.1、Kv9.2、Kv9.3mRNA均明显降低(P均<0.05);
短句来源
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  potassium channel
     Characteristics of KCNQ2/3 potassium channel current and its modulation by M_1 receptor
     KCNQ2/3钾通道电流特征及M_1受体的调节作用
短句来源
     ②Different kinds of potassium channel blockers, such as 1 mmol/L tetraethylammonium, 1 mmol/L 4-aminopyridine, 100 nmol/L Iberiotoxin(IBTX) or 100 μmol/L paxilline were added into culture medium ten minutes before the treatment of SNP (0.1 mmol/L) and neuronal survival rate were estimated 18 hours later.
     ②硝普钠(0.1mmol/L)处理前10min培养液内预先分别给予钾通道阻断剂四乙铵1mmol/L、4-氨基砒碇1mmol/L、Iberiotoxin(IBTX)100nmol/L或paxilline100μmol/L,18h后测定神经元存活率。
短句来源
     Conclusion The expression of potassium channel Kv9.1,Kv9.2 and Kv9.3 mRNA after myocardial infarction exhibits the time-dependent downregulation.
     结论心肌梗死后钾通道Kv9.1、Kv9.2、Kv9.3 mRNA表达呈时间依赖性下调。
短句来源
     A Study of Kir2.1, Kir3.4 and Kv4.3 Potassium Channel GeneExpression in Atria of Patients with Chronic Atrial Fibrillation
     慢性心房颤动患者心房肌细胞Kir2.1、Kir3.4和Kv4.3钾通道重构的研究
短句来源
     Kir2.1 and Kir3.4 potassium channel gene expression in atria of patients with atrial fibrillation
     心房颤动患者心房组织钾通道Kir2.1和Kir3.4的基因表达
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  k + channel
     Objective To investigate the changes and Significance of Ca 2+ activated K + channel(K Ca) in mesenteric arterial smooth muscle(MASM) cells of essential hypertension.
     目的 研究高血压病 (essential hypertension,EH )患者肠系膜动脉平滑肌 (mesenteric arterial sm oothmuscle,MASM)细胞钙激活钾通道 (Ca2 + activated K+ channel,KCa)改变及意义。
短句来源
     The animals received isoflurane(1.2%~1.5%) preconditioning 60 minutes before I/R in ISO group,received 5-HD(10 mg/kg),the mitochondrial ATP-sensitive K+ channel(mitoKATP) blocker,and ISO before I/R in 5-HD+ISO group,and received 5-HD 30 minutes before I/R in 5-HD group.
     ISO组,I/R前60min接受1.2%~1.5%的ISO预处理; 5-HD+ISO组,线粒体ATP敏感性钾通道(mitoKATP)阻滞剂5-HD 10mg/kg腹腔内注射30min后同ISO组;
短句来源
     The results showed that ①In whole cell recordings with voltage ramp protocol,a Voltage dependent K +(Kv) channels was recorded in Daudi cells,which was sensitive to quinine 100 μmol·L -1 and charybdotoxin (CTX) 100 nmol·L -1 ,K + channel blockers.
     结果表明,①全细胞voltagerampprotocol 证实有Kv 通道的存在,该Kv 通道对钾通道阻断剂quinine 100μmol·L- 1 及charybdotoxin(CTX)100nmol·L- 1 敏感。
短句来源
     (5) In 12 neurons, the large-conductance Ca2+-activated K+ channel blocker tetraethylammoniumchloride (TEA) 1 mmol/L significantly increased the discharge rate of 10/12 (83.3%) neurons. Resveratrol (5 μmol/L) inhibited theincreased discharges of 9/10 (90%) neurons.
     (5)灌流大电导钙激活性钾通道阻断剂tetraethylammoniumchloride(TEA)1mmol/L后,大多数神经元(10/12,83.3%)放电增加,在此基础上灌流白藜芦醇(5μmol/L)2min,(9/10,90%)放电频率明显减低。
短句来源
     Results: A big conductance calcium activated K + channel was observed,with symmetrical 140mol/L across patches,in ward conductance was (171±12) pS and change little as different [Ca 2+ ] was included in the bath solution. Outward conductance was regulated by and was (76±14)pS with 500 μmol/L [Ca 2+ ].
     结果 :记录到一种大电导钙激活钾通道 (KCa) ,在对称 140mmol/L [K+ ]时内向电导为 (171± 12 )pS ,不随 [Ca2 + ]变化而改变 ,而外向电导可受 [Ca2 + ]调控 ,当 [Ca2 + ]为 5 0 0 μmol/L时 ,外向电导为 (76± 14)pS。
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  k ~ + channel
     The HeLa cells were prevented partially from As_2O_3-induced cell death by co-application for 24h with typical voltage- dependent K~+ channel blockers,4-aminopyridine (3mmol·L~(-1)) or tetraethylammonium (5mmol· L~(-1)).
     As_2O_3诱导的 HeLa 细胞死亡可被共同孵育钾通道阻滞剂四氨基吡啶(3mmol·L~(-1))或四乙基铵(5mmol·L~(-1))所部分抑制。
短句来源
     Conclusion Chronic treatment with As_2O_3 increased voltage-dependent K~+ currents in HeLa cells and the cell death induced by As_2O_3 was reduced partially by voltage-dependent K~+ channel blockers,4-aminopyridine or tetraethylammonium.
     结论 As_2O_3长期处理增加 HeLa 细胞的电压依赖性钾电流。 As_2O_3诱导的 HeLa 细胞死亡可被钾通道阻滞剂四氨基吡啶或四乙基铵部分抑制。
短句来源
     Effect of BQ123 on the expression of voltage-gated K~+ channel of rat pulmonary artery smooth muscle cells
     BQ123对大鼠肺动脉平滑肌细胞电压门控钾通道表达的影响
短句来源
     Objective To investigate the effect of voltage dependent K~+ channel blocker(4-aminopyridine,4-AP) and calcium activated K~+ channel blocker(tetraethylammonia,TEA)on human pulmonary adenocarcinoma AGZY-83-a cell line and to explore its mechanism.
     目的研究电压依赖性延迟整流钾通道阻断剂4-氨基吡啶(4-aminopyridine,4-AP)、钙离子激活钾通道阻断剂四乙铵(tetraethylammonia,TEA)对人肺腺癌细胞(AGZY-83-a)的影响,并探讨人肺腺癌细胞株凋亡的机制。
短句来源
     ACTIVATION OF CA~(2+)-ACTIVATED K~+ CHANNEL IN PORCINE CORONARY ARTERIAL SMOOTH MUSCLE CELLS BY MG~(2+)
     Mg~(2+)对猪冠脉平滑肌细胞钙激活钾通道的激活作用的初步研究
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  potassium channels
Role of Opiate Receptors and ATP-Dependent Potassium Channels of Mitochondria in the Formation of Myocardial Adaptive Resistance
      
The Role of Potassium, Potassium Channels, and Symporters in the Apoptotic Cell Volume Decrease: Experiment and Theory
      
Potential RNase targets in a tumor cell and the role of modulation of calcium-dependent potassium channels and the ras oncogene in RNase-induced cell damage are considered.
      
The conductance of outward- and inward-rectifying potassium channels, as well as the currents of excitable chloride channels, decreased after 2-3 days of heat treatment.
      
By the 15th-17th days, the conductance of potassium channels was reduced by a factor of 3-5, whereas the peak values of the chloride current, associated with the action potential, was reduced by a factor of 8-10.
      
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  potassium channel
Guanidine-type compounds that reached clinical status include amongst others the potassium channel opener, pinacidil and the histamine H2-receptor antagonists (e.g.
      
Permeation mechanism of a two-state potassium channel
      
The regulatory mechanism of the mitochondrial ATP-dependent potassium channel by the adenine nucleotides
      
Potassium Channel Modulation by Acetylcholinesterase Inhibitor Donepezil
      
Study of the interaction of unaggregated and aggregated amyloid β protein (10-21) with outward potassium channel
      
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  k + channel
These systems are localized in the plasma membrane of the corresponding cells and are composed of O?2-generating NADPH-oxidase and an H2O2-activated K+ channel.
      
Analysis of the diffusion and accumulation of potassium in mouse zygotes mediated by the cyclic activity of 240 pS K+ channel
      
This system is likely to represent a K+ channel that catalyzes the electrogenic transfer of potassium ions to the mitochondrial matrix.
      
Double-gating mechanism and diversity of an adenosine triphosphate (ATP)-sensitive K+ channel in neurons acutely dissociated fro
      
voltagedependent fast and slow K+ channels, Ca2+-activated K+ channel and ATP-sensitive K+ channel.
      
更多          
  k ~ + channel
These systems are localized in the plasma membrane of the corresponding cells and are composed of O?2-generating NADPH-oxidase and an H2O2-activated K+ channel.
      
Analysis of the diffusion and accumulation of potassium in mouse zygotes mediated by the cyclic activity of 240 pS K+ channel
      
This system is likely to represent a K+ channel that catalyzes the electrogenic transfer of potassium ions to the mitochondrial matrix.
      
Double-gating mechanism and diversity of an adenosine triphosphate (ATP)-sensitive K+ channel in neurons acutely dissociated fro
      
voltagedependent fast and slow K+ channels, Ca2+-activated K+ channel and ATP-sensitive K+ channel.
      
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