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致敏物质
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  “致敏物质”译为未确定词的双语例句
     Common contact sensitizers in the Czech Republic. Patch test results in 12,058 patients with suspected contact dermatitis
     捷克共和国的常见接触性致敏物质:疑有接触性皮炎的12058例患者的斑贴试验结果
短句来源
     Allergic Purpura is also called Schonlein-Henoch purpura which is a frequent blood vessel allergia hemorrhagic disease. It is thought that what leads to this disease is the allergy caused by organism happening to some sensitizing substance . This allergy causes the augment of capillary fragility and permeability and blood extravasation which creates skin purpura , mucosa and some organic bleeding .
     过敏性紫癜(Allergic Purpura),又称出血性毛细血管中毒症,为一种常见的血管变态反应性出血性疾病,因机体对某些致敏物质发生变态反应,导致毛细血管脆性及通透性增加,血液外渗,产生皮肤紫癜,黏膜及某些器官出血。
短句来源
     (4) bronchial asthma may be also relatedto allergic individual and impurity of the drug abused.
     (4)哮喘发作的原因可能与过敏体质和毒品中含有致敏物质有关。
短句来源
     Objective To study whether illers exists substance increasing tile sensitivityto endoloxin in burn rat serum and its role ill the endotoxill-induced liver injury.
     目的探讨烧伤血清中是否存在内毒素致敏物质及其在内毒素诱导肝细胞损伤中的作用。
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  相似匹配句对
     On Substance
     论物质
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     Basic knowledge of analytical reagents-biological material of high molecular weight
     生物高分子物质
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     CONCLUSION: Cough response in sensitized guinea pigs is enhanced and finally develops into wheezing with a progress in eosinophilic airway inflammation.
     结论 :致敏豚鼠?
短句来源
     ② anaphylaxis test;
     ②致敏试验。
短句来源
     (4) bronchial asthma may be also relatedto allergic individual and impurity of the drug abused.
     (4)哮喘发作的原因可能与过敏体质和毒品中含有致敏物质有关。
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Pigmentedcosmeticdermatitiscomposesamajorsortofundesirablereactionsinducedbycosmetics,butthemelanogenicefectofcosmeticscanscarcelybedetectedwithpatchtestsinhuman.Inthepresentstudy,10cosmeticproductsmostfrequentlyusedbythepatientswithpigmentedcosmet-icdermatitisand8ingredientscontainedinthecosmeticswerepaintedontoseparateareasofthedorsalflankofbrownishguineapigsfolowingthemethodreportedbyImokawaetal,thenskinspecimenswereremoved,theparafinimbededsectionswerestainedwithH.E.,Schmorl′sreactionwasusedtoshowmelanin,andBacher,PraverandThatcher′sdopa-oxidasereactiontostudythemelanogenicef-fectofthecosmeticsandtheingredients.Theresultsshowedthatalofthe10cosmeticstestedsignifi-cantlyincreasedthenumberofcelscontainingmelaninintheepidermalbasallayeroftheguineapigs,whileamongthe8ingredientstested,only5(carmine,parabens,limocitrin,resinandlanolin)re-vealedthiskindofefect.Thissuggeststhattheguineapigmodel,beingsensitiveinresponsetothemelanogenicactivityofcosmeticsontheonehandandreactingdiferentialytovariouskindsofingredi-entsontheother,mightbeusedinassayingthemelanogeniceffectofcosmeticproducts.Theexistenceandthedegreeofthehistologicalinflammatoryreactioninducedbythetestedmaterialsdidnotgoparal-leltothoseofhyperpigmentation,suggestingthatcosmeticinducinghyperpigmentationmaynotbesim-plyasequenceofdermatitis.Therefore,tosolvetheproblemofcosmetichyperpigmentationitshouldnotonlyavoidthesensitizers,butalsothemelanogenicagentscontainedinthecosmeticproducts....

Pigmentedcosmeticdermatitiscomposesamajorsortofundesirablereactionsinducedbycosmetics,butthemelanogenicefectofcosmeticscanscarcelybedetectedwithpatchtestsinhuman.Inthepresentstudy,10cosmeticproductsmostfrequentlyusedbythepatientswithpigmentedcosmet-icdermatitisand8ingredientscontainedinthecosmeticswerepaintedontoseparateareasofthedorsalflankofbrownishguineapigsfolowingthemethodreportedbyImokawaetal,thenskinspecimenswereremoved,theparafinimbededsectionswerestainedwithH.E.,Schmorl′sreactionwasusedtoshowmelanin,andBacher,PraverandThatcher′sdopa-oxidasereactiontostudythemelanogenicef-fectofthecosmeticsandtheingredients.Theresultsshowedthatalofthe10cosmeticstestedsignifi-cantlyincreasedthenumberofcelscontainingmelaninintheepidermalbasallayeroftheguineapigs,whileamongthe8ingredientstested,only5(carmine,parabens,limocitrin,resinandlanolin)re-vealedthiskindofefect.Thissuggeststhattheguineapigmodel,beingsensitiveinresponsetothemelanogenicactivityofcosmeticsontheonehandandreactingdiferentialytovariouskindsofingredi-entsontheother,mightbeusedinassayingthemelanogeniceffectofcosmeticproducts.Theexistenceandthedegreeofthehistologicalinflammatoryreactioninducedbythetestedmaterialsdidnotgoparal-leltothoseofhyperpigmentation,suggestingthatcosmeticinducinghyperpigmentationmaynotbesim-plyasequenceofdermatitis.Therefore,tosolvetheproblemofcosmetichyperpigmentationitshouldnotonlyavoidthesensitizers,butalsothemelanogenicagentscontainedinthecosmeticproducts.

用Imokawa等的实验方法,用10种化妆品及8种化妆品化学成分涂于黄棕色豚鼠侧背部皮肤,然后取材,进行HE染色、黑素染色及Dopa氧化酶染色,以探讨化妆品及其化学成分的致色素作用。结果:10种化妆品均显著增加黄棕色豚鼠表皮基底细胞中含黑素的细胞数,而8种化妆品成分中仅胭脂红、对羟基苯甲酸、柠檬黄、松香、羊毛脂等5种引起此种改变。提示此动物模型易于显示化妆品的致色素作用,又可显示化学品致色素作用的差异。并观察到组织有无炎症反应及反应程度与色素沉着不完全平行,提示化妆品及其成分所致的色素沉着并非全是炎症的后果,因此,在化妆品生产中不仅要避免化妆品中所含的致敏物质,还须设法避免致色素的物质

In clinic, brochial asthma caused by chaising heroin is not rare. In or-der to find out its morbidity and clinical characteristics, we made a retrospective studyon the patients who volunteered to accept heroin detoxification in our hospital, fromJune, 1993 to August, 1994. The results showed that: (1) morbidity of bronchial asthmacaused by heroin abuse was 4%; (2) the relationship between occurrence of asthma andtime and quantity the patients abused heroin was not obvious; (3) It was the abuse routechaising...

In clinic, brochial asthma caused by chaising heroin is not rare. In or-der to find out its morbidity and clinical characteristics, we made a retrospective studyon the patients who volunteered to accept heroin detoxification in our hospital, fromJune, 1993 to August, 1994. The results showed that: (1) morbidity of bronchial asthmacaused by heroin abuse was 4%; (2) the relationship between occurrence of asthma andtime and quantity the patients abused heroin was not obvious; (3) It was the abuse routechaising dragon that caused bronchial asthma; (4) bronchial asthma may be also relatedto allergic individual and impurity of the drug abused.

在临床实践中,烫吸海洛因所致支气管哮喘并非罕见,为了弄清其发病率和临床表现,我们进行了回顾研究。研究对象为1993年6月-1994年8月连续自愿入院戒毒的300名海洛因依赖者。研究结果表明:(1)滥用海洛因所致支气管哮喘的发病率占自愿戒毒者的4%。(2)哮喘同滥用时间长短和剂量关系不大,除五例外,其他均在1a内发生哮喘;(3)只有以呼吸道吸食海洛因的方式即烫吸可导致哮喘发作;(4)哮喘发作的原因可能与过敏体质和毒品中含有致敏物质有关。

Objective To study whether illers exists substance increasing tile sensitivityto endoloxin in burn rat serum and its role ill the endotoxill-induced liver injury. MethodsKupffer cells and hepatocytes were isolated from Wistar rats by a two-step method, andtumor necrosis factor (TNF) and lactate dehydrogenatase (LDH) were detected in the supernatant of Kupffer cells cocultured with hepatocytes. Results The burned rat serum couldsignificantly increase the sensitivity of Kupffer cells to elldotoxin, and increase...

Objective To study whether illers exists substance increasing tile sensitivityto endoloxin in burn rat serum and its role ill the endotoxill-induced liver injury. MethodsKupffer cells and hepatocytes were isolated from Wistar rats by a two-step method, andtumor necrosis factor (TNF) and lactate dehydrogenatase (LDH) were detected in the supernatant of Kupffer cells cocultured with hepatocytes. Results The burned rat serum couldsignificantly increase the sensitivity of Kupffer cells to elldotoxin, and increase the concentration of endotoxin in burn serum group, inducing Kupffer cells to release LDH. And the level of TNF was higher than that of the control group. And the burn serum could enhance LDHlevel after hepatocyte injury induced by Kupffer cells. Such phenomenon was not found in thenormal serum group. Conclusion it suggests that there exists substance increasing thesensitivity to endotoxin ill the burned rat serum, which can significantly increase thesensitivity of Kupffer cells to elldotoxin and induce hepatocyte injury by TNF and othermediators released from KuDffer cells. evefl in very low endotoxin concentration. The lipolysaccharide-binding protein (LBP) afld soluble CD,. are accessed to important roles.

目的探讨烧伤血清中是否存在内毒素致敏物质及其在内毒素诱导肝细胞损伤中的作用。方法以Wistar大鼠为对象,采用二步灌注法分离肝细胞及枯否细胞,分别与内毒素和烧伤血清培养后,观察培养上清中乳酸脱氨酸(LDH)及肿瘤坏死因子(TNF)的变化。结果烧伤血清可显著提高枯否细胞对内毒素的敏感性,诱导枯否细胞释放LDH及TNF明显高于对照组,同时可显著增加枯否细胞与肝细胞联合培养时LDH水平。结论在烧伤血清中存在内毒素数敏物质,可显著提高机体对内毒素的敏感性,在内毒素浓度较低时,仍可诱导枯否细胞释放TNF,从而损伤肝细胞,其中内毒素结合蛋白sCD14可能起重要作用。

 
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