The incidence of heart damage in group 1(52%) was higher than that in group 2(27%)(t=2.48,P<0.05).
The frequency of symptoms in turn was myotonia (99%), muscle weakness (97%), muscle atrophy (85%), cataract(63%), hair losing or bald(57%) and gonadal atrophy (37%), sexuality disfunction (33%), heart damage (11%), intelligence impairment(11%), hypothyroid or disfunction of adrenal gland (8%), mental state disorders (8%).
常见症状依次为肌强直 (99% ) ,肌无力 (97% ) ,肌萎缩 (85 % ) ,白内障(6 3% ) ,脱发或秃顶 (5 7% ) ,性腺萎缩 (37% ) ,性欲障碍 (33% ) , 心脏损害 (11% ) ,智力减退 (11% ) ,甲状腺或肾上腺功能低下 (8% )和精神发育缺陷 (8% )。
Results The rate of heart damage was 39.6 in 103 AL patients, 9.4 when complete remission(CR), 21.6 after six therapy cycles,only 12.8 in treatment group, 31.4 in control group, the difference was significant.
Results:(1)The heart damage occurred within 1 week after SARS onset in mild cases; the rates of LDH,HBDH,CK and CK MB returning to normal were 74.2%,80.6%,100% and 100%,respectively.
结果 :(1) SARS轻型患者 心脏损害多在 1周内出现 ,L DH、HBDH、CK、CK- MB复常率分别为 74 .2 %、80 .6 %、10 0 %和 10 0 % ;
Clinical Analysis of Heart Damage of 219 Cases of Chronic Renal Failure
Rate of cardiac damage is 87.5% in the anti-J0-1 positive PM/DM(P<0.05).
clinical manifestations were different among the cases of cardiac damage,in which 60.0% arrhythmia,57.8% ST-T abnomality,28.9% pericardium were involved.
Increasing of CK-MB in the cases of PM/DM were significant related to having cardiac damage(P<0.01).
Conclusion Cardiac damage is common in paitients with PM/DM.
Methods 97 cases of PM/DM from 2001 to 2005 were retrospectively reviewed. Results Cardiac damage(46.4%)occurred in 45 out of 97 cases of PM/DM.
Cardiac involvement was the main complication (5/12).
Results 89 of 185 cases (48.12%) appeared cardiac involvement,among of them,51 cases (57.30%) companied with electrocardiography change and 34 cases (38.20%) abnormal ultrasonic cardiogram.
结果185例患者中89例出现 心脏损害(48.12%)。 其中心电图改变51例(57.30%),超声心动检查异常34例(38.20%)。
Results There were 47 cases with cardiac involvement in 91 patients and only 3 of them had cardiac symptoms (6.38%).
Clinical Analysis for 185 Cases of Dermatomyositis with Cardiac Involvement
Clinical analysis of cardiac involvement in 100 cases of SLE
Clinical analysis of cardiac injury of acute carbon monoxide poisoning
Clinical Study on Cardiac Injury in Infants of Diabetic Mother
CARDIAC INJURY OF ACUTE CARBON MONOXIDE POISONING ∶APPENDIX CLINICAL ANALYSIS OF 147 PATIENT
Mechanism of cardiac injury in uremia patients
Objective To investigate cardiac injury after biliary obstruction and its mechanism.
目的 探讨胆道梗阻后 心脏损害及损害机制。
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A working hypothesis on pathogenesis of ischemic heart damage has been proposed.
Significantly greater heart damage resulting from CVB1 and EV9 was observed in Se-deficient than in Se-adequate mice, and the Se status had no influence on CVA9-induced myocarditis.
In contrast, heart damage caused by HSV1 was significantly milder in Se-deficient than in Se-adequate mice.
Heart damage also occurred in ATM-treated mice but did not correlate with mortality.
There was a significant difference (P >amp;lt; 0.001, by a Wilcoxon rank sum test) based upon the severity of the ischaemic heart damage in the two groups.
It is concluded that the PAF-induced shock is primarily based on direct cardiac damage.
It emphasizes the importance of the cellular arm of the immune response in cardiac damage, as wall as possible genetic disposition to the disease.
Multiple mechanisms have been proposed for the beneficial effects of the delivered cells; however, true reversal of cardiac damage implies the generation of new contractile myocytes.
It was assumed that local deviations from normal electrolyte levels indicate the degree of cardiac damage.
Severe cardiac damage was introduced by the so-called Ca paradox.
Endocarditis based on valves with a preexisting damage is the most common manifestation of cardiac involvement.
Under this therapy the patient showed prompt remission of the pulmonary and cardiac involvement and could be extubated on day 11 after admission.
Presently, a fortunate course of a patient with fulminant Churg-Strauss syndrome was demonstrated with particularly the rarely observed cardiac involvement, which is recognized as a leading cause of mortality.
Cardiac involvement is well known in a number of skeletomuscular diseases but not in facio-scapulohumeral muscular dystrophy (FSHD).
Cardiac involvement has been documented in a number of primary MDs and is even the dominant feature in some of them.
Noninvasive tests that may support the diagnosis of fulminant or acute myocarditis include cardiac magnetic resonance imaging and biomarkers of cardiac injury.
Blunt Cardiac Injury in Patients with Isolated Sternal Fractures: the Importance of Fracture Grading
Ischemic cardiac injury is a complication of atherosclerosis, which remains a major contributor to morbidity and mortality throughout much of the world.
Cardiac injury may occur following many types of brain injury, although the most widely investigated form of neurocardiogenic injury is subarachnoid hemorrhage (SAH).
Cardiac injury that occurs following an SAH appears to be reversible.