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  waken
     RESULTS Naloxone treated group show better effects no matter in aspects of spur waken or shorten therapy time than control group.
     结果表明 :采用纳洛酮治疗组无论是在催效果 ,还是在缩短治疗时间均优于常规治疗组。
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     Naloxone can waken patients with pulmonary encephalopathy reduce respiratory inhibition and casefatality rate without side-effect.
     研究显示纳络酮可有效解除肺性脑病患者的呼吸抑制、促并明显降低其死亡率 ,且无明显毒副作用。
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  “醒”译为未确定词的双语例句
     STUDIES ON THE METABOLISM OF CUI XING NING:Ⅰ.A SPECTROFLUOROMETRIC DETERMINATION OF CUI XING NING
     催宁的代谢Ⅰ:催宁萤光测定法
短句来源
     STUDIES ON THE METABOLISM OF CUI XING NING:Ⅲ.THE PHARMACOKINETICS OF CUI XING NING
     催宁的代谢Ⅲ:催宁药物动力学观察
短句来源
     THE PHARMACOLOGICAL STUDY OF CUIXINGNING(催醒宁)
     催宁的药理学研究
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     MODEL EXPERIMENT OF ANESTHETIC REVERSAL IN ANIMAL BY USING CUI XING NING (催醒宁)
     关于催宁催作用的动物实验模型
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     Experimental Studies of Antagonist for Anaesthetic Compound Agent 846
     846麻醉合剂的催剂的实验研究
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  相似匹配句对
     A FLUOROMETRIC METHOD FOR THE DETERMINATION OF CUI XING AN
     催安的萤光测定法
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     THE METABOLISM OF CUI XING AN
     催安的代谢
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  waken
Durch Waken oder Pressen kann eine Struktur der Anisotropieklasse IV (Walzstruktur) erzeugt werden.
      
However, Ton has to be long enough for a waken-up node to completely receive one RTS.
      
If children are sleeping when you approach them, use a light touch to waken them gently.
      
Simple tasks like rolling over in bed may waken the person with pain.
      
There fore, the digital section is not waken-up by any unwanted input signal.
      


Menadione sodium bisulfite (Ⅱ),its heat transformation isomer sodium 2-me- thyl-1,4-naphthohydroquinone-3-sulfonate(Ⅲ),as well the oxidized form of the latter sodium 2-methyl-1,4-naphthoquinonate-3-sulfonate(Ⅶ),undergoes desulfonation with the recovery of menaione (Ⅰ) .In acid medium,I is recovered in 80,75,and 25 per cents respectively. Diacetoxy-2-methyl-naphthohydroquinone (Ⅹ) has been pepered from Ⅱ,Ⅲ, and Ⅶ by desulfonation in acetic acid,followed by acetylation or reductive acetyla- tion with acctic anhydride...

Menadione sodium bisulfite (Ⅱ),its heat transformation isomer sodium 2-me- thyl-1,4-naphthohydroquinone-3-sulfonate(Ⅲ),as well the oxidized form of the latter sodium 2-methyl-1,4-naphthoquinonate-3-sulfonate(Ⅶ),undergoes desulfonation with the recovery of menaione (Ⅰ) .In acid medium,I is recovered in 80,75,and 25 per cents respectively. Diacetoxy-2-methyl-naphthohydroquinone (Ⅹ) has been pepered from Ⅱ,Ⅲ, and Ⅶ by desulfonation in acetic acid,followed by acetylation or reductive acetyla- tion with acctic anhydride in yields over 90 per cent. When Ⅲ or Ⅶ is treated with acetic acid,acetic anhydride,fused sodium acetate (and zinc powder in the case of Ⅶ),the aceylation reaction predominates, and,as a result,potassium diacetoxy-2-methyl-1.4-naphthohydroquinone-3-sulfonate (Ⅺ) is obtained instead of X.

亚硫酸氢钠甲萘(Ⅱ),及其热变化生成物2-甲基-1,4-萘氢醌-3-磺酸盐(Ⅲ),和后者的氧化产物2-甲基-1,4-萘醌-3-磺酸盐(Ⅶ),可以进行显著的脱磺酸基作用。在酸性溶液中,甲萘醌(Ⅰ)的回收率分别为80,75及25%。脱磺酸基作用的速度,亦依序遞减。Ⅱ、Ⅲ及Ⅶ,先于醋酸溶剂中脱磺酸基后,再用醋酐等使行乙酰化。可以制得二乙酰萘氢(X),收得率都达90%以上。但Ⅲ及Ⅶ如不先行脱磺酸基,而使与乙酰化或还原乙酰化同时进行,则优先乙酰化,不再脱磺酸基,得到的产物是二乙酰萘氢醌-3-磺酸盐(Ⅺ),而不是X。

Cupric glycerine is a deep blue colored complex salt. It may be used as an analytical reagent for aldehydes and reducing sugars. It possesses certain advantages over Fehling’s reagent in that: its color is more deep, its reaction with aldehydes and reducing sugars exhibits higher sensitivity and is faster, and it is more stable on heating. Cupric glycerine is also much cheaper than Fehling’s reagent. All these properties make it a good substitute for Febling’s reagent in the qualitative analysis of aldehydes...

Cupric glycerine is a deep blue colored complex salt. It may be used as an analytical reagent for aldehydes and reducing sugars. It possesses certain advantages over Fehling’s reagent in that: its color is more deep, its reaction with aldehydes and reducing sugars exhibits higher sensitivity and is faster, and it is more stable on heating. Cupric glycerine is also much cheaper than Fehling’s reagent. All these properties make it a good substitute for Febling’s reagent in the qualitative analysis of aldehydes and both qualitative and quantitative analysis of reducing sugars. Preparation of Cupric glycerine reagent: solution A—34. 64 g. Of CuSO_4·5H_2O dissolved in 500 ml. of distilled water, solution B—60 g. of NaOH and 20 ml. glycerine dissolved in 500 ml. of distilled water. Mix equal volumes of the two solutions before use.

甘油铜是一种深蓝色的络盐,可用为醛类和还原醣的分析试剂,它与Fehling试剂相比较,有其特殊的优点,例如:颜色较深,与还原醣和作用时灵敏度高且反应较快单独加热时也较稳定等。甘油铜的价值比Fehling试剂便宜的多,所以用来作醛类和还原醣的鉴定及还原醣的定量分析是值得推广使用的试剂,甘油的制备方法:甲液——34.64结晶硫酸铜溶于蒸溜水,配成500毫升;乙液——60克氢氧化钠和20毫升甘油溶于蒸溜水,配成500毫升临用前取等容量的二液混合即可。

It is well known that β-diethylaminoethyl diphenylpropylacetate HCl(SKF 525A) is a potent inhibitor of hepatic microsomal drug-metabolizing enzymes. However, our present study indicates that the effect of SKF 525A on these enzyme systems is actually biphasic. Sodium pentobarbital sleeping-time was used as an indirect criterion of the activity of the drug-transforming enzymes in mice and rats. The rate of disappearance of pentobarbital from whole mouse was determined to assess the effect of SKF 525A on the in...

It is well known that β-diethylaminoethyl diphenylpropylacetate HCl(SKF 525A) is a potent inhibitor of hepatic microsomal drug-metabolizing enzymes. However, our present study indicates that the effect of SKF 525A on these enzyme systems is actually biphasic. Sodium pentobarbital sleeping-time was used as an indirect criterion of the activity of the drug-transforming enzymes in mice and rats. The rate of disappearance of pentobarbital from whole mouse was determined to assess the effect of SKF 525A on the in vivo metabolism of the hypnotic. The amount of pentobarbital metabolized after incubating with rat liver slices was also determined. Goldbaum's method was used for the assay. It was found that either prolongation or shortening of pentobarbital sleeping-time could occur depending upon the length of the time interval between the administrations of SKF 525A(40-80 mg/kg, ip.) and pentobarbital(60 mg/kg, ip.). When pentobarbital was administered to mice within 12 hours after SKF 525A, prolongation of sleeping time was observed. On the other hand, if the mice were treated with SKF 525A 48 hours beforehand, a shortening of sleeping time was noted. Similar results were obtained for both rats and mice receiving daily injections of SKF 525A(10-40 mg/kg/day) for 3-10 days. Further studies showed that the decrease in pentobarbital sleeping time was associated with an increase in the rate of metabolism of the hypnotic. In mice receiving SKF 525A 48 hours prior to injection of pentobarbital, the whole body concentrations of the hypnotic were found to be significantly lower than those of control mice. Furthermore, liver slices from rats treated with a dose of SKF 525A 48 hours previously metabolized more pentobarbital than slices from control animals. However, the minimal body pentobarbital levels required to maintain the mice asleep seemed to be similar in both the experimental and the control groups. This fact indicates that the change in sleeping time cannot be attributed to changes in sensitivity of the central nervous system to pentobarbital. With diethylbarbital(a drug which is not metabolized in vivo), neither sleeping time nor the rate of disappearance of the drug from whole mouse was changed by SKF 525A pretreatment. The ascorbic acid contents of rat urine and mouse liver were also determined by the method of Roe and Kuther. It was found that pretreatment of animals with SKF 525A 12-24 hours before sacrifice led to elevated excretion and higher hepatic levels of ascorbic acid. This effect of SKF 525A disappeared 48 hours after injection. These results show that the enhancing effect of SKF 525A on ascorbic acid metabolism occurs before the stimulatory effect on liver drug-metabolizing enzymes.

已知二苯基丙基乙酸β-二乙基氨基乙酯(SKF 525A)为肝微粒体药物轉化酶的抑制剂。我們以戊巴此妥鈉睡眠时間为指标,发現SKF 525A的作用是双相的。小鼠注射一剂SKF 525A(40—80毫克/公斤)后0—12小时为莉酶受抑制期,表現为戊巴比妥鈉睡眠时間明显延长;但注射后48小时,小鼠戊巴比妥鈉睡眠时間不仅不延长,反而縮短。SKF 525A的双相效应在雌雄小鼠均能看到。小鼠及大鼠經連續多次注射SKF 525A后48小时,同样也出現第二相效应。下述进一步的实驗表明第二相效应是由于肝脏药物轉化酶活性加強的結果:(1)48小时前接受过一剂SKF 525A的小鼠,戊巴比妥鈉自体內消失的速率此正常动物者明显加快。(2)不論48小时前是否接受过SKF 525A,小鼠戊巴比妥鈉睡眠刚时,体內催眠药含量无显著差別。(3)48小时前曾經注射SKF 525A的大鼠肝切片轉化戊巴比妥鈉的速率比正常动物肝切片者快。(4)48小时前注射SKF525A并不改变二乙基巴比妥鈉(一个在体內不經轉化的莉物)引起的小鼠睡眠时間及其自体內消失的速度。 給小鼠(或大鼠)注射相当剂量的SKF 525A后12—24小时,肝脏(及尿)...

已知二苯基丙基乙酸β-二乙基氨基乙酯(SKF 525A)为肝微粒体药物轉化酶的抑制剂。我們以戊巴此妥鈉睡眠时間为指标,发現SKF 525A的作用是双相的。小鼠注射一剂SKF 525A(40—80毫克/公斤)后0—12小时为莉酶受抑制期,表現为戊巴比妥鈉睡眠时間明显延长;但注射后48小时,小鼠戊巴比妥鈉睡眠时間不仅不延长,反而縮短。SKF 525A的双相效应在雌雄小鼠均能看到。小鼠及大鼠經連續多次注射SKF 525A后48小时,同样也出現第二相效应。下述进一步的实驗表明第二相效应是由于肝脏药物轉化酶活性加強的結果:(1)48小时前接受过一剂SKF 525A的小鼠,戊巴比妥鈉自体內消失的速率此正常动物者明显加快。(2)不論48小时前是否接受过SKF 525A,小鼠戊巴比妥鈉睡眠刚时,体內催眠药含量无显著差別。(3)48小时前曾經注射SKF 525A的大鼠肝切片轉化戊巴比妥鈉的速率比正常动物肝切片者快。(4)48小时前注射SKF525A并不改变二乙基巴比妥鈉(一个在体內不經轉化的莉物)引起的小鼠睡眠时間及其自体內消失的速度。 給小鼠(或大鼠)注射相当剂量的SKF 525A后12—24小时,肝脏(及尿)中的維生素C含量比对照动物者显著增加。但在給药后48小时,此作用即已消失。可見SKF525A对动物体內維生素C合成的促进作用出現在对肝脏莉物轉化酶的刺激作用之前。

 
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