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完全逆转
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     At 15 min after LTP induction,PD98059(100 μmol/L) or SL327(200 μmol/L) reversed LTP completely,and at 30 min after LTP induction,the inhibitory rate of spinal LTP inhibited by PD98059 or SL327 was 62.5% and 75.0%, respectively.
     在LTP诱导后15m in,脊髓局部给予PD98059(100μmol/L)或SL327(200μmol/L)可完全逆转LTP。 在LTP诱导后30 m in,单独给予PD98059或SL327,LTP的抑制率分别为62.5%与75.0%。
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     TTD, at the concentration of 10 μmol·L -1 , completely reversed ADR resistance in MCF 7/adr cells.
     其中10μmol·L-1TTD能完全逆转MCF7/Adr细胞对ADR的抗药性。
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     Moreover, captopril (30 μmol·L -1 ) also reversed the impairment of vasorelaxation induced by LPC (3 μmol·L -1 ).
     3 0 μmol·L-1卡托普利也可完全逆转LPC( 3 μmol·L-1)对内皮依赖性血管舒张反应的损害。
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     α-Helical CRF9-41, of 10-7 and 10-6 mol/L could remarkably stimulate hCG release (P<0. 001) and α-Helical CRF9-41 of 2 × 10-7 and 2 × 10-6 mol/L could completely reverse the inhibitory effect of CRH on hCG release.
     2×10-7mol/L和2×10-6mol/L的α-HelicalCRF9-41可完全逆转CRH对hCG释放的抑制作用;
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     Results:LY980503at4.0μmol L -1 mostly reversed resistance against ADM in mul tidrug resistant MCF/ADM and K562/ADM cells,and10.0μmol L -1 LY980503could com-plete ly re verse the resistance against ADM in the two resistant cell lines.
     结果:细胞毒实验显示,4μmol·L-1LY980503能大部分逆转多药耐药细胞株MCF/ADM和K562/ADM对阿霉素的耐药性,10μmol·L-1LY980503能完全逆转MCF/ADM和K562/ADM细胞对阿霉素的耐药性;
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     fmk, a selective inhibito r of caspase 3, only induced partial reversion of the apoptotic effects.
     fmk可完全逆转此作用。
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     These changes were abolished in 2K1C rats treated with captopril.
     卡托普利可完全逆转 2K1C组大鼠的上述改变。
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     Completely Normal Spaces
     完全正规空间
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     The Household Registering System Reform Reverse
     户籍改革逆转
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     Complete Infinite Modules
     完全无限模
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  full reversal
The percentages of reversed dementia varied widely: from 0 to 23% for partial and from 0 to 10% for full reversal.
      
The percentage of both partial and full reversal of dementia has fallen in recent years, to less than 1% for both in the four most recent studies.
      
The antagonists were equally effective, leading to full reversal of all effects with a potency ratio of approximately 2.5 mg Ro 15-1788 for 1 mg Ro 15-3505.
      
At eight weeks, functional graft effects were not evident but after 11 weeks small effects on motor asymmetry could be monitored and rats tested 19-21 weeks after grafting exhibited full reversal of the lesion-induced rotational behaviour.
      
However, exogenous supply of a mixture of phenylalanine, tyrosine and tryptophan failed to achieve full reversal of cell growth inhibition, yet the occurrence of other target(s) cannot be ruled out.
      
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The effect of thioprolinc (TP) on the proliferation of bone marrow cells of L6565 leukemic mice towards granulocytic and monocytic cells in diffusion chamber cultures (DCs) was observed. It was found that the proliferation levels of L6565 mice's bone marrow ceils in DCs were low, After treatment with TP the proliferation levels were considerably elevated to nearly normal levels. The proliferation cells were mainly granulocytic and monocytic series. TP had no promoting effect on the proliferation of normal bone...

The effect of thioprolinc (TP) on the proliferation of bone marrow cells of L6565 leukemic mice towards granulocytic and monocytic cells in diffusion chamber cultures (DCs) was observed. It was found that the proliferation levels of L6565 mice's bone marrow ceils in DCs were low, After treatment with TP the proliferation levels were considerably elevated to nearly normal levels. The proliferation cells were mainly granulocytic and monocytic series. TP had no promoting effect on the proliferation of normal bone marrow cells in DCs, and did not increase the peripheral leucocytes in normal mice. The authors consider that although TP could not completely reverse L6565 leukemic cells to normal ones, yet it could change the inhibitory effect of the leukemic cells on the normal CFU-c.

本文观察了硫杂脯氨酸(TP)对L6565小鼠骨髓细胞在体内扩散盒培养中向粒一单系细胞增殖的影响。实验结果发现,L6565小鼠骨髓细胞在扩散盒培养中增殖水平低,经TP治疗后其增殖水平明显提高,接近于正常小鼠,增殖的细胞主要为粒一单系细胞。TP对正常小鼠骨髓细胞在扩散盒中的增殖无促进作用,外周白细胞数亦不增加。我们认为TP虽不能使L6565白血病细胞完全逆转为正常细胞,但可能使其对正常CFu-c的抑制作用发生改变。

The relationship of cellulase to detached leaf senescence of rice seedlings was investigated by examining the effect of ABA and 6-BA on changes in the level of cellulase of leaf segm- ents during senescence.It was shown that the rise in cellulase activity increased with declin- ing chlorophyll content,which was used as the senescence indicator during the senescence of detached rice leaves caused by ABA.The action of ABA took place only after a 48h lag period. ABA enhanced the cellulase secretion and increased...

The relationship of cellulase to detached leaf senescence of rice seedlings was investigated by examining the effect of ABA and 6-BA on changes in the level of cellulase of leaf segm- ents during senescence.It was shown that the rise in cellulase activity increased with declin- ing chlorophyll content,which was used as the senescence indicator during the senescence of detached rice leaves caused by ABA.The action of ABA took place only after a 48h lag period. ABA enhanced the cellulase secretion and increased the permeability of plasma membrane. A high level of cellulase activity in cell wall closely related to membrane permeability changes. The action of cellulase in the cell wall may cause depolymerization of β-1,4-glucan in situ,thus speeding senescence.The 6-BA reverses completely or partly the increase in cellulase activity and the permeability caused by ABA duriug the first two day,but it antagonized hardly any of the ABA effect from the third day on,suggesting the onset of an irreversible stage in the senescence of detached rice leaves.

在脱落酸(ABA)促进离体水稻叶片衰老过程中,叶绿素含量日渐下降,纤维素酶活力日益升高。ABA 作用的滞后期为48小时。衰老叶片的胞壁附有大量的纤维素酶。酶从胞内分泌到胞壁,是与质膜结构受到损害,使透性增大有关。6-苄氨基嘌呤(6-BA)与 ABA 的相互作用对纤维素酶活力的影响表明,ABA 引起的水稻叶片衰老可分为两个阶段:在0到1天,6-BA近乎完全逆转 ABA 的效应,第二天有所减弱,这是可逆期;从第三天起,6-BA 对 ABA 几无拮抗作用,为不可逆期开始。

In this study,we observed that PGE2 3×10-7~3×10-6 mol/L & nifedipine 1 ×10-5mol/L inhibited lymphocyte proliferation markedly.The effect of nifedipine was completely reversible, however the effects of PGE2(3 ×10-6mol/L)was only partly reversible .In combination of PGE2(3×10-8mol/L) with nifedipine (1 ×10-6mol/L) ,the effect was additive,no significant antagonism and synergism were observed .PGE26× 10-6mol/L and nifedipine 1×10-3 mol/L directly inhibited maerbphage migration but no significant effect on PHA-induced...

In this study,we observed that PGE2 3×10-7~3×10-6 mol/L & nifedipine 1 ×10-5mol/L inhibited lymphocyte proliferation markedly.The effect of nifedipine was completely reversible, however the effects of PGE2(3 ×10-6mol/L)was only partly reversible .In combination of PGE2(3×10-8mol/L) with nifedipine (1 ×10-6mol/L) ,the effect was additive,no significant antagonism and synergism were observed .PGE26× 10-6mol/L and nifedipine 1×10-3 mol/L directly inhibited maerbphage migration but no significant effect on PHA-induced MIF .Preincuba-tation with nefidipine did not influonce macrophage phagocytosis, but PGE2 slightly enhanced it. The results stated above strongly suggests that PGE2 may regulate cells interaction and Ca2+ is required during lymphocyte proliferation.

PGE_23×10~(-7)~3×10~(-6)mol/L及硝苯吡啶5×10~(-6)~1×10~(-5)mol/L对ConA诱导的淋巴细胞增殖有明显抑制作用。药物作用48h后洗去药物,加入致裂原后继续培养细胞,结果发现硝苯吡啶的抑制作用已完全逆转,而PGE_23×10~(-6)mol/L的抑制作用则部分逆转。当低浓度PGE_2(3×10~(-3)mol/L)与硝苯吡啶(1×10~(-6)mol/L)同时作用于脾细胞时,可见两者作用正巧相加,无明显拮抗与协同作用。PGE_26×10~(-6)mol/L及硝苯吡啶1×10~(-5)mol/L对巨噬细胞移动有直接抑制作用。用~(51)Cr标记调理的绵羊红细胞方法证明低浓度PGE_2对巨噬细胞吞噬功能有增强,而硝苯吡啶预培养对吞噬功能影响不大。

 
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