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粗糙纤维
相关语句
  相似匹配句对
     Special Style Fibres
     特殊风格纤维
短句来源
     rough capsuls on tumor;
     包膜粗糙
短句来源
     fibre trachieds;
     纤维管胞;
短句来源
     Rough Subalgebra
     粗糙子代数
短句来源
     The results show that the fibre surface treatment make the surface coarser;
     结果表明,纤维表面处理使碳纤维的表面更粗糙;
短句来源
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Objective To explore the regularity of variation of the injured cartilage after intra-articular fracture. Methods Intra-articular fracture was induced by an impact to the rabbit knee joint. Autoradiography, light and scanning electron microscopy were applied to study the changes of morphology and synthesizing function of the injured cartilage in different intervals from 1 week to 6 months after intra-articular fracture. Results The number of chondrocytes in the injured cartilage reduced dramatically and...

Objective To explore the regularity of variation of the injured cartilage after intra-articular fracture. Methods Intra-articular fracture was induced by an impact to the rabbit knee joint. Autoradiography, light and scanning electron microscopy were applied to study the changes of morphology and synthesizing function of the injured cartilage in different intervals from 1 week to 6 months after intra-articular fracture. Results The number of chondrocytes in the injured cartilage reduced dramatically and the residual chondrocytes failed to incorporate tritiated proline 2 weeks after the fracture. One month later, assembling chondrocytes were present and a part of chondrocytes could incorporate proline. Form 3 to 6 months, there were few residual chondrocytes in the injured cartilage. Assembling chondrocytes were strikingly apparent, and some chondrocytes demonstrated inability to uptake proline. The cartilage cracks weren't repaired. The cartilaginous surface became coarse and fibrillated, and began to flake off. There were double tidemarks above the calcified cartilage. Conclusion The injury to the cartilage induced by intra-articular fracture is probably irreversible, and deterioration is progressive. This may be one of the causes of inducing traumatic osteoarthritis.

目的探讨关节软骨损伤后的变化规律。 方法利用冲击伤造成兔膝关节内骨折 ,在术后 1周至6月的不同时间处死动物 ,利用放射自显影、光镜及扫描电镜等观察软骨形态及细胞合成功能的改变。 结果损伤后 2周 ,软骨细胞明显减少 ,残存细胞不能掺入脯氨酸。 1月后 ,软骨细胞开始集群性聚居 ,部分细胞恢复掺入脯氨酸。 3~ 6月后 ,残存的软骨细胞较少 ,细胞集群性聚居更明显 ,部分细胞不能掺入脯氨酸 ;损伤软骨裂隙未修复 ,表面粗糙纤维化并开始脱落 ,出现双潮标。 结论关节内骨折后 ,软骨损伤难以修复并呈进行性发展 ,可能是创伤后骨关节炎发生的原因之一。

Oral submucous fibrosis(OSF) is a chronic oral mucous membrane disease characterized with progressive limitation of the opening of mouth caused by inflammation and mucous membrane disorder.Data from recent epidemiological studies provide overwhelming evidence that areca nut is the main aetiological factor for OSF.A clear dose-dependent relationship was observed for both frequency and duration of chewing areca nut in the development of OSF.Areca chewing may exert a strong mechanical stimulation on the oral mucous...

Oral submucous fibrosis(OSF) is a chronic oral mucous membrane disease characterized with progressive limitation of the opening of mouth caused by inflammation and mucous membrane disorder.Data from recent epidemiological studies provide overwhelming evidence that areca nut is the main aetiological factor for OSF.A clear dose-dependent relationship was observed for both frequency and duration of chewing areca nut in the development of OSF.Areca chewing may exert a strong mechanical stimulation on the oral mucous membrane as the robust fibre in the nut,the areca-specified alkaloids and tannin which may have a synergistic role,seem to interfere with the molecular processes of deposition and/or degradation of extracellular matrix molecules such as collagen;the copper content of areca nut is high and may play a role as a mediator in fibrosis.Furthermore,it has been postulated that areca nut may also induce the development of the disease by increased levels of cytokines in the lamina propria.Therefore,this article aims at analyzing the recent developments that may lead to our understanding of the aetiology and pathogenesis of OSF with special reference to areca nut as the major aetiological factor and making it known to the public what a role areca nut may have played in human OSF.

口腔黏膜下纤维性变(O ra l Subm ucous F ibrosis,O SF)是一种以炎症和渐进性黏膜改变导致进行性张口困难为特征的口腔黏膜慢性病症.近年来相当多的流行病学研究资料指出槟榔是O SF的主要病源因子,在O SF的发病上,不论是嚼食槟榔的频率还是持续的时间都呈现了剂量依赖性.在嚼食槟榔时粗糙纤维对口腔黏膜产生的强烈机械刺激作用、槟榔中的生物碱与单宁对细胞外基质分子的影响以及槟榔中的铜在纤维化疾病中的作用等方面对槟榔在O SF发病原因与发病机制中的作用加以综述,旨在分析迄今为止国内外学者对槟榔在O SF上所做的研究,以期让基础科研人员、公共卫生工作者以及广大槟榔嗜好者对槟榔在人类O SF中的作用有更深入的了解.

 
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