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   细胞毒效应 在 中药学 分类中 的翻译结果: 查询用时:0.01秒
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细胞毒效应
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  cellulotoxic effect
    Results: The LP (0. 1 and 0.5 mg/ml) can increase the lymphocyte proliferating rate, cellulotoxic effect of niacrophage, and the level of NO in normal mice ( P <0. 01 ) and can increase the immunological function of immunological suppression mice (0.5 mg/ml, P < 0. 01).
    结果:LP 0.1,0.5 mg/ml组明显提高淋巴细胞增殖率,增强巨噬细胞细胞毒效应,提高巨噬细胞和脾细胞分泌NO的水平(P<0.01),0.5 mg/ml明显提高免疫抑制小鼠的免疫功能(P<0.01)。
短句来源
    Methods: Different doses of LP were givent to stomach of normal or immunological suppression mice for 7 days and the effects of LP on lymphocyte proliferating, cellulotoxic effect of macrophagc, and level of NO were determined.
    方法:LP不同剂量给正常或免疫抑制小鼠灌胃7 d,观察LP对小鼠淋巴细胞增殖反应,巨噬细胞细胞毒效应以及巨噬细胞和脾细胞产生NO的影响。
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  “细胞毒效应”译为未确定词的双语例句
    2 Method To test the cytotoxic effects of vincristine(VCR) and the arteannuin (dihydroartemisinin,artesμnate and extracts of abrotani herba)to KBV200. and calculate the corresponding IC50 throught MTT method ;
    2方法采用MTT法分别测定长春新碱(VCR)、青蒿素、二氢青蒿素、青蒿琥酯及青蒿提取物(乙醇粗提物)对KBV200细胞毒作用,计算相应的IC50,并以上述药物对KBV200的抑制率<30﹪的实验用药浓度与VCR配伍使用时对VCR细胞毒效应(IC50),依据公式计算实验用药的增敏指数。
短句来源
    The Experiment Studies for the Effect of Fufan Polygonum Multiflorum(PMC)on Lymphocytes Proliferation, IL-2 production and Macrophages Cytotoxicity
    复方首乌水提液诱导淋巴细胞增生、白细胞介素2生成及巨噬细胞细胞毒效应的实验研究
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    In recent years, many research show that there is a kind of new material , nitric oxide ( NO ) , which is a kind of small inorganic molecule.
    近年来的研究表明有一种新的M细胞毒效应分子,即一氧化氮(nitric oxide NO),是一种无机小分子,它在活化的M杀伤肿瘤细胞的效应中起着重要的作用,是一种新发现的抗肿瘤分子。
短句来源
    4. Cell viability analysis showed that AGE inhibited the proliferation of tumor cells without affecting their viability. Treatment with AGE, the cell cycle was arrested at the G0/G1stage without inducing apoptosis or necrosis of tumor cells.
    在五加皮蛋白作用下,使肿瘤细胞停止在细胞周期的G0/G1期,未显示直接的细胞毒效应
短句来源
    In the present study,vascular dementia was induced by a permanent bilateral ligation of common carotid arteries (2V0). After operation,it caused cell death by releasing free radicals,NO, inflammatory and cellular factors.
    本实验采用的是双侧颈总动脉永久性结扎(2VO)的血管性痴呆模型,造模后,通过释放氧自由基、NO、炎症细胞因子而行使细胞毒效应,引起细胞死亡,所以iNOS和自由基的过量产生可能是引起痴呆的重要机制。
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After injecting the mice with staphylococcal protein A(SpA) or ApS containing staphylococci via different routs, i. e, subcutaneous or intraperitoneal, dynamic changes of ultrastructure and cytotoxic effect of maeorophages in the intraperitoneal cavity were observed by the electron microscope and the release test of ~(125)Ⅰ-udR labeled tumor target Cell DNA. It was fount that, 5-14 days after injecting the mice with SpA containing staphylococci intraperitoneally, volume of the murine peritoneal cavity macrophages...

After injecting the mice with staphylococcal protein A(SpA) or ApS containing staphylococci via different routs, i. e, subcutaneous or intraperitoneal, dynamic changes of ultrastructure and cytotoxic effect of maeorophages in the intraperitoneal cavity were observed by the electron microscope and the release test of ~(125)Ⅰ-udR labeled tumor target Cell DNA. It was fount that, 5-14 days after injecting the mice with SpA containing staphylococci intraperitoneally, volume of the murine peritoneal cavity macrophages was augmented remarkably, being 2-3 times as large as that of the control group. The surface folds of macrophages were significantly increased in comparison with control group. SpA containing staphylococci-activated macrophages were very irregular, with many pseudopodiums and finger-like process, starfishlike in shape. There was marked enhancement in cell organs-especilly, rough endoplasmic retieulars and lysosomes and there was abundant phagosomes in the cytoplasm. Furthermoren, the results showed that, 5 days after injecting SpA or SpA containing staphylococci into the mice intraperitoneally, cytotoxic effect of the mae rophages in the peritoneal cavity was increased more significantly than that of the control group(cytotoxicity 32.0±13.8%), reaching 42.0±7.8% and 46.3±11.7%. The cytotoxic effect reached peak levels(50.4±17.3% and 56.9±13.4% respectively)on 7 the day. Based on the experimental results, possible mechanism in the cytotoxic effect of macrophage increased by SpA was diseeused.

通过电镜和~(125)Ⅰ—udR标记肿瘤靶细胞DNA释放试验,对腹腔或皮下注射SpA或SpA菌体后小鼠腹腔巨噬细胞的超微结构、及其对靶细胞的细胞毒效应进行了动态观察。结果首次证明,小鼠腹腔注射SpA菌体后第5、14d,其腹腔巨噬细胞体积明显增大,可达对照组的2—3倍;皱褶明显增多,有众多的伪足和指状突起,外形极不规则,形似海星;胞质内细胞器明显增多,尤以粗面内质网和溶酶体为显著,胞质内充满吞噬体。实验进一步证明,小鼠腹腔注射SpA或SpA菌体后第5d,其腹腔巨噬细胞杀伤效应(细胞毒分别为42.0±7.8%和46.3±11.7%)比对照组(细胞毒为32.0±13.8%)明显增强,至第7d,细胞毒效应达峰值水平(分别为50.4±17.3%和56.9±13.4%)。根据实验结果,对SpA增强巨噬细胞杀伤效应的可能机理进行了讨论。

The purpose of this study is to observe how SPA (Staphylococcal protein A) and SAC (Staphylococcus aureus Cowan I) injected directly into tumorbearing mice in vivo have effects on morphological changes of their peritoneal macrophages, on cytotoxic activaties of theri splenic natural killer cells (NK cells), on macrophages against tumor cells and on direct cytotoxicity against tumor cells, through both release test of 125I—udR labeled tumor cell DNA and scanning electron microscopy. Our results demonstrated that...

The purpose of this study is to observe how SPA (Staphylococcal protein A) and SAC (Staphylococcus aureus Cowan I) injected directly into tumorbearing mice in vivo have effects on morphological changes of their peritoneal macrophages, on cytotoxic activaties of theri splenic natural killer cells (NK cells), on macrophages against tumor cells and on direct cytotoxicity against tumor cells, through both release test of 125I—udR labeled tumor cell DNA and scanning electron microscopy. Our results demonstrated that the macrophages were apparently activated after injecting SAC: they became 2—3 times in size as large as those of the control group; The surface folds and processes of these macrophages were remarkably enhanced. It seems to be that the cytotoxic activaties of the macrophages and NK cells augmented significantly by both SPA and SAC were consistent with the strength of their antitumor responses. these data sugest that the antitumor mechanisms of SPA injected directly in vivo may be related to the enhancement of macrophage and NK cell activaties, perhaps without the direct cytotoxicity of SPA against tumor cells.

本研究通过~(125)I-udR标记肿瘤细胞DNA释放试验和扫描电镜,观察了SPA和SAC直接体内注射对带瘤小鼠腹腔巨噬细胞细胞毒及其超微结构和脾脏NK细胞细胞毒的影响;SPA和SAC对瘤细胞的直接毒效应。结果表明,SAC治疗后巨噬细胞明显活化,其体积可达对照组的2—3倍,表面皱褶增多、增厚,有许多粗大的伪足;SPA和SAC均能使巨噬细胞、NK细胞、细胞毒活性显著增强,与抗肿瘤效应的强弱似呈一致关系;二者无直接瘤细胞毒效应。这些结果提示,葡萄球菌A蛋白体内注射的抗肿瘤机制与其增强巨噬细胞和NK细胞活性有关,而与直接瘤细胞毒作用无关。

PMC extracted has been shown to be a stimulus for murine spleen cell tested by cell proliferation and IL-2 production in vitro. The cell proliferation and IL-2 production of mice spleeen cell was also promoted by injection ip PMC 300 mg/kg/day for 5 day. Macrophage cytolytic and cytostatic of mice treated with PMC was higher than those control mice. The results indicated that stimulation effect of PMC may be the basis of its beneficial antagonize effect human elderly.

复方首乌水提液(PMC)300mg/kg/天ip连续5天,可使大鼠脾细胞对ConA、PHA的增殖反应明显增强,IL-2生成显著提高,在体外PMC 1mg/ml与亚剂量0.25μgConA诱导的大鼠脾淋巴细胞增殖反应及IL-2生成也均见明显增强。用药大鼠腹腔Mφ超微结构显示活化,Mφ细胞毒效应亦明显升高。表明PMC可增加T细胞数量,提高T细胞功能,诱导Mφ活化。

 
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