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    INDUCTION OF EXPRESSION OF PROTO-ONCOGENE C FOS AND ORNITHINE DECARBOXYLASE GENES BY TRANSIENT CEREBRAL ISCHEMIA
    暂时性脑缺血诱导c-fos原癌基因和鸟氨酸脱羧酶基因表达
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    Expression of c—fos Protooncogene Protein(Fos)In The Rat Hypothalamus Induced By Transient Global Cerebral Ischemia
    全脑暂时性缺血诱导c—fos癌基因蛋白(FoS)在大鼠下丘脑的表达
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    Experimental Study on the Mechanisms of Transient Increase of Intraocular Pressure after Argon Laser Iridectomy
    氩激光虹膜切除术后暂时性眼压升高机制的实验研究
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    ConclusionThe transient rise of IOP after argon laser iridectomy was correlated with the decrease of the activity of SOD in aqueous.
    结论氩激光虹膜切除术后暂时性眼压升高与术后房水内SOD活性下降有关,术前肌注VitE、VitC对兔眼氩激光虹膜切除术后暂时性眼压升高有防治作用。
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    Intercellular adhesion molecule 1 (ICAM 1) expression was studied with rat transient cerebral ischemic model.
    本文用暂时性脑缺血的大鼠模型对血管内皮细胞间粘附分子1(ICAM1)的表达进行了研究。
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Experiments were performed in 14 anesthetized open-chest dogs to study the

本实验在14只麻醉开胸狗心脏上观察了氟碳乳剂与右旋糖酐稀释血液对心肌耗氧量与供应缺血心肌氧量关系的影响。以左室压力-时间指数(SPTI)作为心肌耗氧量的指标,根据冠脉有效侧支血流量(ECF)、PaO_2和 Hb 浓度计算供应缺血心肌的氧量。实验结果表明,低分子右旋糖酐稀释血液后,SPTI 暂时性轻度增加(稀释后30min 时较对照增加7.1±2.7%,P<0.05,稀释后60min 时增加2.8±1.2%,P>0.05),ECF 明显增多(稀释后30min 时较对照增加58.5±6.1%,P<0.01),缺血区边缘心肌氧供需关系未发生明显变化。氟碳乳剂稀释血液后,SPTI 的变化规律与右旋糖酐稀释后相同(稀释后30min 和60min 时分别较对照增加2.5±0.7%和1.9±0.8%)ECF 和 PaO_2升高(稀释后30min 时分别较对照增加53.9±6.7%和93±8.9%),供应缺血心肌的氧量显著增加,缺血区边缘心肌氧供需矛盾明显改善。

Temporary and permanent effects of altered neonatal nutrition on the growth of cardiac myocyte were studied by calculating every dimension of isolated myceyte. We found that:malneutrition could delay the differentiation and development of cardiac myocyte in the very early stage after birth; but it did not reduce the myocyte number in left ventricle of those 12 weeks rats. Malnutrition slowed myocyte hyperplasia in right ventricle, and eventually decreased the myocyte number in right ventricle of the adult rats....

Temporary and permanent effects of altered neonatal nutrition on the growth of cardiac myocyte were studied by calculating every dimension of isolated myceyte. We found that:malneutrition could delay the differentiation and development of cardiac myocyte in the very early stage after birth; but it did not reduce the myocyte number in left ventricle of those 12 weeks rats. Malnutrition slowed myocyte hyperplasia in right ventricle, and eventually decreased the myocyte number in right ventricle of the adult rats.

通过测量游离心肌细胞的各项数值,研究断奶前期不同营养条件对心肌细胞生长发育的暂时性和持久性影响。发现:低营养延缓左心空心肌细胞的发育,阻碍右心室心肌细胞的分化和减少成熟期右心室心肌细胞的数量。高营养能限制成熟期心肌细胞体积的过度肥大,并促进左心室心肌细胞厚度的适当增加。

The present work indicated that during duodenal ulceration induced by cysteamine(400 mg/kg×2, po), the pH of proximal duodenum decreased markedly; the content of binding mucus decreased; while DNA synthetic rate of duodenal tissue did not reduce but increased transiently. These results suggest that the duodenal acidification and decrease in mucus-HCO3- barrier function may contribute to the pathogenesis of ulceration. Since the gastric total acid output observed on Shay rats treated with cysteamine remained...

The present work indicated that during duodenal ulceration induced by cysteamine(400 mg/kg×2, po), the pH of proximal duodenum decreased markedly; the content of binding mucus decreased; while DNA synthetic rate of duodenal tissue did not reduce but increased transiently. These results suggest that the duodenal acidification and decrease in mucus-HCO3- barrier function may contribute to the pathogenesis of ulceration. Since the gastric total acid output observed on Shay rats treated with cysteamine remained unchanged or reduced, the acidification in duodenum seems not to be a result of increase in acid load to the duodenum, but may be that of reduction in acid disposal ability of the duodenum.

本工作观察到在半胱胺(400mg/kg×2,po)诱发大鼠十二指肠溃疡过程中,十二指肠近端pH明显降低,十二指肠壁结合粘液量减少,而十二指肠纽织DNA合成速率出现暂时性升高。这些结果提示:溃疡的生成可能与十二指肠酸化以及粘液-HCO_3~-盐屏障作用的降低有关,而与十二指肠组织修复能力的大小似无明显关系。但致溃疡剂量的半胱胺(250mg/kg,sc)并不增加shay大鼠的胃总酸排出量。因此,半胱胺导致十二指肠酸化的主要原因似不是由于其酸负荷的增加,而可能由于十二指肠对酸处置能力的降低。

 
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