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暂时性
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  “暂时性”译为未确定词的双语例句
    THE CHARACTERISTICS OF C-FOS PROTOONCOGENE PROTEIN (FOS) EXPRESSION IN RAT HIPPOCAMPUS INDUCED BY TRANSIENT GLOBAL CEREBRAL ISCHEMIA
    全脑暂时性缺血诱导原癌基因蛋白(Fos)在大鼠海马结构表达的特征
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    IN SITU DETECTION OF APOPTOSIS IN RAT HIPPOCAMPUS AFTER TRANSIENT GLOBAL ISCHEMIA
    大鼠暂时性全脑缺血再灌后海马区凋亡检测
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    7 nitro indazole decreases cerebral infarction area during transient focal cerebral ischemia in Rats
    7-nitro-indazole 减小大鼠暂时性局灶性脑梗塞灶范围
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    Transplantation of gene-transfected neural stem cells for transient cerebral ischemia in rats
    转基因神经干细胞移植治疗暂时性脑缺血的实验研究
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    The tMCAO models were randomly divided into control group, PBS transplantation group, neural stem cells transplantation group and VEGF-secreting neural stem cells transplantation group.
    建立大鼠暂时性大脑中动脉阻塞模型(tMCAO),按神经损害严重程度评分(NSS)对缺血后神经功能进行评定,并随机分成对照组、细胞悬液(PBS)移植组、神经干细胞移植组和转基因神经干细胞移植组。
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1) We have measured the CSF contents of CAMP of 27 normal Chinese adults and established a normal value of 12.66±1.0 pmole/ml.2) We have measured the CSF contents of CAMP of 154 patients with various diseases of the nervous system and found a significant increase in carotid occlusion (within 14 days), intracerebral hemorrhage, subarachnoid hemorrhage, active epilepsy, tuberculous meningitis, cysticercosis cerebralis and compressions on the spinal cord as compared to normal value. Among them the increase in tuberculous...

1) We have measured the CSF contents of CAMP of 27 normal Chinese adults and established a normal value of 12.66±1.0 pmole/ml.2) We have measured the CSF contents of CAMP of 154 patients with various diseases of the nervous system and found a significant increase in carotid occlusion (within 14 days), intracerebral hemorrhage, subarachnoid hemorrhage, active epilepsy, tuberculous meningitis, cysticercosis cerebralis and compressions on the spinal cord as compared to normal value. Among them the increase in tuberculous meningitis is the most striking one. As to vertebral occlusion (within 14 days), TIAs static epilepsy, encephalitis, cysticercosis cellulosae cutis, degenerative and functional diseases the values are within normal range.3) Some possible mechanisms responsible for the increase of CAMP content in CSF in some pathological conditions have been briefly discussed.

(一)测定了27例正常中国人脑脊液中CAMP的浓度,正常值为12.66±1.00微微克分子/毫升。 (二)测定了154例各类神经系统疾病脑脊液中CAMP的浓度,发现颈内动脉梗塞后14天内,脑内出血、蛛网膜下腔出血、癫痫发作期、结核性脑膜炎、脑囊虫、脊髓压迫症患者脑脊液中CAMP的浓度与正常人相比有明显升高,其中结核性脑膜炎尤为显著。至于椎动脉梗塞后14天内、暂时性脑缺血样发作、癫痫间歇期、脑炎、皮下囊虫、变性疾病、功能性疾病则属正常。 (三)对某些病理情况下脑脊液CAMP升高的可能的机制进行了简要的讨论。

This report deals with the changes of the cerebral blood flow when mock cere-brospinal fluid was injected into the cisterna magna in different ways to raisethe intracranial pressure.The result shows:1.The reduction of the cerebral blood flow did not have significant statisticalchanges until the intracranial pressure was raised by steps to an average of over50 mmHg.Afterwards,the higher the intracranial pressure was raised,the moreobvious the reduction of the cerebral blood flow was.2.Compared with reference...

This report deals with the changes of the cerebral blood flow when mock cere-brospinal fluid was injected into the cisterna magna in different ways to raisethe intracranial pressure.The result shows:1.The reduction of the cerebral blood flow did not have significant statisticalchanges until the intracranial pressure was raised by steps to an average of over50 mmHg.Afterwards,the higher the intracranial pressure was raised,the moreobvious the reduction of the cerebral blood flow was.2.Compared with reference data,the reduction of the cerebral blood flowhad significant statistical changes when the intracranial pressure was raised to reducealthe cerebral perfusion pressure to less than 60 mmHg.Afterwards,the reductionof the cerebral perfusion pressure was related to the reduction of the cerebral bloodflow.It suggests that the autoregulation of the cerebral circulation would belost when the intracranial pressure reduced the cerebral perfusion pressure toa point below 60mm Hg.3.The abrupt rise of the intracranial pressure caused the obvious rise in thearterial pressure,which could cause a temporary rise of the cerebral perfusionpressure and a temporary increase in the cerebral blood flow at the initial stage.Then the cerebral blood flow was reduced commensurably to the reduction ofthe cerebral perfusion pressure.4.After the raised intracranial pressure was reduced,there could be tem-porary increase of the cerebral blood flow.This phenomenon is often seen in theanimal whose intracranial pressure can cause obvious reduction of the cerebralblood flow.

本文报告了用不同方式向小脑延髓池注入人工脑脊液升高颅内压时脑血流量的变化,结果表明:(1)阶梯性升高颅内压至其均值为50 mmHg 以上时,脑血流量的减少才具有统计学意义的差别,此后颅内压愈高,脑血流量的减少愈明显。(2)颅内压升高使脑灌注压降低至60mmHg 以下时,脑血流量的下降与对照相比,开始有统计学差异,此后脑灌注压的下降与脑血流量的减少有密切关系。这说明颅内高压使脑灌注压降到60mmHg 以下时,脑循环的主动调节可能丧失。(3)急骤升高颅内压引起血压明显增高时,初期可以发生血压和脑灌注压的暂时性升高及脑血流量的暂时增加,然后脑血流量随着血压和脑灌注压的降低而逐渐减少。(4)颅内压升高后降低,可出现暂时的脑血流量增加,这种现象常见于颅內高压引起脑血流量明显下降的动物。

A clinical analysis of 47 cases of pulseless disease is reported. Of these cases 26 (55.3%) revealed neurologic symptoms and signs, including recurrent attacks of syncope and vertigo, transient loss of vision, monocular or binocular, hemiparesis, sensory impairment and various disturbances of the cranial nerves. The local neurological deficits were due to cerebral ischemia.Hemodynamic change especially the subclavian steal was the chief factor regional ischemia of the brain. Platelet hyperaggregation was detected...

A clinical analysis of 47 cases of pulseless disease is reported. Of these cases 26 (55.3%) revealed neurologic symptoms and signs, including recurrent attacks of syncope and vertigo, transient loss of vision, monocular or binocular, hemiparesis, sensory impairment and various disturbances of the cranial nerves. The local neurological deficits were due to cerebral ischemia.Hemodynamic change especially the subclavian steal was the chief factor regional ischemia of the brain. Platelet hyperaggregation was detected under an electronic microscope in two cases. This suggested that the disorder of blood coagulation might be an important cause of the disease. Etiology and treatment of the illness arc also discussed in this article.

47例无脉症(主动脉弓综合征)的临床分析。有神经系统体征者26例(55.3%),主要为发作性晕厥和眩晕、一或两眼的暂时性视力丧失、偏瘫、感觉障碍及颅神经损害等。限局性神经系统体征系由脑缺血引起。血液动力学障碍尤其是“锁骨下偷漏”是导致脑缺血的主要因素。两例以电镜测出血小板高聚集性,提示凝血机制障碍也是本病的重要原因。对本病病因及治疗进行了讨论。

 
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