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   炎症介质 的翻译结果: 查询用时:1.308秒
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炎症介质     
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  inflammatory mediators
     Effect of treatment with Xuebijing injection (血必净注射液) on serum inflammatory mediators and Th1/2 of spleen in rats with sepsis
     血必净注射液对脓毒症大鼠血清炎症介质及Th1/2的影响
短句来源
     Methods: Inflammatory mediators including IL-1β, IL-6, IL-8, TNF-α,and LTB_4 were measured,which were released from polymorphonuclear (PMN) cells collected from normal subjects and pretreated by homoeriodictyol-7-O-β-D-glycoside with PAF stimulating.
     方法:利用HG预处理人体主要的炎症细胞多形核白细胞(PMN),测定PAF刺激下PMN产生的炎症介质IL-1β、IL-6、IL-8、TNF-α和白三烯B4(LTB4)。
短句来源
     Production of several inflammatory mediators including IL 1β,IL 6,IL 8,TNF α,LTB 4 and superoxide free radicals were measured.
     结果 :PAF可刺激PMN产生炎症介质IL 8、TNF α、LTB4 及超氧自由基 ;
短句来源
     This study has been designed to compare the contralateral lung injury by injecting HC1 and Pepsin to single lung of rabbits, and the patho-genesis was investigated by examining the changes in inflammatory mediators TXB2, 6K - PGF_(1a), in order to provide theoretical evidence for its treatment.
     本研究通过给兔单侧肺注入盐酸与胃蛋白酶,比较对侧肺损伤的轻重程度,以评价盐酸与胃蛋白酶在误吸中的作用,同时通过炎症介质血栓素B_2(TXB_2),6-酮-前列腺素F_(1a)(6-K-PGF_(1a))变化,初步探讨继发肺损伤的发病机理,为今后治疗误吸提供理论依据。
短句来源
     Among them, high volume hemofiltration (HVHF)is more valid in renal replacement and inflammatory mediators removement.
     其中高容量血液滤过(high volume hemofiltration,HVHF)对替代肾脏功能,稳定内环境,清除炎症介质和中分子物质更为有效。
短句来源
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  inflammatory mediator
     The result showed that the moxibustion alleviated the inflammatory reaction and significantly reduced the level of the chemical inflammatory mediator(Hm, 5-HT) and the inflammatory cytokine (TNF, IL-1β) (P < 0. 05 or P < 0. 01. comparing with control group).
     对炎症介质的观察表明,灸治能降低炎症化学介质(Hm、5-TH)和炎性细胞因子(TNF、 IL-1β)的含量,与炎症对照组比较,差异显著(P< 0.05或 P<0.01)。
短句来源
     RESULTS: The powder solution could inhibit the increased permeability induced by inflammatory mediator histamine with the alteration of Pa from 9 32±1.56 to 4 38±0 65 ( P <0 05).
     结果:微元素粉末对炎症介质组胺介导的高通透性有明显的抑制作用,通透系数(10-6cm/s,下同)从932±156下降到438±06。
短句来源
     Study of Platelet Aggregation Induced by Inflammatory Mediator (PAF) in Allergic Rhinitis
     炎症介质(PAF)致变应性鼻炎血小板聚集的研究
短句来源
     Study of Platelet Aggregation Induced byInflammatory Mediator (PAF) in Allergic Rhinitis
     炎症介质(PAF)致变应性鼻炎血小板聚集的研究
短句来源
     Objective: To investigate the clinical therapeutic effect of Qingfeihuatan Quyu prescription(清肺化痰祛瘀方,苇茎汤加碱) on treatment of chronic obstructive pulmonary disease(COPD) in acute(deteriorated) stage and its intervention effect on inflammatory mediator in plasma.
     目的:观察清肺化痰祛瘀方(苇茎汤加减)治疗慢性阻塞性肺疾病(COPD)急性加重期患者的临床疗效以及对血浆炎症介质的影响。
短句来源
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  inflammatory medium
     Nuclear factor - κ B is an important transcription factor and the key step of inflammatory medium.
     核因子κB(nuclear factor-κB NF-κB)是一种重要的转录因子,参与许多基因的表达调控,是炎症介质的中心环节。
短句来源
     CONCLUSION: Homocysteine might play an important role in the occurrence of ACS by mediating expression of CD11b/CD18 on leukocyte as an inflammatory medium.
     结论:同型半胱氨酸作为炎症介质可能通过调节白细胞黏附分子CD11b/CD18的表达,在ACS发生和发展中起重要作用。
短句来源
     This study was designed to observe the association of the polymorphisms of inflammatory medium interleukin 6 gene promoter region in circulation with interleukin 6,high sensitive C reactive protein and fibrinogen in circulation among the healthy population in Beijing city,and assess the race difference of the polymorphisms of interleukin 6 gene.
     观察北京市区健康人循环中炎症介质白细胞介素6启动子区基因多态性与循环中白细胞介素6、超敏C反应蛋白和纤维蛋白原的关系,并评价白细胞介素6基因多态性的种族差异。
短句来源
     Results The PYKS obviously raised the expression of Caspase-3 and MMP-2, but decreased the expression of NF-κB. Conclusion The anti-inflammatory mechanism of the PYKS possibly raised the expression of Caspase-3 and MMP-2 of mice endometrial cells, decreased the expression of NF-κB, so as to reduce inflammation soakage in excess and formation of inflammatory medium, accelerate apoptosis and reduce local inflammation.
     结论盆炎康栓对模型大鼠的抗炎作用机理可能是通过抑制模型大鼠子宫内膜细胞NF-κB活性,增加Caspase-3、MMP-2的表达,从而减少炎细胞过度浸润及炎症介质的形成,促进炎细胞的凋亡,减轻局部炎症反应。
短句来源
     Vascular activation ,lipid metabolism, inflammatory medium and activation of leucocyte were obviously increased in group B(P<0.05,P<0.01).
     免疫损伤高脂组ET-1、脂质代谢成分、炎症介质含量、白细胞活性变化显著,与对照组比较差异显著(P<0.05);
更多       
  inflammation mediators
     Conclusion:It demonstrated the inflammation mediators TNF-α and IL-1β maybe play an important role in the biological damage of rat lung induced by repeated +Gz exposure.
     结论:炎症介质TNF-α和IL-1β在+Gz暴露导致大鼠肺组织的生物性损伤中可能起着重要的作用。
短句来源
     With the role of causative agent, the increase and interaction of various kinds of digestive enzymes and inflammation mediators such as cytokines, reactive oxygen species (ROS), endotoxin, NF-κB resulted in the injury of pancreas and distant organ.
     在致病因素的作用下,各种消化酶和细胞因子、活性氧簇、内毒素、NF-κB等炎症介质的生成增加以及它们之间的相互作用,共同造成胰腺和远处器官的损伤。
短句来源
     Objective To elucidate the effect of inflammation mediators on the mRNA expression of hyaluronan synthases and hyalaronan synthesis in human peritoneal mesothelial cells(HPMCs) .
     目的 了解炎症介质对人腹膜间皮细胞(HPMCs)透明质酸合成酶mRNA的调节作用及对透明质酸合成的影响。
短句来源
     Background and objectives: Bronchial asthma (abbreviating asthma) is a chronic disease that seriously threaten human health over the world, it is a kind of chronic inflammation of airway, which is induced by a series of inflammation mediators, such as mast cells, eosinophils , T lymphocytes and so on.
     背景与目的:支气管哮喘(Bronchial asthma BA以下简称哮喘)是全球范围内严重威胁公众健康的慢性疾病,是由肥大细胞、嗜酸性粒细胞(Eosinophil EOS)及T淋巴细胞等效应细胞分泌一系列炎症介质而诱发的气道慢性炎症。
短句来源
     Change of Inflammation Mediators in Sputum from Asthma Patients Treated by Seretide
     舒立迭吸入治疗24例支气管哮喘的疗效及诱导痰中炎症介质的变化
短句来源
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  inflammatory mediators
Upon stimulation by inflammatory mediators, the gelatinase granules are secreted before the common peroxidase-negative granules.
      
Either factor alone, be it respiratory virus infection or inflammatory mediators (chemokines, neutrophils, or reactive oxygen species), is insufficient to cause desquamation.
      
In addition, many factors such as microcirculation disturbance, ischemical reperfusion injury, excessive release of inflammatory mediators and apoptosis may also play important roles in the damage of intestinal mucosa barrier.
      
Study on the action mechanism of inflammatory mediators generated by the severe acute pancreatitis (SAP) in multiple organ injury is a hotspot in the surgical field.
      
Among many pathogenic factors, the changes of vasoactive substances, participation of inflammatory mediators as well as OFR (oxygen free radical), endotoxin, etc.
      
更多          
  inflammatory mediator
Nitric oxide (NO) is a nonspecific inflammatory mediator that has been involved in the pathogenesis of multiple sclerosis (MS).
      
Among them, tumor necrosis factor (TNF)-α, a multipotent pro-inflammatory mediator, plays a critical role in immunoregulation of asthma by contributing to bronchopulmonary inflammation and airway hyperresponsiveness.
      
H4R is involved in chemotaxis and inflammatory mediator release by eosinophils, mast cells, monocytes, dendritic cells, and T cells.
      
The platelet-activating factor (PAF) is an inflammatory mediator and it may exert some of its effects by reactive oxygen species (ROS).
      
Reactions are often tissue- and cell-specific, and provide a spectrum of inflammatory mediator release in which many of the molecular details remain to be elucidated.
      
更多          
  inflammatory medium
Effect of acupuncture to the ultra-micro structure of the injured rat lumbar nerve and inflammatory medium
      
Objective: To establish the animal model of the compressed lumbar nerve and investigate the effect of acupuncture to the ultra-micro structure of the injured rat lumbar nerve and inflammatory medium.
      
  inflammation mediators
Compromised barrier function of the endothelium in response to inflammation mediators, such as thrombin, is accompanied by reversible cell rounding and interendothelial gap formation.
      
Sequential release of lysosomal inflammation mediators and chemiluminescence response of stimulated polymorphonuclear leukocytes
      
However, the further course seems to be determined by liberated inflammation mediators like prostaglandins and leukotrienes.
      
Intraocular inflammation was induced in the rat by footpad injection of salmonella endotoxin in order to study the influence of chemical inflammation mediators in this uveitis model.
      
Osteoarthritic chondrocytes (OC) produce excessive prostaglandin E2 (PGE2) and nitric oxide (NO), which function as inflammation mediators in the pathogenesis of osteoarthritis (OA).
      
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