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肾传入神经
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  renal afferent nerve
     1. Intrarenal artery injection of L-arginine inhibits spontaneous activity of renal afferent nerve fibersFifty-two rabbits were used to determine the effect of intrarenal artery injection of L-arginine on multi- and single-unit spontaneous discharges of renal afferent nerve fibers in anesthetized rabbits.
     所得结果如下:1、肾动脉注射L-精氨酸抑制麻醉家兔肾传入纤维自发性活动在52只麻醉家兔,应用记录肾传入神经多单位和单位放电方法,观察了肾动脉内注射L-精氨酸(L-arginine)对肾传入神经活动(ARNA)的影响。
短句来源
     The present work was undertaken to study the effects of intrarenal artery injection of adenosine on spontaneous discharges of renal afferent nerve fibers, and further analyze the possible mechanisms.
     所得结果如下:在65只麻醉家兔,应用记录肾传入神经多单位和单位放电方法,观察了肾动脉内注射腺苷(adenosine)对肾传入神经活动(ARNA)的影响。
短句来源
     The purpose of this study was to determine the effect of intrarenal artery injection of L-arginine on multi- and single-unit spontaneous discharges of renal afferent nerve fibers in anesthetized rabbits.
     研究旨在应用记录肾传入神经多单位和单位放电的方法 ,观察肾动脉内注射L 精氨酸对麻醉家兔肾神经传入纤维自发放电活动的影响。
短句来源
  afferent renal nerve
     The Role of Afferent Renal Nerve in Pathogenesis of 2K1C Renovascular Hypertension
     肾传入神经在2K1C肾血管性高血压发病中的作用
短句来源
     Aim To study the role of afferent renal nerve(ARN) pathway in two kidney, one clip(2K1C) renovascular hypertension.
     目的研究肾传入神经(ARN)及其中枢投射通路在二肾一夹型(2K1C)肾血管性高血压发病中的作用。
短句来源
     The effect of intrarenal artery injection of bradykinin (BK, 5 0 μg/kg) on multi and single unit recordings of afferent renal nerve activity (ARNA) was examined in anesthetized 48 rabbits.
     在48只麻醉家兔,应用记录肾传入神经多单位和单位放电方法,观察了肾动脉内注射缓激肽(bradykinin,BK,50g/kg)对肾传入神经活动(ARNA)的影响。
短句来源
     (4)The units of afferent renal nerve with no spontaneous activity wereactivated markedly by elevation of UP,while the units with spontaneous activityshowed no change.
     (4)输尿管压增高可诱发无自发活动的肾传入神经出现明显的放电,而有自发放电的单位对此种刺激不敏感。
短句来源
  “肾传入神经”译为未确定词的双语例句
     (2)KCl(0.15 mol/L)and NaCl(1 mol/L)perfused retrogradely intopclvis via ureter route resulted in increase of integral value of ARNA by 253.79±21.64% and 172.17±15.19%(P<0.001),respectively.
     (2)经输尿管向肾盂内逆向灌注0.15mol/L KCl 和 1mol/L NaCl 溶液时,肾传入神经放电积分值分别增加253.79±21.64%和172.17±15.19%(P<0.001)。
短句来源
     (5)In response to the retrograde perfusion of KCl(0.15mol/L)and NaCl(1 mol/L)into pelvis,the activity in units with spontancousdischarge increased markedly by 210.70±23.40% and 140.07±15.72%(P<0.001),respectively,and the other units may be recruited concomitantly.
     (5)向肾盂内逆向灌流0.15mol/L KCl 和1mol/L NaCl 溶液时,肾传入神经自发放电单位的电活动分别增加210.70±23.40%,6和140.07±15.72%(P<0.001),并有新的单位被激活。
短句来源
     (4) During 10 min renal ischemia, the averaged multi unit discharge rate of the renal afferent was increased from 0 14±0 08 to 0 65±0 12 imp/s ( P <0 01).
     (4 )肾缺血 (10min)时 ,肾传入神经放电活动由 0 14± 0 0 8增至 0 6 5± 0 12imp/s (P <0 0 1)。
短句来源
     The results obtained were as follows:(1)Elevation of ureteral pressure(UP)could induce an increase in integral value of ARNA by 175.13±22.41%(P<0.001).
     结果如下:(1)输尿管压增高20.20±1.09mmHg 引起肾传入神经放电的积分值增加175.13±22.41,(P<0.001)。
短句来源
     Conclusion ARN plays an important role in the pathogenesis of 2K1C renovascular hypertension.
     结论肾传入神经在2K1C肾血管性高血压的发病中起着重要作用
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  afferent renal nerve
These afferent renal nerve inputs alter the activity of central integrative neuronal circuits that normally give rise to command signals that influence the function of effector organs.
      
There was a 28-s latency for initiation of the hindlimb vasoconstriction, which is consistent with recent evidence for renal autocoid stimulation of the afferent renal nerve receptors.
      
The 2 and unit recordings of afferent renal nerve activity were adopted to observe the response to intrarenal artery injection of BK.
      


Hemodynamic effects of renal mechano-and chemoreceptor activation andafferent renal nerve stimulation were observed in 39 anesthetized rabbits.Themean arterial pressure(MAP)and heart rate(HR)were decreased followingelveation of ureteral pressure(13—22 mmHg),and retrograde perfusion of NaCl(1.0 mol/L)or KCl(0.15 mol/L)into pelvis Via ureter route.The decreasein MAP was enhanced by sinoarotic denervation.HR,MAP,mesentery arterialperfusion pressure(MPP),hindlimb arterial perfusion pressure(HLPP),left ventricular...

Hemodynamic effects of renal mechano-and chemoreceptor activation andafferent renal nerve stimulation were observed in 39 anesthetized rabbits.Themean arterial pressure(MAP)and heart rate(HR)were decreased followingelveation of ureteral pressure(13—22 mmHg),and retrograde perfusion of NaCl(1.0 mol/L)or KCl(0.15 mol/L)into pelvis Via ureter route.The decreasein MAP was enhanced by sinoarotic denervation.HR,MAP,mesentery arterialperfusion pressure(MPP),hindlimb arterial perfusion pressure(HLPP),left ventricular pressure(LVP),left ventricular dp/dt_(max)(LV dp/dt_(max),cardiac output(CO)and total peripheral resistance(TPR)were decreased duringelectrical stimulation of afferent renal nerve.By sinoaortic denervation,theaforementioned hemodynamic parameters,except HR,CO and TPR,werereduced more markedly by electric stimulation of afferent renal nerves.There-fore it appears that sinoaortic depressor reflex plays a buffer role in the cardio-vascular responses to the stimulation of afferent renal nerves.

在39只麻醉家兔观察刺激肾脏机械和化学感受器以及电刺激肾传入神经的血流动力学效应。增加输尿管压8—22mmHg 及经输尿管向肾盂内逆向灌注 NaCl(1.0 mol/L)及 KCl(0.15mol/L)溶液时,引起平均动脉压(MAP)和心率(HR)下降;切断双侧缓冲神经后,MAP 降低更为显著。电刺激肾传入神经时,HR 减慢,MAP、肠系膜动脉和后肢动脉灌流压降低,左心室收缩压及其微分值下降,心输出量(CO)和总外周阻力(TPR)减小;切断双侧窦神经和减压神经后,除 HK、CO 和 TPR 外,其余各血流动力学指标的减弱更为显著。由此提示,动脉压力感受器反射对肾传入神经激活的心血管效应有缓冲作用。

The cffects of stimulation of renal mechano-and chemoreceptors on theafferent renal nerve activity(ARNA)were observed in 44 anesthetized rabbits.The results obtained were as follows:(1)Elevation of ureteral pressure(UP)could induce an increase in integral value of ARNA by 175.13±22.41%(P<0.001).(2)KCl(0.15 mol/L)and NaCl(1 mol/L)perfused retrogradely intopclvis via ureter route resulted in increase of integral value of ARNA by 253.79±21.64% and 172.17±15.19%(P<0.001),respectively.(3)Four patternsof afferent...

The cffects of stimulation of renal mechano-and chemoreceptors on theafferent renal nerve activity(ARNA)were observed in 44 anesthetized rabbits.The results obtained were as follows:(1)Elevation of ureteral pressure(UP)could induce an increase in integral value of ARNA by 175.13±22.41%(P<0.001).(2)KCl(0.15 mol/L)and NaCl(1 mol/L)perfused retrogradely intopclvis via ureter route resulted in increase of integral value of ARNA by 253.79±21.64% and 172.17±15.19%(P<0.001),respectively.(3)Four patternsof afferent unit discharge were found:no spontaneous activity,regular spontane-ous activity,regular spontaneous activity with burst and irregular spontancousactivity.(4)The units of afferent renal nerve with no spontaneous activity wereactivated markedly by elevation of UP,while the units with spontaneous activityshowed no change.(5)In response to the retrograde perfusion of KCl(0.15mol/L)and NaCl(1 mol/L)into pelvis,the activity in units with spontancousdischarge increased markedly by 210.70±23.40% and 140.07±15.72%(P<0.001),respectively,and the other units may be recruited concomitantly.(6)The units with no spontaneous discharge were activated by renal artery occlusion.The results implied that there are mechanoreceptor,R_1 and R_2 chemoreceptorsin the kidney of the rabbit,and they may sense the change in UP,renal ischemiaand ionic(K~+,Na~+)concentration of the solute within pelvis.

在44只麻醉家兔观察肾机械和化学感受器刺激对肾传入神经放电活动的影响。结果如下:(1)输尿管压增高20.20±1.09mmHg 引起肾传入神经放电的积分值增加175.13±22.41,(P<0.001)。(2)经输尿管向肾盂内逆向灌注0.15mol/L KCl 和 1mol/L NaCl 溶液时,肾传入神经放电积分值分别增加253.79±21.64%和172.17±15.19%(P<0.001)。(3)肾传入神经纤维的单位放电至少有四种类型:无自发放电活动,自发规则放电,自发规则的猝发放电和不规则放电。(4)输尿管压增高可诱发无自发活动的肾传入神经出现明显的放电,而有自发放电的单位对此种刺激不敏感。(5)向肾盂内逆向灌流0.15mol/L KCl 和1mol/L NaCl 溶液时,肾传入神经自发放电单位的电活动分别增加210.70±23.40%,6和140.07±15.72%(P<0.001),并有新的单位被激活。(6)夹闭肾动脉可诱发无自发活动的肾传入神经单位的放电活动。以上结果提示,家兔肾脏内存在机械和 R_1, R_2化...

在44只麻醉家兔观察肾机械和化学感受器刺激对肾传入神经放电活动的影响。结果如下:(1)输尿管压增高20.20±1.09mmHg 引起肾传入神经放电的积分值增加175.13±22.41,(P<0.001)。(2)经输尿管向肾盂内逆向灌注0.15mol/L KCl 和 1mol/L NaCl 溶液时,肾传入神经放电积分值分别增加253.79±21.64%和172.17±15.19%(P<0.001)。(3)肾传入神经纤维的单位放电至少有四种类型:无自发放电活动,自发规则放电,自发规则的猝发放电和不规则放电。(4)输尿管压增高可诱发无自发活动的肾传入神经出现明显的放电,而有自发放电的单位对此种刺激不敏感。(5)向肾盂内逆向灌流0.15mol/L KCl 和1mol/L NaCl 溶液时,肾传入神经自发放电单位的电活动分别增加210.70±23.40%,6和140.07±15.72%(P<0.001),并有新的单位被激活。(6)夹闭肾动脉可诱发无自发活动的肾传入神经单位的放电活动。以上结果提示,家兔肾脏内存在机械和 R_1, R_2化学感受器,分别感受输尿管压、肾缺血和肾盂浸浴液中 Na~+,K~+浓度的变化。

Aim To study the role of afferent renal nerve(ARN) pathway in two kidney, one clip(2K1C) renovascular hypertension. Methods The 2K1C renovascular hypertensive rat model was established in this experiment.\ The ARN pathway was blocked by the dorsal rhizotomy and electrical lesion of paraventricular nucleus of the hypothalamus(PVN).\ Systolic blood pressure of the rat was measured by tail culf method. Results Dorsal rhizotomy and destruction of PVN induced decrease of the systolic blood pressure of 2K1C...

Aim To study the role of afferent renal nerve(ARN) pathway in two kidney, one clip(2K1C) renovascular hypertension. Methods The 2K1C renovascular hypertensive rat model was established in this experiment.\ The ARN pathway was blocked by the dorsal rhizotomy and electrical lesion of paraventricular nucleus of the hypothalamus(PVN).\ Systolic blood pressure of the rat was measured by tail culf method. Results Dorsal rhizotomy and destruction of PVN induced decrease of the systolic blood pressure of 2K1C rats obviously.\ No apparent hypotensive effect of dorsal rhizotomy and destruction of PVN was found on the sham 2K1C control group. Conclusion ARN plays an important role in the pathogenesis of 2K1C renovascular hypertension.

目的研究肾传入神经(ARN)及其中枢投射通路在二肾一夹型(2K1C)肾血管性高血压发病中的作用。方法立2K1C肾血管性高血压大鼠模型,应用脊髓背根切除术及电损毁下丘脑室旁核(PVN)的方法以阻断肾传入神经通路,用尾套法测尾动脉收缩压。结果脊髓背根切除以及下丘脑室旁核损毁可导致2K1C大鼠收缩压明显降低,但对假2K1C动物并无明显影响。结论肾传入神经在2K1C肾血管性高血压的发病中起着重要作用

 
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