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钙负载
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  calcium load
     Methods Calcium load and cyclic AMP content were measured in vivo and in vitro after incubation with 5.6mmol/L and 16.7mmol/L glucose concentration. Atom spectrum absorption assay for calcium and radioimmunoassay for cAMP were employed.
     方法 体内实验直接测定糖尿病和正常大鼠骨骼肌钙负载和 c AMP含量 ,体外实验用比目鱼肌与 5 .6 mmol/ L和 16 .7mmol/ L葡萄糖保温后测定钙负载和 c AMP,二者分别采用原子光谱吸收法和放免法测定。
短句来源
     The effects of hyperglycemia on calcium load and cyclic AMP content in rat skeletal muscle
     高血糖对大鼠骨骼肌钙负载和cAMP含量的影响
短句来源
     Objective To investigate the effect of hyperglycemia on calcium load and cyclic AMP content in normal and STZ diabetic rat skeletal muscle.
     目的 探讨高浓度葡萄糖对正常和糖尿病大鼠骨骼肌钙负载和环磷酸腺苷 (c AMP)含量的影响。
短句来源
     But diabetes discounted the reduction of calcium load induced by 16.7mmol/L glucose.
     同时糖尿病倾向于阻碍高血糖引起的钙负载下降。
短句来源
     Conclusion The effects of hyperglycemia on calcium load and cyclic AMP content in skeletal muscle may accord with its anti insulin action,which may be involved in the mechanism of glucose toxicity.
     结论 高血糖对大鼠骨骼肌钙负载和 c AMP含量的影响与其抗胰岛素作用相一致 ,可能与葡萄糖毒性的发生有关
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  “钙负载”译为未确定词的双语例句
     Several signal pathways including ROS and calcium signaling, cytochrome C, p38 MAPK, PI3K/Akt are involved in the development of doxorubicin-induced cardiotoxicity, which may play a critical role though the precise biochemical mechanism still remains unclear.
     Dox可明显上调心脏组织中ROS及TNF-α水平,引起心肌细胞内钙负载及线粒体细胞色素C释放,活化钙信号、p38MAPK、PI3K/Akt等多条信号途径。
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  相似匹配句对
     Catalytic Reduction of NO by Chars Loaded with Ca and Fe
     半焦负载和铁催化还原NO的研究
短句来源
     But diabetes discounted the reduction of calcium load induced by 16.7mmol/L glucose.
     同时糖尿病倾向于阻碍高血糖引起的负载下降。
短句来源
     Calcium Nutrition of Apple Trees
     苹果树的营养
短句来源
     Load Equalization Summary
     负载均衡综述
短句来源
     2.Load balance;
     2.负载均衡;
短句来源
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  calcium load
The calcium content in woven bone was predicted to increase by 10-11% in response to this calcium load.
      
Seventeen patients who recurrently formed idiopathic calcium kidney stones (SF) and 25 age- and sex-matched healthy blood donors (H) were challenged by an oral calcium load (1 g) after an overnight fast.
      
Urine samples were taken before, 2 1/2, and 4 h after the calcium load.
      
Ninetenths of the NCSF had higher serum ionic calcium levels than H after calcium load (P>amp;lt;0.001), whereas HCSF were not different from H.
      
Depending on the calciuria or calcemia 4 h after an oral calcium load, 16 of 17 SF showed a metabolic abnormality (hypercalcemia or hypercalciuria).
      
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Objective To investigate the effect of hyperglycemia on calcium load and cyclic AMP content in normal and STZ diabetic rat skeletal muscle.Methods Calcium load and cyclic AMP content were measured in vivo and in vitro after incubation with 5.6mmol/L and 16.7mmol/L glucose concentration.Atom spectrum absorption assay for calcium and radioimmunoassay for cAMP were employed.Results High concentration glucose elevated cyclic AMP content and lessened calcium load significantly.Cyclic AMP also elevated in diabetic...

Objective To investigate the effect of hyperglycemia on calcium load and cyclic AMP content in normal and STZ diabetic rat skeletal muscle.Methods Calcium load and cyclic AMP content were measured in vivo and in vitro after incubation with 5.6mmol/L and 16.7mmol/L glucose concentration.Atom spectrum absorption assay for calcium and radioimmunoassay for cAMP were employed.Results High concentration glucose elevated cyclic AMP content and lessened calcium load significantly.Cyclic AMP also elevated in diabetic skeletal muscle incubated with even 5.6mmol/L glucose.Diabetes was necessary for high concentration glucose to elevate cyclic AMP in vitro,because cyclic AMP did not change in normal rat muscle even incubated with 16.7mmol/L glucose.But diabetes discounted the reduction of calcium load induced by 16.7mmol/L glucose.Conclusion The effects of hyperglycemia on calcium load and cyclic AMP content in skeletal muscle may accord with its anti insulin action,which may be involved in the mechanism of glucose toxicity.

目的 探讨高浓度葡萄糖对正常和糖尿病大鼠骨骼肌钙负载和环磷酸腺苷 (c AMP)含量的影响。方法 体内实验直接测定糖尿病和正常大鼠骨骼肌钙负载和 c AMP含量 ,体外实验用比目鱼肌与 5 .6 mmol/ L和 16 .7mmol/ L葡萄糖保温后测定钙负载和 c AMP,二者分别采用原子光谱吸收法和放免法测定。结果 高血糖对骨骼肌具有升高c AMP、降低钙负载的作用。糖尿病本身可升高 c AMP,并且高血糖是升 c AMP必要的辅助因素 ;同时糖尿病倾向于阻碍高血糖引起的钙负载下降。结论 高血糖对大鼠骨骼肌钙负载和 c AMP含量的影响与其抗胰岛素作用相一致 ,可能与葡萄糖毒性的发生有关

The solid base of NaOH adsorbed on MgO or CaO was used as condensation catalyst, and Pd/C was used as hydrogenation catalyst. 2 - Heptanone was synthesized from n - butyl aldehyde and propanone via cross al-dol condensation, dehydration, catalyzed hydrogenation. The advantage of solid base catalyst and the conditions of dehydration and hydrogenation were investigated. The results showed that the side reaction was reduced and the selectivity was improved when solid base was used.The yield and purity of the product...

The solid base of NaOH adsorbed on MgO or CaO was used as condensation catalyst, and Pd/C was used as hydrogenation catalyst. 2 - Heptanone was synthesized from n - butyl aldehyde and propanone via cross al-dol condensation, dehydration, catalyzed hydrogenation. The advantage of solid base catalyst and the conditions of dehydration and hydrogenation were investigated. The results showed that the side reaction was reduced and the selectivity was improved when solid base was used.The yield and purity of the product were 95.0% and 99.5% respectively under 70 ℃ of temperature,0.7 MPa of pressure and 10.0 h of reaction time.

用氧化镁或氧化钙负载氢氧化钠(NaOH)固体碱作缩合催化剂,钯/炭(Pd/C)作为加氢催化剂,由丁醛和丙酮经交叉酮醛缩合、脱水、催化加氢制得2-庚酮。研究了固体碱催化剂的优点及催化加氢条件。结果表明,固体碱作缩合催化剂,副反应少,选择性高;在温度70℃、压力0.7 MPa、反应时间10.0 h催化加氢条件下,加氢产品收率达95%,产品纯度达99.5%。

Doxorubicin (Dox) is a broad spectrum and powerful anticancer drug while its use is severely restricted by its cardiotoxicity. Dox can upregulate the expression of reactive oxygen species(ROS) and TNF-α in vitro and in vivo, as well as elevate intracellular calcium level. Several signal pathways including ROS and calcium signaling, cytochrome C, p38 MAPK, PI3K/Akt are involved in the development of doxorubicin-induced cardiotoxicity, which may play a critical role though the precise biochemical mechanism still...

Doxorubicin (Dox) is a broad spectrum and powerful anticancer drug while its use is severely restricted by its cardiotoxicity. Dox can upregulate the expression of reactive oxygen species(ROS) and TNF-α in vitro and in vivo, as well as elevate intracellular calcium level. Several signal pathways including ROS and calcium signaling, cytochrome C, p38 MAPK, PI3K/Akt are involved in the development of doxorubicin-induced cardiotoxicity, which may play a critical role though the precise biochemical mechanism still remains unclear. In this review, we will discuss the potential signal events in doxorubicin-induced cardiotoxicity. We hope it may be of great value in new drug discovery for preventing and curing doxorubicin-induced cardiotoxicity.

多柔比星(Dox)为临床常用的广谱、高效抗肿瘤药物,用于恶性淋巴瘤、实体瘤等多种癌症的治疗。然而,心脏毒性严重限制其临床应用。Dox诱发心脏毒性的作用机制十分复杂,其具体机制至今尚不清楚。Dox可明显上调心脏组织中ROS及TNF-α水平,引起心肌细胞内钙负载及线粒体细胞色素C释放,活化钙信号、p38MAPK、PI3K/Akt等多条信号途径。充分理解这些信号途径在Dox介导心脏毒性过程中的作用对于防治Dox心脏毒性具有重要的指导意义。本文就近年来Dox致心脏毒性的可能信号途径作简要综述。

 
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