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效应细胞
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  effector cells
    RADIORESISTIVITY OF CYTOSTATIC EFFECTOR CELLS AGAINST TUMOR
    抗肿瘤的细胞静止效应细胞耐放射性研究
短句来源
    Multiple Myeloma Cell Line XG-7 Can Induce CD8 +TCRαβ-bearing T Cell to Proliferate and Differentiate into Cytotoxic Effector Cells
    多发性骨髓瘤细胞XG-7诱导CD8和TCRαβ阳性的T细胞增殖分化成为细胞毒效应细胞
短句来源
    Enhancement of Killer Activity of Immune Effector Cells Caused by HLA-G Antisense Oligodeoxynucleotides
    HLA-G反义基因增强免疫效应细胞杀伤活性的研究
短句来源
    Influence of HSP70 on combined method of hyperthermia and immunologic effector cells to treat cancer
    热疗促进HSP70表达对免疫效应细胞杀伤肿瘤的影响
短句来源
    Anti-P185~(erbB2) scFv-Fc-IL-2 modulates tumor cell surface molecules and activates immune effector cells
    抗人P185~(erbB2) scFv-Fc-IL-2调变肿瘤细胞表面分子和激活免疫效应细胞
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  “效应细胞”译为未确定词的双语例句
    Activation of systemic immune effectors and induction of cytokine production by direct intratumoral injection of IL-2 DNA-liposome
    瘤内注射脂质体包裹的IL-2基因激活免疫效应细胞及诱导细胞因子
短句来源
    The cytotoxicities of CD8 positive CTLs,CD8 negative CTLs and T lymphocytes(TLs)to U937 cells were(66.36±12.43)%,(34.47±8.19)% and(15.79±4.64)% respectively under the same effector target ratio(40∶1). Among them,the anti-leukemia cytotoxicity of the CD8 positive group was highest.
    3组效应细胞CD8+CTL、CD8-CTL和T淋巴细胞(TL)组在相同效靶比(40∶1)对U937细胞株的杀伤率分别为(66.36±12.43)%、(34.47±8.19)%和(15.79±4.64)%,以CD8+CTL组最高;
短句来源
    And AdIL-2-HepG2/DCs could secrete high level of IL-2 in vitro (6.78±0. 18) mg/L and also could stimulate T cell proliferation markedly (CPM 21 878±1089). Compared with other DC groups, the AdIL-2-HepG2/DCs induced higher cytotoxicity against HepG2 cells (74.5±3.8)% in vitro.
    AdIL-2-HepG2/DC能非常显著地刺激自体T细胞增殖(CPM值为21 878±1089),当靶细胞为HepG2时,其诱导的CTL杀伤活性(74.5±3.8)%显著高于其他各组,并且其杀伤能力与效应细胞数量成正比。
短句来源
    Results The killing rate in vitro of 3 groups were 53.14% ,30.10% and 31.49% ,respectively.
    结果3组效应细胞在体外对NCI-H460细胞的杀伤率分别为53.1%,30.1%和31.5%;
短句来源
    Method:Peripheral blood mononuclear cells (PBMNC) isolated from healthy donors were induced to obtain DC and CIK cells by conventional methods, the DC pulsed with or without idiotype (Id) precipitated from MM cell line OPM-2 culture supernatant was co-cultured with CIK (CIK, DC + CIK and Id-DC+CIK). Cells phenotypes were analyzed by flow cytometry.
    方法:采集健康供者外周血单个核细胞(PBMNC)用常规方法诱导DC和CIK细胞,将骨髓瘤OPM-2细胞培养上清提取的Id冲击或未冲击的DC与CIK细胞共培养(CIK、DC加CIK、Id-DC加CIK),用流式细胞术分析细胞表型,MTT法检测体外效应细胞杀伤活性。
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  effector cells
And their suppression on the proliferation of CD4+CD25- T effector cells was analyzed by cell proliferation assay in vitro.
      
Opioidergic mechanisms are involved in responses to nociceptive and antigenic stimuli at all levels and stages (from peripheral nociceptors to the cerebral cortex and from the precursors of immunocompetent cells to mature effector cells).
      
Nitric oxide (NO) is a mobile, highly reactive signal molecule, and changes the expression of specific genes in effector cells.
      
The regulatory controls comprise the receptor cells recognizing the external and internal signals, the tissues of connection channels, the effector cells, and the feedback loop elements.
      
A novel cytokine fusion protein was constructed by fusing granulocyte macrophage colony stimulating factor (GM-CSF) with monocyte chemotactic activating factor (MCAF), which acts as a factor directing effector cells (monocytes) to a target site.
      
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The human macrophage cytotoxicity in vitro was observed by inverted microscope with photographical arrangement and time-lapse cinemicrography.This observation revealed dramatic changes in both effectors and target cells. The macrophage cytotoxicity, tumor cell killing, should go through the tumout cell-macrophage membrane contact. The in vitro cytotoxicity of macrophages from patients with stomach cancer, benign stomach diseases and normal adults were studied and different degrees of cytotxicity effect observed....

The human macrophage cytotoxicity in vitro was observed by inverted microscope with photographical arrangement and time-lapse cinemicrography.This observation revealed dramatic changes in both effectors and target cells. The macrophage cytotoxicity, tumor cell killing, should go through the tumout cell-macrophage membrane contact. The in vitro cytotoxicity of macrophages from patients with stomach cancer, benign stomach diseases and normal adults were studied and different degrees of cytotxicity effect observed. It was found that pre-cultivation of macrophages from stomach cancer patients still had cytotoxicic effects on gastric carcinoma cell line. In cultivation of macrophages from stomach cancer patients, the cell-free exudate had no influence on the target cells.The experimental results are discussed.

本文应用了附有摄影装置的倒置显微镜与缩时定格显微电影摄影观察了人巨噬细胞体外细胞毒作用。这观察发现了效应细胞与靶细胞之间戏剧性的变化:巨噬细胞细胞毒对肿瘤细胞的杀伤必须通过这两种细胞的膜接触这一步骤。本实验分别观察了胃癌患者、良性胃疾患患者以及正常人的巨噬细胞体外细胞毒作用,发现这些细胞毒作用有强弱程度的不同。实验还发现胃癌患者的巨噬细胞经预培养后对胃癌细胞株仍有细胞毒作用。还看到胃癌患者巨噬细胞培养上清液不影响靶细胞的生长。本文最后对实验观察的结果进行了讨论。

Natural Killer(NK)cytotoxicity of human PML coulcb be markedly increased by human umbilical-corld-blood-derived interferon-α (P-IFN) and Astragalus membranacesu (AIMember.)but slightly inhibited by high concentration of P-IFN (105u/ml) or A-Membr. (10mg/ml) .P-IFN and A.Membr., on the other hand, could protect target cells from NK cytotoxicity.However, this effect was lower than that of their enhancement of NK cytotoxicity.Moreover, P-IFN and A.Membr .could enhance each other.NK cytotoxicity might be increased...

Natural Killer(NK)cytotoxicity of human PML coulcb be markedly increased by human umbilical-corld-blood-derived interferon-α (P-IFN) and Astragalus membranacesu (AIMember.)but slightly inhibited by high concentration of P-IFN (105u/ml) or A-Membr. (10mg/ml) .P-IFN and A.Membr., on the other hand, could protect target cells from NK cytotoxicity.However, this effect was lower than that of their enhancement of NK cytotoxicity.Moreover, P-IFN and A.Membr .could enhance each other.NK cytotoxicity might be increased by 5 to 6 times when the both agents were com-bined ia treatment of effector cells. A.Membr could induce interferon-r which mediated the enhancing effect of A.Membr NK cytotoxicity.There was a relationship be-tween enhancing effect of P-IFN on NK activity and large molecular metabolism of effector cells. It was supposed that interferon, except for activating NK cytotoxicity directly, might stimulate lymphocytes producing other kinds of factors by which interferon might further amplify its enhancing effect on NK cytotxicity.

本文应用~(125)IudR标记的人胃癌细胞SGC-7991作靶,研究了部分纯合的人脐血α干扰素及黄芪对人外周血NK细胞毒的影响,并对其促进NK活性的机理做了初步探讨。结果指出,人脐血干扰素和黄芪对NK细胞毒均有明显的促进作用,促进活性前者大于后者,但大剂量的干扰素或黄芪则可轻度抑制NK细胞毒。另外,干扰素和黄芪还可保护靶细胞抵抗NK细胞毒,其程度不及对NK活性的促进作用。干扰素和黄芪具有协同作用,两者联合处理效应细胞,可使NK细胞毒提高5~6倍。实验进一步表明,黄芪可诱生γ干扰素,黄芪对NK细胞毒的促进作用与其诱导的抗病毒活性相平行。干扰素对NK细胞毒的促进作用与细胞的大分子代谢有关,推测,干扰素除能直接迅速激活NK细胞毒外,还可诱导淋巴细胞产生其它因子,进一步扩大干扰素对NK细胞毒的激活效应。

That alpha-foetopretoin(AFP) has suppressive effect in vitro on lymphocytes response to stimulation of mitogen or alloantigen has been well reported.Our study here exhibited the inhibitory effect of AFP on interleukin-2 (IL-2) production in vitro which has not yet been reported before.After treatment of MLA-144 with AFP, IL-2 activity was measured by3H-TdR incorporation in activated lymphocytes and expressed by growth index or per cent of control. The activity of IL-2 in the medium with AFP was significantly...

That alpha-foetopretoin(AFP) has suppressive effect in vitro on lymphocytes response to stimulation of mitogen or alloantigen has been well reported.Our study here exhibited the inhibitory effect of AFP on interleukin-2 (IL-2) production in vitro which has not yet been reported before.After treatment of MLA-144 with AFP, IL-2 activity was measured by3H-TdR incorporation in activated lymphocytes and expressed by growth index or per cent of control. The activity of IL-2 in the medium with AFP was significantly lower than that of the control. The inhibitory effect could be partially eliminated by addition of exogenous IL-2 preparation after 24 hr incubation, and thus the activity was raised from about 30% to 80% of the control.After treatment of rat mixed lymphocyte culture with 50 μg/ml or 100μg/ml of AFP in the medium, the activity of IL-2 was 33% or 38% of the control respectively. Similar effect was also observed in the production of IL-2 induced by rat lymphocytes activated by ConA in vitro. The activity of IL-2 reduced to 63% or 71% in the medium with 20μg/ml or 50μg/ml of AFP respectively.These results show that one o?the immunoregulatory actions of AFP is probably through the inhibitory effect on the production of IL-2.

本文报道了用MLA-144 T细胞瘤株的培养、大鼠混合淋巴细胞反应及刀豆素A(ConA)刺激淋巴细胞等三种方式制备白细胞间介素-2(IL-2)。以~3H-胸苷掺入效应细胞值作为测定IL-2活性的指标,分别测定甲胎蛋白(AFP)对IL-2产生的影响,与无AFP对照相比较,观察到AFP对体外三种方式产生IL-2均有明显的抑制作用。

 
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