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   急性肾损伤 在 中医学 分类中 的翻译结果: 查询用时:0.507秒
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急性肾损伤
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  acute renal injury
After acute renal injury, renal expression of several growth factors becomes transiently altered.
      
The data indicate thatCCr is not an accurate indicator of GFR in children who have had acute renal injury.
      
It is reported that urinary annexin V concentration may be an indicator of apoptosis and acute renal injury related to the urinary protein level.
      
Reactive oxygen species (ROS) have been postulated to play a major role in postischemic acute renal injury.
      
Insulin-like growth factor-I increases p21 expression and attenuates cisplatin-induced acute renal injury in rats
      
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In order to evaluate the effect of Cordycops sinensis(CS)on aminoglycoside (AG) induced nephrotoxicity,gentamycin was imposed on the young and old rats with CS adiministration.The renal tubular injury was ameliorated as evidenced by less prominent increment of BUN,SCr,sodium excretion,urinary NAGase and less severity of histopathological changes as compared with control.In addition,the use of CS could promote an earlier recovery of renal oxygen consumption insulin clearance,and sodium absorption in isolated...

In order to evaluate the effect of Cordycops sinensis(CS)on aminoglycoside (AG) induced nephrotoxicity,gentamycin was imposed on the young and old rats with CS adiministration.The renal tubular injury was ameliorated as evidenced by less prominent increment of BUN,SCr,sodium excretion,urinary NAGase and less severity of histopathological changes as compared with control.In addition,the use of CS could promote an earlier recovery of renal oxygen consumption insulin clearance,and sodium absorption in isolated perfused kidney from CS treated intoxicated rat than that from control.Possible mechanisms of CS on drug-induced nephrotoxicity include:(1)Accelerating the regeneration of tubular cells,(2) Protecting the sodium pump activity of tubular cells,(3)Attenuating the tubular cell lysosome hyperfunction stimulated by phagocytosis of AG as well as decreasing the tubular cell lipoperoxidation in response to toxic injury;(4) Reducing the tissue Ca++ content.

为阐明冬虫夏草(简称虫草)对氨基糖甙肾毒性损伤的防治作用,我们进行了一系列的实验研究,结果表明:在庆大霉素急性肾损伤模型中,接受虫草治疗的大鼠尿NAG酶、血肌酐水平低于对照组,肾小球滤过和保钠功能优于对照组。离体肾灌注(IPK)研究证明,虫草可提高IPK代谢率,增加肾小球滤过,保护肾小管正常运转。此外虫草还可减轻体外培养的肾小管细胞对庆大霉素损伤的易感性。虫草作用的机理可能包括:(1)拮抗氨基糖甙所致肾脏氧耗下降,提高肾小管Na+-K+-ATP酶活力;(2)减轻氨基糖甙溶酶体损伤和脂质过氧化损伤;(3)降低组织钙含量;(4)通过诱导肾小管细胞c-myc基因表达,以及对损伤状态下肾内肾组织表皮生长因子(EGF)调节的保护,促进肾小管的再生修复。

objective: To study the protective effects of Shen Huang Cao-mixture on acute renal injury caused by gentamicin in rats.. Methods: 40 rats were randomly divided into four groups (10/group) : group A (normal group), group B(Shen Huang Cao-mixture to be convinced), group C(treated with coated oxidized strach) and group D (pathologic group). The later three groups were made to become acute renal injury models. Group A & group D used physiologic saline only. After ten days treatment, blood urea nitrogen(BUN), Serum...

objective: To study the protective effects of Shen Huang Cao-mixture on acute renal injury caused by gentamicin in rats.. Methods: 40 rats were randomly divided into four groups (10/group) : group A (normal group), group B(Shen Huang Cao-mixture to be convinced), group C(treated with coated oxidized strach) and group D (pathologic group). The later three groups were made to become acute renal injury models. Group A & group D used physiologic saline only. After ten days treatment, blood urea nitrogen(BUN), Serum creatinine (Scr) and p-N-acetyglocosamidase (NAG) were monitored. And renal histological changes were observed. Results: In decreasing BUN, Scr & NAG, Shen Huang Cao is superior to group C(P<0.01); It was also found that Shen Huang Cao-mixture could reduce the edema of renal tubular by lightscope, the damage of mitochondria is lighter. Conclusion: Shen Huang Cao-mixture can not only improve the renal function of the rats with damaged kidney, but also alleviate the edema and prevent the epithelial cells of the renal tubular from being damaged by gentamicin anymore.

目的:研究保肾康合剂对庆大霉素所致的急性肾损伤大鼠肾功能的保护作用。方法:取大鼠40只,其中10只作为正常对照组,其余30只制成急性肾损伤模型,并随机分为三组:保肾康组用保肾康溶液灌胃;包醛组用包醛氧淀粉灌胃;病模组(同正常组)用0.85%生理盐水灌胃。治疗10天,观察各组血肌酐(Scr)、尿素氮(BuN)和尿酶(NAG)的变化;将大鼠处死后观察其肾组织的病理改变。结果:保肾康组治疗后(Scr)、(BuN)和(NAG)较病模组明显下降,并优于包醛组(P<0.01);光镜检查保肾康合剂可使肾小管上皮细胞肿胀,坏死等组织病理改变明显减轻,电镜下可见线粒体损伤等变化亦显著减轻。结论:保肾康合剂可改善急性肾损伤大鼠的肾功能,减轻水肿,阻止庆大霉素对肾小管上皮细胞的进一步损害。

 
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