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脂蛋白酯酶基因
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  lipoprotein lipase gene
     Relationship between High Density Lipoprotein Subclasses Distribution and Lipoprotein Lipase Gene HindⅢ Polymorphism in Obese Subjects
     肥胖者高密度脂蛋白亚类组成与脂蛋白酯酶基因多态性的关系
短句来源
     Breeding of Lipoprotein Lipase Gene Knockout Mice
     脂蛋白酯酶基因敲除小鼠的繁育
短句来源
     Relationship between polymorphism of the lipoprotein lipase gene and pregnancy induced hypertension
     妊高征与脂蛋白酯酶基因PvuⅡ多态性与血脂指标关系
短句来源
     Objective This study inquired into the relationship between single nucleotide polymorphisms(SNPs) of lipoprotein lipase gene (LPL) and coronary atherosclerotic heart disease(CHD).
     目的 探讨脂蛋白酯酶基因 (lipoprotein lipase,L PL)单核苷酸多态性 (single nucleotidepolymorphisms,SNP)与冠状动脉粥样硬化性心脏病 (简称冠心病 )的关系。
短句来源
     AIM: To investigate lipoprotein lipase gene HindⅢ polymorphism and its relationship with serum lipids and apolipoprotein, serum HDL subclasses in patients with hyperlipoidemia.
     目的:探讨高脂血症患者脂蛋白酯酶基因内含子8HindⅢ酶切位点多态性与HDL亚类组成的变化关系。
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  “脂蛋白酯酶基因”译为未确定词的双语例句
     Relationship between subclasses of serum HDL and LPL gene HindⅢ polymorphism in hyperlipidemia
     高脂血症患者HDL亚类组成与脂蛋白酯酶基因内含子8 HindⅢ多态性的关系
短句来源
     Conclusions H~+H~+ genotype was associated with higher plasma TG levels of obesity.
     结论脂蛋白酯酶基因H+H+基因型与肥胖者增高的甘油三酯水平有关。
短句来源
     Conclusion:The Hind Ⅲ polymorphism at intron 8 of LPL gene is associated with subclasses of HDL and mature metabolism of HDL in type Ⅳ hypertriglyceridemic subjects.
     结论:ⅣHL患者脂蛋白酯酶基因多态性与HDL亚类的组成及成熟代谢相关。
短句来源
     Investigation of the Relationship between Polymorphism of Lipoprotein Lipase(Se r447-Thr) and Pregnancy Induced Hypertension Syndrome
     妊高征与脂蛋白酯酶基因Ser447-Thr多态性与血脂指标关系
短句来源
     Objective:The present study explored the association between lipoprotein lipase (LPL) gene Hind Ⅲ polymorphism and serum high density lipoprotein (HDL) subclasses in patients with type Ⅳ hypertriglyceridemia.
     目的:探讨Ⅳ型高脂血症(ⅣHL)患者高密度脂蛋白(HDL)亚类组成与脂蛋白酯酶基因多态性的关系。
短句来源
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  相似匹配句对
     Breeding of Lipoprotein Lipase Gene Knockout Mice
     脂蛋白酯酶基因敲除小鼠的繁育
短句来源
     4 new genes were obtained.
     U基因
短句来源
     Objective To explore the relationship between polymorphism of the lipoprotein lipase gene and pregnancy induced hypertension.
     目的探讨妊娠高血压综合征与脂蛋白酯酶(LPL)PvuII基因多态性的关系。
短句来源
     A preliminary study of single nucleotide polymorphisms of lipoprotein lipase gene in coronary atherosclerotic heart disease *
     冠状动脉粥样硬化性心脏病患者脂蛋白酯酶基因的单核苷酸多态性的初步研究
短句来源
     Lipoprotein Lipase and Atherosclerosis
     脂蛋白酯酶与动脉粥样硬化(英文)
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  lipoprotein lipase gene
Association of the HindIII Polymorphism of the Lipoprotein Lipase Gene with Myocardial Infarction
      
Association of the HindIII Polymorphism of the Lipoprotein Lipase Gene with Myocardial Infarction and the Life Span in Elderly P
      
Common mutations of the lipoprotein lipase gene and their clinical significance
      
Furthermore, they decrease triglycerides by increasing lipoprotein lipase gene expression and by decreasing apolipoprotein C-III gene expression.
      
Sequencing of this proband's lipoprotein lipase gene showed no alteration compared to the wildtype sequence.
      
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Objective This study inquired into the relationship between single nucleotide polymorphisms(SNPs) of lipoprotein lipase gene (LPL) and coronary atherosclerotic heart disease(CHD). Methods Exon 6 and fragment of intron 6 in LPL gene from 102 cases of CHD and 110 normal subjects were analysed by polymerase chain reaction amplification, temperature modulated high performance liquid chromatography (TmHPLC) and DNA sequencing. Results Three new SNPs (4212 t/c, 4509 t/c, 4576 a/c) were detected in the intron...

Objective This study inquired into the relationship between single nucleotide polymorphisms(SNPs) of lipoprotein lipase gene (LPL) and coronary atherosclerotic heart disease(CHD). Methods Exon 6 and fragment of intron 6 in LPL gene from 102 cases of CHD and 110 normal subjects were analysed by polymerase chain reaction amplification, temperature modulated high performance liquid chromatography (TmHPLC) and DNA sequencing. Results Three new SNPs (4212 t/c, 4509 t/c, 4576 a/c) were detected in the intron 6 of LPL gene. There was significant difference in frequencies of 4212t/c,and 4576 a/c between health group and CHD group( P <0 05). Conclusion This study has accumulated additional data on SNPs in LPL gene and provided new data for exploring the mechanism of CHD.

目的 探讨脂蛋白酯酶基因 (lipoprotein lipase,L PL)单核苷酸多态性 (single nucleotidepolymorphisms,SNP)与冠状动脉粥样硬化性心脏病 (简称冠心病 )的关系。方法 利用聚合酶链反应扩增110名健康人和 10 2例冠心病患者 L PL 基因第 6外显子和第 6内含子的片段 ,采用温度调控高效液相色谱技术对扩增的片段进行了筛查 ,对筛查到的杂合片段进行序列测定 ,并与正常序列进行对照分析。结果 在L PL 基因第 6内含子中发现了 3个尚未报道的 SNP(42 12 t/ c、45 0 9t/ c、45 76 a/ c)。它们的频率在被检测的冠心病患者和健康人群中存在差异 ,其中 42 12 t/ c、45 76 a/ c两组间差异显著 (P<0 .0 5 )。结论 补充了 L PL基因 SNPs的数据库信息。L PL基因 SNPs的研究可能对冠心病等复杂疾病机理的认识有重要价值

Objective To establish the credible examination methods of LPL(-/-) knock-out mice and to find out their breeding rule. Methods The mice were bred in different ways,including crossbreed,backcross and intercross. In order to determine the genotype of the mice by PCR,genomic DNA of LPL(-/-) knock-out mice was extracted from tails. Results LPL(-/-) knock-out mice was made successfully. The credible breeding process and examination methods were established as well.Conclusion The animal model of LPL(-/-) knock-out...

Objective To establish the credible examination methods of LPL(-/-) knock-out mice and to find out their breeding rule. Methods The mice were bred in different ways,including crossbreed,backcross and intercross. In order to determine the genotype of the mice by PCR,genomic DNA of LPL(-/-) knock-out mice was extracted from tails. Results LPL(-/-) knock-out mice was made successfully. The credible breeding process and examination methods were established as well.Conclusion The animal model of LPL(-/-) knock-out mice was developed.

目的 摸清脂蛋白酯酶基因敲除小鼠的繁殖规律,建立可靠的检测方法。方法 采用杂交、回交、互交的方法进行培育;采用从LPL(- / - )基因敲除小鼠的个体组织(鼠尾)中提取基因组DNA进行PCR分析,对其LPL(- / - )基因型作出鉴定。结果 繁育出脂蛋白酯酶基因敲除小鼠新品系,并建立了可靠的繁殖方法和检测方法。结论 建立了脂蛋白酯酶基因敲除小鼠动物模型。

AIM: To investigate lipoprotein lipase gene HindⅢ polymorphism and its relationship with serum lipids and apolipoprotein, serum HDL subclasses in patients with hyperlipoidemia. METHODS: Lipoprotein lipase gene HindⅢ polymorphism was assayed by polymerase chain reaction-restriction fragment length polymorphism (PCR-RFLP). The subclasses of serum HDL in 152 hyperlipoidemia patients and 128 healthy subjects were determined by two-dimensional gel electrophoresis conjunction with immunodetection method. RESULTS:...

AIM: To investigate lipoprotein lipase gene HindⅢ polymorphism and its relationship with serum lipids and apolipoprotein, serum HDL subclasses in patients with hyperlipoidemia. METHODS: Lipoprotein lipase gene HindⅢ polymorphism was assayed by polymerase chain reaction-restriction fragment length polymorphism (PCR-RFLP). The subclasses of serum HDL in 152 hyperlipoidemia patients and 128 healthy subjects were determined by two-dimensional gel electrophoresis conjunction with immunodetection method. RESULTS: H+H+ genotype and allele H+ in hyperlipoidemia and control groups were both the highest. In hyperlipidemia group, H+H+ genotypes tended to be higher than that in control group, while H+H- and H-H- genotypes were significantly lower (P<0.05). In hyperlipidemia group allele H+ carriers' frequency tended to be higher than that in control group (P<0.05). In hyperlipoidemia group, the genotype of H+H+ showed higher serum TG, apoB100 levels, TG/HDL-C ratio, preβ_1-HDL, HDL_(3b) and lower HDL_(2a), HDL_(2b) compared with H-H- (P<0.05). In control group, the genotype of H+H+ had higher serum TG,HDL_(3c) and lower HDL_(2a) compared with H-H- (P<0.05). CONCLUSION: The HindⅢ polymorphism at intron 8 of LPL gene is associated with the general shift toward smaller size of HDL particle size in hyperlipoidemia, and the change of HDL subclasses distribution profile may be closely related to the pathogenesis of atherosclerosis in Chinese patients with hyperlipoidemia.

目的:探讨高脂血症患者脂蛋白酯酶基因内含子8HindⅢ酶切位点多态性与HDL亚类组成的变化关系。方法:采用聚合酶链反应-限制性片段长度多态性和双向电泳-免疫印迹检测法,分析152例高脂血症患者和128例血脂正常者的脂蛋白酯酶基因内含子8HindⅢ多态性、HDL亚类组成及相对百分含量。结果:高脂血症组和对照组均以H+H+基因型为主,但高脂血症组H+H+基因型频率显著高于对照组,而H+H-和H-H-基因型频率显著低于对照组(P<0.05)。高脂血症组H+等位基因频率显著高于对照组,而H-等位基因频率显著低于对照组(P<0.05)。高脂血症组和正常对照组H+H+基因型者血清TG明显高于H-H-者;高脂血症组H+H+基因型者apoB100、TG/HDL-C比值明显高于H-H-者(P<0.05)。高脂血症患者H+H+基因型小颗粒的preβ1-HDL、HDL3b相对含量多于H-H-者,大颗粒的HDL2a、HDL2b相对含量少于H-H-者;正常对照组H+H+基因型HDL3c多于H-H-者,而HDL2a少于H-H-者,其差异均显著(P<0.05)。结论:中国人高脂血症患者脂蛋白酯酶基因Hi...

目的:探讨高脂血症患者脂蛋白酯酶基因内含子8HindⅢ酶切位点多态性与HDL亚类组成的变化关系。方法:采用聚合酶链反应-限制性片段长度多态性和双向电泳-免疫印迹检测法,分析152例高脂血症患者和128例血脂正常者的脂蛋白酯酶基因内含子8HindⅢ多态性、HDL亚类组成及相对百分含量。结果:高脂血症组和对照组均以H+H+基因型为主,但高脂血症组H+H+基因型频率显著高于对照组,而H+H-和H-H-基因型频率显著低于对照组(P<0.05)。高脂血症组H+等位基因频率显著高于对照组,而H-等位基因频率显著低于对照组(P<0.05)。高脂血症组和正常对照组H+H+基因型者血清TG明显高于H-H-者;高脂血症组H+H+基因型者apoB100、TG/HDL-C比值明显高于H-H-者(P<0.05)。高脂血症患者H+H+基因型小颗粒的preβ1-HDL、HDL3b相对含量多于H-H-者,大颗粒的HDL2a、HDL2b相对含量少于H-H-者;正常对照组H+H+基因型HDL3c多于H-H-者,而HDL2a少于H-H-者,其差异均显著(P<0.05)。结论:中国人高脂血症患者脂蛋白酯酶基因HindⅢ酶切位点多态性与HDL亚类的组成相关。

 
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