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环境干预
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  environmental intervention
    Objective To observe the effect of early enriched environmental intervention on synaptic ultrastructure and the synaptophysin expression (p38) in hippocampal CA1 area after hypoxia-ischemic brain damage(HIBD) in neonatal rats.
    目的探讨早期丰富环境干预对缺氧缺血性脑损伤(HIBD)新生大鼠海马CA1区神经元突触超微结构和突触素(p38)表达的影响。
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    Effects of enriched environment intervention on cerebral ultramicrostructure in rats after hypoxic-ischemic brain damage
    丰富环境干预后HIBD大鼠脑超微结构改变
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  environmental intervention
A successful, simple and widely applicable environmental intervention could have major benefits.
      
Such environmental intervention may be more widespread than usually considered, triggering some apparent predator-prey cycles.
      
Determinants of the Willingness-to-Participate in an Environmental Intervention in a Beirut Neighborhood
      
An analysis of determinants of the willingness-to-participate in an environmental intervention in a Beirut neighborhood is conducted.
      
Environmental intervention: The monitoring paradigm
      
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Objective To observe the effect of early enriched environmental intervention on synaptic ultrastructure and the synaptophysin expression (p38) in hippocampal CA1 area after hypoxia-ischemic brain damage(HIBD) in neonatal rats. Methods The rat model of HIBD was made by the method of Rice in 20 Sprague-Dawley rats, and divided randomly into 2 groups: the intervention group, non-intervention group. Another 10 rats underwent sham-operation as control. From the second day after HIBD, the rats of the intervention...

Objective To observe the effect of early enriched environmental intervention on synaptic ultrastructure and the synaptophysin expression (p38) in hippocampal CA1 area after hypoxia-ischemic brain damage(HIBD) in neonatal rats. Methods The rat model of HIBD was made by the method of Rice in 20 Sprague-Dawley rats, and divided randomly into 2 groups: the intervention group, non-intervention group. Another 10 rats underwent sham-operation as control. From the second day after HIBD, the rats of the intervention group were kept in an enriched environment for 20 d, the rats of non-intervention group and sham-operation group were raised in standard enviroment instead. The hippocampal ultrastructure was observed with electron microscope and the p38 expression was detected immunohistochemically in all rats. Results The ultrastructure of hippocampus CA1 pyramidal neuron in the non-intervention group were distorted as compared with that in the intervention group and control group. The corrected optical densities (COD) of immunoreactive products of the hippocampal p38 were significantly decreased in non-intervention group as compared with intervention group and control group (P<0.01). There were no marked difference of either ultrastructure or immunohistochemically reactived COD of p38 between intervention group and control group (P>0.05). Conclusion Early enriched environment intervention could improve the synaptic reconstruction and reduce the ultrastructural damage induced by hypoxic-ischemia.

目的探讨早期丰富环境干预对缺氧缺血性脑损伤(HIBD)新生大鼠海马CA1区神经元突触超微结构和突触素(p38)表达的影响。方法按Rcie法制作HIBD新生动物模型,共计20只,随机分为干预组和非干预组。同时设假手术对照组10只。干预组大鼠于术后第2天开始给予丰富环境刺激,持续20d。干预结束后用透射电镜观察各组海马CA1区锥体神经元超微结构,免疫组化和图像分析技术检测p38的表达。结果与对照组比较,非干预组海马CA1区锥体细胞见部分核膜消失、线粒体嵴模糊、神经元突触数量减少,突触间隙增宽,突触囊泡减少,突触后致密物变薄;干预组神经元和突触无明显异常。非干预组海马CA1区突出素光密度值明显低于干预组和对照组(P<0.01),而干预组和对照组之间无显著差异(P>0.05)。结论阻止或减轻HI后突触超微结构的损伤、促进突触的重建可能是丰富环境发挥作用的物质基础。

Objective To investigate the effect of enriched environment intervention on cerebral ultrastructure of hypoxic-ischemic brain damage (HIBD) rats. Methods Neonatal SD rats of 7 d were established into HIBD model according to the methods of Rice, and divided into enriched environment (EE) group in which touch and enriched environment stimulus were carried out, and standard environment (SE) group. Hippocampal ultramicrostructure, nerve plexus and synapse were observed by transmission electron microscope (TEM)....

Objective To investigate the effect of enriched environment intervention on cerebral ultrastructure of hypoxic-ischemic brain damage (HIBD) rats. Methods Neonatal SD rats of 7 d were established into HIBD model according to the methods of Rice, and divided into enriched environment (EE) group in which touch and enriched environment stimulus were carried out, and standard environment (SE) group. Hippocampal ultramicrostructure, nerve plexus and synapse were observed by transmission electron microscope (TEM). Results The neuronal shrinkage, swellen mitochondria, reduced nerve plexus and synapse, changed synapse structure were observed in SE group, while neuron and synapse in EE group were of no apparent abnormality. Conclusion Touch and enriched environment stimulus can promote the ultramicrostructure’s recovery of HIBD rats, in which the re-establishment of neural network and the enhancement of plasticity was one of the mechanism.

目的研究丰富环境刺激对缺氧缺血性脑损伤(HIBD)大鼠脑超微结构的影响。方法大鼠分为缺氧缺血组及丰富环境干预组,干预组予早期抚触和丰富环境刺激共28d,利用透射电镜观察两组大鼠海马神经元超微结构、神经丝及突触情况。结果缺氧缺血组海马神经细胞固缩改变,线粒体肿胀,神经丝数量减少,排列稀疏,突触数量减少,突触间隙增宽,突触囊泡减少,突触后致密物变薄。干预组海马神经元和突触无明显异常。结论早期抚触及丰富环境刺激可以促进缺血缺氧的脑组织超微结构恢复,脑组织神经网络重建及脑的可塑性增加是其可能的机制之一。

Objective To investigate effect of enriched environment on neurofilament protein (NF) expression in the rats with hypoxic-ischemic brain injury (HIBI). Methods Totally 52 7-day-aged rats were divided into normal control group (n=12), and the left 40 into HI non-intervention group(n=20) and EE intervention group(n=20) after establishment of HIBI model. EE intervention group received touch and enriched environment stimulus for 28 d. After determination of ethology, the expression of NF protein was observed in...

Objective To investigate effect of enriched environment on neurofilament protein (NF) expression in the rats with hypoxic-ischemic brain injury (HIBI). Methods Totally 52 7-day-aged rats were divided into normal control group (n=12), and the left 40 into HI non-intervention group(n=20) and EE intervention group(n=20) after establishment of HIBI model. EE intervention group received touch and enriched environment stimulus for 28 d. After determination of ethology, the expression of NF protein was observed in the hippocampus and the results were analyzed quantitatively with image analysis system. Results Compared with normal control group and EE intervention group, the NF expression and the ratio of integrated optical density of NF in right to left brain (L∶R_ NF ) were decreased in HI non-intervention group, while no difference was seen in the normal control group and EE intervention group. Morris water maze indicated that long-term memory of HI non-intervention group was much lower than that of normal control group and EE intervention group (P<0.01) , but EE intervention group was similar in normal control group. Conclusion Touch and enriched environment stimulus conduce to attenuate HIBI, probably by the mechanism of promoting neural network’s re-establishment.

目的研究丰富环境刺激对缺氧缺血性脑损伤(hypoxic-ischemicbrain injury,HIBI)大鼠脑神经丝(neurofila-ment,NF)蛋白的影响。方法大鼠分为正常对照组12只、缺氧缺血非干预组及丰富环境干预组各20只,干预组予早期抚触和丰富环境刺激共28d,行为学测定后,利用免疫组织化学方法观察各组海马神经丝蛋白的染色情况,并借助自动图像分析系统对其进行定量分析。结果非干预组左侧脑组织NF表达较干预组及正常对照组明显减少,左侧与右侧脑组织NF-H积分光密度值之比(L∶RNF)非干预组明显小于对照组及干预组(P<0.01),干预组与对照组无显著差异(P>0.05)。Morris水迷宫测试表明远期学习记忆能力非干预组明显低于对照组及干预组(P<0.01),干预组与对照组无显著差异(P>0.05)。结论早期抚触和丰富环境刺激有助于促进缺血缺氧脑损伤恢复,促进脑组织神经网络重建是其可能的机制之一。

 
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