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肾近曲小管
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  renal proximal tubules
     Methods To use Ce3+ or Pb2+ as capture agents to detect carbonic anhydrase, Na+ - K+ - ATPase, Mg2+ -ATPase, 5'-nucleotidase and NADPH oxidase activities on endosome membrane in epithelial cells of renal proximal tubules,hepatic cell of rat and human leukocytes, with cytochemical method.
     方法用细胞化学方法,以Ce3+或Pb2+作捕捉剂,分别检测碳酸酐酶,Mg2+-ATP酶,Na+-K+-ATP酶,5’-核苷酸酶,NADPH oxidase在大鼠肾近曲小管上皮细胞、肝细胞以及人体嗜中性白细胞内吞体膜上的酶活性。
短句来源
     Sensitivity of Various Indicators Reflecting Mercurials Cytotoxicity in Isolated Rat Renal Proximal Tubules
     汞化合物对大鼠游离肾近曲小管细胞毒性及其指标的探讨
短句来源
     The renal proximal tubules damage due to chronic cadmium poisoning usually showed increase in urine alkaline phosphatase(U-AKP) activity, urine β2-microglobulin quantity and urine cadmium quantity. The changes in U-AKP activity were more sensitive than the latter two.
     慢性镉中毒性的肾近曲小管损害时,尿中碱性磷酸酶活性及尿β_2-微球蛋白、尿镉排泄量增加,前者要比后两项指标更为敏感。
短句来源
     Conclusion Ultrastructural changes in renal proximal tubules induced by low dose of cadmium had a dose response relationship,and were possibly reversible.
     【结论】 低剂量镉引起肾近曲小管超微结构的早期变化呈明显的剂量 效应关系 ,具有可逆性。
短句来源
     Histopathological examination showed tumefaction and degeneration in renal proximal tubules epithelia in all animals from high dose group sacrificed on week 13 and 26.ConclusionFrom these results, the safe-dose of Rhubarb total anthraquinones, was estimated to be 794 mg/kg under the present conditions, and the toxic target organ was estimated to be kidney (esp. the proximal tubules).
     高剂量组肾脏均可见近曲小管上皮细胞不同程度肿胀变性。 结论在该试验条件下 ,大黄总蒽醌对SD大鼠的毒性反应靶器官可能主要是肾脏 (特别是肾近曲小管 ) ,这种毒性反应是可逆的 ,对SD大鼠的安全剂量为 794mg/kg。
短句来源
  renal proximal convoluted tubules
     Results Cadmium ions could enter the epithelial cells of renal proximal convoluted tubules.
     结果镉离子可进入到肾近曲小管上皮细胞内。
短句来源
     DISTRIBUTION OF LEAD IN RENAL PROXIMAL CONVOLUTED TUBULES OF LEAD-POISONING RATS
     铅中毒大鼠肾近曲小管内铅的分布
短句来源
     Excessive production of oxygen free radical was found in the kidney tissues, which located mainly in the endothelial cells of blood vessels, mitochondria of epithelial cells of renal proximal convoluted tubules, microvilli and basement membrane.
     肾组织内有氧自由基的过量产生,其主要位于血管内皮细胞,肾近曲小管上皮细胞的线粒体,微绒毛及基底膜等部位。
短句来源
     The results showed that fluoride could significantly increase the contents of renal lipid peroxides and result in degeneration and necrosis of epithelia of renal proximal convoluted tubules. Fluoride could induce an increase of mitochondria and a decrease of mitochondrial cristae, locally thickening of basement membrane, markedly de- crease the activities of succinate dehydrogenase (SDH) and alkaline phosphatase (ALP), but elevate the activi- ties of lactate dehydrogenase (LDH) and acid phosphatase (ACP) in epithelia of renal proximal convoluted tubules.
     结果表明:氟可使肾脏脂质过氧化物含量明显增加,肾近曲小管上皮细胞变性、坏死,线粒体数目增多,线粒体嵴减少,基底膜局部增厚,并可使肾近曲小管上皮细胞的琥珀酸脱氢酶(SDH)和碱性磷酸酶(ALP)活性明显降低,乳酸脱氢酶(LDH)和酸性磷酸酶(ACP)活性明显增高。
短句来源
     Swelling of epithelial cells and mitochondria and drop of epithelial cells could occur in renal proximal convoluted tubules.
     肾近曲小管上皮细胞及线粒体可发生肿胀及内皮细胞脱落等病变。
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  renal proximal tubule
     Study on Mechanisms of Apoptosis Cadmium-induced in Porcine Renal Proximal Tubule Epithelial Cells (LLC-PK_1)
     镉诱导猪肾近曲小管上皮细胞(LLC-PK_1)凋亡及其机理的研究
短句来源
     Objective To explore the effect of cadmium on the apoptosis of porcine renal proximal tubule epithelial cells(LLC-PK_1)and the relationship between the apoptosis and the bcl-2,p53,c-myc,c-fos and ras gene expression.
     目的探讨镉诱导猪肾近曲小管上皮细胞(LLC-PK1)凋亡对bcl-2、p53(mtp53)、c-mycc、-fos与ras基因表达的影响。
短句来源
     Objective: To identify the effect of porcine renal proximal tubule epithelial cells LLC-PK_1 induced by cadmium in medium with or without serum.
     目的 :探讨细胞培养液中有、无小牛血清对镉诱导猪肾近曲小管上皮细胞 (L L C-PK1 )损伤的影响 ,为研究镉对 L L C- PK1 细胞毒性的方法提供依据。
短句来源
     After 24 hours of reoxygenation the levels of ATP, ADP, AXP and EC increased to 67%, 85%, 74% and 95% of control′s, respectively. Most of mitochondria in renal proximal tubule swelled with fragmentized cristae, and some appeared vacuolar degeneration.
     恢复供氧24小时后,ATP、ADP、AXP、EC含量分别上升至正常对照组的67%、85%、74%及95%,肾近曲小管线粒体肿胀,嵴模糊、断裂,基质出现部分透明区,少数线粒体空泡变性。
短句来源
     In present experiment, we used cytochemical methods, EDX microanalysis and biochemical analysis technique to evaluate the changes of calcium content in rat renal proximal tubule cells during ischemia and reperfusion period.
     本实验采用细胞化学方法结合EDX微区分析及生化测试方法,从细胞水平研究了大鼠肾近曲小管上皮细胞内钙含量在缺血-再灌流损伤过程中的改变情况,并探讨了其机理。
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  “肾近曲小管”译为未确定词的双语例句
     Methods Cultured HKC were treated by high concentration of glucose and JAK inhibitor AG490 to detect the expression of α-SMA, E-Cadherin, STAT1, STAT3, p-STAT1 and p-STAT3 by Western blot and to determine the contents of TGF-β1, fibronectin and type Ⅰ by ELISA and to measure TGF-β1 mRNA expression by RT-PCR.
     方法体外培养人肾近曲小管上皮细胞株(HKC),分别给予高糖和JAK抑制剂AG490干预,Western印迹检测α-SMA、E-Cadherin及STAT1、STAT3、p-STAT1和p-STAT3的表达; ELISA法测定上清液中TGF-β1、I型胶原的分泌,RT-PCR检测TGF-β1mRNA表达。
短句来源
     The numbers of cells undergoing apoptosis in the cadmium group 152.25±5.19/mm2) were significantly higher than those of the control group 112.50±3.54/mm2)(P<0.01).
     镉处理组的肾近曲小管的凋亡细胞数(52.25±5.19个/mm2)明显高于对照组(12.50±3.54个/mm2),两者之间差异有显著性意义(P<0.01)。
短句来源
     TNF-α regulation of C_3 gene expression and protein biosynthesis in human proximal tubular epithelial cells
     肿瘤坏死因子α诱导人肾近曲小管上皮细胞C_3mRNA表达和蛋白合成及其意义
短句来源
     Objective To study the effect of two TGF-β1 shRNA expression plasmids (pcDU6-A1-A2 and pcDU6-B1-B2) on proliferation, TGF-β1, CTGF and FN synthesis induced by human serum albumin (HSA) in HK2 cells and to explore the mechanism concerned.
     目的研究pcDU6载体质粒介导的转化生长因子β1(TGF-β1)短发夹RNA (shRNA)对人血清白蛋白(HSA)致人肾近曲小管上皮细胞(HK2细胞)增殖,TGF-β1、结缔组织生长因子(CTGF)和纤连蛋白(FN)表达的影响,并探讨有关机制。
短句来源
     Se 2mg/L can antagonism injured by Fluoride 150mg/L,Se 0. 5mg/Land 4mg/L can't obviously protect kidney.
     提示:饮水中含过量的氟明显损伤肾近曲小管,而含2mg/L剂量的硒能拮抗氟对肾近曲小管的破坏作用,剂量为0.5mg/L和4mg/L的硒对肾脏无明显保护作用.
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  renal proximal tubules
Organic anions of diverse chemical structures are secreted in renal proximal tubules.
      
Immunofluorescence data demonstrated that NHE-3 is located in the renal proximal tubules.
      
The effect of pretreatment with cadmium (Cd) complexes on the distribution and accumulation of Cd in the liver and kidneys was examined for the Cd complexes that are reabsorbed site-selectively at the renal proximal tubules of rats.
      
These increases were probably a result of damage to the renal proximal tubules.
      
Since the above-mentioned symptoms suggested dysfunction of the renal proximal tubules, the activity of urinary N-acetyl-β-D-glucosaminidase, a marker enzyme of injury to the proximal tubules, was measured.
      
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  renal proximal convoluted tubules
Positive staining was noted in the lining epithelium of the renal proximal convoluted tubules, but ultrastructural studies indicated that this was probably related to reabsorption, and not to synthesis.
      
Despite negative findings for TTR-mRNA in the gut and the kidney, endocrine cells of the gut and epithelial cells of the renal proximal convoluted tubules showed TTR-IR in some of the fetuses.
      
Lipid metabolism in the cells of the renal proximal convoluted tubules (PCT) was investigated in healthy fowls and in fowls with the Fatty Liver and Kidney Syndrome (FLKS).
      
Lysosomal glycogen storage was absent from renal proximal convoluted tubules and from the neurons examined (dorsal root ganglia, supraoptic nucleus).
      
Strong positive reactions were seen in hepatic cells, renal proximal convoluted tubules, pancreatic acinar cells, and epithelium of the small intestine.
      
更多          
  renal proximal tubule
Dopamine synthesized in non-neural tissues, eg, renal proximal tubule, functions in an autocrine or paracrine manner.
      
The term familial tumoral calcinosis encompasses a number of rare recessive diseases, often associated with increased reabsorption of phosphate through the renal proximal tubule, which manifests with periarticular or acral calcium deposition.
      
Our animal model is an excellent one for determining the effect of cadmium on renal proximal tubule damage.
      
More recent work with rabbit renal proximal tubule cells suggests that the 12,13-metabolites of linoleic acid are more toxic than the 9,10-isomers.
      
Ion exchangers mediating NaCl transport in the renal proximal tubule
      
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This paper reports the research results of pathology of derma-ulcer caused by Aeromonas hydrophila subsp. hydrophila during the overwinteringseason of Tilapia nilotica ~ The histopathological changese were characterized by the alternative inflamma-tion. In ulcerated focus, the skin was inflammatory oedema, necrosis and finally fellaway; uncleus of skeletal muscles ewelled, moved to the conter, and finally necro-sised; muscles fibres swelled and necrosised. Liver cells cloudy swelled and fatdegenerated. The epithelial...

This paper reports the research results of pathology of derma-ulcer caused by Aeromonas hydrophila subsp. hydrophila during the overwinteringseason of Tilapia nilotica ~ The histopathological changese were characterized by the alternative inflamma-tion. In ulcerated focus, the skin was inflammatory oedema, necrosis and finally fellaway; uncleus of skeletal muscles ewelled, moved to the conter, and finally necro-sised; muscles fibres swelled and necrosised. Liver cells cloudy swelled and fatdegenerated. The epithelial cells in the proximal .convoluted tubules of the kindneycloudy swelled, glassy degenerated boundary lines of cells became unclear, but thenucleus still remained. The healing of skeletal muscles in focus was mended throughthe formation of granulative tissue and regenerationt. The healing of epidermis wasmended by regeneration. The biochemi-pathological changes were characterized by the significant changesof the activities and contents of LDH, MDH and a-GPDH of white muscle of diseasedfish. Number of bands of e1ectropherotograms decreased, activities decreased ordisappeared completely. The contents of muscle protein also decreased significantly.

本文报道了尼罗罗非鱼(Tilapia nilotica)越冬期间由嗜水产气单孢菌嗜水亚种(Aeromonas hydrophila subsp.hydrophila subsp.hydrophila)引起的溃烂病的病理研究结果。 组织病理变化主要表现为变质性炎症。病性部位皮肤炎性水肿、坏死、脱落。骨胳脱脱核肿大、移行、乃至坏死;肌纤维肿胀及坏死。肝细胞颗粒变性、脂肪变性。肾近曲小管上皮细胞颗粒变性,胞浆内充满玻璃样小滴,使细胞界限不清,但胞核仍存在。病灶部位骨胳肌的愈合,是通过形成肉芽组织及再生进行修补,而表皮的愈合则是借助再生予以修补。与健康鱼相比,病鱼的生化病理变化主要表现为白肌的乳酸脱氢酶(LDH)、苹果酸脱氢酶(MDH)、甘油-3-磷酸脱氢酶(α-GPDH)等同工酶的活性与含量均有显著变化,谱带减少,活性减弱或消失。肌蛋白量显著减少。

Investigation of lead-induced renal changes has been done using rats as the experimental animals. The 55 Wistar male rats were divided into 4 groups. Group Ⅰ was given dosage of 10mg/kg lead acetate via ip per week for 3 weeks and sacrificed immediately after treatment. Groups Ⅱ and Ⅲ were put on a diet contaning 0.5% and 2% lead acetate respectively. Group Ⅳ was designed as control. Animals from Groups Ⅱ and Ⅲ were killed 2 weeks,1 month and 3 months after treatment and their kidneys were samples for pathological...

Investigation of lead-induced renal changes has been done using rats as the experimental animals. The 55 Wistar male rats were divided into 4 groups. Group Ⅰ was given dosage of 10mg/kg lead acetate via ip per week for 3 weeks and sacrificed immediately after treatment. Groups Ⅱ and Ⅲ were put on a diet contaning 0.5% and 2% lead acetate respectively. Group Ⅳ was designed as control. Animals from Groups Ⅱ and Ⅲ were killed 2 weeks,1 month and 3 months after treatment and their kidneys were samples for pathological examination.Under light microscope, the lesions were characterzed by foci of degeneration and necrosis of the proximal tubules. Some tubules were abnormally dilated and lined by flattened epithelium after 3 months. Numerous lead-conataining crystals were found kidney tubules of the treated rats. A few focal areas with early iaterstitial fibrosis adjacent to altered tubular profiles were preseut. lutranuclear inclusion bodies could be observed on rats that were killed 1 month and a months after treated with lead. The number and size of intranuclear inclusion bodies got incteased in the rats killed 3 months after the treatment.Extensive ultrastructural changes were revealed by electronic microscope. Major early damages to epitheliunl of proximal tubules were swelling of mitochondria and accumulation of lysosomes. Besides typical intranuclear lead includion bodies, pseudonuclear inclusion bodies were also noticed in some epithelium. The leadinduced pathological changes in the kidney of rats can be provided as an animal model for future studies of plumbism

雄性Wistar大鼠55只分为4组,观察实验性铅中毒肾脏的病变,Ⅰ组10只,以2%醋酸铅腹腔注射,染毒3周后处死;Ⅱ和Ⅲ组各15只,分别以0.5%和2%醋酸铅饲料口服3个月;Ⅳ组15只为对照组。后3组均于实验后2周,1月及3月各处死5只。光镜检查:大鼠肾近曲管上皮细胞核内见伊红色边界清楚的核内包函体、口服3月组大鼠肾可见近曲小管上皮萎缩、灶性纤维组织曾生;集合管及远曲管腔内有较多灰黄色无定形结晶状沉淀物。电镜观察:肾近曲小管上皮细胞核内可见包函体、呈圆形或椭圆形,有致密浓染的核心,周围为纤维丝状结构;近曲小管上皮细胞中线粒体肿胀、溶酶体增多。此外,尚可见髓样小体,有的镶嵌于核膜的皱折内,形成假包函体。近曲小管上皮细胞浆及管腔内尚可见电子密度深的无定形物质。作者认为铅在大鼠中所引起的肾脏病理改变,早期为曲管上皮细胞浆内细胞器的变性及核内包函体形成。核内包函体数量随染毒时间延长而增多。肾单位萎缩、纤维化和集合管及远近曲管腔内无定形结晶均为后期病变。

SM and inosine have a similarly protective effect on the epithelial cells of the proximal convoluted tubules, demonstrated in this study that the microvilli and the mitochondria were less damaged than those of control rabbits after 60 and 120 rain of warm ischemia. However, the ischemie injury of the endothelial cells lining the intertubular capillarries of inosine-proteeted kianey were much evident than those of SM-protected group. In both inosine and SM groups the microvilli and mitochondria recovered fundamentally...

SM and inosine have a similarly protective effect on the epithelial cells of the proximal convoluted tubules, demonstrated in this study that the microvilli and the mitochondria were less damaged than those of control rabbits after 60 and 120 rain of warm ischemia. However, the ischemie injury of the endothelial cells lining the intertubular capillarries of inosine-proteeted kianey were much evident than those of SM-protected group. In both inosine and SM groups the microvilli and mitochondria recovered fundamentally after 30 rain of blood fellow, while these structures cannot return in the control group. The results of this study indicate that one of the chief effects of SM may be the suppling of energy for the repair of localised damage during warm ischemia.

丹参和肌苷对实验兔肾近曲小管上皮细胞具有相似的保护作用。在热缺血60min和120min后,该处微绒毛和线粒体的缺血性损害比对照组兔为轻。但用肌苷保护的肾小管间毛细血管内皮细胞的缺血性损害比用丹参保护的要重。缺血肾恢复血流30min后,丹参与肌苷两组的微绒毛和线粒体均基本恢复正常,而对照组的这些结构未能恢复正常。丹参的主要作用之一,可能与提供维持近曲小管细胞的微绒毛有规则地排列,以及修复热缺血期间引起的局部损害所需的能量有关。

 
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