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细胞反应性
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  cell responses
     Mesenchymal Stem Cells Suppress Allogeneic T Cell Responses by Secretion of TGF-β1
     间充质干细胞分泌TGF-β1抑制异体T细胞反应性
短句来源
  “细胞反应性”译为未确定词的双语例句
     The inhibitory effect of hyporesponsive peptide on human leukocyte antigen-DRβ1 specific T cell activation
     人类白细胞抗原-DRβ1特异性低T细胞反应性肽对T细胞激活的抑制作用
短句来源
     The number of DC and the expression levels of HLA-DR and CD80,CD86 on DC were detected by CMRF-44 mAb labeling and flow cytometer.
     通过流式细胞仪检测各组的DC数量 (CMRF 44标记法 )及DC表面HLA DR、CD80、CD86表达水平以及DC诱导的T细胞反应性增殖。
短句来源
     Objective:To investigate the distribution and cytotoxicity of T cell receptor V domain β chain (TCR Vβ) subfamily T cells in patients with M2a subtype of acute myelogenous leukemia (AMLM2a).
     目的 :研究急性髓细胞性白血病 M2 a亚型 (AML M2 a)患者外周血白血病细胞反应性 T细胞受体V区 β链 (TCR Vβ)亚家族 T细胞体内外优势表达情况及其体外细胞毒性分析。
短句来源
     The elicitors from V. dahliae kleb which were treated by low concentration Salicylic acid(SA) (1mmol/L) and H 2O 2 (0.2mmol/L)can induce higher oxidative burst comparing with the control.
     使用显著低于杀菌浓度的剂量的水杨酸 (SA)和H2 O2 作用上述大丽轮枝菌 ,发现水杨酸和H2 O2 都能影响微生物 ,经 1mmol/L水杨酸和 0 2mmol/LH2 O2 作用后的微生物 ,其所产生的诱导物对植物细胞反应性氧的诱导作用要高于未被作用的微生物的诱导物。
短句来源
     The protective effect of VEGF was inhibited in part by SU1498. CONCLUSION:Hypoxia could induce reactive activity of the Müller cells and the expressions of GFAP,VEGF,Flk-1 and Flt-1. The exogenous VEGF may exert neuroprotective effects partially on the Müller cells via VEGF receptor-Flk-1 and Flt-1 in vitro.
     加入SU1498后能部分抑制VEGF对Müller细胞的作用。 结论:短时间缺氧使Müller细胞反应性增生,VEGF和Flk-1,Flt-1表达增加,外源性VEGF可能部分地借助于受体Flk-1和Flt-1对缺氧的Müller细胞起到保护作用。
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  相似匹配句对
     Autoreactive T Lymphocytes and Alopecia Areata
     自身反应性T细胞与斑秃
短句来源
     T cell lymphoma with distinct reactive lymphoid follicles
     具有显著反应性滤泡的T细胞淋巴瘤
短句来源
     Cellular Spectro-Analysis
     细胞光谱分析
短句来源
     The phenotype of those cells was NKH1+ CD16-.
     _-的细胞
短句来源
     On the Stability and Reactivity of the Carbocations
     碳正离子的稳定性和反应性
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  cell responses
The level and duration of the humoral and cell responses were assayed.
      
Thus, DNA immunization with pDNAgD induced both B- and T-cell responses and completely protected mice from a lethal doze of HSV1.
      
The quantitative changes in the major organelles, chloroplasts and mitochondria, were followed in order to evaluate plant cell responses to prolonged atmospheric pollution.
      
The regulation of cell responses to Al stress may involve extensin expression.
      
The comparison with other heavy metals is used to disclose the general mechanisms that disturb plant mineral nutrition, water regime, photosynthesis, and morphogenesis as well as the common cell responses aimed at detoxification of heavy metals.
      
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The normal level of serum glucose and insulin of mice are showed 110.0(60.4-177.5)mg/100 ml and 8.67 (0.88-22.50)μu/ml. The relatio- nship of glucose and insulin in normal and alloxon-diabetic mice is linear chiefly

本文观察了小白鼠血清萄葡糖和胰岛素的正常值为110.0(60.4-177.5)mg/100m和8.67(0.88-22.50)μu/ml,发现了两者在正常和四氧嘧啶糖尿病时的相互关系是以线性成分为主,并且糖尿病时β细胞反应性较正常时低(回归系数较小)。

The immnnoreactivity of Alzheimer type I (AI) cells were strongly positive and type II (AII) cells negative to GFAP stain in Marchiafava-Bignami disease. The- cytogenesis of AI and A II cells in non-hepatic and non-hyperarnmonemic encophalopathy was discussed. Observations suggest: (1) a close interrelationship between the development of AI abnormality -and reactive astrogliosis, (2) the increase in the abiliiy of mitotic division of early A II cells in response to injury.

本文报道原发性胼胝体坏死灶中出现ALzhEimer Ⅰ型(AⅠ)和2型(AⅡ)细胞的胶质纤维酸性蛋白(GFAP)免疫组织化学染色反应。AⅠ细胞呈强阳性染色,AⅡ细胞呈阴性染色。讨论了非高血氨性脑病中AⅠ和AⅡ细胞的形成机理。结果提示AⅠ细胞的发生与星形胶质细胞反应性增生的密切关系,以及AⅡ细胞在其形成的早期比正常的星形胶质细胞有更高的分裂和增殖能力。

The normal levels of serum glucose and insulin of mice fasted for 7-9hours were 6.11 (3.36-9.86) mmol/L and 8.67 (0.88-22.50) μu/ml respectively. The insulin release response was an increase of insulin from 9.81 to 21.58 μu/ml in normal mice (p<0.001) 30-60 minutes after meals, but absent in alloxan-diabe-tic mice (p>0.05).The relationship between blood glucose and insulin in normal and alloxan-diabetic mice was linear chiefly, if any. The regression coefficients were: 1.098 (p<0.05. compared with 0.252)in normal...

The normal levels of serum glucose and insulin of mice fasted for 7-9hours were 6.11 (3.36-9.86) mmol/L and 8.67 (0.88-22.50) μu/ml respectively. The insulin release response was an increase of insulin from 9.81 to 21.58 μu/ml in normal mice (p<0.001) 30-60 minutes after meals, but absent in alloxan-diabe-tic mice (p>0.05).The relationship between blood glucose and insulin in normal and alloxan-diabetic mice was linear chiefly, if any. The regression coefficients were: 1.098 (p<0.05. compared with 0.252)in normal fasted mice; 2.160 in normal fasted mice mixed with normal feeded mice (30-60 min after meal). 0.252 or 0.378(P>0.05, compared with 0.252) in fasted mice into which alloxan was injected 4 or 9 days ago. The response of the p cell of pancreas to the serum glucose increase was less in alloxan-diabetic mice than that of the normal. Responses in both groups of alloxan diabetic mice (alloxan injected 4 or 9 days ago) are about the same.Because the mouse blood may be taken only once, the delay of the glucose-insulin feedback system can not be shown here.

本文观察了小鼠血清葡萄糖和胰岛素的正常值分别为6.11(3.36~9.86)mmol/L和8.67(0.88~22.50)μu/ml,发现两者在正常和四氧嘧啶所致糖尿病时的相互关系均为线性,并且患糖尿病时β细胞反应性较正常为低(回归系数较小)。

 
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