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酯酶基因
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  lipase gene
    Relationship between lipoprotein lipase gene Hind Ⅲ polymorphism and subclasses of serum HDL in type Ⅳ hypertriglyceridemia
    Ⅳ型高脂血症患者高密度脂蛋白亚类组成与脂蛋白酯酶基因多态性的关系
短句来源
    Relationship between High Density Lipoprotein Subclasses Distribution and Lipoprotein Lipase Gene HindⅢ Polymorphism in Obese Subjects
    肥胖者高密度脂蛋白亚类组成与脂蛋白酯酶基因多态性的关系
短句来源
    AIM: To investigate lipoprotein lipase gene HindⅢ polymorphism and its relationship with serum lipids and apolipoprotein, serum HDL subclasses in patients with hyperlipoidemia.
    目的:探讨高脂血症患者脂蛋白酯酶基因内含子8HindⅢ酶切位点多态性与HDL亚类组成的变化关系。
短句来源
    METHODS: Lipoprotein lipase gene HindⅢ polymorphism was assayed by polymerase chain reaction-restriction fragment length polymorphism (PCR-RFLP).
    方法:采用聚合酶链反应-限制性片段长度多态性和双向电泳-免疫印迹检测法,分析152例高脂血症患者和128例血脂正常者的脂蛋白酯酶基因内含子8HindⅢ多态性、HDL亚类组成及相对百分含量。
短句来源
    Method:Lipoprotein lipase gene HindⅢ polymorphism was assayed by polymerase chain reaction-restriction fragment length polymorphism (PCR-RFLP), the subclasses of serum HDL were determined by two-dimensional gel electrophoresis conjunction with immunodetection method in 104 type Ⅳ hypertriglyceridemic patients and 107 healthy subjects.
    方法:采用聚合酶链反应限制性片段长度多态性和双向电泳免疫印迹检测法,分析104例ⅣHL患者和107例血脂正常者的脂蛋白酯酶基因内含子8HindⅢ酶切位点多态性、HDL亚类组成及相对百分含量。
短句来源
  “酯酶基因”译为未确定词的双语例句
    The association of paraoxonase 2 gene 311Cys/Ser polymorphism and type 2 diabetes mellitus complicated by coronary heart disease
    Ⅱ型对氧磷酯酶基因311Cys/Ser多态性与2型糖尿病合并冠心病关系的研究
短句来源
    Relationship between paraoxonase 2 polymorphism and diabetic retinopathy
    对氧磷酯酶基因多态性与糖尿病视网膜病变的关系(附236例分析)
短句来源
    Relationship between subclasses of serum HDL and LPL gene HindⅢ polymorphism in hyperlipidemia
    高脂血症患者HDL亚类组成与脂蛋白酯酶基因内含子8 HindⅢ多态性的关系
短句来源
    Relationship between Paraoxonase Gene Polymorphism and Type 2 Diabetes Mellitus Accompanying Hypertension
    对氧磷酯酶基因多态性与2型糖尿病合并高血压的关系
短句来源
    Conclusion:The Hind Ⅲ polymorphism at intron 8 of LPL gene is associated with subclasses of HDL and mature metabolism of HDL in type Ⅳ hypertriglyceridemic subjects.
    结论:ⅣHL患者脂蛋白酯酶基因多态性与HDL亚类的组成及成熟代谢相关。
短句来源
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  lipase gene
Association of the HindIII Polymorphism of the Lipoprotein Lipase Gene with Myocardial Infarction
      
Association of the HindIII Polymorphism of the Lipoprotein Lipase Gene with Myocardial Infarction and the Life Span in Elderly P
      
Isolation and characterization of a homologous to lipase gene from Brassica napus
      
To analyze its function and roles during seed germination and growth, a full-length cDNA encoding a homologous to lipase gene named BnLIP1 was cloned from Brassica napus, cv.
      
Common mutations of the lipoprotein lipase gene and their clinical significance
      
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AIM: To investigate lipoprotein lipase gene HindⅢ polymorphism and its relationship with serum lipids and apolipoprotein, serum HDL subclasses in patients with hyperlipoidemia. METHODS: Lipoprotein lipase gene HindⅢ polymorphism was assayed by polymerase chain reaction-restriction fragment length polymorphism (PCR-RFLP). The subclasses of serum HDL in 152 hyperlipoidemia patients and 128 healthy subjects were determined by two-dimensional gel electrophoresis conjunction with immunodetection method. RESULTS:...

AIM: To investigate lipoprotein lipase gene HindⅢ polymorphism and its relationship with serum lipids and apolipoprotein, serum HDL subclasses in patients with hyperlipoidemia. METHODS: Lipoprotein lipase gene HindⅢ polymorphism was assayed by polymerase chain reaction-restriction fragment length polymorphism (PCR-RFLP). The subclasses of serum HDL in 152 hyperlipoidemia patients and 128 healthy subjects were determined by two-dimensional gel electrophoresis conjunction with immunodetection method. RESULTS: H+H+ genotype and allele H+ in hyperlipoidemia and control groups were both the highest. In hyperlipidemia group, H+H+ genotypes tended to be higher than that in control group, while H+H- and H-H- genotypes were significantly lower (P<0.05). In hyperlipidemia group allele H+ carriers' frequency tended to be higher than that in control group (P<0.05). In hyperlipoidemia group, the genotype of H+H+ showed higher serum TG, apoB100 levels, TG/HDL-C ratio, preβ_1-HDL, HDL_(3b) and lower HDL_(2a), HDL_(2b) compared with H-H- (P<0.05). In control group, the genotype of H+H+ had higher serum TG,HDL_(3c) and lower HDL_(2a) compared with H-H- (P<0.05). CONCLUSION: The HindⅢ polymorphism at intron 8 of LPL gene is associated with the general shift toward smaller size of HDL particle size in hyperlipoidemia, and the change of HDL subclasses distribution profile may be closely related to the pathogenesis of atherosclerosis in Chinese patients with hyperlipoidemia.

目的:探讨高脂血症患者脂蛋白酯酶基因内含子8HindⅢ酶切位点多态性与HDL亚类组成的变化关系。方法:采用聚合酶链反应-限制性片段长度多态性和双向电泳-免疫印迹检测法,分析152例高脂血症患者和128例血脂正常者的脂蛋白酯酶基因内含子8HindⅢ多态性、HDL亚类组成及相对百分含量。结果:高脂血症组和对照组均以H+H+基因型为主,但高脂血症组H+H+基因型频率显著高于对照组,而H+H-和H-H-基因型频率显著低于对照组(P<0.05)。高脂血症组H+等位基因频率显著高于对照组,而H-等位基因频率显著低于对照组(P<0.05)。高脂血症组和正常对照组H+H+基因型者血清TG明显高于H-H-者;高脂血症组H+H+基因型者apoB100、TG/HDL-C比值明显高于H-H-者(P<0.05)。高脂血症患者H+H+基因型小颗粒的preβ1-HDL、HDL3b相对含量多于H-H-者,大颗粒的HDL2a、HDL2b相对含量少于H-H-者;正常对照组H+H+基因型HDL3c多于H-H-者,而HDL2a少于H-H-者,其差异均显著(P<0.05)。结论:中国人高脂血症患者脂蛋白酯酶基因Hi...

目的:探讨高脂血症患者脂蛋白酯酶基因内含子8HindⅢ酶切位点多态性与HDL亚类组成的变化关系。方法:采用聚合酶链反应-限制性片段长度多态性和双向电泳-免疫印迹检测法,分析152例高脂血症患者和128例血脂正常者的脂蛋白酯酶基因内含子8HindⅢ多态性、HDL亚类组成及相对百分含量。结果:高脂血症组和对照组均以H+H+基因型为主,但高脂血症组H+H+基因型频率显著高于对照组,而H+H-和H-H-基因型频率显著低于对照组(P<0.05)。高脂血症组H+等位基因频率显著高于对照组,而H-等位基因频率显著低于对照组(P<0.05)。高脂血症组和正常对照组H+H+基因型者血清TG明显高于H-H-者;高脂血症组H+H+基因型者apoB100、TG/HDL-C比值明显高于H-H-者(P<0.05)。高脂血症患者H+H+基因型小颗粒的preβ1-HDL、HDL3b相对含量多于H-H-者,大颗粒的HDL2a、HDL2b相对含量少于H-H-者;正常对照组H+H+基因型HDL3c多于H-H-者,而HDL2a少于H-H-者,其差异均显著(P<0.05)。结论:中国人高脂血症患者脂蛋白酯酶基因HindⅢ酶切位点多态性与HDL亚类的组成相关。

Objective:The present study explored the association between lipoprotein lipase (LPL) gene Hind Ⅲ polymorphism and serum high density lipoprotein (HDL) subclasses in patients with type Ⅳ hypertriglyceridemia. Method:Lipoprotein lipase gene HindⅢ polymorphism was assayed by polymerase chain reaction-restriction fragment length polymorphism (PCR-RFLP), the subclasses of serum HDL were determined by two-dimensional gel electrophoresis conjunction with immunodetection method in 104 type Ⅳ hypertriglyceridemic patients...

Objective:The present study explored the association between lipoprotein lipase (LPL) gene Hind Ⅲ polymorphism and serum high density lipoprotein (HDL) subclasses in patients with type Ⅳ hypertriglyceridemia. Method:Lipoprotein lipase gene HindⅢ polymorphism was assayed by polymerase chain reaction-restriction fragment length polymorphism (PCR-RFLP), the subclasses of serum HDL were determined by two-dimensional gel electrophoresis conjunction with immunodetection method in 104 type Ⅳ hypertriglyceridemic patients and 107 healthy subjects. Result:Both in type Ⅳ hypertriglyceridemic group and control group, the H+H+ homozygote was the major alleltype.The H+ allelic frequency of type Ⅳ hypertriglyceridemic group was higher than that of control group,while the H- allelic frequency was significantly lower (P<0.05). In type Ⅳ hypertriglyceridemic group and control group, the plasma triglyceride (TG) levels and TG/HDL-C ratio of H+H+ genotype were significantly higher than H-H- genotype (P<0.05). In type Ⅳ hypertriglyceridemic group, compared with the H-H- genotype the H+H+ genotype had higher serum preβ1-HDL, HDL3a and lower serum HDL2a , HDL2b (P<0.05). Conclusion:The Hind Ⅲ polymorphism at intron 8 of LPL gene is associated with subclasses of HDL and mature metabolism of HDL in type Ⅳ hypertriglyceridemic subjects.

目的:探讨Ⅳ型高脂血症(ⅣHL)患者高密度脂蛋白(HDL)亚类组成与脂蛋白酯酶基因多态性的关系。方法:采用聚合酶链反应限制性片段长度多态性和双向电泳免疫印迹检测法,分析104例ⅣHL患者和107例血脂正常者的脂蛋白酯酶基因内含子8HindⅢ酶切位点多态性、HDL亚类组成及相对百分含量。结果:ⅣHL组和对照组均以H+H+纯合子基因型为主,ⅣHL患者H+等位基因频率较对照组增加,而H-H-等位基因频率显著降低(P<0.05)。ⅣHL组和对照组H+H+基因型者与H-H-者比较,血浆TG、TG/HDLC比值明显升高,Ⅳ型HL组H+H+基因型者与H-H-者比较,HDL小颗粒的preβ1HDL、HDL3a相对含量增多,大颗粒的HDL2a、HDL2b相对含量减少,其差异有统计学意义(P<0.05)。结论:ⅣHL患者脂蛋白酯酶基因多态性与HDL亚类的组成及成熟代谢相关。

Aim To elucidate the relationship between lipoprotein lipase(LPL) gene HindⅢ polymorphism and plasma lipid profiles and HDL subclasses in obesity. Methods LPL gene HindⅢ polymorphism was assayed by polymerase chain reaction-restriction fragment length polymorphism(PCR-RFLP).The subclasses of serum HDL in 95 obese subjects and 144 nonobese subjects were determined by two-dimensional gel electrophoresis conjunction with immunodetection method. Results Both in obese group and control group,the H~+H~+ homozygote...

Aim To elucidate the relationship between lipoprotein lipase(LPL) gene HindⅢ polymorphism and plasma lipid profiles and HDL subclasses in obesity. Methods LPL gene HindⅢ polymorphism was assayed by polymerase chain reaction-restriction fragment length polymorphism(PCR-RFLP).The subclasses of serum HDL in 95 obese subjects and 144 nonobese subjects were determined by two-dimensional gel electrophoresis conjunction with immunodetection method. Results Both in obese group and control group,the H~+H~+ homozygote and H~+ allele were the major allelotype.There was no statistically significant difference in frequencies of genotypes or alleles between two groups.Obese group had higher plasma triglycerides,apoB100,apoCⅡ,apoCⅢ,apoE,preβ1-HDL levels and TG/HDLC,but lower HDLC,apoAI,HDL2b,apoE/CⅢ compared with control group(P<0.05).In obese group,HindⅢ H~+H~+ had higher plasma triglycerides than H~-H~-genotype.Compared with H~-H~-genotype,the subjects with H~+H~+ and H~+H~-genotypes had higher apoB100,preβ1-HDL,HDL3b levels but lower apoE/CⅢI,HDL2a,HDL2b.In control group,the genotypes of H~+H~+ and H~+H~-showed higher apoCⅡ,apoCⅢ,HDL3b levels,but lower apoE/CⅢ,HDL2a,HDL2b levels compared with the genotype of H~-H~-(P<0.05). Conclusions H~+H~+ genotype was associated with higher plasma TG levels of obesity.LPL gene polymorphism was associated with plasma levels as well as associated with changes of HDL subclasses distribution in Chinese population.And the particle size of HDL tended to be smaller in H~+ allele carrier' subjects,furthermore,the tendency was more obvious in obese group,which indicated that HDL maturation might be abnormal in subjects with H~+ allele.

目的探讨肥胖者高密度脂蛋白亚类组成与脂蛋白酯酶基因内含子8 HindⅢ酶切位点多态性的关系。方法采用聚合酶链反应限制性片长多态性和双向电泳免疫印迹检测法,分析95例肥胖者和144例体质指数正常者的脂蛋白酯酶基因内含子8 HindⅢ多态性、高密度脂蛋白亚类组成及相对百分含量。结果肥胖组和对照组均以H+H+基因型为主。肥胖组血浆甘油三酯、载脂蛋白B100、CⅡ、CⅢ、E、前β1高密度脂蛋白水平及甘油三酯/高密度脂蛋白胆固醇较对照组增加,而高密度脂蛋白胆固醇、载脂蛋白AI、高密度脂蛋白2b水平及载脂蛋白E/CⅢ明显降低(P<0.05)。肥胖组H+H+基因型者与H-H-者比较,血清甘油三酯、载脂蛋白B100、前β1-高密度脂蛋白、高密度脂蛋白3b含量显著升高,而载脂蛋白E/载脂蛋白CⅢ、高密度脂蛋白2a和高密度脂蛋白2b显著降低;肥胖组H+H-基因型者与H-H-者比较,载脂蛋白B100、前β1高密度脂蛋白和高密度脂蛋白3b含量显著升高,而载脂蛋白E/CⅢ和高密度脂蛋白2a显著降低(P<0.05)。对照组H+H+及H+H-基因型者血清载脂蛋白CⅡ、载脂蛋白CⅢ和高密度脂蛋白3a含量均较H-H-者...

目的探讨肥胖者高密度脂蛋白亚类组成与脂蛋白酯酶基因内含子8 HindⅢ酶切位点多态性的关系。方法采用聚合酶链反应限制性片长多态性和双向电泳免疫印迹检测法,分析95例肥胖者和144例体质指数正常者的脂蛋白酯酶基因内含子8 HindⅢ多态性、高密度脂蛋白亚类组成及相对百分含量。结果肥胖组和对照组均以H+H+基因型为主。肥胖组血浆甘油三酯、载脂蛋白B100、CⅡ、CⅢ、E、前β1高密度脂蛋白水平及甘油三酯/高密度脂蛋白胆固醇较对照组增加,而高密度脂蛋白胆固醇、载脂蛋白AI、高密度脂蛋白2b水平及载脂蛋白E/CⅢ明显降低(P<0.05)。肥胖组H+H+基因型者与H-H-者比较,血清甘油三酯、载脂蛋白B100、前β1-高密度脂蛋白、高密度脂蛋白3b含量显著升高,而载脂蛋白E/载脂蛋白CⅢ、高密度脂蛋白2a和高密度脂蛋白2b显著降低;肥胖组H+H-基因型者与H-H-者比较,载脂蛋白B100、前β1高密度脂蛋白和高密度脂蛋白3b含量显著升高,而载脂蛋白E/CⅢ和高密度脂蛋白2a显著降低(P<0.05)。对照组H+H+及H+H-基因型者血清载脂蛋白CⅡ、载脂蛋白CⅢ和高密度脂蛋白3a含量均较H-H-者显著升高,而载脂蛋白E/载脂蛋白CⅢ、高密度脂蛋白2a、高密度脂蛋白2b显著降低(P<0.05)。结论脂蛋白酯酶基因H+H+基因型与肥胖者增高的甘油三酯水平有关。中国人脂蛋白酯酶基因HindⅢ酶切位点多态性与高密度脂蛋白亚类的组成和分布相关,H+等位基因携带者高密度脂蛋白颗粒有变小的趋势,且肥胖者更为明显,表明H+等位基因携带者高密度脂蛋白成熟代谢障碍。

 
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