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混合型高脂血症
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  combined hyperlipidemia
     Linkage of familial combined hyperlipidemia to chromosome 1q21-23 in Chinese and German families
     家族性混合型高脂血症与人类染色体1q21-23连锁
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     Detection of lipoprotein lipase S447X polymorphisms in combined hyperlipidemia
     混合型高脂血症患者脂蛋白脂酶基因S447X的多态性
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     Linkage analysis between familial combined hyperlipidemia and lipoprotein lipase gene in Chinese families
     家族性混合型高脂血症与脂蛋白脂酶基因的连锁分析
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     Objective To study the interaction between hepatic lipase gene-250G/A polymorphism and combined hyperlipidemia.
     目的:探讨混合型高脂血症与肝脂酶基因启动子250G/A多态性的关系。
短句来源
     Methods\ DNA polymorphisms of LPL were studied by using polymerase chain reaction (PCR) in 156 combined hyperlipidemia patients from a population of Chinese Han nationality in Guangdong area. The loci studied included -93T→G, Asp9→Asn and Asn291→Ser of LPL.
     方法 应用多聚酶链反应 (PCR)对广东地区 15 6例混合型高脂血症患者脂蛋白脂酶基因的多态性进行了研究 ,检测的LPL基因突变位点有 :-93T→G、Asp9→Asn及Asn2 91→Ser突变。
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  mixed hyperlipemia
     Conclusion Acipimox combined with Fluvastatin can significantly lower TC,LDL-C,TG and raise HDL-C in patients with mixed hyperlipemia,the adverse reaction of Acipimox and Fluvastatin are mild and can be well tolerated.
     3组患者TC、LDL-C和TG分别下降31%、37%和40%,HDL-C升高,联合用药组降低TC、LDL-C、TG,升高HDL-C的疗效优于单药治疗。 结论阿昔莫司和氟伐他汀联用可全面调节血脂,有效治疗混合型高脂血症
短句来源
     Effect of Atorvastatin on Endothelium-Dependent Diastolic Function of Patients with Mixed Hyperlipemia
     阿托伐他汀对混合型高脂血症患者血管内皮依赖性舒张功能的影响
短句来源
     Curative Effects and Safety Study of Acipimox Combined with Fluvastatin Treating the Aged with Mixed Hyperlipemia
     阿昔莫司联合氟伐他汀治疗老年混合型高脂血症的疗效和安全性研究
短句来源
     Objective To observe the effect of short term treatment with atorvastatin on brachial endothelium dependent diastolic function of patients with mixed hyperlipemia.
     目的 观察阿托伐他汀对混合型高脂血症患者血管内皮依赖性舒张功能的影响。
短句来源
     Method 60 patients with mixed hyperlipemia were treated with atorvastatin(10mg/d;po) for 8 weeks. 60 healthy adults were as control group. Blood lipids were measured,and the baseline of brachial angiobore,blood flow and the changes of these at response to nitroglycerin and reactive hyperemia were measured by Dopple ultrasound before and after treatment.
     方法  60例混合型高脂血症患者进行 8周阿托伐他汀片 10mg/d ,口服治疗 ,60例健康人群为对照组 ,治疗前后测定血脂以及用超声多普勒测定肱动脉血管内径、血流量以及反应性充血和含服硝酸甘油片后肱动脉内径和血流量变化。
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  mixed hyperlipaemia
     ObjectiveTo observe the influence of lipid - regulating treatment on the brachial endothelium - dependent diastolic function of patients with mixed hyperlipaemia, and the variation of the content of Endothelin within the plasma, and to clarifythe effect of Atorvastatin on the function of vascular endothecium.
     混合型高脂血症患者的内皮依赖性舒张功能明显受损(P<0.01),而对硝酸甘油反应两组无差异(P>0.05),血浆内皮素明显增高(P<0.01)。
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  “混合型高脂血症”译为未确定词的双语例句
     mixed type 27/400,6.75%.
     混合型高脂血症占6·75%(27/400)。
短句来源
     mixed type 6.00%.
     混合型高脂血症占6.00%。
短句来源
     Conclusions Atorvastatin 10 mg/d can significantly lower TC,LDL C,TG and (TC HDL C)/HDL C in combined dyslipidemia.
     结论 阿托伐他汀 10mg d治疗混合型高脂血症可以明显降低TC、LDL C ,TG和 (TC HDL C) HDL C ;
短句来源
     Interaction between hepatic fipase gene promoter 250G/A polymorphism and combined hyperfipidemia
     混合型高脂血症与肝脂酶基因启动子250G/A多态性的关系
短句来源
     10.5% cases(42/400) had decreased high density lipoprotein cholesterol(HDL-C) and 6.75% cases(27/255) had high lipid of mixed types.
     低HDL-C血症占10.5%(42/400); 混合型高脂血症占6.75%(27/255)。
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  combined hyperlipidemia
They all have acquired combined hyperlipidemia and increased Lp(a), hyperhomocysteinemia, decreased physical activity, pychosocial stress, insulin resistance, procoagulant factors, left ventricular hypertrophy, and increased oxidative stress.
      
Gene therapy could also be used to increase expression of certain proteins, such as apolipoprotein A-I as a strategy to raise HDL cholesterol levels or apoE as a strategy for severe combined hyperlipidemia.
      
Familial combined hyperlipidemia (FCHL) is characterized by elevated levels of serum total cholesterol, triglycerides, or both.
      
A moderate-fat diet for combined hyperlipidemia and metabolic syndrome
      
However, low-density lipoprotein (LDL) responses depend on the type of hyperlipidemia (ie, simple hypercholesterolemia or combined hyperlipidemia).
      
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The author has made an investigation and statistics of the serum cholesterol and triglyceride of 595 persons from the middle and old aged teachers, clerks and workers in Nanchong Teachers' College. It has been found that 31 of them have high cholesterol in blood, making up 5.2% of the total, 104 of them have high triglyceride in blood, making up 17.5% of the total, and 30 of them have mixture type of high lipemia, amounting to 5% of the total. All the diseases mentioned above increase with ageing, and the percentage...

The author has made an investigation and statistics of the serum cholesterol and triglyceride of 595 persons from the middle and old aged teachers, clerks and workers in Nanchong Teachers' College. It has been found that 31 of them have high cholesterol in blood, making up 5.2% of the total, 104 of them have high triglyceride in blood, making up 17.5% of the total, and 30 of them have mixture type of high lipemia, amounting to 5% of the total. All the diseases mentioned above increase with ageing, and the percentage of female's lipemia is higher than that of male's.

本文对595名40岁以上知识分子的血清胆固醇和甘油三酯进行了调查统计,其中31人为高胆固醇血症,占5.2%,104人为高甘油三酯血症,占17.5%,30人为混合型高脂血症,占5%。高脂血症的发病率有随年龄增高而增高趋势,且女性高于男性。

Familial Combined Hyperlipidemia(FCHL)was first described as a new genetic disorder in 1973 by Goldstein and Rose.Among survivors premature myocardial infarction (below the age of 60 yr),FCHL was the most common familial lipid disorder(11~15%).FCHL may be present in 0.5~2% of the population.Apparently,FCHL is clustered in families and pedigree studies have suggested that it is inhertited as an autosomal dominant disorder.This article reviews the general character and pathogenesis of FCHL and new advances of...

Familial Combined Hyperlipidemia(FCHL)was first described as a new genetic disorder in 1973 by Goldstein and Rose.Among survivors premature myocardial infarction (below the age of 60 yr),FCHL was the most common familial lipid disorder(11~15%).FCHL may be present in 0.5~2% of the population.Apparently,FCHL is clustered in families and pedigree studies have suggested that it is inhertited as an autosomal dominant disorder.This article reviews the general character and pathogenesis of FCHL and new advances of transgenic mice in FCHL

家族性混合型高脂血症的发病机理及转基因动物研究进展(中国医学科学院中国协和医科大学阜外心血管病医院心血管病研究所,北京100037)裴卫东黄尚志*惠汝太*AbstractFamilialCombinedHyperlipidemia(FCHL)wasf...

Aim To determine the contribution of body mass index(BMI) and family genetic factors(FGFs) to the development of hyperlipidemia and the interaction of these two factors. Methods 25 pedigrees with familial hyperlipidemia[familial combined hyperlipidemia(FCHL) and familial hypercholesterolemia(FH)] and control families were investigated and analysed in Beijing area during 1996 ̄1997. Results There are significantly higher levels of total cholesterol(TC),triglycide(TG),low density lipoprotein cholesterol(LDLC)...

Aim To determine the contribution of body mass index(BMI) and family genetic factors(FGFs) to the development of hyperlipidemia and the interaction of these two factors. Methods 25 pedigrees with familial hyperlipidemia[familial combined hyperlipidemia(FCHL) and familial hypercholesterolemia(FH)] and control families were investigated and analysed in Beijing area during 1996 ̄1997. Results There are significantly higher levels of total cholesterol(TC),triglycide(TG),low density lipoprotein cholesterol(LDLC) in hyperlipidemia families than that of control families. After adjustment of other risk factors,the serum levels of TC,TG,LDLC from high to low is: overweight in hyperlipidemia families,nonoverweight in hyperlipidemia families, overweight in control families and nonoverweight in control families. Conclusion These findinds suggest that FGFs and BMI are both important risk factors for hyperlipidemia . They influence on serum lipid synergically.

目的探讨遗传因素、超重及其相互作用对血脂水平的影响。方法采用家系调查法,于1996~1997年在北京地区搜集家族性高脂血症(混合型高脂血症家系和家族性高胆固醇血症家系)及正常对照家系共25个进行统计分析。结果高脂血症家系血清总胆固醇、甘油三酯、低密度脂蛋白胆固醇水平显著高于正常对照家系;调整其他危险因素后的血脂水平,高脂血症家系超重组>高脂血症家系非超重组>对照家系超重组>对照家系非超重组。结论家族遗传因素和超重都是高脂血症重要的危险因素,二者具有协同作用。

 
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