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急性肝功能不全
相关语句
  acute hepatic failure
     Protective effects of glutamine on rats with acute hepatic failure
     谷氨酰胺对急性肝功能不全大鼠的防治作用
短句来源
     OBJECTIVE The present experiments were designed to study the protective effects of glutamine on rats with acute hepatic failure.
     目的探讨谷氨酰胺对大鼠实验性急性肝功能不全的防治作用。
短句来源
  acute hepatic dysfunction
     Acute hepatic dysfunction and the application of Ⅱ score in surgery critical illness
     急性肝功能不全与外科危重病监测中急性病理生理和慢性健康评分的应用
短句来源
  “急性肝功能不全”译为未确定词的双语例句
     The action of endotoxin on acute hepatic insufficiency
     内毒素对实验性急性肝功能不全的作用
短句来源
     These results suggest that endotoxin is an important factor in the development and progress of acute hepatic insufficiency.
     上述结果表明,内毒素血症是诱发急性肝功能不全发生发展的重要因素。
短句来源
  相似匹配句对
     The action of endotoxin on acute hepatic insufficiency
     内毒素对实验性急性肝功能不全的作用
短句来源
     Protective effects of glutamine on rats with acute hepatic failure
     谷氨酰胺对急性肝功能不全大鼠的防治作用
短句来源
     Acute Infectious Hepatitis
     急性传染性肝炎
短句来源
     Acute Posterior Ischemic Optic Neuropathy
     急性后部缺血性视神经病变
短句来源
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  acute hepatic failure
A molecular adsorbent recycling system in treating posthepatectomy acute hepatic failure patients with hepatocellular carcinoma:
      
Differential gene expression profiles in acute hepatic failure model in mice infected with MHV-3 virus intervened by anti-hepati
      
Differential gene expression profiles in Balb/cJ mouse model of acute hepatic failure infected with MHV-3 virus intervened by anti-hepatic failure compound (AHFC) and the changes of cytokines regulated by genes were investigated.
      
Acute hepatic failure model of Balb/cJ mice infected with MHV-3 virus was established.
      
Changes of high mobility group box 1 in serum of pig acute hepatic failure model and significance
      
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  acute hepatic dysfunction
Classical galactosemia, characterized clinically by acute hepatic dysfunction, sepsis, cataract, and failure to thrive, is caused by deficiency of galactose-1-phosphate uridyltransferase (GALT).
      
No apparent effect of acute hepatic dysfunction was observed on both the pharmacokinetic and pharmacodynamic behavior of the drug.
      


The effects of E. coli endotoxin on acute hepatic insufficiency induced by galactosamine were studied. Results indicated that endotoxin exhibited significant effects of promoting the nerosis of hepatocyte, prolonging prothrombin time, elevating the serum levels of billrubin and urea nitrogen on acute hepatic insufficiency in rats induced by galactosamtne. Lethargy and coma in the latter rats treated with endotoxin occured more quickly and more seriously than those in the controls, but no significant difference...

The effects of E. coli endotoxin on acute hepatic insufficiency induced by galactosamine were studied. Results indicated that endotoxin exhibited significant effects of promoting the nerosis of hepatocyte, prolonging prothrombin time, elevating the serum levels of billrubin and urea nitrogen on acute hepatic insufficiency in rats induced by galactosamtne. Lethargy and coma in the latter rats treated with endotoxin occured more quickly and more seriously than those in the controls, but no significant difference of amino acids in plasma and brain was found except for taurine which showed a significant increase in brain. These results suggest that endotoxin is an important factor in the development and progress of acute hepatic insufficiency.

本文对大肠杆菌内毒素在半乳糖胺引起急性肝功能不全中的作用进行了研究。实验结果表明,内毒素加重半乳糖胺所致的肝细胞坏死,使大鼠血浆尿素氮、胆红素水平升高,凝血酶原时间延长,嗜睡时间提前与昏迷加重。经血浆、脑匀浆游离氨基酸分析提示,内毒素可使脑组织牛磺酸水平增高。上述结果表明,内毒素血症是诱发急性肝功能不全发生发展的重要因素。

Objective To investigate the pathological characteristics of hepatic energy metabolism changes due to biliary sepsis. Methods The hepatic mitochondrial respiratory function and content of ATP was dynamically measured in the self controlled rabbit model of biliary sepsis. Results The mitochondrial S 3, respiration control rate (RCR) and phosphorus/oxygen (P/O) were significantly dropping in the infective hepatic lobe 12 hrs after operation with S 4 increasing markedly, and the oxidative phosphorylation...

Objective To investigate the pathological characteristics of hepatic energy metabolism changes due to biliary sepsis. Methods The hepatic mitochondrial respiratory function and content of ATP was dynamically measured in the self controlled rabbit model of biliary sepsis. Results The mitochondrial S 3, respiration control rate (RCR) and phosphorus/oxygen (P/O) were significantly dropping in the infective hepatic lobe 12 hrs after operation with S 4 increasing markedly, and the oxidative phosphorylation was uncoupled from 48 hrs after operation onward. The hepatic mitochondrial RCR showed early ascending and then dropping in the non infective hepatic lobe. The content of ATP and mitochondrial respiratory activity decreased synchronously in both hepatic lobes.Conclusion The hepatic energy metabolic failure was induced in the early stage by biliary sepsis. This is probably the pathological basis of biliary sepsis that is highly critical and always lead to MOF following acute liver function failure.

目的 探讨胆源性脓毒症时肝脏能量代谢改变的病理特征。方法 借助兔自身对照性动物模型 ,对胆源性脓毒症状态下肝脏线粒体呼吸功能及肝组织ATP含量进行动态检测。结果 自术后 12小时起 ,感染肝叶线粒体Ⅲ态呼吸率 (S3 )、呼吸控制比 (RCR)及磷氧比呈进行性显著降低 ;至术后 48小时氧化与磷酸化脱耦联。非感染肝叶线粒体RCR呈先升后降的双相改变。感染和非感染肝叶组织内ATP含量与其线粒体呼吸活性同步降低。结论 胆源性脓毒症早期即发生肝脏能量代谢障碍 ,这可能是此症常呈现以急性肝功能不全为先导的多器官衰竭的高危病程的病理基础

OBJECTIVE The present experiments were designed to study the protective effects of glutamine on rats with acute hepatic failure.METHODS Establishing the acute hepatic failure models by intraperitoneal injection of galatosamine.Biochemical indexes and histopathological examination were determined. RESULTS At 24 hours,48 hours after intraperitoneal injection of galatosamine,biochemical indexes(ALT,AST,TBIL) level in the glu-treated group were significantly lower than those of the model group(P<(0.05)).Histopathological...

OBJECTIVE The present experiments were designed to study the protective effects of glutamine on rats with acute hepatic failure.METHODS Establishing the acute hepatic failure models by intraperitoneal injection of galatosamine.Biochemical indexes and histopathological examination were determined. RESULTS At 24 hours,48 hours after intraperitoneal injection of galatosamine,biochemical indexes(ALT,AST,TBIL) level in the glu-treated group were significantly lower than those of the model group(P<(0.05)).Histopathological examinations indicated that the glu-treated group had much slighter lesion compared with the model group(P<0.05).CONCLUSION The present results showed that glutamine can effectively lighten galatosamine-induced liver necrosis.

目的探讨谷氨酰胺对大鼠实验性急性肝功能不全的防治作用。方法腹腔注射D-氨基半乳糖(GalN)建立急性肝功能不全大鼠模型,并测定血清生化指标和肝组织病理检查。结果肝功能检测显示:造模后24,48h肝脏损伤程度,与模型组相比,防治组的肝损较轻(P<0.05)。肝脏病理切片结果:防治组的损伤程度明显轻于模型组,两者差异显著(P<0.05)。结论谷氨酰胺对GalN致急性肝功能不全大鼠有明显的保护作用。

 
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