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慢性重型病毒性肝炎    
相关语句
  chronic severe viral hepatitis
    Clinical Analysis of 56 Cases of Biopsy Proved Subacute and Chronic Severe Viral Hepatitis
    亚急性、慢性重型病毒性肝炎56例临床分析
短句来源
    Ultrastructural observation on the neutrophils of patients with chronic severe viral hepatitis
    慢性重型病毒性肝炎中性粒细胞超微结构的初步观察
短句来源
    Clinical Studies on Treatment of Chronic Severe Viral Hepatitis with PGE1
    PGE1治疗慢性重型病毒性肝炎的临床研究
短句来源
    A Clinical Analysis on Chronic Severe Viral Hepatitis Complicated With Spontaneous Bacterial Peritonitis
    慢性重型病毒性肝炎并自发性细菌性腹膜炎的临床分析
短句来源
    Conclusion Our studies suggested that PGE1 were effective for chronic severe viral hepatitis and corrected remarkedly pathological disturbance ,it can act as a effective drug in comprehensive treatment for chronic severe viral hepatitis.
    结果发现PGE1治疗有效患者血浆内毒素、TNFα、总胆红素(SB)水平下降,PGE2水平上升。 结论应用PGE1治疗慢性重型病毒性肝炎可部分缓解病情和纠正重型肝炎的病理障碍,故可作为慢性重型病毒性肝炎综合治疗的一种有效药物。
短句来源
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  viral chronic severe hepatitis
    Treatment of Viral Chronic Severe Hepatitis with Hyponatremia
    慢性重型病毒性肝炎并低钠血症治疗的动态观察
短句来源
    Objective: To discuss the treatment of viral chronic severe hepatitis with hyponatremia.
    目的 :探讨慢性重型病毒性肝炎患者低钠血症的治疗。
短句来源
    Methods: 46 cases of viral chronic severe hepatitis with hyponatremia and ascites were supplied with sodium, and the changes of concertration of sodium in blood,ascites, urine and its volume were observed during 24 hours.
    方法 :对 4 6例慢性重型病毒性肝炎并低钠血症及腹水患者给予补钠治疗 ,观察血钠、尿钠、腹水钠、2 4h尿量的变化。
短句来源
    Conclusion: It is an important assist treatment to supply sodium to the patients with viral chronic severe hepatitis with hyponatremia.
    结论 :对慢性重型病毒性肝炎低钠血症患者及时足量补钠是重要的辅助治疗措施
短句来源
  chronic severe viral hepatitis
    Clinical Analysis of 56 Cases of Biopsy Proved Subacute and Chronic Severe Viral Hepatitis
    亚急性、慢性重型病毒性肝炎56例临床分析
短句来源
    Ultrastructural observation on the neutrophils of patients with chronic severe viral hepatitis
    慢性重型病毒性肝炎中性粒细胞超微结构的初步观察
短句来源
    Clinical Studies on Treatment of Chronic Severe Viral Hepatitis with PGE1
    PGE1治疗慢性重型病毒性肝炎的临床研究
短句来源
    A Clinical Analysis on Chronic Severe Viral Hepatitis Complicated With Spontaneous Bacterial Peritonitis
    慢性重型病毒性肝炎并自发性细菌性腹膜炎的临床分析
短句来源
    Conclusion Our studies suggested that PGE1 were effective for chronic severe viral hepatitis and corrected remarkedly pathological disturbance ,it can act as a effective drug in comprehensive treatment for chronic severe viral hepatitis.
    结果发现PGE1治疗有效患者血浆内毒素、TNFα、总胆红素(SB)水平下降,PGE2水平上升。 结论应用PGE1治疗慢性重型病毒性肝炎可部分缓解病情和纠正重型肝炎的病理障碍,故可作为慢性重型病毒性肝炎综合治疗的一种有效药物。
短句来源
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  chronic severe hepatitis
Discriminant function for prognostic indexes and probability of death in chronic severe hepatitis B
      
We aimed to determine a discriminant function for prognosis in chronic severe hepatitis B (CSHB), by discriminant analysis of prognostic indexes and probability of death.
      
The discriminant function is an objective, convenient, and practical method to assess the prognosis of chronic severe hepatitis B.
      
  其他


56 cases of biopsy proved subacute and chronic severe viral hepatitis are analysed. It showed that the serum bilirubin and the degree of prothrombin activity were important prognostic factors. The prognosis of the subacute severe types were better than that of chronic severe types. When fall of blood oxygen pressure and the degree of prothrombin activity appeared, one should watch out for subacute liver necrosis. Early diagnosis and prompt treatment might increase survival.The pathological recovery was later...

56 cases of biopsy proved subacute and chronic severe viral hepatitis are analysed. It showed that the serum bilirubin and the degree of prothrombin activity were important prognostic factors. The prognosis of the subacute severe types were better than that of chronic severe types. When fall of blood oxygen pressure and the degree of prothrombin activity appeared, one should watch out for subacute liver necrosis. Early diagnosis and prompt treatment might increase survival.The pathological recovery was later than that of clinical symptoms and the laboratory finding improvements.Some of the cases had converted into chronic aggressive hepatitis or cirrhosis of liver in the 3rd or 4th months of the disease.

本文对经组织学证实的亚急性及慢性重型病毒性肝炎56例进行了临床分析,发现患者血清胆红素及凝血酶原活动度为影响患者预后的重要指标。亚急性重型预后明显优于慢性重型。血氧分压明显下降、凝血酶原活动度低下时,应警惕亚急性肝坏死。早期诊断及时治疗可提高存活率。病理学恢复晚于临床症状及血清生化指标的好转,在病程的第三、四个月,已有部分患者转变为慢活肝或肝硬变。

The plasma and urinary lipid peroxides (LPO)levels were measured by fluorometric assay in 38 patients with chronic severe viral hepatitis(CSVH), 22 patients with acute hepatitis (AH)and 20 healthy controls. The levels of plasma cyclic nucleotide, prostacyclin(PGI_2) and thromboxane A_2(TXA_2) were simultaneously meassured by RIA in patients with CSVH. The results showed that: 1. In comparision with AH and controls, the plasma and urinary LPO levels in patients with CSVH were significantly elevated(P<0.01). 2....

The plasma and urinary lipid peroxides (LPO)levels were measured by fluorometric assay in 38 patients with chronic severe viral hepatitis(CSVH), 22 patients with acute hepatitis (AH)and 20 healthy controls. The levels of plasma cyclic nucleotide, prostacyclin(PGI_2) and thromboxane A_2(TXA_2) were simultaneously meassured by RIA in patients with CSVH. The results showed that: 1. In comparision with AH and controls, the plasma and urinary LPO levels in patients with CSVH were significantly elevated(P<0.01). 2. In patients with CSVH, the plasma LPO levels were positively related to plasma TXA_2 and urinary LPO, and negatively related to PGI_2 and cAMP. The important role of lipid peroxidation in hepatic injury were discussed.

采用荧光法检测38例慢性重型病毒性肝炎(慢重肝)、22例急性肝炎和20名正常人血浆和尿液脂质过氧化物(LPO)含量。同时用放免法检测慢重肝血浆环核苷酸、前列环素(PGI_2)和血栓素A发_2(TXA_2、含量,并与血浆LPO作相关分析。结果发现:①慢重肝血浆和尿液LPO含量较急性肝炎和正常人均非常显著升高(P均<0.01)。②血浆LPO与尿液LPO及血浆TXA_2呈正相关,与血浆cAMP及PGI_2呈负相关。提示脂质过氧化在慢重肝的肝损害中可能起重要作用。

This paper reports the studies of platelet parameters and

采用血细胞计数仪和电镜对50例慢性重型病毒性肝炎(慢重肝)患者进行了血小板分析三项参数和超微结构改变的对比观察研究。结果发现慢重肝患者血小板计数(PLT)、平均血小板体积(MPV)和血小板压积(PCT)均显著降低,PLT及MPV与PCT之间呈显著正相关;电镜观察结果显示血小板形态不规则、活化聚集。颗粒释放以及退化衰变等变化。上述改变可能与慢重肝时体内血小板激活所致血小板退行性变及骨髓巨核细胞系统成熟不良有关。

 
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